Search results for "Lymph"

showing 10 items of 4590 documents

Trimeric Hemibastadin Congener from the Marine Sponge Ianthella basta

2012

The first naturally occurring trimeric hemibastadin congener, sesquibastadin 1 (1), and the previously reported bastadins 3, 6, 7, 11, and 16 (2-6) were isolated from the marine sponge Ianthella basta, collected in Indonesia. The structure of 1 was elucidated on the basis of 1D and 2D NMR measurements and by HRMS. Among all the isolated compounds, the linear sesquibastadin 1 (1) and bastadin 3 (2) showed the strongest inhibition rates for at least 22 protein kinases (IC(50) = 0.1-6.5 μM), while the macrocyclic bastadins (3-6) demonstrated a strong cytotoxic potential against the murine lymphoma cell line L5178Y (IC(50) = 1.5-5.3 μM).

StereochemistryPharmaceutical ScienceMarine BiologySesquibastadinAnalytical ChemistryMiceIanthella bastaDrug DiscoveryHalogenated Diphenyl EthersAnimalsNuclear Magnetic Resonance BiomolecularProtein Kinase InhibitorsPharmacologyMolecular StructurebiologyMurine lymphomaOrganic Chemistrybiology.organism_classificationPoriferaSpongeCongenerComplementary and alternative medicineIndonesiaMolecular MedicineDrug Screening Assays AntitumorTwo-dimensional nuclear magnetic resonance spectroscopyJournal of Natural Products
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Mutations affecting MHC class II binding of the superantigen streptococcal erythrogenic toxin A.

1993

Streptococcal pyrogenic exotoxin A (SPEA) is an important pathogenicity factor of group A streptococci. It is a member of the family of 'superantigens' produced by Staphylococcus aureus and Streptococcus pyogenes, and its T lymphocyte stimulating activity is involved in the pathogenesis of certain diseases caused by pyogenic streptococci. In this study we have generated nine mutant SPEA molecules by substituting amino acids in the regions of homology between different streptococcal and staphylococcal superantigens. An additional mutant was created by deletion of the 10 N-terminal amino acids. The mutants were expressed as fusion proteins. Several mutations led to a loss of function due to a…

Streptococcus pyogenesT-LymphocytesImmunologyMutantMolecular Sequence DataExotoxinsBiologymedicine.disease_causeLymphocyte ActivationMicrobiologyMiceStructure-Activity Relationshipstomatognathic systemBacterial ProteinsSuperantigenmedicineImmunology and AllergyAnimalsHumansAmino Acid SequencePeptide sequencechemistry.chemical_classificationMice Inbred BALB CSuperantigensBase SequenceHistocompatibility Antigens Class IIMembrane ProteinsGeneral Medicinebiology.organism_classificationFusion proteinAmino acidchemistrySpeaStreptococcus pyogenesMutationExotoxinInternational immunology
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Role of beta3-adrenergic receptors in intestinal diseases

2022

The Inflammatory Bowel Diseases (IBD), characterized by a gastro-intestinal tract inflammation, are a global health-care problem due to their morbidity, mortality and the constant increase in their worldwide incidence. The available therapies are still not curative only providing temporary symptomatic relief and reduction in complications.Although etiology is still unclear, IBD results in an interplay of various factors among which impaired and/or inappropriate immune response toward intestinal bacteria. Increasing disease incidence link environmental triggers occurring during socioeconomic development to IBD, especially stress. In this work, we aimed to investigated the role of adrenergic …

Stress oxydantOxidative stress[SDV.OT] Life Sciences [q-bio]/Other [q-bio.OT]MacrophagesIbdAdrb3LymphocytesMici
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Human interleukin-4 enhances stromal cell-dependent hematopoiesis: costimulation with stem cell factor

