Search results for "L-lactate"

showing 10 items of 93 documents

Diversity of Lactobacillus species in deep carious lesions of primary molars

2010

AIM: This was to determine the prevalence of Lactobacilli (LB) species in different stages of caries progression and are considered as secondary invaders of existing carious lesions and specialists for caries progression. METHODS: Carious dentine samples were collected from 70 primary molars (M) during step-wise (S1, S2: n = 35 M) or one-step (O1: n = 35 M) caries treatment and after 11 months of temporary restorations (S3, O2). LB were identified by selected physiological and biochemical characteristics, ratio of lactic acid isomers, electrophoretic mobilities of lactic acid dehydrogenases, and shotgun mass mapping by MALDI mass spectrometry. RESULTS: LB were isolated from 46% of soft dent…

MolarDental CariesBacterial countsDental Pulp CappingMicrobiologyCalcium Hydroxidechemistry.chemical_compoundIsomerismstomatognathic systemHumansMedicineDentistry (miscellaneous)Electrophoretic mobilitiesLactic AcidTooth DeciduousLactobacillus speciesChildCaries treatmentL-Lactate DehydrogenaseLacticaseibacillus rhamnosusbusiness.industryfood and beveragesTemporary restorationMolarBacterial LoadLactic acidDental Restoration TemporaryLactobacillusstomatognathic diseasesCaries excavationchemistrySpectrometry Mass Matrix-Assisted Laser Desorption-IonizationDentinPediatrics Perinatology and Child HealthDisease ProgressionElectrophoresis Polyacrylamide GelDental Cavity PreparationbusinessPulp Capping and Pulpectomy AgentsFollow-Up StudiesEuropean Archives of Paediatric Dentistry
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Lactate Dehydrogenase Isozymes in the Developing Rat Brain

1962

THE immaturity and the lack of differentiation of the rat brain at birth has been shown some fifty years ago by Sugita1 in a classic paper on the postnatal morphogenesis of the brain in this animal. The sequence of physiological and biochemical changes which parallel the histological maturation of the rat brain has been investigated, however, only recently.

MultidisciplinaryL-Lactate DehydrogenaseMorphogenesisBrainL-Lactate dehydrogenaseBiologyRat brainIsozymeRatsIsoenzymeschemistry.chemical_compoundBiochemistrychemistryLactate dehydrogenaseAnimalsSequence (medicine)Nature
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Changes of energy metabolism, myosin light chain composition, lactate dehydrogenase isozyme pattern and fibre type distribution of denervated fast-tw…

1985

The influence of low frequency (8-10 Hz) electrical stimulation on denervated fast-twitch muscle from rabbit was investigated. Prolonged direct stimulation of denervated muscle resulted in higher oxidative enzyme activities. Furthermore, single fibre analyses for succinate dehydrogenase showed a more uniform distribution of activity in stimulated-denervated muscle when compared to normal muscle. As was also the case following stimulation of innervated muscle, glycolytic enzymes were decreased in activity and the LDH-isozyme pattern was also shifted towards heart type. No change of the myosin light chain pattern could be observed after 56 days of stimulation.

Myosin light-chain kinaseChemical PhenomenaPhysiologyClinical BiochemistryStimulationMyosinschemistry.chemical_compoundPhysiology (medical)Lactate dehydrogenaseMyosinAnimalsDenervationMuscle DenervationLagomorphaL-Lactate DehydrogenasebiologyHistocytochemistryChemistryMusclesSuccinate dehydrogenasebiology.organism_classificationElectric StimulationMuscle DenervationIsoenzymesChemistrybiology.proteinBiophysicsRabbitsEnergy MetabolismPfl�gers Archiv European Journal of Physiology
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c-erbB-2 expression in small-cell lung cancer is associated with poor prognosis.

2001

Small-cell lung cancer (SCLC) carries a bad prognosis despite good initial response to chemotherapy. It is therefore important to identify molecular markers that influence survival as potential new therapeutic targets. In our study, expression of the tyrosine kinase c-erbB-2 (HER2/neu) receptor in tumor tissues of 107 consecutive newly diagnosed patients with primary SCLC was quantified using a monoclonal antibody directed against the c-terminal domain of c-erbB-2. A clear-cut positive expression of c-erbB-2 was observed in 13% of patients. Surprisingly, c-erbB-2 was an independent prognostic factor (RR = 2.16; p = 0.014) when a proportional-hazard model was adjusted to stage (limited vs. e…

