Search results for "Immunity"

showing 10 items of 1537 documents

Recent Advances on the Innate Immune Response to Coxiella burnetii.

2021

Coxiella burnetii is an obligate intracellular Gram-negative bacterium and the causative agent of a worldwide zoonosis known as Q fever. The pathogen invades monocytes and macrophages, replicating within acidic phagolysosomes and evading host defenses through different immune evasion strategies that are mainly associated with the structure of its lipopolysaccharide. The main transmission routes are aerosols and ingestion of fomites from infected animals. The innate immune system provides the first host defense against the microorganism, and it is crucial to direct the infection towards a self-limiting respiratory disease or the chronic form. This review reports the advances in understanding…

Microbiology (medical)LipopolysaccharidesImmunologyexperimental modelcytokine—immunological termsQ feverimmunotherapeuticReviewMicrobiologyMicrobiologyImmune systemCellular and Infection MicrobiologyToll-like receptorinflammasomeautophagiamedicineAnimalsHumansPathogeninnate immunityInnate immune systemObligatebiologyTransmission (medicine)MacrophagesInflammasomeCoxiella burnetiibiology.organism_classificationmedicine.diseasebacterial infections and mycosesImmunity InnateQR1-502Toll-like receptorsimmunotherapeuticsInfectious DiseasesCoxiella burnetiibacteriaQ Fevercytokine—immunological termmedicine.drug
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Efficacy of a genetically engineered Candida albicans tet-NRG1 strain as an experimental live attenuated vaccine against hematogenously disseminated …

2009

ABSTRACT We report on the efficacy of the genetically engineered Candida albicans tet-NRG1 strain as an experimental live, attenuated vaccine against disseminated candidiasis in both immunocompetent and immunodeficient mice mostly dependent on T-cell immunity. This experimental vaccination model may represent an important tool to unravel the mechanisms of protective immunity during candidiasis.

Microbiology (medical)Neuregulin-1T-LymphocytesClinical BiochemistryImmunologyBiologyVaccines AttenuatedMicrobiologyMiceImmunityCandida albicansImmunology and AllergyAnimalsCandida albicansFungal vaccineVaccines SyntheticAttenuated vaccineStrain (biology)Candidiasisbiochemical phenomena metabolism and nutritionDisseminated Candidiasisbiology.organism_classificationVaccine ResearchVirologySurvival AnalysisVaccinationImmunizationFungal VaccinesClinical and vaccine immunology : CVI
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Chitin Deacetylase, a Novel Target for the Design of Agricultural Fungicides

2021

Fungicide resistance is a serious problem for agriculture. This is particularly apparent in the case of powdery mildew fungi. Therefore, there is an urgent need to develop new agrochemicals. Chitin is a well-known elicitor of plant immunity, and fungal pathogens have evolved strategies to overcome its detection. Among these strategies, chitin deacetylase (CDA) is responsible for modifying immunogenic chitooligomers and hydrolysing the acetamido group in the N-acetylglucosamine units to avoid recognition. In this work, we tested the hypothesis that CDA can be an appropriate target for antifungals using the cucurbit powdery mildew pathogen Podosphaera xanthii. According to our hypothesis, RNA…

Microbiology (medical)chitin-triggered immunityQH301-705.5Plant ImmunityPlant SciencechitinChelating ActivityArticleMicrobiologychitin; chitin deacetylase; chitin-triggered immunity; EDTA; <i>Podosphaera xanthii</i>; powdery mildews; RNAi silencingchemistry.chemical_compoundPodosphaera xanthiiChitinChelationchitin deacetylaseBiology (General)Ecology Evolution Behavior and Systematics<i>Podosphaera xanthii</i>powdery mildewsRNAi silencingEDTAElicitorChitin deacetylaseFungicidechemistryPowdery mildewJournal of Fungi
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COVID-19—Importance for Patients on the Waiting List and after Kidney Transplantation—A Single Center Evaluation in 2020–2021

2021

(1) Background: Dialysis patients and recipients of a kidney allograft are at high risk for infection with SARS-CoV-2. It has been shown that the development of potent neutralizing humoral immunity against SARS CoV-2 leads to an increased probability of survival. However, the question of whether immunocompromised patients develop antibodies has not yet been sufficiently investigated; (2) Methods: SARS-CoV-2 antibodies were examined in hemodialysis patients on the waiting list for kidney transplantation as well as patients after kidney transplantation. Patients were interviewed about symptoms and comorbidities, BMI, and smoking history; (3) Results: SARS-CoV-2 antibodies were found in 16 out…

Microbiology (medical)medicine.medical_specialtyCoronavirus disease 2019 (COVID-19)medicine.medical_treatmentkidney transplantationlcsh:MedicineCOVID-19; waiting list; hemodialysis; kidney transplantationSingle CenterArticleInternal medicinemedicineImmunology and AllergyMolecular BiologyKidney transplantationKidneyhemodialysisGeneral Immunology and Microbiologybiologybusiness.industrylcsh:RCOVID-19waiting listmedicine.diseaseVaccinationInfectious Diseasesmedicine.anatomical_structureHumoral immunitybiology.proteinHemodialysisAntibodybusinessPathogens
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Immune evasion, immunopathology and the regulation of the immune system.

