Search results for "MESH : Male"

showing 10 items of 84 documents

Discrete and effortful imagined movements do not specifically activate the autonomic nervous system.

2009

International audience; BACKGROUND: The autonomic nervous system (ANS) is activated in parallel with the motor system during cyclical and effortful imagined actions. However, it is not clear whether the ANS is activated during motor imagery of discrete movements and whether this activation is specific to the movement being imagined. Here, we explored these topics by studying the baroreflex control of the cardiovascular system. METHODOLOGY/PRINCIPAL FINDINGS: Arterial pressure and heart rate were recorded in ten subjects who executed or imagined trunk or leg movements against gravity. Trunk and leg movements result in different physiological reactions (orthostatic hypotension phenomenon) whe…

MaleMESH : MovementPhysiologyBlood PressureMESH: MovementElectromyographyMESH : Autonomic Nervous SystemMESH: Autonomic Nervous SystemOrthostatic vital signs0302 clinical medicineHeart RateNervous System Physiological PhenomenaMESH: Heart RateMESH: Nervous System Physiological Phenomena0303 health sciencesNeuroscience/Behavioral NeuroscienceMultidisciplinarymedicine.diagnostic_testMovement (music)QRMESH: Blood PressureAnatomyMESH : AdultMESH : Nervous System Physiological PhenomenaMESH : ElectromyographyMedicine[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Research ArticleAdultScienceMovementMESH : MaleBaroreflexBiologyAutonomic Nervous SystemMESH: Electromyography03 medical and health sciencesMotor imageryMotor systemmedicineMESH : Blood PressureHumans[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]030304 developmental biologyNeuroscience/Cognitive NeuroscienceMESH: HumansElectromyographyMESH : Heart RateMESH : HumansMESH: AdultTrunkMESH: MaleAutonomic nervous system[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neuroscience030217 neurology & neurosurgeryNeuroscience
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Grape seed and skin extract reduces pancreas lipotoxicity, oxidative stress and inflammation in high fat diet fed rats.

2016

IF 2.326; International audience; Obesity is related to an elevated risk of diabetes and the mechanisms whereby fat adversely affects the pancreas are poorly understood. We studied the effect of a high fat diet (HFD) on pancreas steatosis, oxidative stress and inflammation as well as the putative protection afforded by grape seed and skin extract (GSSE). HFD induced body weight gain, without affecting insulinemia, nor glycemia and dropped adiponectemia. HFD also provoked the ectopic deposition of cholesterol and triglyceride, and an oxidative stress characterized by increased lipoperoxidation and carbonylation, inhibition of antioxidant enzyme activities such as CAT, GPx and SOD, depletion …

MaleMESH : Oxidative Stress0301 basic medicineMESH: InflammationAntioxidantmedicine.medical_treatmentMESH: Grape Seed Extractmedicine.disease_causeAntioxidantschemistry.chemical_compound0302 clinical medicineMESH: ObesityVitisMESH: AnimalsMESH : Pancreas2. Zero hungerMESH: Oxidative StressMESH : Grape Seed ExtractMESH : RatsMESH : Diet High-Fatfood and beveragesGeneral MedicineMESH: Pancreas3. Good healthLipotoxicity030220 oncology & carcinogenesisSeedsMESH : AntioxidantsMESH : Obesitymedicine.symptommedicine.medical_specialtyGrape seed and skin extractMESH: RatsMESH : MaleInflammationBiologyDiet High-FatMESH : Rats WistarMESH: VitisMESH : Vitis03 medical and health sciencesDiabetes mellitusInternal medicinemedicineAnimalsObesityRats WistarPancreasPharmacologyInflammationMESH : InflammationGrape Seed ExtractTriglycerideCholesterolMESH: Antioxidants[ SDV.SP.PHARMA ] Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologyMESH: Rats Wistarmedicine.diseaseMESH : SeedsMESH: MaleRatsMESH: Diet High-Fat030104 developmental biologyEndocrinologychemistryMESH: SeedsOxidative stress[SDV.SP.PHARMA]Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologyMESH : AnimalsSteatosisOxidative stress
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General oxidative stress during doxorubicin-induced cardiotoxicity in rats: Absence of cardioprotection and low antioxidant efficiency of alpha-lipoi…