OncologyMaleCancer Researchmedicine.medical_specialtyPathologyLung NeoplasmsTime FactorsReceptor ErbB-2medicine.medical_treatmentSmall-cell carcinomaDisease-Free SurvivalSex FactorsInternal medicinemedicineCarcinomaHumansCarcinoma Small CellLung cancerneoplasmsAgedProportional Hazards ModelsChemotherapyPerformance statusL-Lactate DehydrogenaseProportional hazards modelbusiness.industryAge FactorsCancerAntibodies MonoclonalMiddle Agedmedicine.diseasePrognosisImmunohistochemistryProtein Structure TertiaryTreatment OutcomeOncologyPhosphopyruvate HydrataseImmunohistochemistryFemalebusinessInternational journal of cancer
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PPAR-γ Agonist GW1929 But Not Antagonist GW9662 Reduces TBBPA-Induced Neurotoxicity in Primary Neocortical Cells

2013

Tetrabromobisphenol A (2,2-bis(4-hydroxy-3,5-dibromophenyl)propane; TBBPA) is a widely used brominated flame retardant. TBBPA induces neuronal damage, but the mechanism by which this occurs is largely unknown. We studied the possible involvement of peroxisome proliferator-activated receptor gamma (PPAR-γ) in TBBPA-induced apoptosis and toxicity in mouse primary neuronal cell cultures. TBBPA enhanced both, caspase-3 activity and lactate dehydrogenase (LDH) release in neocortical cells after 6 and 24 h of exposition. These data were supported at the cellular level with Hoechst 33342 staining. Immunoblot analyses showed that, compared with control cells, 10 μM TBBPA decreased the expression of…

PPAR-γTime FactorsNeuroscience(all)Polybrominated BiphenylsPeroxisome proliferator-activated receptorGW1929Caspase 3ApoptosisNeocortexPharmacologyBiologyToxicologyNeuroprotectionBenzophenonesMicemedicineNeurotoxicityAnimalsAnilidesReceptorCells Culturedchemistry.chemical_classificationNeuronsDose-Response Relationship DrugL-Lactate DehydrogenaseCaspase 3General NeuroscienceNeurotoxicityApoptotic bodymedicine.diseasePPAR gammaTBBPANeuroprotective AgentschemistryCell cultureApoptosisTyrosineNeurotoxicity SyndromesOriginal ArticleCentral Nervous System AgentsNeurotoxicity Research
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The effect of β-mercaptoethanol on lactate dehydrogenase from rat brain

1963

Abstract Catalytic studies on lactate dehydrogenase from rat brain in the presence of β-mercaptoethanol have been performed. It has been found that pyruvate reduction and lactate dehydrogenation were differently affected by the addition of the sulfhydryl compound to the reaction mixture. The effect of β-mercaptoethanol on the catalyzed reaction has been shown to be dependent on the hydrogen ion concentration. When lactate dehydrogenation is measured, β-mercaptoethanol causes a net enhancement of the reaction rate at the more acidic pH values.

PharmacologyHydrogen ionL-Lactate DehydrogenaseResearchBrainL-Lactate dehydrogenaseRat brainBiochemistryRatsCatalysisReaction ratechemistry.chemical_compoundchemistryBiochemistryLactate dehydrogenaseDehydrogenationSulfhydryl CompoundsMercaptoethanolNuclear chemistryBiochemical Pharmacology
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Influence of carboxylic acids on the stereospecific nicotinamide adenine dinucleotide-dependent and nicotinamide adenine dinucleotide-independent lac…

1971

Leuconostoc mesenteroides increased its lactic acid production from glucose threefold when malic acid was added to the culture. This increase resulted also in a reduction of the ratio of d -lactic acid to l -lactic acid (31.5 to 1.23). Addition of malic acid increased 6.5-fold the specific activity of nicotinamide adenine dinucleotide (NAD)-linked l -lactate dehydrogenase and increased 3.2-fold that of NAD-linked d -lactate dehydrogenase. The Michaelis constant ( K m ) for NAD of the NAD-linked l -lactate dehydrogenase increased with the addition of malate, but no change was observed in the K m values for the respective d -enzyme. The effect of carboxylic acids on the NAD-linked l -lactate…

Physiology and MetabolismCarboxylic AcidsMalatesDehydrogenaseNicotinamide adenine dinucleotideBiologyMicrobiologyMalate dehydrogenasechemistry.chemical_compoundMolecular BiologyCell-Free SystemL-Lactate DehydrogenaseStereoisomerismElectrophoresis DiscNADMolecular biologyStimulation ChemicalLactic acidCulture MediaCitric acid cycleGlucosechemistryBiochemistryLactatesNAD+ kinaseBranched-chain alpha-keto acid dehydrogenase complexOxoglutarate dehydrogenase complexAcidsLeuconostocJournal of bacteriology
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Nicotinamide Adenine Dinucleotide-Dependent and Nicotinamide Adenine Dinucleotide-Independent Lactate Dehydrogenases in Homofermentative and Heterofe…

