Search results for "Mesh"

showing 10 items of 885 documents

Global, regional, and national comparative risk assessment of 79 behavioural, environmental and occupational, and metabolic risks or clusters of risk…

2015

Summary Background The Global Burden of Disease, Injuries, and Risk Factor study 2013 (GBD 2013) is the first of a series of annual updates of the GBD. Risk factor quantification, particularly of modifiable risk factors, can help to identify emerging threats to population health and opportunities for prevention. The GBD 2013 provides a timely opportunity to update the comparative risk assessment with new data for exposure, relative risks, and evidence on the appropriate counterfactual risk distribution. Methods Attributable deaths, years of life lost, years lived with disability, and disability-adjusted life-years (DALYs) have been estimated for 79 risks or clusters of risks using the GBD 2…

MaleFine particulate matterNutrition and DiseaseMESH : SanitationHealth BehaviorDiseasesMESH: Metabolic DiseasesMESH: Global Health030204 cardiovascular system & hematologyMESH: Risk AssessmentGlobal HealthMESH : Nutritional StatusMESH: Occupational Exposure0302 clinical medicineUnsafe SexMESH: Risk FactorsRisk FactorsVoeding en ZiekteMedicineAir-pollutionMESH : Female030212 general & internal medicineMESH : Risk AssessmentSanitationWasting2. Zero hungerFactors de risc en les malaltiesMedicine (all)[ SDV.SPEE ] Life Sciences [q-bio]/Santé publique et épidémiologiePublic Health Global Health Social Medicine and EpidemiologyGeneral MedicineMESH : Occupational DiseasesMESH: Nutritional StatusAll-cause mortalityMESH : Risk FactorshumanitiesEnvironmental Exposure; Female; Global Health; Health Behavior; Humans; Male; Metabolic Diseases; Nutritional Status; Occupational Diseases; Occupational Exposure; Risk Assessment; Risk Factors; Sanitation; Medicine (all)Tobacco smoking3. Good healthOccupational DiseasesNutritional StatuMESH : Occupational ExposureMESH : Metabolic DiseasesCohortFemalemedicine.symptomRisk assessmentBlood-pressureHumanMESH: Occupational DiseasesRisk factors in diseasesCoronary-heart-diseaseMESH : MaleMESH: Health BehaviorMESH: Environmental ExposureNutritional StatusPopulation healthBody-mass indexRisk Assessment03 medical and health sciencesHousehold cookingMetabolic DiseasesCardiovascular-diseaseEnvironmental healthGeneral & Internal MedicineOccupational Exposureparasitic diseasesLife ScienceMESH: SanitationHumansRisk factorMESH : Health BehaviorVLAGGBD2013MESH: Humansbusiness.industryRisk FactorGlobal Burden of Disease Study; 79 behavioural environmental and occupational and metabolic risksLong-term exposureMESH : HumansCAUSE-SPECIFIC MORTALITYEnvironmental ExposureMESH: MaleMetabolic DiseaseOccupational DiseaseFolkhälsovetenskap global hälsa socialmedicin och epidemiologiMALE BRITISH DOCTORSYears of potential life lostRelative riskMalaltiesMESH : Global HealthOUTDOOR AIR-POLLUTION[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologiebusinessMESH : Environmental ExposureMESH: FemaleLancet
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A novel DFNB1 deletion allele supports the existence of a distant cis-regulatory region that controls GJB2 and GJB6 expression

2010

Contains fulltext : 87760_1.pdf (author's version ) (Open Access) Contains fulltext : 87760_2.pdf (Publisher’s version ) (Closed access) Eleven affected members of a large German-American family segregating recessively inherited, congenital, non-syndromic sensorineural hearing loss (SNHL) were found to be homozygous for the common 35delG mutation of GJB2, the gene encoding the gap junction protein Connexin 26. Surprisingly, four additional family members with bilateral profound SNHL carried only a single 35delG mutation. Previously, we demonstrated reduced expression of both GJB2 and GJB6 mRNA from the allele carried in trans with that bearing the 35delG mutation in these four persons. Usin…