2012

International audience; To evaluate the effects of alpha-lipoic acid (AL) in a model of doxorubicin (DOX)-induced cardiotoxicity, male Wistar rats were treated with DOX (1 mg/kg/d; 10 d) in combination or not with AL (50 mg/kg/d; 15 d). Plasma oxidative stress was determined by hydroperoxides (ROOH) and the ascorbyl radical/ascorbate ratio. One and two months later, the functional parameters of the hearts were determined in vivo by catheterization and cardiac oxidative stress was assessed by malonedialdehyde (MDA) and O₂*⁻ (dihydroethidium fluorescence) content in tissue. After two months, body weight was higher in the DOX-AL group than in DOX (+16%), but this was due to ascites. Histologic…

MaleMESH : Oxidative StressAntioxidantmedicine.medical_treatmentMESH : HematocritMESH : Thioctic AcidBiochemistryAntioxidants0302 clinical medicineSuperoxidesAscitic FluidMESH: AnimalsMESH : Body WeightComputingMilieux_MISCELLANEOUS0303 health sciencesThioctic AcidCumulative doseMESH: Heart DiseasesHeartGeneral Medicine3. Good healthMESH: Ascitic Fluid[SDV] Life Sciences [q-bio]030220 oncology & carcinogenesisMESH : Ascitic FluidMESH: Hydrogen PeroxideMESH : AntioxidantsMESH: Thioctic Acidmedicine.medical_specialtyCardiotonic AgentsCardiotoxinsMESH: Hematocrit03 medical and health sciencesMESH: Doxorubicin[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemIn vivoRats Wistar[ SDV ] Life Sciences [q-bio]MyocardiumMESH: AntioxidantsHydrogen PeroxideMESH: Cardiotonic AgentsMESH : Organ SizeMESH: Body WeightMESH: Heartcarbohydrates (lipids)EndocrinologyMESH: LiverMESH : SuperoxidesMESH: Organ Size[SDV]Life Sciences [q-bio]MESH : Cardiotonic AgentsAscorbic AcidMESH: Superoxidesmedicine.disease_causeMESH: EatingEatingpolycyclic compoundsMESH : MyocardiumMESH: Thiobarbituric Acid Reactive SubstancesMESH: Ascorbic AcidAntibiotics AntineoplasticMESH: Oxidative StressChemistryMESH : RatsOrgan SizeMESH : Antibiotics Antineoplastic[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemBiochemistryHematocritLiverMESH : Cardiotoxinsmedicine.drugMESH : EatingMESH: MyocardiumHeart DiseasesMESH: RatsMESH : MaleMESH : Thiobarbituric Acid Reactive SubstancesMESH : Rats WistarThiobarbituric Acid Reactive SubstancesContractilityMESH : HeartInternal medicinemedicineTBARSAnimalsMESH : DoxorubicinDoxorubicinMESH: Antibiotics AntineoplasticMESH : Ascorbic Acid030304 developmental biologyCardiotoxicityBody WeightMESH : LiverMESH : Heart DiseasesMESH: Rats WistarMESH: MaleRatsOxidative StressMESH: CardiotoxinsDoxorubicinMESH : AnimalsMESH : Hydrogen PeroxideOxidative stress
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Smoking and FOS expression from blood leukocyte transcripts in patients with coronary artery disease.

2011

International audience; OBJECTIVE: Analysis of the leukocyte transriptome, in particular the Finkel-Biskis-Jinkins Osteosarcoma (c-Fos) gene, which has a prominent role in inflammation, provides new insights into atherosclerosis mechanisms. Although smoking is a major risk factor, the links between smoking status and coronary artery disease (CAD) remains unclear. We aimed to analyze the relationship between smoking status and c-Fos expression in circulating leukocytes of patients with CAD. METHODS: c-Fos expression was measured by RT-Q-PCR, from blood leukocytes of 239 consecutive patients after acute myocardial infarction (MI). The patients were asked about their smoking status and stratif…