1994

Abstract Interleukin-4 (IL-4) has distinct hematopoietic activities, primarily as a costimulant with other cytokines to enhance colony formation of hematopoietic progenitors. We investigated the influence of IL-4 on stromal cell-supported long-term cultures (LTCs) of normal human bone marrow. Addition of IL-4 to LTCs of unseparated bone marrow or highly enriched CD34+ cells resulted in a significant increase of myeloid progenitors in the nonadherent, as well as in the stromal cell-adherent cell populations. In contrast, the total cell number was not influenced by IL-4, suggesting a selective effect on primitive progenitor cells. Cord blood cells or CD34+ bone marrow cells were incubated wit…

Stromal cellImmunologyCD34Stem cell factorCell BiologyHematologyBiologyBiochemistryCell biologyEndothelial stem cellImmunologyLymph node stromal cellProgenitor cellInterleukin 3Adult stem cellBlood
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Stromal niche communalities underscore the contribution of the matricellular protein SPARC to B-cell development and lymphoid malignancies

2014

Neoplastic B-cell clones commonly arise within secondary lymphoid organs (SLO). However, during disease progression, lymphomatous cells may also colonize the bone marrow (BM), where they localize within specialized stromal niches, namely the osteoblastic and the vascular niche, according to their germinal center-or extra-follicular-derivation, respectively. We hypothesized the existence of common stromal motifs in BM and SLO B-cell lymphoid niches involved in licensing normal B-cell development as well as in fostering transformed B lymphoid cells. Thus, we tested the expression of prototypical mesenchymal stromal cell (MSC) markers and regulatory matricellular proteins in human BM and SLO u…

Stromal cellImmunologylymphomalymphomasBiologybone marrow nicheB cell development; SPARC; bone marrow niches; lymphomas; microenvironmentStromaB cell developmentmedicineImmunology and AllergyLymphopoiesisB cellOriginal ResearchMesenchymal stem cellMatricellular proteinGerminal centerSPARCmedicine.diseasemicroenvironmentLymphomamedicine.anatomical_structureOncologyImmunologyCancer researchbone marrow nichesOncoImmunology
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Microenvironment-centred dynamics in aggressive B-cell lymphomas.

2012

Aggressive B-cell lymphomas share high proliferative and invasive attitudes and dismal prognosis despite heterogeneous biological features. In the interchained sequence of events leading to cancer progression, neoplastic clone-intrinsic molecular events play a major role. Nevertheless, microenvironment-related cues have progressively come into focus as true determinants for this process. The cancer-associated microenvironment is a complex network of nonneoplastic immune and stromal cells embedded in extracellular components, giving rise to a multifarious crosstalk with neoplastic cells towards the induction of a supportive milieu. The immunological and stromal microenvironments have been cl…

Stromal cellMicroenvironmentHematologyReview ArticleBiologyMicroenvironment; aggressive B-cell lymphomasCrosstalk (biology)Immune systemmedicine.anatomical_structureImmunologymedicineDiseases of the blood and blood-forming organsRC633-647.5Indolent lymphomasB cellaggressive B-cell lymphomas
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REGULATORY ELEMENTS OF THE LEUKAEMIA INHIBITORY FACTOR (LIF) PROMOTER IN MURINE BONE MARROW STROMAL CELLS

1999

Leukaemia inhibitory factor (LIF) plays an important role as a haematopoietically active cytokine. As described earlier in a murine model, interleukin 1 (IL-1) induced LIF mRNA and protein expression. We utilized the murine cell line +/+-1.LDA11 to further define regulatory mechanisms of LIF expression in bone marrow stromal cells. The production of LIF mRNA is stimulated by IL-1beta, TNF-alpha, and the cAMP analogue 8-bromoadenosine 3':5'-monophosphate (8BrcAMP). LIF mRNA expression is controlled at the transcriptional level. Different fragments from -542 to -45 bp 5' upstream of the transcriptional start site of the murine LIF gene were fused to the luciferase gene. All LIF-promoter lucif…