MaleGenetics and epigenetic pathways of disease [NCMLS 6][SDV]Life Sciences [q-bio]PenetranceMESH: Base SequenceRegulatory Sequences Nucleic Acidsensorineural hearing lossConnexinsMESH: GenotypeMESH: Hearing Loss Sensorineural/diagnosisMESH: PenetranceGenotypeCopy-number variationGenetics (clinical)Sequence DeletionGeneticsComparative Genomic Hybridization0303 health sciencesMESH: Genetic TestingMESH: Gene Expression Regulation*030305 genetics & heredityPenetranceGJB2PedigreeConnexin 26MESH: Sequence Deletion*MESH: Hearing Loss Sensorineural/geneticsFemaleChromosome DeletionFunctional Neurogenomics [DCN 2]GJB6GenotypeMESH: PedigreeMESH: Chromosome DeletionHearing Loss SensorineuralMolecular Sequence Dataconnexin 26connexin 30DFNB1gene expression regulationGJB2GJB6sensorineural hearing losssequence deletionBiologyMESH: Connexin 30MESH: Connexins/genetics*MESH: Sequence Homology Nucleic AcidArticleGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesMonoallelic MutationGJB6MESH: Connexin 26Sequence Homology Nucleic AcidConnexin 30otorhinolaryngologic diseasesGeneticsHumansGenetic TestingAlleleGeneMESH: Regulatory Sequences Nucleic Acid/genetics*AllelesDFNB1030304 developmental biologyFamily HealthMESH: HumansMESH: Molecular Sequence DataBase SequenceChromosomes Human Pair 13MESH: AllelesBreakpointMESH: MaleMESH: Comparative Genomic HybridizationGene Expression RegulationMESH: Family Healthbiology.proteinHuman medicineMESH: Chromosomes Human Pair 13/geneticsMESH: FemaleClinical Genetics
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Attenuation of disease phenotype through alternative translation initiation in low-penetrance retinoblastoma

2006

Hereditary predisposition to retinoblastoma (RB) is caused by germline mutations in the retinoblastoma 1 (RB1) gene and transmits as an autosomal dominant trait. In the majority of cases disease develops in greater than 90% of carriers. However, reduced penetrance with a large portion of disease-free carrier is seen in some families. Unambiguous identification of the predisposing mutation in these families is important for accurate risk prediction in relatives and their genetic counseling but also provides conceptual information regarding the relationship between the RB1 genotype and the disease phenotype. In this study we report a novel mutation detected in 10 individuals of an extended fa…

MaleGenotypeDNA Mutational AnalysisGreen Fluorescent ProteinsMolecular Sequence DataPenetranceBiologyRetinoblastoma ProteinFrameshift mutationExonGermline mutationGeneticsmedicineHumansGenetic Predisposition to DiseaseAmino Acid SequenceRNA MessengerChildFrameshift MutationPeptide Chain Initiation TranslationalGenetics (clinical)GeneticsRetinoblastomaRetinoblastomaInfantAutosomal dominant traitExonsmedicine.diseasePenetranceAlternative SplicingPhenotypeCodon NonsenseHereditary RetinoblastomaMutation (genetic algorithm)FemaleHuman Mutation
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Stability of pedalling mechanics during a prolonged cycling exercise performed at different cadences.

2005

The aim of this study was to analyse the effect of pedalling rate on the pattern of mechanical torque application and on neuromuscular fatigue during prolonged cycling exercise. Eleven well-trained individuals performed three 1-h pedalling sessions, at 50 rev.min-1, 110 rev.min-1 and a freely chosen cadence, at an intensity corresponding to 65% of their maximal aerobic power. The mechanical torque applied on the right pedal was recorded for 30 s every 5 min while pedalling. Contractile and neural properties of the quadriceps and hamstring muscles were analysed before and immediately after each of the three pedalling sessions. The post-exercise reduction in knee extensors maximal voluntary c…

MaleHamstring muscles[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH: Muscle ContractionMuscle RelaxationMESH : Analysis of VarianceElectromyographyCohort Studies0302 clinical medicineVoluntary contractionMESH: Risk FactorsRisk FactorsOrthopedics and Sports MedicineMESH: Oxygen ConsumptionMESH : Oxygen ConsumptionMESH: Cohort StudiesKnee extensorsmedicine.diagnostic_test[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]BiomechanicsMESH: Comparative StudyMESH : AdultMESH : Risk FactorsMESH: Muscle FatigueMuscle FatigueMESH : ElectromyographyCyclingCadenceMESH: Physical EnduranceMESH : Physical EnduranceMuscle ContractionAdultmedicine.medical_specialtyMESH: ProbabilityMESH : ProbabilityMESH : MalePhysical ExertionMESH : ExertionMESH : Cohort StudiesPhysical Therapy Sports Therapy and RehabilitationSensitivity and SpecificityMESH : Muscle RelaxationMESH: BicyclingMESH: Electromyography03 medical and health sciencesPhysical medicine and rehabilitationOxygen ConsumptionMESH: Analysis of VariancemedicineMESH: Exertion[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]HumansProbabilityAnalysis of VarianceMESH : SMESH: Humansbusiness.industryElectromyographyMESH : HumansMESH : Comparative StudyMESH: Adult030229 sport sciencesNegative workMESH : Muscle FatigueMESH: MaleBicyclingbody regionsMESH : Exercise TestTorqueMESH : BicyclingMESH: SMESH: Muscle RelaxationPhysical therapyExercise TestPhysical EnduranceMESH : Muscle ContractionbusinessMESH: Exercise Testhuman activities030217 neurology & neurosurgeryJournal of sports sciences
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Intra-arterial idarubicin_lipiodol without embolization can provide prolonged complete response in hepatocellular carcinoma: A case report.

