Search results for "Insulin"

showing 10 items of 1360 documents

Chronic exposure of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) induces an obesogenic effect in C57BL/6J mice fed a high fat diet

2017

IF 3.582; International audience; Contaminant involvement in the pathophysiology of obesity is widely recognized. It has been shown that low dose and chronic exposure to endocrine disruptor compounds (EDCs) potentiated diet- induced obesity. High and acute exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD), a persistent organic pollutant (POP) and an EDC with anti-estrogenic property, causes wasting syndrome . However at lower doses, the TCDD metabolic effects remain poorly understood. We investigated the obesogenic effect during chronic exposure of TCDD at 1μg/kg body weight (bw)/week in adult C57BL/6J mice fed with a high fat diet (HFD) and exposed from 10 to 42 weeks old to TCDD or e…

Blood GlucoseLeptinMale0301 basic medicineTCDDPolychlorinated DibenzodioxinsTime FactorsAdipose tissue010501 environmental sciencesToxicology01 natural sciencesBasic Helix-Loop-Helix Transcription FactorsInsulinAdiposity2. Zero hunger[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism3. Good healthLiverEndocrine disruptorReceptors AndrogenCytokinesEnvironmental PollutantsFemaleInflammation Mediatorsmedicine.symptomStearoyl-CoA Desaturasemedicine.medical_specialtyLipolysisInflammationchronic exposureIntra-Abdominal FatDiet High-FatRisk Assessment03 medical and health sciencesSex FactorsobesogenInternal medicinemedicineAnimalsEndocrine systemObesityRNA MessengerWasting SyndromeTriglycerides0105 earth and related environmental sciencesbusiness.industrymedicine.diseaseObesityMice Inbred C57BL030104 developmental biologyEndocrinologyReceptors Aryl HydrocarbonInsulin ResistancebusinessBiomarkersObesogenDrug metabolism
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Asperuloside Enhances Taste Perception and Prevents Weight Gain in High-Fat Fed Mice

2021

Asperuloside is an iridoid glycoside found in many medicinal plants that has produced promising anti-obesity results in animal models. In previous studies, three months of asperuloside administration reduced food intake, body weight, and adipose masses in rats consuming a high fat diet (HFD). However, the mechanisms by which asperuloside exerts its anti-obesity properties were not clarified. Here, we investigated homeostatic and nutrient-sensing mechanisms regulating food intake in mice consuming HFD. We confirmed the anti-obesity properties of asperuloside and, importantly, we identified some mechanisms that could be responsible for its therapeutic effect. Asperuloside reduced body weight …

Blood GlucoseLeptinMalecannabinoid (CB) receptor 10301 basic medicineTastePro-Opiomelanocortinfood intakeEndocrinology Diabetes and MetabolismAdipose tissueWeight Gainnutrient-sensing mechanismslcsh:Diseases of the endocrine glands. Clinical endocrinologyCyclopentane MonoterpenesEnergy homeostasisMiceEndocrinology0302 clinical medicineGlucosidesWeight lossInsulinasperuloside; cannabinoid (CB) receptor 1; CD36; FFAR1-4; food intake; nutrient-sensing mechanisms; TAS1R2-3; weight lossReceptorOriginal ResearchLeptindigestive oral and skin physiologyTaste PerceptionGhrelinTAS1R2-3Ghrelinmedicine.symptommedicine.medical_specialtyHypothalamusBiologyDiet High-Fatasperuloside03 medical and health sciencesInternal medicinemedicineAnimalsPyranslcsh:RC648-665Body WeightFFAR1-4030104 developmental biologyEndocrinologyAnti-Obesity Agentsweight lossEnergy IntakeCD36Weight gain030217 neurology & neurosurgery
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Hepatic and very low-density lipoprotein fatty acids in obese offspring of overfed dams.