Stromal cellRecombinant Fusion Proteinsmedicine.medical_treatmentImmunology8-Bromo Cyclic Adenosine MonophosphateBone Marrow CellsStimulationRegulatory Sequences Nucleic AcidBiologyLeukemia Inhibitory FactorBiochemistryMiceGenes ReportermedicineAnimalsHumansImmunology and AllergyLuciferaseRNA MessengerNuclear proteinPromoter Regions GeneticMolecular BiologyCells CulturedLymphokinesMessenger RNAInterleukin-6Tumor Necrosis Factor-alphaInterleukinHematologyMolecular biologyGrowth InhibitorsRecombinant ProteinsCytokinemedicine.anatomical_structureGene Expression RegulationBone marrowStromal CellsInterleukin-1Cytokine
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Intra-tumor heterogeneity of Diffuse Large B-cell Lymphoma involves the induction of diversified stroma-tumor interfaces

2020

ABSTRACTIntra-tumor heterogeneity in lymphoid malignancies is articulated around several fundamentals, encompassing selection of genetic subclonal events and epigenetic regulation of transcriptional programs. Clonally-related neoplastic cell populations are unsteadily subjected to immune editing and metabolic adaptations within different tissue microenvironments. How tissue-intrinsic mesenchymal determinants impact on the diversification of aggressive lymphomas is still unknown. In this study we adopted the established A20 line-based model of Diffuse Large B-cell Lymphoma (DLBCL), to investigate the intra-tumor heterogeneity associated with the infiltration of different tissue microenvironm…

Stromal cellStromahemic and lymphatic diseasesMatricellular proteinMesenchymal stem cellmedicineCancer researchNeoplastic cellEpigeneticsBiologymedicine.diseaseDiffuse large B-cell lymphomaLymphoma
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The thymus at the crossroad of neuroimmune interactions.

2006

The numerous relationships existing between the nervous and the immune systems suggest that the neural networks present in the intrathymic microenvironment may influence T-cell development. We previously reported that thymic neural-crest-derived stromal cells are involved in a neural differentiation pathway and are able to produce neurotrophic factors and neurokines that are in turn able to increase and/or modulate thymic-stromal cell neuronal phenotype. We also showed that EGF promotes a neural phenotype in thymic epithelial cells by enhancing the expression of neuronal-specific markers, neurotransmitters, and neuropoietic cytokines, such as IL-6 and CNTF. More recently we showed that the …

Stromal cellbiologyNeuroimmunomodulationGeneral NeuroscienceT-LymphocytesThymus GlandCiliary neurotrophic factorCell fate determinationPhenotypeGeneral Biochemistry Genetics and Molecular BiologyCell biologyThymocyteImmune systemHistory and Philosophy of ScienceNeurotrophic factorsImmunologybiology.proteinAnimalsHumansNeurotrophinAnnals of the New York Academy of Sciences
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Evidence against a key role for transforming growth factor-beta1 in cytomegalovirus-induced bone marrow aplasia.

1998

During immunodeficiency after sublethal haematoablative treatment, cytomegalovirus (CMV) infection interferes with haematopoietic reconstitution and can cause lethal bone marrow (BM) aplasia. The in vivo model of murine CMV infection has identified the BM stroma as the principal target site of CMV in the haematopoietic cord. The infected cell type is the reticular stromal cell which forms the stromal network and produces essential haemopoietins, such as stem-cell factor (SCF). The expression of SCF was found to be reduced in the infected stroma, but the stromal network was not disrupted and the number of infected stromal cells was too low to explain the functional deficiency. These facts ca…

Stromal cellmedicine.medical_treatmentCytomegalovirusGene ExpressionBone Marrow CellsBone Marrow AplasiaCD8-Positive T-LymphocytesKidneyVirus ReplicationMiceTransforming Growth Factor betaVirologymedicineAnimalsCytotoxic T cellBone Marrow DiseasesBone Marrow TransplantationMice Inbred BALB CbiologyTransforming growth factor betaVirologyHematopoiesisHaematopoiesisCytokinemedicine.anatomical_structureLiverCytomegalovirus Infectionsbiology.proteinFemaleImmunotherapyBone marrowStromal CellsTransforming growth factorJournal of General Virology
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