Search results for "Mediators"

showing 10 items of 245 documents

Hypothermic preservation of lung allograft inhibits cytokine-induced chemoattractant-1, endothelial leucocyte adhesion molecule, vascular cell adhesi…

2007

Summary Organ dysfunction is a major clinical problem after lung transplantation. Prolonged cold ischaemia and reperfusion injury are believed to play a central role in this complication. The influence of cold preservation on subsequent warm reperfusion was studied in an isolated, ventilated and perfused rat lung. Rat lungs were flushed with cold Perfadex-solution and stored at 4°C for different time periods. Thereafter lungs were perfused and ventilated for up to 3 h. Physiological parameters, production of inflammatory mediators and leucocyte infiltration were measured before and after perfusion. Lungs subjected to a cold ischaemia time of up to 6 h showed stable physiological conditions …

ChemokinePathologymedicine.medical_specialtymedicine.medical_treatmentImmunologyIntercellular Adhesion Molecule-1Vascular Cell Adhesion Molecule-1Blood PressurePulmonary EdemaPulmonary ArteryBasic ImmunologyHypothermia InducedmedicineImmunology and AllergyAnimalsRespiratory systemRats WistarLungChemokine CCL2LungbiologyCell adhesion moleculemedicine.diseaseIntercellular Adhesion Molecule-1RatsEndothelial stem cellCytokinemedicine.anatomical_structureReperfusion InjuryImmunologybiology.proteinLeukocytes MononuclearTissue PreservationInflammation MediatorsE-SelectinReperfusion injuryCell Adhesion MoleculesLung Transplantation
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An inducible mouse model of colon carcinogenesis for the analysis of sporadic and inflammation-driven tumor progression.

2007

Colorectal cancer is a life-threatening disease that can develop spontaneously or as a complication of inflammatory bowel diseases. Mouse models are essential tools for the preclinical testing of novel therapeutic options in vivo. Here, we provide a highly reliable protocol for an experimental mouse model to study the development of colon cancers. It is based on the mutagenic agent azoxymethane (AOM), which exerts colonotropic carcinogenicity. Repeated intraperitoneal administration of AOM results in the development of spontaneous tumors within 30 weeks. As an alternative option, inflammation-dependent tumor growth can be investigated by combining the administration of AOM with the inflamma…

Colorectal cancerAzoxymethaneInflammationDiseaseTumor initiationBiologyBioinformaticsGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundMiceIn vivomedicineAnimalsCarcinogenAzoxymethaneDextran Sulfatemedicine.diseaseDisease Models AnimalchemistryTumor progressionColonic NeoplasmsCancer researchCarcinogensDisease Progressionmedicine.symptomInflammation MediatorsMutagensNature protocols
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Impaired Mast Cell-Driven Immune Responses in Mice Lacking the Transcription Factor NFATc2

2009

Abstract The three calcium-dependent factors NFATc1, c2, and c3 are expressed in cells of the immune system and play pivotal roles in modulating cellular activation. With regard to NFATc2, it was reported that NFATc2-deficient mice display increased immune responses in several models for infection and allergy in vivo. This led to the assumption that NFATc2 is involved in the maintenance of immune homeostasis. Using the synthetic TLR7 agonist imiquimod as an adjuvant in epicutaneous peptide immunization, we observed that both the inflammatory reaction and the peptide-specific CTL response are severely impaired in NFATc2-deficient mice. Detailed analyses revealed that early production of proi…

Cytotoxicity ImmunologicImmunologyMice TransgenicInflammationBiologyProinflammatory cytokineMiceImmune systemAdjuvants ImmunologicCell MovementmedicineAnimalsImmunology and AllergyMast CellsLymph nodeInflammationNFATC Transcription FactorsReverse Transcriptase Polymerase Chain ReactionTLR7Flow CytometryMast cellAcquired immune systemCTL*medicine.anatomical_structureLangerhans CellsImmunologyInflammation Mediatorsmedicine.symptomThe Journal of Immunology
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Traditionally used Thai medicinal plants: in vitro anti-inflammatory, anticancer and antioxidant activities.

