Search results for "M2"

showing 10 items of 256 documents

Murine embryonic stem cell line CGR8 expresses all subtypes of muscarinic receptors and multiple nicotinic receptor subunits: Down-regulation of α4- …

2015

Non-neuronal acetylcholine mediates its cellular effects via stimulation of the G-protein-coupled muscarinic receptors and the ligand-gated ion channel nicotinic receptors. The murine embryonic stem cell line CGR8 synthesizes and releases non-neuronal acetylcholine. In the present study a systematic investigation of the expression of nicotinic receptor subunits and muscarinic receptors was performed, when the stem cells were grown in the presence or absence of LIF, as the latter condition induces early differentiation. CGR8 cells expressed multiple nicotinic receptor subtypes (α3, α4, α7, α9, α10, β1, β2, β3, β4, γ, δ, e) and muscarinic receptors (M1, M3, M4, M5); M2 was detected only in 2 …

PharmacologyImmunologyMuscarinic acetylcholine receptor M3Down-RegulationMuscarinic acetylcholine receptor M2Cell DifferentiationMuscarinic acetylcholine receptor M1BiologyReceptors NicotinicReceptors MuscarinicCell biologyCell LineMiceProtein SubunitsNicotinic agonistGanglion type nicotinic receptorGene Expression RegulationMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptorImmunology and AllergyAnimalsAlpha-4 beta-2 nicotinic receptorEmbryonic Stem CellsInternational immunopharmacology
researchProduct

Acetylcholine release at motor endplates and autonomic neuroeffector junctions: a comparison.

1996

Acetylcholine released at motor endplates and at autonomic neuroeffector junctions binds to nicotinic and muscarinic receptors to affect the activity of the corresponding target cells. Additionally, nicotonic and muscarinic receptors modulate various intracellular regulatory pathways (second messengers, gene expression) and mediate trophic effects. To maintain homeostasis of the individual cell and of the whole organism the release of acetylcholine has to be strictly controlled within both nervous systems. The basic events of synthesis, storage, and release are comparable at motoneurones and autonomic neurones, but mechanisms regulating transmitter release appear to differ. The motor endpla…

PharmacologyMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2BiologyMotor EndplateReceptors MuscarinicAcetylcholineNeuroeffector junctionNicotinic agonistMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptorMuscarinic acetylcholine receptor M4medicineNeuroeffector JunctionAnimalsNeuroscienceAcetylcholinemedicine.drugPharmacological research
researchProduct

Non-neuronal acetylcholine, a locally acting molecule, widely distributed in biological systems: expression and function in humans.

1998

Acetylcholine acts as a neurotransmitter in the central and peripheral nervous systems in humans. However, recent experiments demonstrate a widespread expression of the cholinergic system in non-neuronal cells in humans. The synthesizing enzyme choline acetyltransferase, the signalling molecule acetylcholine, and the respective receptors (nicotinic or muscarinic) are expressed in epithelial cells (human airways, alimentary tract, epidermis). Acetylcholine is also found in mesothelial, endothelial, glial, and circulating blood cells (platelets, mononuclear cells), as well as in alveolar macrophages. The existence of non-neuronal acetylcholine explains the widespread expression of muscarinic …

Pharmacologymedicine.medical_specialtyMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2BiologyAcetylcholineCell biologyCholine O-AcetyltransferaseCircadian RhythmEndocrinologyNicotinic agonistInternal medicineMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptormedicineMuscarinic acetylcholine receptor M4CholinergicHumansPharmacology (medical)Acetylcholinemedicine.drugPharmacologytherapeutics
researchProduct

Pho85 and PI(4,5)P(2) regulate different lipid metabolic pathways in response to cold

2019

Lipid homeostasis allows cells to adjust membrane biophysical properties in response to changes in environmental conditions. In the yeast Saccharomyces cerevisiae, a downward shift in temperature from an optimal reduces membrane fluidity, which triggers a lipid remodeling of the plasma membrane. How changes in membrane fluidity are perceived, and how the abundance and composition of different lipid classes is properly balanced, remain largely unknown. Here, we show that the levels of phosphatidylinositol 4,5-bisphosphate [PI(4,5)P2], the most abundant plasma membrane phosphoinositide, drop rapidly in response to a downward shift in temperature. This change triggers a signaling cascade trans…