2013

21 pages; International audience; Costs and benefits of the immune response have attracted considerable attention in the last years among evolutionary biologists. Given the cost of parasitism, natural selection should favor individuals with the most effective immune defenses. Nevertheless, there exists huge variation in the expression of immune effectors among individuals. To explain this apparent paradox, it has been suggested that an over-reactive immune system might be too costly, both in terms of metabolic resources and risks of immune-mediated diseases, setting a limit to the investment into immune defenses. Here, we argue that this view neglects one important aspect of the interaction…

Microbiology (medical)medicine.medical_treatmentlcsh:MedicineReviewBiologymedicine.disease_causehygiene hypothesisAutoimmunity03 medical and health sciences0302 clinical medicineImmune systemHygiene hypothesisImmunopathologymedicineImmunology and Allergy[ SDV.IMM ] Life Sciences [q-bio]/Immunologymolecular mimicryMolecular Biology030304 developmental biologyimmune evasion0303 health sciencesNatural selectionimmunosuppressionGeneral Immunology and Microbiologylcsh:Rautoimmunityimmune regulationImmunosuppressionbiochemical phenomena metabolism and nutritionEvasion (ethics)Molecular mimicryInfectious DiseasesImmunology[SDV.IMM]Life Sciences [q-bio]/ImmunologyTreg cells030215 immunology
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Social evolution of innate immunity evasion in a virus

2019

Antiviral immunity has been studied extensively from the perspective of virus−cell interactions, yet the role of virus−virus interactions remains poorly addressed. Here, we demonstrate that viral escape from interferon (IFN)-based innate immunity is a social process in which IFN-stimulating viruses determine the fitness of neighbouring viruses. We propose a general and simple social evolution framework to analyse how natural selection acts on IFN shutdown and validate it in cell cultures and mice infected with vesicular stomatitis virus. Furthermore, we find that IFN shutdown is costly because it reduces short-term viral progeny production, thus fulfilling the definition of an altruistic tr…

Microbiology (medical)virusesImmunologyBiologyApplied Microbiology and BiotechnologyMicrobiologyAntiviral AgentsModels BiologicalArticleVirusVesicular stomatitis Indiana virus03 medical and health sciencesMiceViral ProteinsInterferonImmunityGeneticsmedicineAnimals030304 developmental biologyImmune Evasion0303 health sciencesMice Inbred BALB CInnate immune systemNatural selection030306 microbiologyBrainCell BiologyDNA-Directed RNA Polymerasesbiology.organism_classificationAltruismVirologyBiological EvolutionImmunity Innate3. Good healthDisease Models AnimalVesicular stomatitis virusViral evolutionHost-Pathogen InteractionsFemaleInterferonsSocial evolutionmedicine.drugNature Microbiology
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Role of HLA-B α-3 domain amino acid position 194 in HIV disease progression

2013

HLA class I molecules play a role in the regulation of innate immune response. Therefore, the interaction of HLA class I molecules with different activating and inhibitory receptors leads to balancing the immune response. Among the different family of receptors, NK receptors KIR3DL1/S1 and LIR1, play a major role. Aim of this study was to evaluate the role of amino acid polymorphic positions of HLA class I molecules interacting with NK receptors in HIV progression. In order to minimize the influence of viral variability, a cohort of children with a nosocomial monophyletic HIV-1 infection from the Benghazi Children Hospital has been evaluated. To assess the role of single amino acid position…

Models MolecularGene ExpressionKIR3DS1HIV InfectionsPeptide bindingLeukocyte Immunoglobulin-like Receptor B1ModelsImmunologicReceptorsInnateReceptors ImmunologicChildReceptorGeneticschemistry.chemical_classificationCross Infectioneducation.field_of_studyReceptors KIR3DL1Polymorphism Genetic; Models Molecular; Humans; Disease Progression; Gene Expression; HLA-B Antigens; Immunity Innate; Child; Receptors KIR3DL1; Protein Binding; HIV-1; Binding Sites; Receptors KIR3DS1; Receptors Immunologic; HIV Infections; Antigens CD; Protein Structure Tertiary; Signal Transduction; Amino Acid Substitution; Cross InfectionHLA-BCDAmino acidDisease ProgressionKIR3DL1Protein BindingSignal TransductionReceptors KIR3DS1Protein StructureImmunologyPopulationHuman leukocyte antigenBiologyGeneticKIR3DL1Antigens CDHumansPolymorphismAntigenseducationMolecular BiologySettore MED/04 - Patologia GeneralePolymorphism GeneticBinding SitesInnate immune systemImmunityMolecularImmunity InnateProtein Structure TertiaryAmino Acid SubstitutionchemistryHLA-B AntigensImmunologyHIV-1TertiaryMolecular Immunology
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The Molecular Anatomy of Human Hsp60 and its Similarity with that of Bacterial Orthologs and Acetylcholine Receptor Reveal a Potential Pathogenetic R…

