Search results for "MESH : animals"

showing 10 items of 99 documents

An olfactory receptor for food-derived odours promotes male courtship in Drosophila.

2011

International audience; Many animals attract mating partners through the release of volatile sex pheromones, which can convey information on the species, gender and receptivity of the sender to induce innate courtship and mating behaviours by the receiver. Male Drosophila melanogaster fruitflies display stereotyped reproductive behaviours towards females, and these behaviours are controlled by the neural circuitry expressing male-specific isoforms of the transcription factor Fruitless (FRU(M)). However, the volatile pheromone ligands, receptors and olfactory sensory neurons (OSNs) that promote male courtship have not been identified in this important model organism. Here we describe a novel…

MaleOviposition[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH : GenotypeMESH : OvipositionCourtshipMESH: GenotypeSexual Behavior Animal0302 clinical medicineMESH : Drosophila melanogasterMESH: AnimalsMESH : FemaleMatingSex AttractantsMESH: Sexual Behavior AnimalMESH: Ovipositionmedia_commonPhenylacetates0303 health scienceseducation.field_of_studyMultidisciplinaryMESH: Receptors Ionotropic GlutamateMESH : Receptors Ionotropic GlutamateAnatomyMESH: AcetaldehydeMESH : OdorsCell biologymedicine.anatomical_structureDrosophila melanogasterMESH: Sex AttractantsSex pheromonebehavior and behavior mechanismsPheromonefruitlessFemaleDrosophila melanogasterMESH : FoodMESH: FruitMESH: FoodGenotypemedia_common.quotation_subjectMESH : MalePopulationMESH: CourtshipMESH : AcetaldehydeAcetaldehydeMESH : FruitBiologyReceptors Ionotropic GlutamateOlfactory Receptor NeuronsMESH: Drosophila melanogaster03 medical and health sciencesmedicineAnimalseducationMESH : Sexual Behavior Animal030304 developmental biologyMESH : Sex AttractantsOlfactory receptorMESH: OdorsMESH: PhenylacetatesMESH : CourtshipfungiCourtshipMESH : PhenylacetatesMESH: Olfactory Receptor Neuronsbiology.organism_classificationMESH: MaleFoodFruitOdorantsMESH : Olfactory Receptor NeuronsMESH : AnimalsMESH: Female[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgery
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Effects of manganese injected into rat nostrils: implications for in vivo functional study of olfaction using MEMRI.

2011

WOS: 000298212500007; International audience; Manganese-enhanced magnetic resonance imaging (MEMRI) is a powerful tool for visualizing neuronal pathways and mapping brain activity modulation. A potential drawback of MEMRI lies in the toxic effects of manganese (Mn), which also depend on its administration route. The aim of this study was to analyze the effects of Mn doses injected into the nostrils of rats on both olfactory perception and MRI contrast enhancement. For this purpose, doses in the range 0-8 μmol MnCl(2) were tested. Behavioral items were quantified with and without odor stimulation during the first 2 h following Mn injection. The MRI study was performed after 16 h of intermitt…

MalePathologyBrain activity and meditation[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionContrast MediaStimulationPharmacologyMESH : Behavior AnimalMEMRI Manganese030218 nuclear medicine & medical imagingMESH: Magnetic Resonance Imaging0302 clinical medicineMESH: SmellMESH: Behavior AnimalMESH: AnimalsMESH: Administration IntranasalMESH : Olfactory Bulbmedicine.diagnostic_testBehavior AnimalChemistryMESH : RatsMagnetic Resonance ImagingOlfactory BulbSmellDoseToxicityMESH: Image EnhancementMESH: Olfactory Bulbmedicine.medical_specialtyMESH: RatsMESH : MaleBiomedical EngineeringBiophysicsMESH: ManganeseOlfactionMESH : Rats Wistar03 medical and health sciencesPrimary olfactory cortexIn vivoMESH : Magnetic Resonance ImagingMESH: Contrast MediamedicineAnimalsRadiology Nuclear Medicine and imagingRats WistarAdministration IntranasalMESH : Contrast MediaBehaviorManganeseToxicityMESH : Administration IntranasalMagnetic resonance imagingMESH: Rats WistarImage EnhancementOlfactionMESH: MaleRatsOdorRatMESH : SmellMESH : Image EnhancementMESH : AnimalsMESH : Manganese[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgery
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Growth hormone potentiates thyroid hormone effects on post-exercise phosphocreatine recovery in skeletal muscle.

