Search results for "Immunity"

showing 10 items of 1537 documents

Bacteriophage Adherence to Mucus Mediates Preventive Protection against Pathogenic Bacteria

2019

The mucosal surfaces of animals are habitat for microbes, including viruses. Bacteriophages—viruses that infect bacteria—were shown to be able to bind to mucus. This may result in a symbiotic relationship in which phages find bacterial hosts to infect, protecting the mucus-producing animal from bacterial infections in the process. Here, we studied phage binding on mucus and the effect of mucin on phage-bacterium interactions. The significance of our research is in showing that phage adhesion to mucus results in preventive protection against bacterial infections, which will serve as basis for the development of prophylactic phage therapy approaches. Besides, we also reveal that exposure to m…

medicine.medical_treatmentvirusesbacteriophage therapymedicine.disease_causebakteeritBacteriophageFish Diseaseshost-pathogen interactionslimakalvotPathogenOrganism1183 Plant biology microbiology virology11832 Microbiology and virology2. Zero hunger0303 health scienceshostpathogen interactionsbiologyvirulenssimucosal pathogensQR1-5023. Good healthBACTERIOPHAGEResearch ArticleProtein BindingbacteriophagesPhage therapyeducationvirusFlavobacteriumMicrobiologybakteriofagitHost-Microbe BiologyMicrobiologyViral Proteins03 medical and health sciencesImmunityVirologyAntibiosismedicineAnimalsPhage Therapy030304 developmental biologyMucous MembraneBacteria030306 microbiologybacterial virulenceMucinPathogenic bacteriaEditor's Pickkalatauditbiology.organism_classificationMucusfagiterapiaMucusFlavobacterium columnareBacteriamBio
researchProduct

Microbiome: pro-inflammatory Prevotella?

2013

Rheumatoid arthritis (RA) is a prevalent systemic autoimmune disease, caused by a combination of genetic and environmental factors. Animal models suggest a role for intestinal bacteria in supporting the systemic immune response required for joint inflammation. Here we performed 16S sequencing on 114 stool samples from rheumatoid arthritis patients and controls, and shotgun sequencing on a subset of 44 such samples. We identified the presence of Prevotella copri as strongly correlated with disease in new-onset untreated rheumatoid arthritis (NORA) patients. Increases in Prevotella abundance correlated with a reduction in Bacteroides and a loss of reportedly beneficial microbes in NORA subjec…

metagenomicsrheumatoidarthritisMouseinflammationImmunologyautoimmunitymicrobiomeHuman Biology and MedicineResearch ArticleHumanNature reviews. Microbiology
researchProduct

Evaluation of waterborne exposure to heavy metals in innate immune defences present on skin mucus of gilthead seabream (Sparus aurata)

2015

Aquatic animals are continuously exposed to chemical pollutants but the effects evoked in skin surfaces, which receive the most direct contact with them, are poorly investigated. Terminal carbohydrate composition and immunological components present in skin mucus of gilthead seabream (Sparus aurata L.) specimens exposed to waterborne sublethal dosages of heavy metals [arsenic (As2O3), cadmium (CdCl2) and mercury (CH3HgCl) at 5, 5 and 0.04 μM, respectively for 2, 10 and 30 days were analysed. Moreover, the presence of a fucose binding lectin (FBL) was evaluated by western blot and the protein profiles were by SDS-PAGE and HPLC. Results showed little effects of heavy metals in the presence of…

mucosal immunity heavy metals lectins gilthead seabream (Sparus aurata).chemistry.chemical_elementAquatic ScienceBiologyMicrobiologySkin mucus Mucosal immunity Heavy metals Lectins Gilthead seabream (Sparus aurata)chemistry.chemical_compoundRandom AllocationImmune systemWestern blotMetals HeavymedicineEnvironmental ChemistryAnimalsMethylmercuryImmunity MucosalSkinchemistry.chemical_classificationCadmiumInnate immune systemmedicine.diagnostic_testAquatic animalGeneral MedicineMucusImmunity InnatePerciformesEnzymechemistryImmunologyWater Pollutants Chemical
researchProduct