2020

International audience; Hepatocellular carcinoma is the fourth leading cause of cancer death. For unresectable intermediate-stage hepatocellular carcinoma, the standard treatment is transarterial chemoembolization. To date, the overall survival at three years remains low, and there is currently no consensus about the best anticancer agent and optimal treatment regimen. We report the case of a hepatocellular carcinoma patient with a vascular contraindication to embolization who achieved a complete response after four intra-arterial infusions of idarubicin emulsified with lipiodol. The patient maintained his response over a three-year period without any hepatocellular carcinoma treatment, dem…

MaleHepatocellular carcinoma[SDV.IB.IMA]Life Sciences [q-bio]/Bioengineering/Imagingmedicine.medical_treatmentGastroenterologyEthiodized Oil0302 clinical medicineMESH: Infusions Intra-ArterialMESH: Liver NeoplasmsPharmacology (medical)EmbolizationMESH: Carcinoma HepatocellularComplete responseMESH: Treatment OutcomeMESH: AgedStandard treatmentLiver Neoplasms[SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciences3. Good healthTreatment OutcomeOncology030220 oncology & carcinogenesisHepatocellular carcinomaLipiodol030211 gastroenterology & hepatologymedicine.drugmedicine.medical_specialtyIntra-arterial therapyCarcinoma HepatocellularAntineoplastic Agents[SDV.CAN]Life Sciences [q-bio]/Cancer03 medical and health sciencesMESH: Ethiodized OilInternal medicinemedicineHumansInfusions Intra-ArterialIdarubicinContraindicationAgedMESH: Humansbusiness.industryMESH: Idarubicinmedicine.diseasedigestive system diseasesMESH: MaleRegimenMESH: Antineoplastic AgentsbusinessIdarubicin
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H-ficolin (ficolin-3) concentrations and FCN3 gene polymorphism in neonates

2011

Serum H-ficolin (ficolin-3) concentrations (n=613) and FCN3 genotypes (n=529) from a large group of neonates are presented. Both pre-term deliveries and low birthweight (independently of gestational age) were significantly associated with low H-ficolin concentrations but not with heterozygosity for the FCN3 1637delC frameshift mutation. The presence of the variant allele, however, apparently influenced the protein level. No association of FCN3 gene heterozygosity or relative functional H-ficolin insufficiency (determined as serum level ≤8.6 μg/ml) with perinatal infections was found. One premature newborn, with confirmed infection caused by Streptococcus agalactiae, was H-ficolin-deficient …

MaleHeterozygotemedicine.medical_specialtyGenotypeImmunologyGestational AgeBiologymedicine.disease_causeMannose-Binding LectinStreptococcus agalactiaeFrameshift mutationLoss of heterozygosityPolymorphism (computer science)LectinsStreptococcal InfectionsInternal medicineGenotypemedicineHumansImmunology and AllergyFrameshift MutationAllelesGlycoproteinsMannan-binding lectinPolymorphism GeneticHomozygoteInfant NewbornHematologyInfant Low Birth WeightEndocrinologyStreptococcus agalactiaeMannose-Binding Protein-Associated Serine ProteasesImmunologyPremature BirthFemaleGene polymorphismFicolinImmunobiology
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Bone marrow fat quantification of osteoporotic vertebral compression fractures: comparison of multi-voxel proton MR spectroscopy and chemical-shift g…

2011

Background Only a few studies have used in/opposed phase method for a quantitative evaluation of fat fraction in the spine. Purpose To compare multivoxel proton MR spectroscopy and chemical-shift gradient-echo MR imaging for bone marrow fat quantification in vertebral compression fractures (VCF). Material and Methods Vertebral marrow fat quantification in fifteen patients was measured at 3.0-T. Multi-voxel proton spectroscopy (MRS) and in/opposed-phase MR imaging using a fat map build with a triple-echo gradient-echo sequence was used. All the patients had benign vertebral collapse. Bone marrow fat content was evaluated by both techniques in compressed (acute or chronic) and in non-compress…

