Search results for "Knockout"

showing 10 items of 806 documents

Toll-like receptor 4 defective mice carrying point or null mutations do not show increased susceptibility toCandida albicansin a model of hematogenou…

2006

We have studied the role of TLR4 in murine defenses against Candida albicans in two TLR4-defective mouse strains: C3H/HeJ mice which have defective TLR4 signaling, and TLR4-/- knockout mice. Both TLR4-defective mice strains experimentally infected with virulent C. albicans cells showed no significant difference in survival as compared with their respective controls. Recruitment of neutrophils to the peritoneal cavity of i.p. infected mice was not affected in TLR4-/-animals, but significantly enhanced in C3H/HeJ mice, compared with their control mice. In vitro production of TNF-alpha by macrophages from both types of TLR4-defective mice, in response to yeasts and hyphae of C. albicans, was n…

MaleNeutrophilsBiologyMicrobiologyInterferon-gammaMicePeritoneal cavityCandida albicansSplenocytemedicineAnimalsPoint MutationGenetic Predisposition to DiseaseCandida albicansMice KnockoutMice Inbred C3HToll-like receptorTumor Necrosis Factor-alphaCandidiasisGeneral MedicineTh1 CellsFlow Cytometrybiology.organism_classificationInterleukin-12Corpus albicansMice Inbred C57BLToll-Like Receptor 4Infectious Diseasesmedicine.anatomical_structureKnockout mouseMacrophages PeritonealTLR4Femalelipids (amino acids peptides and proteins)Tumor necrosis factor alphaMedical Mycology
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Role of CB2 cannabinoid receptors in the rewarding, reinforcing, and physical effects of nicotine.

2013

This study was aimed to evaluate the involvement of CB2 cannabinoid receptors (CB2r) in the rewarding, reinforcing and motivational effects of nicotine. Conditioned place preference (CPP) and intravenous self-administration experiments were carried out in knockout mice lacking CB2r (CB2KO) and wild-type (WT) littermates treated with the CB2r antagonist AM630 (1 and 3 mg/kg). Gene expression analyses of tyrosine hydroxylase (TH) and α3- and α4-nicotinic acetylcholine receptor subunits (nAChRs) in the ventral tegmental area (VTA) and immunohistochemical studies to elucidate whether CB2r colocalized with α3- and α4-nAChRs in the nucleus accumbens and VTA were performed. Mecamylamine-precipitat…

MaleNicotinemedicine.medical_treatmentNicotinaRecompensa (Psicologia)Self AdministrationPharmacologyNucleus accumbensNucleus AccumbensNicotineReceptor Cannabinoid CB2MiceRewardCannabinoides -- ReceptorsmedicineAnimalsAcetylcholine receptorPharmacologyMice KnockoutTyrosine hydroxylaseVentral Tegmental Areamedicine.diseaseConditioned place preferenceSubstance Withdrawal SyndromeVentral tegmental areaPsychiatry and Mental healthmedicine.anatomical_structureNicotine withdrawalConditioning OperantOriginal ArticleCannabinoidPsychologyReinforcement Psychologymedicine.drugNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
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Cholinergic Responses of Ophthalmic Arteries in M3and M5Muscarinic Acetylcholine Receptor Knockout Mice

2009

PURPOSE. To determine the functional role of M 3 and M 5 muscarinic acetylcholine receptor subtypes in ophthalmic arteries using gene-targeted mice. METHODS. Muscarinic receptor gene expression was quantified in murine ophthalmic arteries using real-time PCR. To test the functional relevance of M 3 and M 5 receptors, ophthalmic arteries from mice deficient in either subtype (M3R -/- , M5R -/- , respectively) and wild-type controls were isolated, cannulated with micropipettes, and pressurized. Changes in luminal vessel diameter in response to muscarinic and nonmuscarinic receptor agonists were measured by video microscopy. RESULTS. With the use of real-time PCR, all five muscarinic receptor …