Phosphatidylinositol 45-DiphosphateSaccharomyces cerevisiae ProteinsMembrane FluiditySphingoid basesAcclimatizationOrm2PhospholipidSaccharomyces cerevisiaePhosphoinositideTriacylglycerideSphingolipidArticle03 medical and health scienceschemistry.chemical_compoundGlycogen Synthase Kinase 3Gene Expression Regulation FungalMembrane fluidityLow temperatureInositolPhosphatidylinositolProtein kinase AMolecular Biology1-IP7030304 developmental biology0303 health sciencesChemistry030302 biochemistry & molecular biologyCell MembraneCell BiologyLipid MetabolismSphingolipidCyclin-Dependent KinasesCell biologyTORC2-Pkh1-Ypk1 signaling moduleCold TemperatureCytosolMetabolic pathwayPhospholipidMetabolic Networks and PathwaysSignal Transduction
researchProduct

Search for multiwavelength emission from the binary millisecond pulsar PSR J1836-2354A in the globular cluster M22

2019

We present a multi-band search for X-ray, optical and $\gamma$-ray emission of the radio binary millisecond pulsar J1836-2354A, hosted in the globular cluster M22. X-ray emission is significantly detected in two Chandra observations, performed in 2005 and 2014, at a luminosity of $\sim$2-3$\times$10$^{30}$ erg s$^{-1}$, in the 0.5-8 keV energy range. The radio and the X-ray source positions are found consistent within 1$\sigma$ error box. No detection is found in archival XMM-Newton and Swift/XRT observations, compatible with the Chandra flux level. The low statistics prevents us to assess if the X-ray source varied between the two observations. The X-ray spectrum is consistent with a power…

PhotonAstrophysics::High Energy Astrophysical PhenomenaFOS: Physical sciencesBinary numberFluxAstrophysicsAstrophysics::Cosmology and Extragalactic Astrophysics01 natural sciencesLuminosityX-rays: binariesMillisecond pulsarpulsars: general0103 physical sciences010303 astronomy & astrophysicsX-rays: individual: PSR J1836-2354AAstrophysics::Galaxy AstrophysicsHigh Energy Astrophysical Phenomena (astro-ph.HE)Physics010308 nuclear & particles physicsglobular clusters: individual: M22 (NGC 6656)Astronomy and AstrophysicsX-rays: binarie13. Climate actionSpace and Planetary ScienceGlobular clusterNo detectionAstrophysics - High Energy Astrophysical Phenomena[PHYS.ASTR]Physics [physics]/Astrophysics [astro-ph]Energy (signal processing)
researchProduct

Mortality risk attributable to wildfire-related PM2·5 pollution: a global time series study in 749 locations

2021

Summary Background Many regions of the world are now facing more frequent and unprecedentedly large wildfires. However, the association between wildfire-related PM2·5 and mortality has not been well characterised. We aimed to comprehensively assess the association between short-term exposure to wildfire-related PM2·5 and mortality across various regions of the world. Methods For this time series study, data on daily counts of deaths for all causes, cardiovascular causes, and respiratory causes were collected from 749 cities in 43 countries and regions during 2000–16. Daily concentrations of wildfire-related PM2·5 were estimated using the three-dimensional chemical transport model GEOS-Chem …

PollutionHealth (social science)all cause mortalitymedia_common.quotation_subjectPopulationMedicine (miscellaneous)610 Medicine & healthPM2.5medical researchwildfirehealth hazard360 Social problems & social servicescardiovascular mortalityEnvironmental healthMedicinecontrolled studyhumaneducation610 Medicine & healthMortality riskCardiovascular mortalitymedia_commonSeries (stratigraphy)education.field_of_studybusiness.industryHealth Policypublic healthPublic Health Environmental and Occupational Healtharticlerisk assessmentPublic Health Global Health Social Medicine and Epidemiologyshort term exposurePollutionFolkhälsovetenskap global hälsa socialmedicin och epidemiologiIncreased riskrisk factorcityRelative risktime series analysisAttributable riskPM 2·5 Pollutionmortality riskDeterminantes da Saúde e da DoençaGenotoxicidade Ambientalbusiness360 Social problems & social servicesGlobal timemeta analysis
researchProduct

Molecular Alterations of the RBI, TP53, and MDM2 Genes in Primary and Xenografted Human Osteosarcomas

1997

We report the status of the RBI, TP53, and MDM2 genes in human osteosarcomas and cell lines established from surgical specimens and transplanted into athymic naked mice. By using reverse transcriptase-polymerase chain reaction (RT-PCR) as a prescreening technique and posterior sequencing, we observe

Primary (chemistry)Cell cultureCancer researchbiology.proteinMdm2Cell BiologyBiologyneoplasmsMolecular BiologyGenePathology and Forensic MedicineDiagnostic Molecular Pathology
researchProduct