MaleIn vivo magnetic resonance spectroscopyMagnetic Resonance Spectroscopy[SDV]Life Sciences [q-bio]Fat quantificationcomputer.software_genreMESH: Magnetic Resonance Imaging030218 nuclear medicine & medical imagingMESH: Aged 80 and over0302 clinical medicineBone MarrowVoxelFractures CompressionMESH: Fractures CompressionAged 80 and overMESH: AgedMESH: Middle Aged[ INFO.INFO-IM ] Computer Science [cs]/Medical ImagingRadiological and Ultrasound TechnologyGeneral MedicineMiddle AgedCompression (physics)LipidsMagnetic Resonance Imagingmedicine.anatomical_structureAcute DiseaseSpinal FracturesMESH: Acute DiseaseFemaleMESH: Bone MarrowMESH: OsteoporosisAdultMESH: Spinal Fractures03 medical and health sciences[INFO.INFO-IM]Computer Science [cs]/Medical ImagingmedicineHumansRadiology Nuclear Medicine and imagingAgedMESH: HumansMESH: Magnetic Resonance Spectroscopybusiness.industryMESH: Chronic DiseaseVertebral compression fractureMESH: Adultmedicine.diseaseMESH: LipidsMr imagingMESH: MaleProton mr spectroscopyChronic DiseaseOsteoporosisBone marrowbusinessNuclear medicineMESH: Femalecomputer030217 neurology & neurosurgeryActa Radiologica
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Effects of typical inducers on olfactory xenobiotic-metabolizing enzyme, transporter, and transcription factor expression in rats.

2010

International audience; Several xenobiotic-metabolizing enzymes (XMEs) have been identified in the olfactory mucosa (OM) of mammals. However, the molecular mechanisms underlying the regulation of these enzymes have been little explored. In particular, information on the expression of the transcriptional factors in this tissue is quite limited. The aim of the present study was to examine the impact of five typical inducers, Aroclor 1254, 3-methylcholanthrene, dexamethasone, phenobarbital, and ethoxyquin, on the activities and mRNA expression of several XMEs in the OM and in the liver of rats. We also evaluated the effects of these treatments on the mRNA expression of transcription factors an…

MaleLIVERMESH : Transcription FactorsMESH: Microsomes Liver[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionPharmaceutical ScienceMESH : CytochromesMESH: Down-RegulationMESH: Membrane Transport ProteinsMESH : Down-RegulationCytosol0302 clinical medicineGlucocorticoid receptorMESH : Membrane Transport ProteinsMESH: CytosolMESH: Reverse Transcriptase Polymerase Chain ReactionGene expressionConstitutive androstane receptorMESH: Up-RegulationMESH: AnimalsReceptorMESH : Up-RegulationMESH: Cytochromes0303 health sciencesPregnane X receptorMESH : Metabolic Detoxication Phase IbiologyReverse Transcriptase Polymerase Chain ReactionMESH : RatsMESH : CytosolINDUCTIONMESH : Reverse Transcriptase Polymerase Chain ReactionMESH: Transcription FactorsUp-Regulation3. Good healthMESH : Microsomes LiverHYDROCARBON HYDROXYLASE-ACTIVITYmedicine.anatomical_structurePHASE-IBiochemistryMESH: Metabolic Detoxication Phase IIEnzyme InductionMicrosomes LiverMESH: Metabolic Detoxication Phase IMESH: XenobioticsMESH: Enzyme InductionMESH: RatsMESH : MaleDown-RegulationMESH : XenobioticsPHENOL SULFOTRANSFERASEMESH : Rats WistarXenobiotics03 medical and health sciencesOlfactory mucosaOlfactory MucosamedicineAnimalsRats WistarMESH: Olfactory MucosaTranscription factor030304 developmental biologyPharmacologyMESH : Olfactory MucosaIDENTIFICATIONRECEPTORMESH : Enzyme InductionMembrane Transport ProteinsMESH : Metabolic Detoxication Phase IIUDP-GLUCURONOSYLTRANSFERASEMESH: Rats WistarAryl hydrocarbon receptorORGANIC ANION TRANSPORTERMolecular biologyMetabolic Detoxication Phase IIMESH: MaleRatsNASAL-MUCOSAbiology.proteinCytochromesMetabolic Detoxication Phase IMESH : Animals[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryTranscription Factors
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Functional reorganization of the attentional networks in low-grade glioma patients: a longitudinal study.

