Search results for "Interleukin-4"

showing 10 items of 128 documents

Interleukin 4 suppresses primary interferon gamma response by T cells immunized in vivo and cultured in vitro with interleukin 2.

1996

This paper describes a novel primary in vivo/in vitro culture system which allows analysis of the effect of IL-4 added to culture 1 day after immunization on the production of IFN-gamma. Mice are immunized epicutaneously with picryl chloride (TNP) and draining lymph node cells were harvested 1 day later. These cells (1 day lymph node cells), when cultured in vitro for 3 days in the presence of IL-2, either continuously or as a pulse, give an IFN-gamma response on reexposure to antigen 3 days later. This production of IFN-gamma is both antigen-specific and genetically (MHC)-restricted and is due to both CD8+ and CD4+ T cells. However, if 1 day lymph node cells are cultured with both IL-2 and…

Interleukin 2MaleT-LymphocytesImmunologyMice Inbred StrainsPicryl ChlorideBiologyMajor histocompatibility complexBiochemistryPicryl chlorideMajor Histocompatibility Complexchemistry.chemical_compoundEpitopesInterferon-gammaMiceAntigenmedicineImmunology and AllergyAnimalsInterferon gammaMolecular BiologyLymph nodeInterleukin 4Cells CulturedHematologyMolecular biologyInterleukin-10Kineticsmedicine.anatomical_structurechemistryDepression ChemicalImmunologybiology.proteinInterleukin-2ImmunizationInterleukin-4Lymph NodesCD8medicine.drugCytokine
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Effect of isoprinosine on IL-2, IFN-γ and IL-4 production in vivo and in vitro

1991

The effects of an immunopotentiating drug, isoprinosine, on the splenocytes of BALB/c mice to produce cytokines were investigated. Isoprinosine enhanced IL-2 production, upregulating the expression of IL-2 receptor in vitro. It also significantly increased the IFN-gamma secretion and decreased the IL-4 production in vivo. The significance of these findings in terms of immune regulation is discussed.

Interleukin 2medicine.drug_classImmunologyIn Vitro TechniquesBiologyImmunostimulantInterferon-gammaMiceT-Lymphocyte SubsetsIn vivoInosine PranobexmedicineSplenocyteAnimalsInterferon gammaReceptorInterleukin 4PharmacologyMice Inbred BALB CIn vitroImmunologyInterleukin-2FemaleInterleukin-3Interleukin-4Spleenmedicine.drugInternational Journal of Immunopharmacology
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IL-13 receptor α 2: A regulator of IL-13 and IL-4 signal transduction in primary human fibroblasts

2005

Background IL-13 and IL-4 share many functional properties as a result of their use of a common receptor complex comprising IL-13 receptor α 1 (IL-13Rα1) and IL-4 receptor α (IL-4Rα). The nonsignaling receptor IL-13 receptor α 2 (IL-13Rα2) binds IL-13 with high affinity and specificity and is believed to be a decoy receptor for IL-13. Objective We sought to examine the inhibitory effects of soluble and membrane-bound IL-13Rα2 on IL-13– and IL-4–mediated effects. Methods Primary human fibroblasts were grown from endobronchial biopsy specimens obtained from volunteers. Upregulation of IL-13Rα2 mRNA was measured by means of RT-PCR, and the level of surface expression was measured by means of F…

Interleukin 2medicine.medical_specialtyReceptor complexmedicine.medical_treatmentImmunologyGene ExpressionBronchiBiologyDownregulation and upregulationInternal medicinemedicineHumansImmunology and AllergyRNA MessengerReceptorCells CulturedInterleukin-13Reverse Transcriptase Polymerase Chain ReactionReceptors Interleukin-13FibroblastsFlow CytometryUp-RegulationCell biologyEndocrinologyCytokineInterleukin 13STAT proteinInterleukin-4Signal transductionSignal Transductionmedicine.drugJournal of Allergy and Clinical Immunology
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Establishment of different T cell sublines using either interleukin 2 or interleukin 4 as growth factors

1990

Purified protein derivative reactive T cell lines were established under identical conditions with the exception that different lymphokines, namely interleukin (IL) 2 and IL 4 were employed as growth factors. IL 2 favored the development of T cell lines (LNC.2) which upon activation by concanavalin A (Con A) secreted predominantly lymphokines characteristic of TH1 cells. By contrast, T cell lines established with the aid of IL 4 as growth factor (LNC.4) produced mainly lymphokines representative of TH2 cells. Apart from their pattern of lymphokine secretion LNC.2 and LNC.4 T cells were found to differ in their proliferative response to lymphokines and Con A. LNC.2 T cells proliferated only …