2009

In order to assess traditional Thai claims about the therapeutic potential of medicinal plants and to select plants for future phytochemical research, nine plant species with anti-inflammatory uses were selected from Thai textbooks and assessed for their in vitro anti-inflammatory, antiproliferative and antioxidant activities.Nuclear factor-kappaB (NF-kappaB) inhibitory effects in stably transfected HeLa cells were determined by luciferase assay, and effects on LPS-induced pro-inflammatory mediators prostaglandin E2 (PGE2), interleukin (IL)-6, IL-1beta, and tumour necrosis factor (TNF)alpha in primary monocytes were assessed by ELISA. Cytotoxic activities were examined against HeLa cells, h…

DPPHmedicine.drug_classCell SurvivalInterleukin-1betaAnti-Inflammatory AgentsPharmacognosyAsteraceaeTransfectionAnti-inflammatoryAntioxidantsDinoprostoneMonocytesHeLachemistry.chemical_compoundInhibitory Concentration 50MagnoliopsidaPhenolsDrug DiscoveryMedicineHumansGynuraPharmacologyPlants MedicinalTraditional medicinebiologyDose-Response Relationship Drugbusiness.industryInterleukin-6Plant ExtractsTumor Necrosis Factor-alphaNF-kappa Bbiology.organism_classificationThailandOroxylum indicumAntineoplastic Agents PhytogenicPolygonaceaeRhinacanthus nasutusPhytochemicalchemistryDrug Resistance NeoplasmBignoniaceaeLipid PeroxidationMedicine TraditionalInflammation MediatorsbusinessHeLa CellsJournal of ethnopharmacology
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Adipokines and Endotoxemia Correlate with Hepatic Steatosis in Non-Alcoholic Fatty Liver Disease (NAFLD)

2020

(1) Background: The etiology of non-alcoholic fatty liver disease (NAFLD) is multifactorial. Dietary composition has been implicated as a factor modulating intestinal barrier and could affect disease severity. The aim of this study was to evaluate dietary intake and markers of intestinal permeability in patients with NAFLD. (2) Methods: We enrolled 63 patients with NAFLD and compared them to age-matched controls. (3) Results: body mass index (BMI) and leptin to adiponectin ratio&mdash

Dietary FiberMale0301 basic medicinemedicine.medical_specialtyAdipokinelcsh:TX341-641GastroenterologyArticle03 medical and health sciences0302 clinical medicineAdipokinesRisk FactorsInternal medicineHumansMedicinehepatic fibrosisNutrition and DieteticsIntestinal permeabilityAdiponectinbusiness.industryLeptinFatty livernon-alcoholic fatty liver diseasedietary fiber consumptionnutritional and metabolic diseasesMiddle Agedmedicine.diseaseEndotoxemiadigestive system diseasesDietFatty Liver030104 developmental biologyCase-Control StudiesFemale030211 gastroenterology & hepatologyDisease Susceptibilitybacterial endotoxinInflammation MediatorsSteatosisbusinessHepatic fibrosislcsh:Nutrition. Foods and food supplyBody mass indexBiomarkersFood ScienceNutrients
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TGF-beta as a T cell regulator in colitis and colon cancer

2005

TGF-beta is a pleiotropic cytokine with powerful immunosuppressive functions. Mice deficient for TGF-beta1 show a dramatic phenotype with severe multiorgan inflammation and die shortly after birth. Recent investigations have highlighted the role of TGF-beta in suppression of T cell mediated autoimmune inflammation and anti-tumor immunity. In addition to its direct anti-inflammatory effects on T cells, TGF-beta has been implicated as central regulator of regulatory T cells. TGF-beta not only mediates the suppression of effector T cells by Tregs, recent evidence also reveals a role for TGF-beta along with TCR stimulation in the peripheral induction of regulatory T cells from naïve CD4+CD25- c…

Endocrinology Diabetes and MetabolismT cellImmunologyBiologyGeneral Biochemistry Genetics and Molecular BiologyTCIRG1Interleukin 21T-Lymphocyte SubsetsTransforming Growth Factor betamedicineAnimalsHumansImmunology and AllergyCytotoxic T cellIL-2 receptorIntestinal MucosaAntigen-presenting cellZAP70Cell DifferentiationColitisNatural killer T cellDisease Models Animalmedicine.anatomical_structureColonic NeoplasmsImmunologyInflammation MediatorsCytokine & Growth Factor Reviews
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Cell-mediated lipoprotein transport: A novel anti-atherogenic concept

2010

Lipoprotein transport is thought to occur in the plasma compartment of the blood, where lipoproteins are modulated by various enzymatic reactions. Subsequently, lipoproteins can migrate through the endothelial barrier to the subendothelial space or are taken up by the liver. The interaction between pro-atherogenic (apoB-containing) lipoproteins and blood cells (especially monocytes and macrophages) in the subendothelial space is well known. This lipoprotein-inflammatory cell interplay is central in the development of the atherosclerotic plaque. In this review, a novel interaction is described between lipoproteins and both leukocytes and erythrocytes in the blood compartment. This lipoprotei…

ErythrocytesApolipoprotein BLipoproteinsComplement receptor 1Blood lipidsLeukocytesInternal MedicinemedicineAnimalsHumansEndothelial dysfunctionComplement ActivationApolipoproteins BbiologyBiological TransportGeneral MedicineLipoprotein(a)Atherosclerosismedicine.diseaseComplement systemCell biologyLiverBiochemistryLipoprotein transportbiology.proteinlipids (amino acids peptides and proteins)Inflammation Mediatorsbiology.geneCardiology and Cardiovascular MedicineLipoproteinAtherosclerosis Supplements
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Pro-Inflammatory Genetic Markers of Atherosclerosis