2012

International audience; OBJECTIVE: The aim of the study was to determine the respective impact of thyroxine and growth hormone on in vivo skeletal mitochondrial function assessed via post exercise phosphocreatine recovery. DESIGN: The hind leg muscles of 32 hypophysectomized rats were investigated using (31)P nuclear magnetic resonance spectroscopy at rest and during the recovery period following a non tetanic stimulation of the sciatic nerve. Each rat was supplemented with hydrocortisone and was randomly assigned to one of the 4 groups: the group Hx was maintained in hypopituitarism., the group HxT was treated with 1 μg/100g/day of thyroxine (T4), the group HxG with 0.2 IU/kg/day of recomb…

MalePhosphocreatineThyroid hormonesEndocrinology Diabetes and MetabolismMESH: Random AllocationThyroid GlandSkeletal muscleHypopituitarismMESH: Physical Conditioning AnimalMESH: Drug SynergismNuclear magnetic resonancechemistry.chemical_compoundRandom Allocation0302 clinical medicineEndocrinologyMESH: Human Growth HormoneMESH: AnimalsMESH : Muscle Skeletal0303 health sciencesMESH: Muscle Skeletal[ INFO.INFO-IM ] Computer Science [cs]/Medical ImagingMESH : RatsHuman Growth HormoneThyroidDrug Synergismmedicine.anatomical_structuremedicine.drugmedicine.medical_specialtyMESH : Drug SynergismMESH: RatsMESH : MaleSomatotropin030209 endocrinology & metabolismMESH: PhosphocreatinePhosphocreatineMESH : Random Allocation03 medical and health sciencesIn vivoInternal medicine[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyPhysical Conditioning AnimalMESH : Thyroxinemedicine[INFO.INFO-IM]Computer Science [cs]/Medical ImagingAnimalsHumansMESH : PhosphocreatineMESH : Human Growth HormoneMitochondrionMESH : Physical Conditioning AnimalMuscle Skeletal030304 developmental biologyHydrocortisoneMESH: HumansMESH : HumansSkeletal muscleMESH : Thyroid GlandMESH: Thyroxinemedicine.diseaseMESH: MaleMESH: Thyroid GlandRatsThyroxineEndocrinologychemistryRatMESH : AnimalsTetanic stimulation[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyHormoneGrowth hormoneIGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society
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Oral homeostasis disruption by medical plasticizer component bisphenol A in adult male rats.

2013

Objectives/Hypothesis Bisphenol A (BPA) is a synthetic estrogen-like chemical mimetic widely used in the manufacture of polycarbonate plastics and epoxy resins found in numerous consumer products including food packaging, medical devices, and dental sealants. Because it is recovered in fluids and it can reach high levels in saliva, this study aimed to evaluate its safety on oral homeostasis by examining its effects on salivary glands, mouth epithelium, water consumption, and salt preference, each parameter being estrogen sensitive. Study Design Randomized controlled trial involving rats. Methods A dose-response study was conducted in adult Wistar rats randomized into five groups (n = 12). B…

MaleSalivaBisphenol A[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH : Dose-Response Relationship DrugMESH : DrinkingMESH: PlasticizersMESH: MouthSalivary GlandsThirstMESH: Dose-Response Relationship Drugchemistry.chemical_compoundMESH: Estrogens Non-SteroidalMESH: PhenolsPlasticizersMESH : MouthHomeostasisMESH: Animalssalt preferencemouth drynessSalivary glandMESH : RatsDose–response relationshipmedicine.anatomical_structureMESH : Salivary Glandsendocrine disruptorsthirstMESH: HomeostasisMESH : Homeostasismedicine.symptomMESH : Estrogens Non-SteroidalMESH: DrinkingMESH : Phenolsmedicine.medical_specialtyMESH: Salivary GlandsMESH: Ratsmedicine.drug_classMESH : MaleDrinkingsalivary glandstomatognathic systemPhenolsInternal medicinemedicineMESH: Benzhydryl CompoundsAnimalsMESH: SalivaEstrogens Non-SteroidalBenzhydryl CompoundsSalivaMouthMESH : Benzhydryl CompoundsDose-Response Relationship Drugbusiness.industryBuccal administrationMESH : Disease Models AnimalMESH: MaleRatsDisease Models AnimalEndocrinologyOtorhinolaryngologychemistryEstrogenMESH : PlasticizersMESH : AnimalsMESH : SalivaMESH: Disease Models Animalbusiness[SDV.AEN]Life Sciences [q-bio]/Food and NutritionHomeostasisThe Laryngoscope
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Expression and differential localization of xenobiotic transporters in the rat olfactory neuro-epithelium.