MaleMESH : RNA MessengerMESH: Chi-Square DistributionMESH : LeukocytesMESH : Prospective StudiesMESH : AgedMyocardial InfarctionSmoking PreventionMESH: Risk Assessmentc-FosMESH : Coronary Angiography0302 clinical medicineMESH : Genetic MarkersProspective StudiesMESH: Coronary Artery DiseaseAged 80 and over0303 health sciencesMESH: Middle AgedGenes fosMESH: Smoking Cessation3. Good healthMESH : SmokingMESH: Myocardial InfarctionOsteosarcomaSmoking statusCardiology and Cardiovascular MedicineGenetic Markersmedicine.medical_specialtyRisk AssessmentMESH: Leukocytes03 medical and health sciences[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemHumansMESH : Middle AgedRNA MessengerMESH : Coronary Artery DiseaseRisk factorMESH : Aged 80 and overAgedChi-Square DistributionMESH: HumansMESH : Chi-Square DistributionMESH : Smoking CessationMESH : Humansmedicine.diseaseMESH: Coronary AngiographyLinear ModelsMESH: FemaleBlood leukocyte transcriptomeMESH : Genes fos030204 cardiovascular system & hematologyMESH: Genetic MarkersBioinformaticsCoronary AngiographyCoronary artery diseaseMESH: Linear ModelsCoronary artery diseaseMESH: Aged 80 and overRisk FactorsMESH: Risk FactorsMESH : Linear ModelsLeukocytesMESH : FemaleMESH : Risk AssessmentMESH: Agedc-FosbiologySmoking[ SDV.MHEP.CSC ] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemMiddle AgedMESH : Risk Factors[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemCardiologyFemaleFrancemedicine.symptomInflammation MediatorsMESH: SmokingMESH : MaleMESH: Inflammation MediatorsInflammationMESH: Genes fosMESH: Multivariate AnalysisMESH : Inflammation MediatorsInternal medicinemedicineIn patientMESH : France030304 developmental biologyMESH: RNA Messengerbusiness.industryMESH : Multivariate AnalysisMESH: MaleMESH: Prospective StudiesMESH: FranceMultivariate Analysisbiology.proteinSmoking CessationMESH : Myocardial Infarctionbusiness
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Dietary polyunsaturated fatty acids reduce retinal stress induced by an elevation of intraocular pressure in rats.

2011

International audience; N-6 and n-3 polyunsaturated fatty acids (PUFAs) have been shown to prevent tissue release of inflammatory molecules. We have shown that a combination of n-6 and n-3 PUFAs is more efficient than single supplementations on the long-term consequences of intraocular pressure elevation. We hypothesized that such an association is also more effective during early retinal stress by modifying retinal proinflammatory prostaglandin and cytokine productions. Rats were supplemented for 3 months with n-6 PUFAs, n-3 PUFAs, or both n-6 and n-3 PUFAs. After 3 months, a surgical elevation of intraocular pressure was induced. Retinal morphometry and glial cell activation were evaluate…

MaleMESH : RNA MessengerMESH: Eicosapentaenoic AcidEndocrinology Diabetes and Metabolismmedicine.medical_treatment[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionInterleukin-1betaMESH: Dietary SupplementsMESH: Rats Sprague-DawleyRats Sprague-Dawleychemistry.chemical_compound0302 clinical medicineEndocrinologyMESH: Interleukin-1betaratMESH: AnimalsProstaglandin E2Prostaglandin E1MESH : Tumor Necrosis Factor-alphaMESH : Intraocular Pressure0303 health sciencesNutrition and DieteticsMESH : RatsMESH : NeurogliaMESH: RetinaMESH: Dinoprostonepression intraoculaireMESH: AlprostadilMESH: Docosahexaenoic AcidsBiochemistryEicosapentaenoic AcidDocosahexaenoic acidlipids (amino acids peptides and proteins)MESH: NeurogliaProstaglandin D2Cell activationNeurogliaMESH : Alprostadilmedicine.drugProstaglandin Emedicine.medical_specialtyMESH : DinoprostoneMESH : Interleukin-6Docosahexaenoic AcidsMESH: RatsMESH : MaleProstaglandinBiologyMESH : Interleukin-1betaDinoprostoneRetinaMESH : Diet03 medical and health sciencesMESH: DietMESH: Intraocular PressureInternal medicinemedicineMESH : Eicosapentaenoic AcidAnimalsMESH : Dietary SupplementsRNA MessengerAlprostadilprostanoids030304 developmental biologyMESH: RNA MessengerInterleukin-6Tumor Necrosis Factor-alphadietary polyunsatured fatty acidretinal stressMESH : RetinaRetinalN-6MESH: Interleukin-6MESH : Rats Sprague-Dawleyeye diseasesMESH: MaleMESH : Docosahexaenoic AcidsDietRatsN-3EndocrinologychemistryMESH: Tumor Necrosis Factor-alphaDietary Supplements030221 ophthalmology & optometryMESH : Animalssense organs[SDV.AEN]Life Sciences [q-bio]/Food and Nutritionintraocular pressure
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Olfactory bulbectomy, but not odor conditioned aversion, induces the differentiation of immature neurons in the adult rat piriform cortex.