MaleNitroprussidemedicine.medical_specialtyCarbacholVasodilator AgentsCholinergic AgentsVideo RecordingGene ExpressionBiologyBradykininArticleMiceOphthalmic ArteryPhenylephrineInternal medicineMuscarinic acetylcholine receptorMuscarinic acetylcholine receptor M4medicineAnimalsRNA MessengerMice KnockoutReceptor Muscarinic M3Receptor Muscarinic M5Reverse Transcriptase Polymerase Chain ReactionMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2Muscarinic acetylcholine receptor M1AcetylcholineVasodilationEndocrinologyCholinergicCarbacholAdrenergic alpha-AgonistsAcetylcholinemedicine.drugInvestigative Opthalmology & Visual Science
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Identification of the Muscarinic Acetylcholine Receptor Subtype Mediating Cholinergic Vasodilation in Murine Retinal Arterioles

2011

To identify the muscarinic acetylcholine receptor subtype that mediates cholinergic vasodilation in murine retinal arterioles.Muscarinic receptor gene expression was determined in murine retinal arterioles using real-time PCR. To assess the functional relevance of muscarinic receptors for mediating vascular responses, retinal vascular preparations from muscarinic receptor-deficient mice were studied in vitro. Changes in luminal arteriole diameter in response to muscarinic and nonmuscarinic vasoactive substances were measured by video microscopy.Only mRNA for the M(3) receptor was detected in retinal arterioles. Thus, M(3) receptor-deficient mice (M3R(-/-)) and respective wild-type controls …

MaleNitroprussidemedicine.medical_specialtyNitric Oxide Synthase Type IIIRetinal ArteryVideo RecordingGene ExpressionBiologyReal-Time Polymerase Chain ReactionMuscle Smooth VascularMiceInternal medicineMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptormedicineMuscarinic acetylcholine receptor M4AnimalsRNA MessengerMice KnockoutReceptor Muscarinic M3Dose-Response Relationship DrugMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2ArticlesMuscarinic acetylcholine receptor M1AcetylcholineVasodilationArteriolesNG-Nitroarginine Methyl EsterEndocrinologyCholinergicCarbacholFemaleEndothelium VascularAcetylcholinemedicine.drugInvestigative Opthalmology & Visual Science
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Contribution of nitric oxide synthase isoforms to cholinergic vasodilation in murine retinal arterioles.

2013

Abstract Nitric oxide synthases (NOSs) are critically involved in regulation of ocular perfusion. However, the contribution of the individual NOS isoforms to vascular responses is unknown in the retina. Because some previous findings suggested an involvement of inducible nitric oxide synthase (iNOS) in the regulation of retinal vascular tone, a major goal of the present study was to examine the hypothesis that iNOS is involved in mediating cholinergic vasodilation responses of murine retinal arterioles. Another subject of this study was to test the contribution of the other two NOS isoforms, neuronal (nNOS) and endothelial NOS (eNOS), to cholinergic retinal arteriole responses. Expression o…

MaleNitroprussidemedicine.medical_specialtyNitric Oxide Synthase Type IIIVasodilator AgentsNitric Oxide Synthase Type IIVasodilationNitric Oxide Synthase Type IBiologyEndothelial NOSReal-Time Polymerase Chain ReactionGene Expression Regulation EnzymologicNitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundMiceInternal medicinemedicineAnimalsRNA MessengerEnzyme InhibitorsMice KnockoutBrainRetinal VesselsRetinalSensory SystemsAcetylcholineNitric oxide synthaseMice Inbred C57BLVasodilationOphthalmologyArteriolesEndocrinologyNG-Nitroarginine Methyl Esterchemistrybiology.proteinCholinergicmedicine.symptomVasoconstrictionAcetylcholinemedicine.drugExperimental eye research
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Role of M1, M3, and M5 muscarinic acetylcholine receptors in cholinergic dilation of small arteries studied with gene-targeted mice

2011

Acetylcholine regulates perfusion of numerous organs via changes in local blood flow involving muscarinic receptor-induced release of vasorelaxing agents from the endothelium. The purpose of the present study was to determine the role of M1, M3, and M5 muscarinic acetylcholine receptors in vasodilation of small arteries using gene-targeted mice deficient in either of the three receptor subtypes (M1R−/−, M3R−/−, or M5R−/− mice, respectively). Muscarinic receptor gene expression was determined in murine cutaneous, skeletal muscle, and renal interlobar arteries using real-time PCR. Moreover, respective arteries from M1R−/−, M3R−/−, M5R−/−, and wild-type mice were isolated, cannulated with mic…

