Search results for "Immune system"

showing 10 items of 2885 documents

Quantitative analysis of specific Th1/Th2 helper cell responses and IgG subtype antibodies in interferon-α-treated patients with chronic hepatitis C

2001

This study aimed to characterise the immune mechanisms relevant to viral clearance in interferon (IFN)-alpha-treated chronic hepatitis C virus (HCV) infection. Proliferative responses of peripheral blood mononuclear cells from sustained complete IFN-alpha therapy responders (n = 8), nonresponders (n = 13), untreated patients (n = 10), and healthy controls (n = 5) were measured retrospectively upon stimulation with recombinant HCV-antigens (core, helicase, NS3, NS4, and NS5) and the secretion of IFN-gamma and interleukins (IL-4, IL-5, IL-10, and IL-12) were tested by ELISA. Furthermore, IFN-gamma as well as IL-10 secreting CD4+ T cells were quantitated by intracellular cytokine staining. Ant…

Interleukin 2biologybusiness.industrymedicine.medical_treatmentvirus diseasesVirologyImmunoglobulin GInterleukin 10Infectious DiseasesImmune systemCytokineInterferonVirologyImmunologybiology.proteinMedicineInterferon gammaAntibodybusinessmedicine.drugJournal of Medical Virology
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Gene Therapy of Human Melanoma — from Animal Experiments to the Clinical Trial

1997

Low immunogenecity of tumour cells is one of the reason that specific immune response is insufficient to destroy malignant cells. In an attempt to augment weakly immunogenic B78H1 mouse melanoma cell line, these cells were transfected with genomic DNA from a line of human melanoma cells expressing a 96kD melanoma associated antigen (MAA) that is intercellular adhesion molecule 1 (ICAM 1). The transfected cells expressed fivefold higher quantities of the melanoma associated antigen from which the DNA was obtained. Human ICAM 1 expressed by mouse melanoma cells appeared to be highly immunogenic leading to the rejection of the modified mouse melanoma cells. The transfected cells appeared to be…

Interleukin 2business.industryMelanomamedicine.diseaseAcquired immune systemImmune systemImmunophenotypingAntigenmedicineCancer researchCytotoxic T cellIL-2 receptorbusinessmedicine.drug
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Innate immunity repairs gut lining

2015

It emerges that innate immune cells called group 3 innate lymphoid cells signal directly to intestinal stem cells to promote the replacement of damaged epithelial cells lining the gut. See Letter p.560 The cellular signals supporting normal epithelial intestine maintenance through regulation of intestinal stem cell (ISC) activity are well characterized, but the signals involved in the regulation of the ISC compartment after damage are still unclear. Alan Hanash and colleagues have found that innate lymphoid cells produce interleukin-22 (IL-22) after injury to increase the growth of mouse intestinal organoids. They further show that recombinant IL-22 promotes ISC expansion in both human and …

Interleukin 22MultidisciplinaryInnate immune systemIntestinal mucosaRegeneration (biology)Innate lymphoid cellImmunologyOrganoidInterleukinStem cellBiologydigestive systemCell biologyNature
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Targeting IL-6 signalling in early rheumatoid arthritis is followed by Th1 and Th17 suppression and Th2 expansion.

2014

OBJECTIVES: To investigate the in vitro and ex-vivo effect of IL-6 inhibition on the balance between Th1, Th2, Th17 and Treg cells. METHODS: Ten consecutive adult patients with active early rheumatoid arthritis (ERA) and ten healthy volunteers were included in the study. The percentages of Th1, Th2, Th17 and Treg cells were analysed by flow cytometry in the peripheral blood mononuclear cells obtained from controls and from RA patients at the time of first evaluation and just before the third TCZ infusion. The in vitro effect of TCZ on the different subsets of CD4+ T cells and the expression levels of Th1, Th2, Th17 and Treg-related cytokines was also assessed. RESULTS: Treatment with TCZ, b…

Interleukin 6; Tocilizumab; Early Rheumatoid ArthritisSettore MED/16 - ReumatologiaEarly Rheumatoid Arthritischemical and pharmacologic phenomenahemic and immune systemsInterleukin 6Tocilizumab
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NHL-ChirEx: An interprofessional cross-border education initiative in the Greater Region with a focus on radiation morbidity and patient safety

2018

NHL-ChirEx is an interprofessional cross-border education project that addresses the potential excess of radiation induced morbidity throughout the radiation planning and treatment process. NHL-ChirEx is supported by ESTRO and the University of the Greater Region and has been recently approved and funded under INTERREG VA Programme.