2011

International audience; The piriform cortex layer II of young-adult rats presents a population of prenatally generated cells, which express immature neuronal markers, such as the polysialylated form of the neural cell adhesion molecule (PSA-NCAM) or doublecortin (DCX), and display structural characteristics of immature neurons. The number of PSA-NCAM/DCX expressing cells in this region decreases markedly as age progresses, suggesting that these cells differentiate or die. Since the piriform cortex receives a major input from the olfactory bulb and participates in olfactory information processing, it is possible that the immature neurons in layer II are affected by manipulations of the olfac…

MaleMESH: Cell DifferentiationMESH: Neural Stem CellsMESH: Olfactory BulbDoublecortin ProteinMESH: RatsNeurogenesisMESH : MaleMESH : Neurogenesis[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH : Rats WistarNeural Stem CellsPiriform cortexAnimalsMESH: AnimalsRats WistarOlfactory memoryMESH : Olfactory BulbbiologyMESH : Olfactory PathwaysMESH : RatsGeneral NeuroscienceNeurogenesisCell DifferentiationOlfactory PathwaysMESH: Rats WistarOlfactory BulbMESH: MaleRatsOlfactory bulbDoublecortinMESH: Neurogenesisnervous systemMESH : Neural Stem Cellsbiology.proteinNeural cell adhesion moleculeOlfactory ensheathing gliaMESH : AnimalsNeuNNeuroscienceMESH : Cell Differentiation[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH: Olfactory Pathways
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Cardiac rehabilitation and 5-year mortality after acute coronary syndromes: The 2005 French FAST-MI study.

2016

IF 2.271; International audience; Background. - Clinical studies have shown a beneficial effect of cardiac rehabilitation (CR) on mortality.Objective. - To study the effect of CR prescription at discharge on 5-year mortality in patients with acute myocardial infarction (AMI).Methods. - Participants, from the 2005 French FAST-MI hospital registry, were 2894 survivors at discharge, divided according to AMI type: ST-segment elevation myocardial infarction (STEMI; n=1523) and non-STEMI (NSTEMI; n=1371). The effect of CR prescription on mortality was analysed using a Cox proportional hazards model.Results. - At discharge, 22.1% of patients had a CR prescription. Patients referred to CR were youn…

MaleMESH: Chi-Square Distributionmedicine.medical_treatmentMESH : Acute Coronary SyndromeMyocardial InfarctionMESH : AgedMESH : Prospective StudiesCardiac rehabilitationMESH: Risk Assessment0302 clinical medicineMyocardial infarctionProspective StudiesReferral and ConsultationMESH: Treatment OutcomeRehabilitationMESH: Middle AgedGeneral MedicineMESH: Follow-Up Studies3. Good healthMESH: Myocardial InfarctionMESH : Patient DischargeCardiology and Cardiovascular Medicinemedicine.medical_specialtyAcute myocardial infarctionMortalitéRisk Assessment03 medical and health sciences[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemHumansMESH : Middle AgedAcute Coronary SyndromeMESH: Kaplan-Meier EstimateAgedChi-Square DistributionMESH: HumansMESH : Chi-Square DistributionProportional hazards modelMESH: Patient DischargeMESH : HumansPatient survivalMESH : Follow-Up Studiesmedicine.diseaseMESH : Proportional Hazards ModelsMESH: Acute Coronary SyndromeST-segment elevation myocardial infarctionMESH: FemaleTime FactorsMESH: RegistriesKaplan-Meier Estimate030204 cardiovascular system & hematologyMESH : Referral and ConsultationMESH: Proportional Hazards ModelsOlder patientsRisk FactorsMESH: Risk FactorsMESH : Female030212 general & internal medicineRegistriesMESH : Risk AssessmentMESH: AgedEjection fractionNon–ST-segment elevation myocardial infarctionMiddle Aged[ SDV.MHEP.CSC ] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemMESH : Risk FactorsPatient DischargeTreatment OutcomeFemaleFranceMESH : Time FactorsMESH : MaleMESH : Treatment OutcomeMESH: Multivariate AnalysisMESH : Kaplan-Meier EstimateMESH: Referral and ConsultationInternal medicineInfarctus du myocarde sans sus-décalage du segment STmedicineIn patientcardiovascular diseasesMedical prescriptionMortalityRéadaptation cardiaqueMESH : FranceProportional Hazards Modelsbusiness.industryInfarctus du myocarde avec sus-décalage du segment STMESH: Time FactorsMESH : Multivariate AnalysisMESH: MaleMESH: Prospective StudiesSurgeryMESH: FranceInfarctus du myocarde aiguMultivariate AnalysisMESH : Myocardial InfarctionbusinessMESH : RegistriesFollow-Up Studies
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Tenectin is a novel alphaPS2betaPS integrin ligand required for wing morphogenesis and male genital looping in Drosophila.