MaleNitroprussidemedicine.medical_specialtyPhysiologyVasodilator AgentsVascular Biology and MicrocirculationSubstance PBiologyKidneyMicePhysiology (medical)Internal medicineMuscarinic acetylcholine receptormedicineAnimalsRNA MessengerMuscle SkeletalSkinAcetylcholine receptorMice KnockoutReceptor Muscarinic M3Receptor Muscarinic M5Dose-Response Relationship DrugReceptor Muscarinic M1Muscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2ArteriesMuscarinic acetylcholine receptor M1Interlobar arteriesAcetylcholineVasodilationmedicine.anatomical_structureEndocrinologyModels AnimalCholinergicCardiology and Cardiovascular MedicineAcetylcholinemedicine.drugAmerican Journal of Physiology-Heart and Circulatory Physiology
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The endocannabinoid system controls food intake via olfactory processes

2014

Comment in Sensory systems: the hungry sense. [Nat Rev Neurosci. 2014] Inhaling: endocannabinoids and food intake. [Nat Neurosci. 2014]; International audience; Hunger arouses sensory perception, eventually leading to an increase in food intake, but the underlying mechanisms remain poorly understood. We found that cannabinoid type-1 (CB1) receptors promote food intake in fasted mice by increasing odor detection. CB1 receptors were abundantly expressed on axon terminals of centrifugal cortical glutamatergic neurons that project to inhibitory granule cells of the main olfactory bulb (MOB). Local pharmacological and genetic manipulations revealed that endocannabinoids and exogenous cannabinoid…

MaleOlfactory systemMESH: Olfactory PerceptionCannabinoid receptorMESH: Feedback Physiological[SDV]Life Sciences [q-bio]medicine.medical_treatmentMESH: Cannabinoid Receptor AgonistsMESH: EndocannabinoidsMESH: Receptor Cannabinoid CB1Synaptic TransmissionMESH: Mice KnockoutMESH: EatingEatingMiceOlfactory bulbReceptor Cannabinoid CB1MESH: AnimalsFeedback PhysiologicalMice Knockoutmusculoskeletal neural and ocular physiologyGeneral Neurosciencedigestive oral and skin physiologyOlfactory PathwaysEndocannabinoid systemMESH: Feeding Behaviorlipids (amino acids peptides and proteins)psychological phenomena and processesMESH: Olfactory BulbBiologyInhibitory postsynaptic potentialGlutamatergicMESH: Mice Inbred C57BLMESH: Synaptic TransmissionmedicineAnimalsMESH: MiceCannabinoid Receptor AgonistsFeeding BehaviorOlfactory PerceptionMESH: MaleOlfactory bulbMice Inbred C57BLnervous systemOdorFeeding behaviourCannabinoid[SDV.AEN]Life Sciences [q-bio]/Food and NutritionNeuroscienceMESH: Olfactory PathwaysEndocannabinoidsNature Neuroscience
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Recognition of Neisseria meningitidis by the long pentraxin PTX3 and its role as an endogenous adjuvant.

2015

Long pentraxin 3 (PTX3) is a non-redundant component of the humoral arm of innate immunity. The present study was designed to investigate the interaction of PTX3 with Neisseria meningitidis. PTX3 bound acapsular meningococcus, Neisseria-derived outer membrane vesicles (OMV) and 3 selected meningococcal antigens (GNA0667, GNA1030 and GNA2091). PTX3-recognized microbial moieties are conserved structures which fulfil essential microbial functions. Ptx3-deficient mice had a lower antibody response in vaccination protocols with OMV and co-administration of PTX3 increased the antibody response, particularly in Ptx3-deficient mice. Administration of PTX3 reduced the bacterial load in infant rats c…