2010

The combined effects of developmental programming and high-fat feeding at weaning on fatty acid metabolism of the offspring are not well known. In the present study, we aim at characterizing the influence of maternal and offspring's own diets on liver and very low-density lipoprotein (VLDL) lipids; fatty acid profiles of VLDL and liver phospholipids, triglycerides, and cholesteryl esters; and hepatic enzyme activities. Twenty obese male rats born to cafeteria diet-fed dams and 20 control rats born to control diet-fed dams were selected. At weaning, 10 rats of each group were fed control or cafeteria diet. Obese rats had a significant increase in serum glucose, insulin, leptin, VLDL apolipop…

Blood GlucoseLeptinMalemedicine.medical_specialtyVery low-density lipoproteinOffspringEndocrinology Diabetes and MetabolismLinoleic acidFatty Acids NonesterifiedLipoproteins VLDLchemistry.chemical_compoundEndocrinologyPregnancyInternal medicinemedicineAnimalsInsulinObesityRats Wistarchemistry.chemical_classificationbiologyFatty acid metabolismBody WeightFatty acidOrgan Sizemedicine.diseaseRatsFatty acid synthaseEndocrinologychemistryLiverMaternal Exposurebiology.proteinlipids (amino acids peptides and proteins)FemaleSteatosisEnergy IntakeEnergy MetabolismPolyunsaturated fatty acidMetabolism: clinical and experimental
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Early adaptive response of the retina to a pro-diabetogenic diet: Impairment of cone response and gene expression changes in high-fructose fed rats

2015

The lack of plasticity of neurons to respond to dietary changes, such as high fat and high fructose diets, by modulating gene and protein expression has been associated with functional and behavioral impairments that can have detrimental consequences. The inhibition of high fat-induced rewiring of hypothalamic neurons induced obesity. Feeding rodents with high fructose is a recognized and widely used model to trigger obesity and metabolic syndrome. However the adaptive response of the retina to short term feeding with high fructose is poorly documented. We therefore aimed to characterize both the functional and gene expression changes in the neurosensory retina of Brown Norway rats fed duri…

Blood GlucoseLeptinMalemedicine.medical_specialtyretinamedicine.medical_treatment[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEukaryotic Initiation Factor-2BiologyDiabetes Mellitus ExperimentalfructoseCellular and Molecular Neurosciencechemistry.chemical_compoundDownregulation and upregulationInternal medicineGene expressionDietary CarbohydratesmedicineAnimalsInsulin[SDV.MHEP.OS]Life Sciences [q-bio]/Human health and pathology/Sensory Organs2. Zero hungerRetinamedicine.diagnostic_testGene Expression ProfilingLeptinInsulinFructoseEndoplasmic Reticulum Stressmedicine.diseaseCrystallinsSensory SystemsRatsOphthalmologyCholesterolmedicine.anatomical_structureEndocrinologyGene Expression Regulationchemistry[ SDV.MHEP.OS ] Life Sciences [q-bio]/Human health and pathology/Sensory OrgansFructosamineRetinal Cone Photoreceptor Cellsgene expressionsense organsMetabolic syndromeelectroretinographydiet[SDV.AEN]Life Sciences [q-bio]/Food and NutritionElectroretinography
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Postnatal overfeeding in rats leads to moderate overweight and to cardiometabolic and oxidative alterations in adulthood.

2011

In contrast to the masses of data on obesity, few data are available concerning the cardiometabolic and oxidative consequences of moderate overweight. The model of postnatal overfeeding (OF) induces an increase in body weight at weaning that remains during adult life. Litters of Wistar rats were either maintained at 12 pups (normal-fed group, NF), or reduced to 3 pups at birth in order to induce OF. At 6 months of age, metabolic parameters, circulating oxidative stress and aortic and coronary vasoreactivity were assessed. Cardiac susceptibility to ischemia-reperfusion injury was also evaluated ex vivo as were markers of cardiac remodeling. OF led to an increase in body weight at weaning (+5…

Blood GlucoseLeptinleft ventricular end-systolic pressuremedicine.medical_treatment030204 cardiovascular system & hematologyOverweight+dP/dtmedicine.disease_causeBiochemistryCardiovascular System0302 clinical medicineOvernutritionHRleft ventricular developed pressureheart rateInsulinhydroperoxidesworking modeComputingMilieux_MISCELLANEOUSmatrix metallo-proteinase-2W0303 health sciencesANOVAMMP-2OFLeptinROOHinternational unitsGeneral MedicineLsuperoxide dismutase[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemleft ventricular maximal pressure developmentFemalemedicine.symptomleft ventricular end-diastolic pressureanalysis of variancemedicine.medical_specialtyLDHNFleft ventricular minimal pressure developmentIschemiaSNPbody mass indexheartReal-Time Polymerase Chain Reactionoxidative stress AchBMI03 medical and health sciences[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemLangendorff modeoverfeedingInternal medicineRLUBKmedicineWeaningAnimalsLVEDPSODRats WistarVentricular remodeling030304 developmental biologyDNA PrimersPostnatal overfeedingBase Sequencebusiness.industryInsulinsodium nitroprussiatelactate dehydrogenaseLVDPLVESPOverweightrelative light unitsmedicine.diseaseacetylcholinearbitrary unitsRatsIUOxidative StressEndocrinology−dP/dtAUnormal-fedbradykininbusinessEx vivoOxidative stressBiochimie
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Bile acid receptor TGR5 is critically involved in preference for dietary lipids and obesity