Role of Human Leukocyte Antigens (HLA) in Autoimmune Diseases

2018

Since the discovery of HLA 60 years ago, it has contributed to the understanding of the immune system as well as of the pathogenesis of several diseases. Aside from its essential role in determining donor-recipient immune compatibility in organ transplantation, HLA genotyping is meanwhile performed routinely as part of the diagnostic work-up of certain autoimmune diseases. Considering the ability of HLA to influence thymic selection as well as peripheral anergy of T cells, its role in the pathogenesis of autoimmunity is understandable. The aim of this paper is to provide a brief overview of the role and current clinical relevance of HLA-B27 in spondyloarthritis and HLA-B51 in Behçet's disea…

musculoskeletal diseases0301 basic medicinemedicine.medical_specialtyReviewBehcet's diseaseDiseaseHuman leukocyte antigenmedicine.disease_causeOrgan transplantationAutoimmune DiseasesAutoimmunityPathogenesisEpitopes03 medical and health sciencesImmune system0302 clinical medicineRheumatologyHLA AntigensInternal medicineHumansImmunology and AllergyMedicineClinical significanceskin and connective tissue diseases030203 arthritis & rheumatologyHLA-B27business.industryHistocompatibility Antigens Class IInutritional and metabolic diseasesmedicine.diseaseRheumatologyeye diseasesstomatognathic diseases030104 developmental biologyRheumatoid arthritisImmunologybusiness
researchProduct

HLA Class II Differentiates Between Thyroid and Polyglandular Autoimmunity.

2015

The HLA class II genes are susceptibility genes for autoimmune endocrine diseases; however, scarce data are available pertaining to the determinants of genetic susceptibility to polyglandular autoimmunity (PGA). A total of 300 consecutive and unselected patients with either PGA or monoglandular autoimmune thyroid disease (AITD) and 100 healthy control subjects were genotyped for the HLA class II DRB1, -DQA1, and -DQB1 alleles. Compared to patients with AITD and controls, the HLA-DRB1*03 (pc =0.001), *04 (pc<0.001), -DQA1*03 (pc<0.001), and -DQB1*02 (pc =0.001) alleles were increased in patients with PGA. When dividing patients with Hashimoto's thyroiditis (HT) into those with PGA (PGA-HT) v…

musculoskeletal diseasesAdultMalemedicine.medical_specialtyendocrine system diseasesAdolescentEndocrinology Diabetes and MetabolismGraves' diseaseClinical BiochemistryThyroid GlandAutoimmunityImmunogeneticsHashimoto Diseasemedicine.disease_causeBiochemistryThyroiditisAutoimmunityYoung AdultEndocrinologyInternal medicineGenetic predispositionMedicineHumansHashimoto DiseaseGenetic Predisposition to DiseaseAlleleskin and connective tissue diseasesChildbusiness.industryBiochemistry (medical)ThyroidHistocompatibility Antigens Class IIInfantGeneral MedicineMiddle Agedmedicine.diseaseGraves DiseaseEndocrinologymedicine.anatomical_structureChild PreschoolImmunologyFemalebusinessHormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme
researchProduct

Deficiency of Nrf2 accelerates the effector phase of arthritis and aggravates joint disease

2011

14 páginas, 8 figuras, 1 tabla.-- et al.

musculoskeletal diseasesGenetically modified mouseMedicinaNF-E2-Related Factor 2PhysiologyChemokine CXCL1Clinical BiochemistryNitric Oxide Synthase Type IIArthritisMice Transgenicmedicine.disease_causeenvironment and public healthBiochemistryNrf2MicemedicineAnimalsMolecular BiologyGeneral Environmental SciencebiologyInterleukin-6Effectorbusiness.industryArthritisInflammation and degenerationCell Biologyrespiratory systemmedicine.diseaseArthritis ExperimentalInfection and autoimmunity Auto-immunity transplantation and immunotherapy [NCMLS 1]Disease Models AnimalOxidative StressEicosanoidCyclooxygenase 2Rheumatoid arthritisTumor Necrosis FactorsImmunologyOsteocalcinbiology.proteinGeneral Earth and Planetary SciencesJointsTumor necrosis factor alphaImmune Regulation Auto-immunity transplantation and immunotherapy [NCMLS 2]businessOxidation-ReductionHeme Oxygenase-1Oxidative stress
researchProduct

Conserved TCR β chain usage in reactive arthritis; evidence for selection by a putative HLA-B27-associated autoantigen