2011

International audience; Transporters, such as multidrug resistance P-glycoproteins (MDR), multidrug resistance-related proteins (MRP) and organic anion transporters (OATs), are involved in xenobiotic metabolism, particularly the cellular uptake or efflux of xenobiotics (and endobiotics) or their metabolites. The olfactory epithelium is exposed to both inhaled xenobiotics and those coming from systemic circulation. This tissue has been described as a pathway for xenobiotics to the brain via olfactory perineural space. Thereby, olfactory transporters and xenobiotic metabolizing enzymes, dedicated to the inactivation and the elimination of xenobiotics, have been involved in the toxicological p…

Male[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH : Multidrug Resistance-Associated Proteinsp glycoproteinATP-binding cassette transporterMESH : HepatocytesReceptors OdorantMESH : P-GlycoproteinMESH: HepatocytesMESH : Lymphatic Vessels0302 clinical medicineMESH : Protein Transportugt2a1MESH: SmellMESH: Receptors OdorantMESH: AnimalsReceptorxenobiotic metabolizingmucosa0303 health sciencesMESH : Gene Expression RegulationMESH : RatsGeneral NeuroscienceMESH : OdorsMESH: Gene Expression RegulationSmellProtein Transportmedicine.anatomical_structureBiochemistryLivertransporterbarrierEffluxMultidrug Resistance-Associated ProteinsMESH: Multidrug Resistance-Associated ProteinsMESH: XenobioticsMESH: Protein TransportMESH: P-GlycoproteinMESH: RatsMESH: Lymphatic VesselsMESH : Maleodorant clearancebrainMESH : XenobioticsxenobioticBiologysystemMESH : Rats WistarOlfactory Receptor NeuronsXenobiotics03 medical and health sciencesbulbOlfactory Mucosamultidrug resistanceMESH : Receptors OdorantmedicineAnimalsATP Binding Cassette Transporter Subfamily B Member 1Rats WistardetoxificationMESH: Olfactory Mucosa030304 developmental biologyLymphatic VesselsMESH : Olfactory MucosaMESH: OdorsMESH : LiverTransporterMESH: Rats WistarMESH: Olfactory Receptor NeuronsEpitheliumMESH: MaleOlfactory bulbRatsenzymeGene Expression RegulationOdorantsHepatocytesMESH : SmellMESH : Olfactory Receptor NeuronsMESH : Animalsolfactory epitheliumOlfactory epitheliumperireceptor event[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryDrug metabolismMESH: Liver
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Is there a role for antioxidant carotenoids in limiting self-harming immune response in invertebrates?

2007

Innate immunity relies on effectors, which produce cytotoxic molecules that have not only the advantage of killing pathogens but also the disadvantage of harming host tissues and organs. Although the role of dietary antioxidants in invertebrate immunity is still unknown, it has been shown in vertebrates that carotenoids scavenge cytotoxic radicals generated during the immune response. Carotenoids may consequently decrease the self-harming cost of immunity. A positive relationship between the levels of innate immune defence and circulating carotenoid might therefore be expected. Consistent with this hypothesis, we show that the maintenance and use of the prophenoloxidase system strongly cor…

MaleantioxidantMESH : Immunity Natural[ SDV.IMM.IA ] Life Sciences [q-bio]/Immunology/Adaptive immunologyAntioxidantsMESH: Linear ModelsMESH: AmphipodaHemolymphMESH : Linear ModelsHemolymphMESH: AnimalsMESH : FemaleCarotenoidchemistry.chemical_classificationbiologyEffectorMonophenol Monooxygenasefood and beveragesProphenoloxidaseMESH : AmphipodaAgricultural and Biological Sciences (miscellaneous)MESH : Monophenol Monooxygenase[SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunologyMESH : AntioxidantsFemaleGeneral Agricultural and Biological SciencesResearch ArticleMESH: Monophenol MonooxygenaseMESH : Maleimmune costsecological immunologyMESH : Hemolymph[ SDV.BBM.BM ] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyImmune systemImmunityAnimalsAmphipodaMESH: Immunity NaturalMESH : CarotenoidsInnate immune systemMESH: HemolymphMESH: Antioxidants[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologybiochemical phenomena metabolism and nutritionbiology.organism_classificationCarotenoidsImmunity InnateMESH: MaleGammarus pulexchemistryImmunologyMESH: CarotenoidsLinear ModelsbacteriaMESH : AnimalsMESH: Female
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Abnormalities of mitochondrial functioning can partly explain the metabolic disorders encountered in sarcopenic gastrocnemius.