Interleukin 2medicine.medical_specialtyT-Lymphocytesmedicine.medical_treatmentT cellImmunologyEnzyme-Linked Immunosorbent AssayIn Vitro TechniquesBiologyCell LineInterferon-gammaMiceInterleukin 21Internal medicinemedicineAnimalsImmunology and AllergyRNA MessengerInterleukin 4Mice Inbred BALB CInterleukin-6LymphokineInterleukinT lymphocyteBlotting NorthernCulture MediaEndocrinologyCytokinemedicine.anatomical_structureInterleukin-2Interleukin-3Interleukin-4Interleukin-5Cell Divisionmedicine.drugEuropean Journal of Immunology
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Molecular and pro-inflammatory aspects of COVID-19: The impact on cardiometabolic health

2022

Obesity, type 2 diabetes (T2DM), hypertension (HTN), and Cardiovascular Disease (CVD) often cluster together as "Cardiometabolic Disease" (CMD). Just under 50% of patients with CMD increased the risk of morbidity and mortality right from the beginning of the COVID-19 pandemic as it has been reported in most countries affected by the SARS-CoV2 virus. One of the pathophysiological hallmarks of COVID-19 is the overactivation of the immune system with a prominent IL-6 response, resulting in severe and systemic damage involving also cytokines such as IL2, IL4, IL8, IL10, and interferon-gamma were considered strong predictors of COVID-19 severity. Thus, in this mini-review, we try to describe the…

Interleukin-6SARS-CoV-2Interleukin-8COVID-19COVID Cardiometabolic Diabetes Inflammation MolecularInterleukin-10Interferon-gammaAdipokinesDiabetes Mellitus Type 2Cardiovascular DiseasesCytokinesHumansInterleukin-2RNA ViralMolecular MedicineInterleukin-4ObesityPandemicsMolecular BiologyBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
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Keratinocytes Determine Th1 Immunity during Early Experimental Leishmaniasis

2010

Experimental leishmaniasis is an excellent model system for analyzing Th1/Th2 differentiation. Resistance to Leishmania (L.) major depends on the development of a L. major specific Th1 response, while Th2 differentiation results in susceptibility. There is growing evidence that the microenvironment of the early affected tissue delivers the initial triggers for Th-cell differentiation. To analyze this we studied differential gene expression in infected skin of resistant and susceptible mice 16h after parasite inoculation. Employing microarray technology, bioinformatics, laser-microdissection and in-situ-hybridization we found that the epidermis was the major source of immunomodulatory mediat…

KeratinocytesCellular differentiationImmunology/Innate ImmunityInterleukin-1betaGene ExpressionInfectious Diseases/Skin InfectionsMiceT-Lymphocyte SubsetsLeishmania majorBiology (General)In Situ HybridizationOligonucleotide Array Sequence AnalysisSkinRegulation of gene expressionMice Inbred BALB CReverse Transcriptase Polymerase Chain ReactionCell DifferentiationImmunohistochemistryInterleukin-12MicrodissectionResearch ArticleQH301-705.5ImmunologyLeishmaniasis CutaneousBiologyMicrobiologyTh2 CellsImmune systemCutaneous leishmaniasisImmunology/Immunity to InfectionsVirologyGeneticsmedicineAnimalsDermatology/Skin InfectionsMolecular BiologyInterleukin 4Epidermis (botany)Interleukin-6Gene Expression ProfilingLasersTh1 CellsRC581-607medicine.diseasebiology.organism_classificationMice Inbred C57BLGene expression profilingDisease Models AnimalImmunology/Immune ResponseImmunologyOsteopontinParasitologyInterleukin-4Immunologic diseases. AllergyPLoS Pathogens
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Local administration of antisense phosphorothioate oligonucleotides to the c-kit ligand, stem cell factor, suppresses airway inflammation and IL-4 pr…

2001

Abstract Background: The c-kit ligand, stem cell factor (SCF), is an important activating and chemotactic factor for both mast cells and eosinophils. These cells are known to play a fundamental role in the pathogenesis of asthma. Objective: Our goal was to analyze the functional role of SCF in the pathogenesis of asthma. Methods: The expression of SCF was targeted in fibroblasts, epithelial cells, and locally in a murine model of asthma in mice induced by ovalbumin sensitization with an antisense DNA strategy. Results: We could suppress SCF expression in NIH 3T3 fibroblasts and SP1 epithelial cells by a specific antisense phosphorothioate oligonucleotide overlapping the translation start si…