2012

Heat-shock protein 60 (Hsp60) is ubiquitous and highly conserved being present in eukaryotes and prokaryotes, including pathogens. This chaperonin, although typically a mitochondrial protein, can also be found in other intracellular sites, extracellularly, and in circulation. Thus, it can signal the immune system and participate in the development of inflammation and immune reactions. Both phenomena can be elicited by human and foreign Hsp60 (e.g., bacterial GroEL), when released into the blood by infectious agents. Consequently, all these Hsp60 proteins become part of a complex autoimmune response characterized by multiple cross reactions because of their structural similarities. In this s…

Models MolecularMolecular Sequence Datachemical and pharmacologic phenomenaAnti-Chaperonin ImmunityBiologymedicine.disease_causecomplex mixturesEpitopeProtein Structure SecondaryHsp60; Myasthenia Gravis; Anti-Chaperonin Immunity; Chlamydia trachomatis; Chlamydia pneumoniae; AChRα1MicrobiologyChaperoninCellular and Molecular NeuroscienceImmune systemChlamydia trachomatiBacterial ProteinsChlamydia pneumoniaeMyasthenia GravisAChRα1medicineHumansReceptors CholinergicAmino Acid SequenceAcetylcholine receptorSequence Homology Amino AcidfungiImmunityCell BiologyGeneral MedicineChaperonin 60Hsp60GroELMyasthenia GraviMolecular mimicryImmunologyHSP60Chlamydia trachomatis
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Identification and relevance of the CD95-binding domain in the N-terminal region of ezrin.

2003

The CD95 (Fas/APO-1) linkage to the actin cytoskeleton through ezrin is an essential requirement for susceptibility to the CD95-mediated apoptosis in CD4+ T cells. We have previously shown that moesin was not involved in the binding to CD95. Here we further support the specificity of the ezrin/CD95 binding, showing that radixin did not bind CD95. The ezrin region specifically and directly involved in the binding to CD95 was located in the middle lobe of the ezrin FERM domain, between amino acids 149 and 168. In this region, ezrin, radixin, and moesin show 60-65% identity, as compared with the 86% identity in the whole FERM domain. Transfection of two different human cell lines with a green …

Moesinchemical and pharmacologic phenomenaApoptosismacromolecular substancesBiologyBiochemistryEzrinRadixinhemic and lymphatic diseasesHumansfas ReceptorMolecular BiologyActinBinding SitesFERM domainhemic and immune systemsCell BiologyTransfectionActin cytoskeletonPhosphoproteinsActinsCell biologyProtein Structure TertiaryCytoskeletal ProteinsMutationbiological phenomena cell phenomena and immunityBinding domainHeLa CellsProtein BindingSignal TransductionThe Journal of biological chemistry
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Bleomycin Exerts Ambivalent Antitumor Immune Effect by Triggering Both Immunogenic Cell Death and Proliferation of Regulatory T Cells

2013

International audience; Bleomycin (BLM) is an anticancer drug currently used for the treatment of testis cancer and Hodgkin lymphoma. This drug triggers cancer cell death via its capacity to generate radical oxygen species (ROS). However, the putative contribution of anticancer immune responses to the efficacy of BLM has not been evaluated. We make here the observation that BLM induces immunogenic cell death. In particular, BLM is able to induce ROS-mediated reticulum stress and autophagy, which result in the surface exposure of chaperones, including calreticulin and ERp57, and liberation of HMBG1 and ATP. BLM induces anti-tumor immunity which relies on calreticulin, CD8(+) T cells and inte…

MouseCancer TreatmentCD8-Positive T-LymphocytesT-Lymphocytes RegulatoryHematologic Cancers and Related DisordersMice0302 clinical medicineTransforming Growth Factor beta[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyCytotoxic T cellImmune Response0303 health sciencesMultidisciplinaryCell DeathbiologyQRFOXP3Animal ModelsHematology3. Good healthCell biologyOncology030220 oncology & carcinogenesisMedicine[SDV.IMM]Life Sciences [q-bio]/ImmunologyImmunogenic cell deathFemaleLymphomasOncology AgentsResearch ArticleTumor Immunologycongenital hereditary and neonatal diseases and abnormalitiesProgrammed cell death[SDV.IMM] Life Sciences [q-bio]/ImmunologyScienceImmunologyAntineoplastic Agentschemical and pharmacologic phenomenaBleomycin03 medical and health sciencesModel OrganismsImmune systemCell Line TumorAnimalsHumansBiologyCell Proliferation030304 developmental biologyHodgkin Lymphomaurogenital systemCell growthImmunitynutritional and metabolic diseasesImmunologic SubspecialtiesChemotherapy and Drug TreatmentImmunity InnateCancer cellbiology.proteinClinical ImmunologyCalreticulinPLoS ONE
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