Molecular dynamics studies on Mdm2 complexes: An analysis of the inhibitor influence

2012

p53 is a powerful anti-tumoral molecule frequently inactivated by mutations or deletions in cancer. However, half of all human tumors expresses wild-type p53, and its activation, by antagonizing its negative regulator Mdm2, might offer a new strategy for therapeutic protocol. In this work, we present a molecular dynamics study on Mdm2 structure bound to two different known inhibitors with the aim to investigate the structural transitions between apo-Mdm2 and Mdm2-inhibitor complexes. We tried to gain information about conformational changes binding a benzodiazepine derivative inhibitor with respect the known nutlin and the apo form. The conformational changes alter the size of the cleft and…

Protein ConformationBiophysicsMolecular Dynamics SimulationMdm2 p53 nutlin benzodiazepine Molecular DynamicsBiochemistryNegative regulatorBenzodiazepineschemistry.chemical_compoundMolecular dynamicsHumansMoleculeEnzyme InhibitorsMolecular BiologyBinding SitesbiologyChemistryProto-Oncogene Proteins c-mdm2Cell BiologyNutlinSettore CHIM/08 - Chimica FarmaceuticaBiochemistryDrug DesignBiophysicsbiology.proteinMdm2LinkerBiochemical and Biophysical Research Communications
researchProduct

Muscarinic M2 receptors in acetylcholine-isoproterenol functional antagonism in human isolated bronchus

2002

The muscarinic functional antagonism of isoproterenol relaxation and the contribution of muscarinic M2 receptors were examined in human isolated bronchus. In intact tissues, acetylcholine (ACh) precontraction decreased isoproterenol potency and maximal relaxation (−log EC50 shift = −1.49 ± 0.16 and Emax inhibition for 100 μM ACh = 30%) more than the same levels of histamine contraction. The M2receptor-selective antagonist methoctramine (1 μM) reduced this antagonism in ACh- but not histamine-contracted tissues. Similar results were obtained for forskolin-induced relaxation. After selective inactivation of M3 receptors with 4-diphenylacetoxy- N-(2-chloroethyl)piperadine hydrochloric acid (3…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyLung NeoplasmsPhysiologyMuscle RelaxationBronchiMuscarinic AntagonistsIn Vitro Techniqueschemistry.chemical_compoundPiperidinesPhysiology (medical)Internal medicineIsoprenalineMuscarinic acetylcholine receptorCyclic AMPmedicineMethoctramineHumansNeurotransmitterAcetylcholine receptorReceptor Muscarinic M2BronchusColforsinIsoproterenolMuscle SmoothMuscarinic acetylcholine receptor M2Cell BiologyReceptors MuscarinicAcetylcholinemedicine.anatomical_structureEndocrinologychemistryAcetylcholineMuscle Contractionmedicine.drugAmerican Journal of Physiology-Lung Cellular and Molecular Physiology
researchProduct

Finite type invariants of knots in homology 3-spheres with respect to null LP-surgeries

2017

We study a theory of finite type invariants for null-homologous knots in rational homology 3-spheres with respect to null Lagrangian-preserving surgeries. It is an analogue in the setting of the rational homology of the Goussarov-Rozansky theory for knots in integral homology 3-spheres. We give a partial combinatorial description of the graded space associated with our theory and determine some cases when this description is complete. For null-homologous knots in rational homology 3-spheres with a trivial Alexander polynomial, we show that the Kricker lift of the Kontsevich integral and the Lescop equivariant invariant built from integrals in configuration spaces are universal finite type i…

Pure mathematicsAlexander polynomialPrimary: 57M27Homology (mathematics)01 natural sciencesHomology sphereMathematics::Algebraic TopologyMathematics - Geometric TopologyKnot (unit)Mathematics::K-Theory and Homologybeaded Jacobi diagramknot[MATH.MATH-GT]Mathematics [math]/Geometric Topology [math.GT]0103 physical sciencesFOS: Mathematics0101 mathematicsInvariant (mathematics)Mathematics::Symplectic Geometry3-manifoldhomology sphereMathematicsBorromean surgerycalculus010102 general mathematicsGeometric Topology (math.GT)Kontsevich integral16. Peace & justiceMathematics::Geometric TopologymanifoldsFinite type invariantnull-move57M27Finite type invariantLagrangian-preserving surgeryEquivariant map010307 mathematical physicsGeometry and Topology3-manifold
researchProduct