KeratinocytesLung DiseasesOvalbuminAdministration TopicalImmunologyInflammationStem cell factorBiology3T3 cellsAllergic inflammationLeukocyte CountMicemedicineImmunology and AllergyAnimalsInterleukin 4InflammationStem Cell FactorOligonucleotide3T3 CellsAllergensFibroblastsOligonucleotides AntisenseThionucleotidesMast cellAsthmaEosinophilsOvalbuminDisease Models Animalmedicine.anatomical_structureembryonic structuresImmunologybiology.proteinInterleukin-4medicine.symptomBronchoalveolar Lavage FluidThe Journal of allergy and clinical immunology
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Epidermal Cells Enhance Interleukin 4 and Immunoglobulin E Production After Stimulation with Protein Allergen

1996

Exposure to certain allergens via epithelial tissues is the primary route for tile induction of immunoglobulin E–dependent allergies of the immediate type associated with atopic diseases. In order to address the question whether and how epithelial cells might contribute to the induction or increase of T H2 -dependent IgE production, we performed co-culture experiments of syngeneic epidermal cells and cells from the associated lymphoid tissue or spleen (responder cells) of BALB/c mice primed with ovalbumin in vivo . In the presence of ovalbumin in vitro , immunoglobulin E but not immunoglobulin G 2a production was significantly enhanced by the addition of epidermal cells, and separation of e…

KeratinocytesLymphoid TissueOvalbuminDermatologyMajor histocompatibility complexImmunoglobulin EBiochemistryImmunoglobulin GMiceAntigenAnimalsRNA MessengerMolecular BiologyInterleukin 4Mice Inbred BALB CDose-Response Relationship Drugintegumentary systembiologyHistocompatibility Antigens Class IIDendritic CellsCell BiologyAllergensImmunoglobulin EMolecular biologycytokinesInterleukin-10Raji cellInterleukin 10Epidermal CellsLangerhans CellsIL-10biology.proteinFemaleImmunizationInterleukin-4EpidermisAntibodyJournal of Investigative Dermatology
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Distinct Roles for IL-1 Receptor Type I Signaling in Early Versus Established Leishmania major Infections

2006

IL-1alpha/beta released by infected dendritic cells (DC) plays a critical role in the development of protective immunity against Leishmania major. Previous studies demonstrated that treatment of susceptible BALB/c mice with IL-1alpha during T-cell priming (days 1-3 post-infection) induced T helper (Th)1-mediated protection. In contrast, we now demonstrate that prolonged treatment with IL-1alpha (for 3 weeks) worsened disease outcome. To characterize the receptor involved, L. major infections in IL-1 receptor type I (IL-1RI) knockout mice were studied. In C57BL/6 IL-1RI-/- mice, the IL-1alpha-mediated protective effect was abrogated. The course of high-dose infection (2 x 10(5) parasites) in…

Leishmaniasis CutaneousPriming (immunology)DermatologyReceptor typeBiochemistryInterferon-gammaMiceTh2 CellsmedicineAnimalsParasite hostingLeishmania majorL-SelectinReceptorMolecular BiologyLeishmania majorMice KnockoutReceptors Interleukin-1 Type IMice Inbred BALB CbiologyReceptors Interleukin-1LeishmaniasisT-Lymphocytes Helper-InducerCell BiologyTh1 Cellsbiology.organism_classificationmedicine.diseaseMice Inbred C57BLGene Expression RegulationCD4 AntigensImmunologyKnockout mouseDisease ProgressionInterleukin-4Ex vivoInterleukin-1Signal TransductionJournal of Investigative Dermatology
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A stage-specific functional role of the leucine zipper transcription factor c-Maf in lung Th2 cell differentiation.

2004

The transcription factor c-Maf controls IL-4 gene expression in CD4(+) T cells, and its expression is up-regulated in human asthmatic airways after allergen challenge. In the present study, we addressed the role of c-Maf in asthma by studying transgenic (Tg) mice overexpressing c-Maf in CD4(+) T cells under the control of the CD2 promoter. As shown, lung CD4(+) T cells of c-maf-Tg mice produced more IL-5 at the early stage (day 2) of culture in the presence of IL-4 than wild-type control cells. Consistently, c-maf-Tg mice spontaneously showed increased IL-5 expression and eosinophils in the bronchial alveolar lavage fluid (BALF) and activated IL-5 signal transduction via Raf-1 and Ras in lu…

Leucine zipperTransgeneCellular differentiationImmunologyMice TransgenicBiologyMiceTh2 CellsProto-Oncogene ProteinsGene expressionmedicineImmunology and AllergyAnimalsTranscription factorLungLeucine ZippersLungCell Differentiationrespiratory systemMolecular biologyrespiratory tract diseasesDNA-Binding ProteinsEosinophilsmedicine.anatomical_structureProto-Oncogene Proteins c-mafInterleukin-4Signal transductionInterleukin-5Proto-Oncogene Proteins c-mafCell DivisionTranscription FactorsEuropean journal of immunology
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