2010

International audience; Morphogenesis of the adult structures of holometabolous insects is regulated by ecdysteroids and juvenile hormones and involves cell-cell interactions mediated in part by the cell surface integrin receptors and their extracellular matrix (ECM) ligands. These adhesion molecules and their regulation by hormones are not well characterized. We describe the gene structure of a newly described ECM molecule, tenectin, and demonstrate that it is a hormonally regulated ECM protein required for proper morphogenesis of the adult wing and male genitalia. Tenectin's function as a new ligand of the PS2 integrins is demonstrated by both genetic interactions in the fly and by cell s…

MaleMESH: Extracellular Matrix ProteinsMESH: DrosophilaMESH : Immunohistochemistry[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionIntegrinLigandsLooping morphogenesisExtracellular matrixchemistry.chemical_compound0302 clinical medicineMESH: Genitalia MaleMorphogenesisMESH: LigandsDrosophila ProteinsWings AnimalMESH: AnimalsTransgenesIn Situ Hybridization0303 health sciencesExtracellular Matrix ProteinsMESH : Genitalia MaleMESH : LigandsIntegrin alpha ChainsCell adhesion moleculeMESH : In Situ HybridizationImmunohistochemistry3. Good healthCell biologyLarvaMESH : Integrin alpha ChainsAdhesionDrosophilaMESH : MutationMESH : TransgenesTenectinIntegrin alpha ChainsDrosophila ProteinEcdysoneEcdysoneMESH: MutationMESH: Drosophila ProteinsMESH : MaleIntegrinMorphogenesisMESH : WingMESH: TransgenesBiologyGenitalia MaleArticle03 medical and health sciencesMESH : Extracellular Matrix ProteinsMESH: In Situ HybridizationAnimalsMESH : DrosophilaCell adhesionMolecular Biology030304 developmental biologyMESH : LarvaMetamorphosisMESH: Integrin alpha ChainsLeft–right asymmetryMESH: ImmunohistochemistryCell BiologyMESH : Drosophila ProteinsMESH: WingMESH: MaleMESH: MorphogenesischemistryMESH : MorphogenesisMutationbiology.proteinMESH : AnimalsMESH: Larva[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryDevelopmental Biology
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Orofacial reactivity to the sight and smell of food stimuli. Evidence for anticipatory liking related to food reward cues in overweight children.

2012

International audience; Whether food liking may be a risk factor of overconsumption and overweight/obesity remains a controversial issue. So far, most studies used subjective reports to assess consummatory behavior, approaches that might overlook subtle or implicit hedonic changes to sensory properties of foods. Therefore, we used a cue-exposure approach by recording different measures of hedonic processes (orofacial reactivity, self-rated pleasantness, food preference) in 6-11 years old overweight (n=20) and normal-weight (n=20) children. Children were exposed to the smell and sight of high and low-energy density food stimuli and to non-food stimuli during pre- and post-prandial states. Th…