2015

International audience; Right brain damage often provokes deficits of visuospatial attention. Although the spatial attention networks have been widely investigated in stroke patients as well as in the healthy brain, little is known about the impact of slow growing lesions in the right hemisphere. We here present a longitudinal study of 20 patients who have been undergoing awake brain surgery with per-operative line bisection testing. Our aim was to investigate the impact of tumour presence and of tumour resection on the functional (re)organization of the attention networks. We assessed patients' performance on lateralized target detection, visual exploration and line bisection before surger…

MaleLongitudinal studyMESH: Attention: physiologySettore M-PSI/02 - PSICOBIOLOGIA E PSICOLOGIA FISIOLOGICAcost function maskingAudiologyFunctional LateralityDevelopmental psychologyMESH: Nerve Net: pathologyphysiopathologyAttentionLongitudinal StudiesMESH: Space Perception: physiologyMESH: Longitudinal Studiesmedia_commonMESH: AgedMESH: Middle AgedBrain Neoplasmsbrain-tumor patientsright-hemisphere damageFunctional recoveryGliomaMiddle AgedMESH: Functional Laterality: physiologyNeuropsychology and Physiological Psychologymedicine.anatomical_structureMESH: Young Adultunilateral neglectFemalevisual neglect[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]medicine.symptomPsychologyAdultmedicine.medical_specialtyAdolescentCognitive Neurosciencemedia_common.quotation_subjectExperimental and Cognitive Psychologyspatial neglectMESH: Brain Neoplasms: complicationspathologyphysiopathologyLateralization of brain functionNeglectWhite matterPerceptual DisordersYoung AdultMESH: Perceptual Disorders: etiologypathologyphysiopathologymedicineDisconnection syndromeHumansSlow growing lesionNeglectAgeddisconnection syndromeMESH: AdolescentMESH: HumansHemispatial neglecthemispatial neglectMESH: AdultMESH: Glioma: complicationspathologyphysiopathologyFunctional recoveryMESH: Malesustained attentionUnilateral neglectSpace PerceptionNerve Netvisuospatial attentionMESH: Female
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Strength training in old age: adaptation of antagonist muscles at the ankle joint.

2005

The purpose of this study was to determine whether strength training could reduce the deficit in plantarflexion (PF) maximal voluntary contraction (MVC) torque observed in previous studies in older subjects relative to young adults. Accordingly, the effects of a 6-month strength training program on the muscle and neural properties of the major muscle groups around the ankle were examined. PF and dorsiflexion (DF) isometric MVC torques were measured and surface electromyographic activity of the triceps surae and tibialis anterior muscles was recorded. The strength training program was very effective in improving strength in PF (+24.5%), and it thus reduced the DF-to-PF MVC torque ratio; in a…

MaleMESH : Ankle Joint[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH : Electric StimulationPhysiologyMESH: Muscle ContractionMESH : AgedMESH: Physical FitnessIsometric exerciseElectromyographyMESH: Research Support Non-U.S. Gov'tMESH : Research Support Non-U.S. Gov't0302 clinical medicineTriceps surae muscleMESH: Ankle JointMESH : FemaleMESH : Muscle SkeletalMESH : Adaptation PhysiologicalMESH : AlgorithmsMESH: AgedMESH: Muscle SkeletalMESH: Middle Agedmedicine.diagnostic_test[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH: Electric StimulationMiddle AgedAdaptation PhysiologicalCoactivationmedicine.anatomical_structureData Interpretation StatisticalMESH : ElectromyographyFemalemedicine.symptomMESH : Physical FitnessAlgorithmsMuscle ContractionMuscle contractionmedicine.medical_specialtyWeight LiftingStrength trainingMESH : MaleJoint stabilityMESH: AlgorithmsMESH: Electromyography03 medical and health sciencesCellular and Molecular NeurosciencePhysical medicine and rehabilitationPhysiology (medical)medicine[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]HumansMESH : Middle AgedMESH : Data Interpretation StatisticalMuscle SkeletalAgedMESH: HumansElectromyographybusiness.industryMESH : Humans030229 sport sciencesMESH: Adaptation PhysiologicalElectric StimulationMESH: MalePhysical FitnessPhysical therapyMESH : Muscle ContractionNeurology (clinical)AnklebusinessMESH: Data Interpretation StatisticalMESH: FemaleAnkle Joint030217 neurology & neurosurgery
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