MaleOvalbuminGene Expressionlcsh:MedicineMeningococcal VaccinesMeningococcal vaccineMeningitis MeningococcalNeisseria meningitidismedicine.disease_causeMicrobiologyMice03 medical and health sciencesImmune systemAdjuvants ImmunologicAntigenImmunitymedicineAnimalsRats Wistarlcsh:Science030304 developmental biologyMice KnockoutAntigens Bacterial0303 health sciencesNeisseria meningitidis PTX3 vaccination protocolsMultidisciplinarybiology030306 microbiologyNeisseria meningitidisVaccinationlcsh:RPTX3Antibodies BacterialVirologyBacterial LoadImmunity InnateImmunity HumoralRats3. Good healthSerum Amyloid P-ComponentC-Reactive ProteinAnimals Newbornbiology.proteinFemalelcsh:QAntibodyBacterial outer membraneResearch ArticlePLoS ONE
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Gene Expression Profiling of Facilitated L-LTP in VP16-CREB Mice Reveals that BDNF Is Critical for the Maintenance of LTP and Its Synaptic Capture

2011

Expression of VP16-CREB, a constitutively active form of CREB, in hippocampal neurons of the CA1 region lowers the threshold for eliciting the late, persistent phase of long-term potentiation (L-LTP) in the Schaffer collateral pathway. This VP16-CREB-mediated L-LTP differs from the conventional late phase of LTP in not being dependent on new transcription. This finding suggests that in the transgenic mice the mRNA transcript(s) encoding the protein(s) necessary for this form of L-LTP might already be present in CA1 neurons in the basal condition. We used high-density oligonucleotide arrays to identify the mRNAs differentially expressed in the hippocampus of transgenic and wild-type mice. We…

MalePatch-Clamp TechniquesTime FactorsTransgeneNeuroscience(all)Long-Term PotentiationNerve Tissue ProteinsDynorphinHippocampal formationCREBHippocampusSynaptic TransmissionMiceNeurotrophic factorsMHC class ImedicineAnimalsRNA MessengerIn Situ HybridizationMice KnockoutNeuronsNeuronal PlasticitybiologyReverse Transcriptase Polymerase Chain ReactionBrain-Derived Neurotrophic FactorGene Expression Profilingmusculoskeletal neural and ocular physiologyGeneral NeuroscienceExcitatory Postsynaptic PotentialsHerpes Simplex Virus Protein Vmw65Long-term potentiationExonsCREB-Binding ProteinMolecular biologyCell biologymedicine.anatomical_structurenervous systemSchaffer collateralSynapsesbiology.proteinFemaleNeuron
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Bone marrow stroma CD40 expression correlates with inflammatory mast cell infiltration and disease progression in splenic marginal zone lymphoma

2014

Splenic marginal zone lymphoma (SMZL) is a mature B-cell neoplasm characterized by rather indolent clinical course. However, nearly one third of patients experience a rapidly progressive disease with a dismal outcome. Despite the characterization of clone geneticsandthe recognition of deregulated immunologic stimulation in the pathogenesis of SMZL, little is known about microenvironment dynamics and their potential biological influence on disease outcome. Here we investigate the effect of stroma-intrinsic features on SMZL disease progression by focusing on the microenvironment of the bone marrow (BM), which represents an elective disease localization endorsing diagnostic and prognostic rele…

MalePathologyBiochemistryMiceTumor MicroenvironmentMast CellMedicineMast CellsInflammation MediatorAged 80 and overMice KnockoutB-LymphocytesMice Inbred BALB CMesenchymal Stromal CellB-LymphocyteCD40 AntigenCell DifferentiationHematologyMiddle AgedPrognosismedicine.anatomical_structureDisease ProgressionCytokinesFemaleInflammation MediatorsClone (B-cell biology)HumanAdultmedicine.medical_specialtyStromal cellPrognosiCD40 LigandImmunologyDisease-Free SurvivalAnimalsHumansSplenic marginal zone lymphomaCD40 AntigensCytokineB cellAgedCell ProliferationAnimalbusiness.industryMesenchymal stem cellMesenchymal Stem CellsLymphoma B-Cell Marginal ZoneCell BiologyGenes p53medicine.diseaseLymphomaSplenic marginal zone lymphoma bone marrow microenvironment CD40Mast cell sarcomaBone marrowbusinessBlood
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