MaleMESH: Facial Expression030309 nutrition & dietetics[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH : Vision OcularMESH : OverweightMESH: Energy IntakeOverweighteating behaviorDevelopmental psychologyBody Mass Index0302 clinical medicineMESH : ChildMESH: ChildMESH: SmellMESH : FemaleOvereatingReactivity (psychology)ChildGeneral Psychology0303 health sciencesNutrition and DieteticsMESH : Food Preferencesdigestive oral and skin physiologyMESH : Feeding BehaviorAnticipationMESH : HyperphagiaFacial ExpressionSmellCategorizationMESH: Feeding BehaviorFemalesensory cueMESH: Hyperphagiamedicine.symptomMESH : FoodPsychologySocial psychologyMESH: FoodhedonicMESH: Anticipation PsychologicalMESH : Maleoverweight childreward sensitivity030209 endocrinology & metabolismpleasureHyperphagiaSatiationMESH : Facial Expressionfacial reactivityMESH: Body Mass Index03 medical and health sciencesFood PreferencesRewardmedicineHumansMESH: Vision OcularMESH: OverweightMESH: Food PreferencesSensory cueVision OcularMESH : Anticipation PsychologicalMESH: RewardFacial expressionMESH: HumansMESH: SatiationMESH : HumansMESH : Energy IntakeFeeding BehaviorOverweightmedicine.diseaseAnticipation PsychologicalObesityMESH: MalebmiMESH : Body Mass IndexFoodfood likingMESH : SmellMESH : RewardEnergy IntakeMESH: Female[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH : Satiation
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A role for the peroxisomal 3-ketoacyl-CoA thiolase B enzyme in the control of PPARα-mediated upregulation of SREBP-2 target genes in the liver.: ThB …

2011

International audience; Peroxisomal 3-ketoacyl-CoA thiolase B (Thb) catalyzes the final step in the peroxisomal β-oxidation of straight-chain acyl-CoAs and is under the transcription control of the nuclear hormone receptor PPARα. PPARα binds to and is activated by the synthetic compound Wy14,643 (Wy). Here, we show that the magnitude of Wy-mediated induction of peroxisomal β-oxidation of radiolabeled (1-(14)C) palmitate was significantly reduced in mice deficient for Thb. In contrast, mitochondrial β-oxidation was unaltered in Thb(-/-) mice. Given that Wy-treatment induced Acox1 and MFP-1/-2 activity at a similar level in both genotypes, we concluded that the thiolase step alone was respons…

MaleMESH: HepatomegalyPalmitatesMESH : PyrimidinesMESH : Gene DeletionBiochemistryelement-binding proteinsMESH : Acetyl-CoA C-AcyltransferaseMiceMESH: Up-RegulationMESH: AnimalsMESH : Up-RegulationMESH: Lipid Metabolism0303 health sciencesMESH : Gene Expression RegulationThiolase030302 biochemistry & molecular biologyGeneral MedicineMESH : HepatomegalyUp-Regulationzellweger-syndromePeroxisome ProliferatorsMESH: Peroxisome ProliferatorsHepatomegalySterol Regulatory Element Binding Protein 2peroxisomal 3-ketoacyl-CoA thiolase BMESH: Mitochondria3-oxoacyl-coa thiolaseLathosterolfatty-acid oxidationrat-liverMESH: Sterol Regulatory Element Binding Protein 203 medical and health sciencesMESH : Sterol Regulatory Element Binding Protein 2HumansPPAR alphaMESH : Peroxisome Proliferators[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyPPARaVLAGMESH : Oxidation-ReductionFatty Acid Oxidation.MESH: HumansCholesterolMESH : HumanscholesterolLipid MetabolismMESH: PeroxisomesSterol regulatory element-binding proteinchemistryMESH: PyrimidinesCholesterol; Micro-array analysis; Peroxisomal 3-ketoacyl-CoA thiolase B; PPARα and SREBP-2; Wy14643Fatty Acid OxidationGene DeletionMESH: LiverMESH: Oxidation-ReductionMESH: Signal TransductionMESH: Mice KnockoutVoeding Metabolisme en Genomicachemistry.chemical_compoundMESH: CholesterolMESH : Lipid MetabolismWy14MESH : PalmitatesMESH: PPAR alphaMESH : CholesterolMice Knockoutneuronal migration643PeroxisomeAcetyl-CoA C-AcyltransferaseMESH: Gene Expression RegulationMetabolism and GenomicsMitochondriaLiverBiochemistryMicro-array analysisMetabolisme en GenomicaACOX1Nutrition Metabolism and GenomicsMESH : MitochondriaOxidation-ReductionSignal Transductionacyl-coa oxidasecholesterol-synthesisMESH : MaleMESH : PPAR alphaPeroxisome ProliferationPPARα and SREBP-2Biologybeta-oxidationVoedingproliferator-activated receptorsMESH : MicePeroxisomesAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyMESH: Mice030304 developmental biologySCP2NutritionMESH : Signal TransductionMESH : LiverMESH: PalmitatesMESH: MalePyrimidinesMESH: Acetyl-CoA C-AcyltransferaseGene Expression RegulationMESH: Gene DeletionMESH : Mice KnockoutMESH : AnimalsMESH : Peroxisomes
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