1971

Three homofermentative ( Lactobacillus plantarum B38, L. plantarum B33, Pediococcus pentosaceus B30) and three heterofermentative ( Leuconostoc mesenteroides 39, L. oenos B70, Lactobacillus brevis ) lactic acid bacteria were examined for the presence or absence of nicotinamide adenine dinucleotide (NAD)-dependent and NAD-independent d - and l -lactate dehydrogenases. Two of the six strains investigated, P. pentosaceus and L. oenos , did not exhibit an NAD-independent enzyme activity capable of reducing dichlorophenol indophenol. The p H optima of the lactic dehydrogenases were determined. The NAD-dependent enzymes from homofermentative strains exhibited optima at p H 7.8 to 8.8, whereas va…

Physiology and MetabolismNicotinamide adenine dinucleotideMicrobiologychemistry.chemical_compoundSpecies SpecificityLactobacillusChemical PrecipitationLeuconostocPediococcusProtaminesMolecular BiologyCell-Free SystemL-Lactate DehydrogenasebiologySulfatesLactobacillus brevisfood and beveragesStereoisomerismHydrogen-Ion ConcentrationNADbiology.organism_classificationCulture MediaLactic acidLactobacillusIndophenolBiochemistrychemistryAmmonium SulfateSpectrophotometryFermentationLactatesPediococcusNAD+ kinaseLeuconostocLactobacillus plantarumJournal of Bacteriology
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Dependence of enzyme reaction mechanism on protonation state of titratable residues and QM level description: lactate dehydrogenase

2005

We have studied the dependence of the chemical reaction mechanism of L-lactate dehydrogenase (LDH) on the protonation state of titratable residues and on the level of the quantum mechanical (QM) description by means of hybrid quantum-mechanical/molecular-mechanical (QM/MM) methods; this methodology has allowed clarification of the timing of the hydride transfer and proton transfer components that hitherto had not been possible to state definitively. Ferrer Castillo, Silvia, Silvia.Ferrer@uv.es, Silla Santos, Estanislao, Estanislao.Silla@uv.es ; Tuñon Garcia de Vicuña, Ignacio Nilo, Ignacio.Tunon@uv.es

ProtonStereochemistryUNESCO::QUÍMICATitratable acidDehydrogenaseProtonationChemical reactionQM/MM:QUÍMICA [UNESCO]CatalysisSubstrate Specificitychemistry.chemical_compoundComputational chemistryLactate dehydrogenaseMaterials ChemistryDependenceEnzyme reaction mechanismchemistry.chemical_classificationL-Lactate DehydrogenaseMolecular StructureChemistryHydrideUNESCO::QUÍMICA::Química analíticaMetals and AlloysTitrimetryGeneral ChemistryNADL-Lactate dehydrogenaseSurfaces Coatings and FilmsElectronic Optical and Magnetic MaterialsDependence ; Enzyme reaction mechanism ; Titratable residues ; L-Lactate dehydrogenase ; QM/MMEnzymeCeramics and Composites:QUÍMICA::Química analítica [UNESCO]Titratable residuesProtons
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Pterostilbene-induced tumor cytotoxicity: a lysosomal membrane permeabilization-dependent mechanism.

2012

The phenolic phytoalexin resveratrol is well known for its health-promoting and anticancer properties. Its potential benefits are, however, limited due to its low bioavailability. Pterostilbene, a natural dimethoxylated analog of resveratrol, presents higher anticancer activity than resveratrol. The mechanisms by which this polyphenol acts against cancer cells are, however, unclear. Here, we show that pterostilbene effectively inhibits cancer cell growth and stimulates apoptosis and autophagosome accumulation in cancer cells of various origins. However, these mechanisms are not determinant in cell demise. Pterostilbene promotes cancer cell death via a mechanism involving lysosomal membrane …

PterostilbeneCancer Treatmentlcsh:MedicineApoptosisResveratrolBiochemistryLung and Intrathoracic Tumorschemistry.chemical_compoundMolecular cell biologyRNA interferenceNeoplasmsPhagosomesStilbenesDrug DiscoveryBreast TumorsBasic Cancer Researchlcsh:ScienceCytotoxicitySkin TumorsApoptotic Signaling CascadeCellular Stress ResponsesMultidisciplinaryMicroscopy ConfocalCell DeathMalignant MelanomaFlow CytometryCellular StructuresSignaling CascadesCell biologyEukaryotic CellsOncologyCaspasesMedicineCellular TypesCell DivisionResearch ArticleSignal TransductionProgrammed cell deathDrugs and DevicesDrug Research and DevelopmentMitosisAntineoplastic AgentsBiologyPermeabilityCell GrowthInhibitory Concentration 50NecrosisComplementary and Alternative MedicineCell Line TumorGastrointestinal TumorsAutophagyHumansHSP70 Heat-Shock ProteinsBiologyCell ProliferationDose-Response Relationship DrugL-Lactate DehydrogenaseCell growthlcsh:RAutophagyProteinsCancers and NeoplasmsRegulatory ProteinschemistrySubcellular OrganellesApoptosisResveratrolCancer celllcsh:QGene expressionLysosomesCytometryPloS one
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