2002

Previous work suggested that expanded CD8+ T-cell clones in the synovial fluid (SF) of HLA-B27+ patients with reactive arthritis (ReA) preferentially use the T-cell receptor variable region (TCRBV) 1, similar CDR3 sequences, and joining region (BJ) 2S3. To determine the range of conservation and disease-specificity of CDR3-sequences, we analyzed the TCRBV1-J2S3 repertoire from 33 healthy HLA-B27+ individuals, patients with various types of spondyloarthropathies (SpA), and with rheumatoid arthritis (RA) by CDR3-spectratyping. After collection and database submission of all available TCRB-CDR3 from HLA-B27-restricted or SpA-derived T cells, we systematically screened the entire human sequence…

musculoskeletal diseasesGeneticsHLA-B27T cellImmunologyT-cell receptorArthritisGeneral MedicineBiologymedicine.disease_causemedicine.diseaseBiochemistryConserved sequenceAutoimmunitymedicine.anatomical_structureAntigenGeneticsmedicineImmunology and Allergyskin and connective tissue diseasesCD8Tissue Antigens
researchProduct

The role of innate and lymphoid IL-22-producing cells in the immunopathology of primary Sjögren's syndrome

2014

In primary Sjögren's syndrome (pSS) a complex of interconnections between epithelial barrier, innate and adaptive immunity occurs. IL-22 is a pleiotropic cytokine that in pSS may be placed at the intersection of the adaptive and innate branches of immunity. Some evidence suggests that, in pSS, IL-22 may play a prominent pro-inflammatory role driving the early phase of tissue and systemic inflammation and participating in the self-perpetuation of disease. Despite contradictory data in literature about the role of NK cells in pSS, recent data also suggest an important contribution of this subset of cells of the innate immune system in the development and perpetuation of inflammation. Here, we…

musculoskeletal diseasesImmunologyInflammationAdaptive ImmunitySystemic inflammationInterleukin 22stomatognathic systemImmunityImmunopathologymedicineHumansImmunology and AllergyInnateKiller CellInnate immune systembusiness.industryInterleukinsInnate lymphoid cellImmunityInterleukinAcquired immune systemeye diseasesImmunity InnateKiller Cells Naturalstomatognathic diseasesSjogren's SyndromeImmunologyNaturalmedicine.symptombusinessHuman
researchProduct

IAP proteins as targets for drug development in oncology.

2013

The inhibitors of apoptosis (IAPs) constitute a family of proteins involved in the regulation of various cellular processes, including cell death, immune and inflammatory responses, cell proliferation, cell differentiation, and cell motility. There is accumulating evidence supporting IAP-targeting in tumors: IAPs regulate various cellular processes that contribute to tumor development, such as cell death, cell proliferation, and cell migration; their expression is increased in a number of human tumor samples, and IAP overexpression has been correlated with tumor growth, and poor prognosis or low response to treatment; and IAP expression can be rapidly induced in response to chemotherapy or …

musculoskeletal diseasesProgrammed cell deathCell growthbusiness.industryCellular differentiationapoptosisCell migrationReviewBioinformaticsbody regionsInternal ribosome entry siteImmune systemOncologyDrug developmentApoptosisCancer researchMedicinePharmacology (medical)Smac mimeticsbiological phenomena cell phenomena and immunitybusinessantitumor therapyOncoTargets and therapy
researchProduct

IAPs: more than just inhibitors of apoptosis proteins.

2008

Inhibitors of apoptosis proteins (IAPs) are a conserved family of proteins identified in species ranging from virus, yeasts, nematodes, fishes, flies and mammals. The common structural feature is the presence of at least one Baculovirus IAP Repeat (BIR) domain. Hence, IAPs are also known as BIR-containing proteins (BIRCs). Most of them display anti-apoptotic properties when overexpressed. In drosophila, IAPs are sufficient and necessary to promote cell survival through a direct regulation of apoptotic proteases called caspases. In mammals, BIRC4/XIAP, the most studied IAP member can directly inhibit the activity of caspase-3, 7 and 9. However, this activity is not conserved in other IAPs an…

musculoskeletal diseasesProteasesCell signalingvirusesCellular differentiationApoptosisModels BiologicalInhibitor of Apoptosis ProteinsCell MovementCellular stress responseMolecular BiologyCaspaseCell ProliferationbiologyCell DifferentiationCell BiologyCell biologyXIAPbody regionsApoptosisCaspasesbiology.proteinbiological phenomena cell phenomena and immunitySignal transductionDevelopmental BiologySignal TransductionCell cycle (Georgetown, Tex.)
researchProduct