2020

International audience; We investigated the implication of Takeda G protein-coupled receptor 5 (TGR5) in fat preference and fat sensing in taste bud cells (TBC) in C57BL/6 wild-type (WT) and TGR5 knock out (TGR5-/-) male mice maintained for 20 weeks on a high-fat diet (HFD). We also assessed the implication of TGR5 single nucleotide polymorphism (SNP) in young obese humans. The high-fat diet (HFD)-fed TGR5-/- mice were more obese, marked with higher liver weight, lipidemia and steatosis than WT obese mice. The TGR5-/- obese mice exhibited high daily food/energy intake, fat mass and inflammatory status. WT obese mice lost the preference for dietary fat, but the TGR5-/- obese mice exhibited n…

Blood GlucoseLipopolysaccharidesMale0301 basic medicine[SDV]Life Sciences [q-bio]Endocrinology Diabetes and MetabolismClinical BiochemistryBiochemistryReceptors G-Protein-CoupledMice0302 clinical medicineInsulinReceptorMice Knockout2. Zero hungerchemistry.chemical_classificationNutrition and DieteticsLipidsG protein-coupled bile acid receptor[SDV] Life Sciences [q-bio]medicine.anatomical_structuremedicine.medical_specialtyMice Transgenic030209 endocrinology & metabolismSingle-nucleotide polymorphismDiet High-FatPolymorphism Single NucleotideBile Acids and SaltsFood Preferences03 medical and health sciencesInternal medicineTaste budmedicineAnimalsObesityMolecular BiologyInflammationbusiness.industryTaste budFatty acidFatty acidmedicine.diseaseDietary FatsObesityIn vitroFatty LiverMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinologychemistryFatCalciumSteatosisbusinessThe Journal of Nutritional Biochemistry
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Lipopolysaccharides-mediated increase in glucose-stimulated insulin secretion: involvement of the GLP-1 pathway.

2013

Lipopolysaccharides (LPS) of the cell wall of gram–negative bacteria trigger inflammation, which is associated with marked changes in glucose metabolism. Hyperglycemia is frequently observed during bacterial infection and it is a marker of a poor clinical outcome in critically ill patients. The aim of the current study was to investigate the effect of an acute injection or continuous infusion of LPS on experimentally induced hyperglycemia in wild-type and genetically engineered mice. The acute injection of a single dose of LPS produced an increase in glucose disposal and glucose-stimulated insulin secretion (GSIS). Continuous infusion of LPS through mini-osmotic pumps was also associated wi…

Blood GlucoseLipopolysaccharidesendocrine systemmedicine.medical_specialtyEndocrinology Diabetes and MetabolismInflammationBiologyCarbohydrate metabolismGlucagon-Like Peptide-1 ReceptorMiceGlucagon-Like Peptide 1Internal medicinePhospholipid transfer proteinInternal MedicinemedicineHyperinsulinemiaReceptors GlucagonAnimalsInsulinSecretionPhospholipid Transfer ProteinsReceptorMice Knockoutmedicine.disease3. Good healthEndocrinologyGlucoseKnockout mousemedicine.symptomAntagonismhormones hormone substitutes and hormone antagonistsDiabetes
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Hepatic steatosis and insulin resistance are associated with severe fibrosis in patients with chronic hepatitis caused by HBV or HCV infection.