Interprofessional Relationsmedia_common.quotation_subjectRadiation inducedMedical OncologyRadiation planning030218 nuclear medicine & medical imaging03 medical and health sciencesPatient safety0302 clinical medicineNursingimmune system diseaseshemic and lymphatic diseasesHumansMedicineRadiology Nuclear Medicine and imagingQuality (business)Radiation InjuriesSimulation Trainingmedia_commonFocus (computing)Education Medicalbusiness.industryTreatment processHematologyInterprofessional educationCross-border educationEuropeOncology030220 oncology & carcinogenesisPatient SafetyMorbidityRadiologybusinessRadiotherapy and Oncology
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Functionally Active T Cell Receptor/CD3 Complexes Are Present at the Surface of Cloned Cytotoxic T Cells without Fluorescence-Immunological Detectabi…

1996

The cytotoxic T cell clone 10BK.1 is activated in response to the ovalbumin peptide OVA257-264 in a major histocompatibility complex class I-restricted manner. Following activation 10BK.1 cells proliferate, secrete lymphokines, and kill syn- and allogeneic target cells. Using immunofluorescence analysis we detected CD8, LFA-1, and ICAM-1 on the surface of 10BK.1 cells, but no CD3 or T cell receptor (TCR). In contrast, the proliferative response of 10BK.1 cells to antigen was efficiently blocked by soluble antibodies directed at CD3 epsilon or TCR alpha beta, but not by antibodies directed at TCR gamma delta. In addition, lysis of target cells was blocked by F(ab')2 fragments of antibodies d…

Intracellular FluidCD40biologyCD3ImmunologyT-cell receptorAntigen presentationAntigen-Presenting CellsMembrane Proteinshemic and immune systemschemical and pharmacologic phenomenaFlow CytometryLymphocyte ActivationMolecular biologyClone CellsMiceFluorescent Antibody Technique DirectReceptor-CD3 Complex Antigen T-Cellbiology.proteinInterleukin 12AnimalsCytotoxic T cellAntigen-presenting cellCD8T-Lymphocytes CytotoxicCellular Immunology
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Has innate immunity evolved through different routes?

2010

Invertebrate self/non-self recognition, defense responses, mating and development share innate immune surveillance and functions challenged by competition and linked to fitness. Independent evolutionary branches of immune responses may use conserved gene traits. On the other hand immunity genes may be conserved due to their role in development. Finally, upregulation of innate immunity genes during ascidian metamorphosis supports the danger hypothesis.

Intrinsic immunityGeneticsInnate immune systemEvolutionanimal diseasesmedia_common.quotation_subjectinvertebrateGeneral Physics and Astronomychemical and pharmacologic phenomenabiochemical phenomena metabolism and nutritionBiologyImmune systemDownregulation and upregulationArtificial IntelligenceImmunityImmunologybacteriaMetamorphosisMatingGeneral Agricultural and Biological SciencesGeneinnate immunitymedia_common
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The echinoderm innate humoral immune response

2015

Abstract: Multicellular organisms have an immune system, which is essential for the survival of living beings. Interest in the immune system has been expanded since common characteristics of innate immunity between Drosophila melanogaster (Meigen, 1830) and mammals were discovered in the 1980. Since then, immunology has mainly focused on the adaptive immune system that seems to be restricted to vertebrates. Unlike the innate immunity, the adaptive one is acquired after exposure to a specific antigen (Ag) and includes: antigen-presenting cells such as macrophages, proliferation of B and T lymphocytes, Ag-specific antibody/cytokine production and immunological memory. Innate immunity is inste…

Intrinsic immunityInnate immune systemDefense moleculeanimal diseasesInnate lymphoid cellinvertebrateCCL18Pattern recognition receptorSettore BIO/05 - Zoologiachemical and pharmacologic phenomenabiochemical phenomena metabolism and nutritionBiologyAcquired immune systemimmunityImmune systemImmunityImmunologyevolutionbacteriaAnimal Science and Zoology
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Portrait of inflammatory response to ionizing radiation treatment

2015

Ionizing radiation (IR) activates both pro-and anti-proliferative signal pathways producing an imbalance in cell fate decision. IR is able to regulate several genes and factors involved in cell-cycle progression, survival and/or cell death, DNA repair and inflammation modulating an intracellular radiation-dependent response. Radiation therapy can modulate anti-tumour immune responses, modifying tumour and its microenvironment. In this review, we report how IR could stimulate inflammatory factors to affect cell fate via multiple pathways, describing their roles on gene expression regulation, fibrosis and invasive processes. Understanding the complex relationship between IR, inflammation and …

Ionizing radiationDNA repairFibrosimedicine.medical_treatmentClinical BiochemistryInflammationReviewCell fate determinationBioinformaticsImmune systemMedicineCytokineRegulation of gene expressionInflammationInvasivenebusiness.industryCancerIonizing radiation Inflammation Cytokine Fibrosis InvasivenessCell Biologymedicine.diseaseFibrosisInvasivenessRadiation therapyCytokineCancer researchmedicine.symptombusinessJournal of Inflammation
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Generation of Cytotoxic T Lymphocytes Against Ly Alloantigen

2008

Cytotoxic T lymphocytes specific for immune alloantigens controlled by alleles of the Ly system have been induced in vivo. These results were obtained either in a secondary type of response or by treating mice before immunization with a single dose of cyclophosphamide (80 mg/kg).

IsoantigensCyclophosphamideT-Lymphocytesanimal diseasesImmunologyMice Inbred Strainschemical and pharmacologic phenomenaBiologyMiceImmune systemIn vivomedicineAnimalsCytotoxic T cellAlleleCyclophosphamideAllelesCells CulturedGeneral Medicinebiochemical phenomena metabolism and nutritionCytotoxicity Tests ImmunologicImmunizationImmunologybacteriaImmunizationmedicine.drugScandinavian Journal of Immunology
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