2007

International audience; Aging triggers several abnormalities in muscle glycolytic fibers including increased proteolysis, reactive oxygen species (ROS) production and apoptosis. Since the mitochondria are the main site of substrate oxidation, ROS production and programmed cell death, we tried to know whether the cellular disorders encountered in sarcopenia are due to abnormal mitochondrial functioning. Gastrocnemius mitochondria were extracted from adult (6 months) and aged (21 months) male Wistar rats. Respiration parameters, opening of the permeability transition pore and ROS production, with either glutamate (amino acid metabolism) or pyruvate (glucose metabolism) as a respiration substr…

Malemuscle atrophyMESH : Cell Aging[SDV]Life Sciences [q-bio]MESH : Reactive Oxygen SpeciesMitochondrion0302 clinical medicineGlycolysisMESH: AnimalsMESH : Muscle SkeletalMESH : Fatty AcidsCellular SenescencePhospholipidsMESH: Superoxide Dismutasereactive oxygen speciesMESH : Free Radicals0303 health sciencesMESH: Muscle SkeletalMESH : RatsFatty Acidsfatty acid profile of mitochondrial lipidsMESH: Reactive Oxygen SpeciesPyruvate dehydrogenase complexMESH: Fatty Acidsmitochondria[SDV] Life Sciences [q-bio]BiochemistryMESH: Cell AgingMESH: CalciumMESH : MitochondriaCell agingPyruvate decarboxylationmedicine.medical_specialtyFree RadicalsMESH: RatsCellular respirationMESH: MitochondriaMESH : MaleCell Respirationchemistry.chemical_elementOxidative phosphorylationBiologyCalciumMESH : Rats WistarMESH : Phospholipids03 medical and health sciencesMESH: Free RadicalsInternal medicinemedicineAnimalsMESH : Superoxide DismutaseRats WistarMuscle SkeletalMESH : Calcium030304 developmental biologyMESH: Phospholipidscalciumpermeability transition poreSuperoxide Dismutaseagingaging;calcium;fatty acid profile of mitochondrial lipids;mitochondria;muscle atrophy;permeability transition pore;reactive oxygen species;Animals;Calcium;Cell Aging;Cell Respiration;Fatty Acids;Free Radicals;Male;Mitochondria;Muscle;Skeletal;Phospholipids;Rats;Wistar;Reactive Oxygen Species;Superoxide DismutaseCell BiologyMESH: Rats WistarMESH: MaleRatsEndocrinologychemistryMESH : Cell RespirationMESH : AnimalsMESH: Cell Respiration030217 neurology & neurosurgery
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Towards understanding the epidemiology of Neisseria meningitidis in the African meningitis belt: a multi-disciplinary overview

2016

International audience; Objectives: Neisseria meningitidis is the major cause of seasonal meningitis epidemics in the African meningitis belt. In the changing context of a reduction in incidence of serogroup A and an increase in incidence of serogroups W and C and of Streptococcus pneumoniae, a better understanding of the determinants driving the disease transmission dynamics remains crucial to improving bacterial meningitis control.Methods: The literature was searched to provide a multi-disciplinary overview of the determinants of meningitis transmission dynamics in the African meningitis belt.Results: Seasonal hyperendemicity is likely predominantly caused by increased invasion rates, spo…

Microbiology (medical)MESH : AfricaBacterial meningitisMeningitis MeningococcalNeisseria meningitidisMESH: AfricaMESH: Neisseria meningitidislcsh:Infectious and parasitic diseasesSeroepidemiologic StudiesDisease control[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesMESH : Seroepidemiologic StudiesAnimalsHumanslcsh:RC109-216MESH: AnimalsMESH : Meningitis MeningococcalComputingMilieux_MISCELLANEOUSResearch prioritiesMESH: HumansMESH: Seroepidemiologic StudiesMESH : Neisseria meningitidisMESH : HumansMESH: Meningitis MeningococcalAfrican belt[SHS.ANTHRO-SE]Humanities and Social Sciences/Social Anthropology and ethnology[ SDV.MHEP.MI ] Life Sciences [q-bio]/Human health and pathology/Infectious diseasesInfectious Diseases[SDU.STU.CL]Sciences of the Universe [physics]/Earth Sciences/Climatology[SDV.SPEE] Life Sciences [q-bio]/Santé publique et épidémiologieAfrica[SDV.MHEP.MI] Life Sciences [q-bio]/Human health and pathology/Infectious diseases[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologieMESH : Animals[ SDU.STU.CL ] Sciences of the Universe [physics]/Earth Sciences/Climatology
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Effects of a high-fat diet on energy metabolism and ROS production in rat liver.