2011

BACKGROUND AND AIMS: Steatosis and insulin resistance (IR) are the major disease modifying in patients with chronic hepatitis C (CHC). Only few studies evaluated these features in patients with chronic hepatitis B (CHB). We aimed to assess the prevalence and the factors related to steatosis and IR in CHB patients, compared with CHC subjects, and to evaluate the potential association between these features and fibrosis severity. MATERIAL AND METHODS: One hundred and seventy consecutive patients with CHB (28 HBeAg positive, 142 HBeAg negative), were evaluated using liver biopsy and metabolic measurements and matched for sex, age and body mass index with 170 genotype 1 CHC patients. IR was def…

Blood GlucoseLiver CirrhosisMaleSettore MED/09 - Medicina InternaBiopsyEnzyme-Linked Immunosorbent AssaySettore MED/08 - Anatomia PatologicaSettore MED/13 - EndocrinologiaBody Mass IndexteatosisHBVPrevalenceHumansInsulinHepatitis B AntibodiesTriglyceridesImmunoassaySettore MED/12 - GastroenterologiaHBV;HCV;steatosisAlanine TransaminaseHepatitis BHepatitis CFatty LiverItalyHCVRegression AnalysisFemaleInsulin ResistanceLiver international : official journal of the International Association for the Study of the Liver
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Chronological Age Interacts with the Circadian Melatonin Receptor 1B Gene Variation, Determining Fasting Glucose Concentrations in Mediterranean Popu…

2020

Gene-age interactions have not been systematically investigated on metabolic phenotypes and this modulation will be key for a better understanding of the temporal regulation in nutrigenomics. Taking into account that aging is typically associated with both impairment of the circadian system and a decrease in melatonin secretion, we focused on the melatonin receptor 1B (MTNR1B)-rs10830963 C&gt

Blood GlucoseMale0301 basic medicinePhysiologyType 2 diabetestype-2 diabetesDiabetis no-insulinodependentMediterranean population0302 clinical medicineRisk FactorsPolymorphism (computer science)MedicineNon-insulin-dependent diabetesmelatonin receptorAged 80 and overeducation.field_of_studyNutrition and DieteticsMediterranean RegionAge FactorsDiabetis en l'embaràsFastingMiddle AgedGestational diabetesMTNR1B polymorphismCohortFemalepregnancywomengestational diabeteslcsh:Nutrition. Foods and food supplyage-interactionhormones hormone substitutes and hormone antagonistsAdultDiabetes riskAdolescentPopulationlcsh:TX341-641030209 endocrinology & metabolismPolymorphism Single NucleotideRisk AssessmentArticleYoung Adult03 medical and health sciencesDiabetes mellitusHumanseducationAgedRetrospective StudiesReceptor Melatonin MT2business.industrymedicine.diseaseCross-Sectional Studies030104 developmental biologyDiabetes Mellitus Type 2Melatonin receptor 1BSpainheterogeneitybusinessDiabetes in pregnancyfasting glucoseFood ScienceNutrients
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Levothyroxine and insulin requirement in autoimmune polyglandular type 3 syndrome: a real-life study

2020

Abstract Purpose To evaluate factors influencing the insulin and levothyroxine requirement in patients with autoimmune polyglandular syndrome type 3 (APS-3) vs. patients with type 1 diabetes mellitus (T1DM) and autoimmune hypothyroidism (AH) alone, respectively. Methods Fifty patients with APS-3, 60 patients with T1DM and 40 patients with AH were included. Anthropometric, clinical and biochemical parameters were evaluated in all patients. Insulin requirement was calculated in patients with APS-3 and T1DM, while levothyroxine requirement was calculated in APS-3 and AH. Results Patients with APS-3 showed higher age (p = 0.001), age of onset of diabetes (p = 0.006) and TSH (p = 0.004) and lowe…

Blood GlucoseMale0301 basic medicineendocrine system diseasesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentAdipose tissueGastroenterologySettore MED/13 - Endocrinologia0302 clinical medicineEndocrinologyAutoimmune hypothyroidism Cardiovascular risk Irisin Type 1 diabetes mellitus Visceral adiposity indexInsulinPolyendocrinopathies AutoimmuneThyroid diseaseMiddle AgedPrognosisFemaleOriginal Articlemedicine.drugAdultIrisinmedicine.medical_specialtyWaistAdolescentLevothyroxineType 1 diabetes mellitus030209 endocrinology & metabolismHashimoto DiseaseYoung Adult03 medical and health sciencesInternal medicineDiabetes mellitusmedicineHumansHypoglycemic AgentsAgedType 1 diabetesAutoimmune hypothyroidismbusiness.industryInsulinThyroiditis AutoimmuneCardiovascular riskmedicine.diseaseVisceral adiposity indexThyroxineDiabetes Mellitus Type 1030104 developmental biologyAge of onsetbusinessBiomarkersFollow-Up StudiesJournal of Endocrinological Investigation
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