2013

Atherosclerosis (AS) is a chronic, progressive, multifactorial disease mostly affecting large and medium-sized elastic and muscular arteries. It has formerly been considered a bland lipid storage disease. Currently, multiple independent pathways of evidence suggest this pathological condition is a peculiar form of inflammation, triggered by cholesterol-rich lipoproteins and influenced both by environmental and genetic factors. The Human Genome Project opened up the opportunity to dissect complex human traits and to understand basic pathways of multifactorial diseases such as AS. Population-based association studies have emerged as powerful tools for examining genes with a role in common mul…

Genetic MarkersSettore MED/09 - Medicina InternaPopulationGenome-wide association studyCoronary Artery DiseaseDiseaseBioinformaticsPolymorphism Single NucleotideCoronary heart disease; genetics; inflammation; meta-analysisSettore MED/05 - Patologia ClinicaHumansSNPMedicineGenetic Predisposition to DiseasePrecision MedicineeducationGenetic associationSettore MED/04 - Patologia GeneraleInflammationGeneticseducation.field_of_studyPolymorphism Geneticbusiness.industryAtherosclerosisPrecision medicineCoronary heart diseasemeta-analysisPersonalized medicinegeneticInflammation MediatorsCardiology and Cardiovascular MedicinebusinessRisk assessmentGenome-Wide Association StudyCurrent Atherosclerosis Reports
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Inhibition of the HER2 pathway by n-3 polyunsaturated fatty acids prevents breast cancer in fat-1 transgenic mice

2013

Overexpression of the tyrosine kinase receptor, ErbB2/HER2/Neu, occurs in 25–30% of invasive breast cancer (BC) with poor patient prognosis. Due to confounding factors, inconsistencies still remain regarding the protective effects of n-3 polyunsaturated fatty acids (PUFAs) on BC. We therefore evaluated whether fat-1 transgenic mice, endogenously synthesizing n-3 PUFAs from n-6 PUFAs, were protected against BC development, and we then aimed to study in vivo a mechanism potentially involved in such protection. E0771 BC cells were implanted into fat-1 and wild-type (WT) mice. After tumorigenesis examination, we analyzed the expression of proteins involved in the HER2 signaling pathway and lipi…

Genetically modified mouseFatty Acid DesaturasesDocosahexaenoic AcidsReceptor ErbB-3Receptor ErbB-2Breast NeoplasmsMice TransgenicQD415-436Biologymedicine.disease_causexenograft preventionBiochemistryReceptor tyrosine kinaseProto-Oncogene Proteins c-mycMiceEndocrinologyDownregulation and upregulationCell Line TumorFatty Acids Omega-3medicineAnimalsHumansCaenorhabditis elegans ProteinsResearch ArticlesCell Proliferationchemistry.chemical_classificationCell growthCell BiologyXenograft Model Antitumor AssaysGene Expression Regulation NeoplasticMice Inbred C57BLchemistryDocosahexaenoic acidImmunologyn-3 tissue enrichmentbiology.proteinCancer researchFemaleSignal transductionCarcinogenesispolyunsaturated fatty acid-derived mediatorsPolyunsaturated fatty acidSignal TransductionJournal of Lipid Research
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CARDIOMETABOLIC SYNDROME AND SARCOPENIC OBESITY IN OLDER PERSONS

2007

The aging of the world's population is a major contributor to the growing prevalence of the cardiometabolic syndrome (CMS) because older persons are more affected by the constellation of cardiovascular risk factors that constitute the syndrome. The prevalence of CMS has been related to the increasing prevalence of obesity, which is growing progressively even among older age groups. Indeed, obesity and aging are 2 overlapping mounting public health problems. It is currently accepted that CMS predicts cardiovascular mortality and/or the development of type 2 diabetes mellitus, and this is also true in studies including older persons. CMS is further complicated by modifications in body composi…

Gerontologymedicine.medical_specialtyAgingEndocrinology Diabetes and MetabolismPopulationAdipokineCardiometabolic syndromeInternal MedicinemedicineHumansSarcopenic obesityObesityeducationhealth care economics and organizationsAgedMetabolic Syndromeeducation.field_of_studybusiness.industryPublic healthType 2 Diabetes Mellitusmedicine.diseaseObesityMuscular AtrophyAdipose TissueCardiovascular DiseasesSarcopeniaLean body massPhysical therapyBody CompositionInflammation MediatorsInsulin ResistanceCardiology and Cardiovascular MedicinebusinessHuman
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