2011

International audience; BACKGROUND & AIMS: A high-fat diet affects liver metabolism, leading to steatosis, a complex disorder related to insulin resistance and mitochondrial alterations. Steatosis is still poorly understood since diverse effects have been reported, depending on the different experimental models used. METHODS: We hereby report the effects of an 8 week high-fat diet on liver energy metabolism in a rat model, investigated in both isolated mitochondria and hepatocytes. RESULTS: Liver mass was unchanged but lipid content and composition were markedly affected. State-3 mitochondrial oxidative phosphorylation was inhibited, contrasting with unaffected cytochrome content. Oxidative…

Mitochondrial ROSMaleTranscription GeneticMESH : Reactive Oxygen SpeciesMitochondria LiverMESH : HepatocytesMitochondrionOxidative PhosphorylationMESH: Hepatocytes0302 clinical medicineMESH: Membrane Potential MitochondrialCitrate synthaseMESH: AnimalsBeta oxidationMESH : Electron Transport2. Zero hungerMembrane Potential Mitochondrial0303 health sciencesMESH : RatsAdenine nucleotide translocatorMESH: Energy MetabolismMESH: Reactive Oxygen SpeciesLipidsBiochemistryLiverMESH: Dietary FatsMitochondrial matrix030220 oncology & carcinogenesisBody CompositionMESH : Oxidative PhosphorylationATP–ADP translocaseMESH: Mitochondria LiverMESH: RatsMESH : Body CompositionMESH : MaleOxidative phosphorylationBiologyMESH : Rats WistarElectron Transport03 medical and health sciencesMESH: Oxidative Phosphorylation[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyRats WistarMESH: Electron Transport[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular Biology030304 developmental biologyHepatologyMESH: Transcription GeneticMESH : Transcription GeneticMESH : LiverMESH : LipidsMESH: Body CompositionMESH: Rats WistarMESH: LipidsDietary FatsMESH: MaleRatsMESH : Energy MetabolismMESH : Membrane Potential MitochondrialMESH : Mitochondria Liverbiology.proteinHepatocytesMESH : AnimalsEnergy MetabolismReactive Oxygen SpeciesMESH : Dietary FatsMESH: Liver
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UDP-glucuronosyltransferases (UGTs) in neuro-olfactory tissues: expression, regulation, and function.

2010

International audience; This work aims to review uridine diphosphate (UDP)-glucuronosyltransferase (UGT) expression and activities along different neuronal structures involved in the common physiological process of olfaction: olfactory epithelium, olfactory bulb, and olfactory cortex. For the first time, using high-throughput in situ hybridization data generated by the Allen Brain Atlas (ABA), we present quantitative analysis of spatial distribution of UGT genes in the mouse brain. The olfactory area is a central nervous system site with the highest expression of UGTs, including UGT isoforms not previously identified in the brain. Since there is evidence of the transfer of xenobiotics to th…

Olfactory systemMESH : RNA Messenger[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH: GlucuronosyltransferaseMESH : Blood-Brain BarrierMESH: Blood-Brain Barrierchemistry.chemical_compound0302 clinical medicineMESH: SmellPharmacology (medical)MESH: AnimalsMESH: Uridine DiphosphateMESH: Nerve Tissue ProteinsGlucuronosyltransferaseGeneral Pharmacology Toxicology and PharmaceuticsMESH : Olfactory BulbMESH : Nerve Tissue Proteins0303 health sciencesMESH: Gene Expression Regulation EnzymologicOlfactory PathwaysOlfactory BulbMESH : OdorsCell biologySmellmedicine.anatomical_structureBlood-Brain BarrierMESH: Olfactory Bulbmedicine.medical_specialtyCentral nervous systemNerve Tissue ProteinsIn situ hybridizationOlfactionBiologydigestive systemGene Expression Regulation EnzymologicOlfactory Receptor NeuronsUridine DiphosphateMESH : Gene Expression Regulation Enzymologic03 medical and health sciencesInternal medicinemedicineAnimalsRNA MessengerMESH : Uridine Diphosphate030304 developmental biologyMESH: RNA MessengerMESH: OdorsMESH : Olfactory PathwaysMESH : GlucuronosyltransferaseMESH: Olfactory Receptor NeuronsOlfactory bulbUridine diphosphateEndocrinologychemistryOdorantsMESH : SmellMESH : Olfactory Receptor NeuronsMESH : AnimalsOlfactory epithelium[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryFunction (biology)MESH: Olfactory Pathways
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