Search results for "Molecular Medicine"

showing 10 items of 3013 documents

Effects of nicotine receptor agonists on acetylcholine release from the isolated motor nerve, small intestine and trachea of rats and guinea-pigs

1992

The effects of nicotine receptor agonists on the release of [3H]acetylcholine from the phrenic nerve, the small intestine and the trachea were investigated to characterize neuronal nicotine receptors within the peripheral nervous system. Contraction of the indirectly-stimulated hemidiaphragm was recorded to investigate desensitization of the postsynaptic muscular nicotine receptors. Nicotine, cytisine, 1,1-dimethyl-4-phenylpiperazinium and 2-(4-aminophenyl)-ethyl-trimethyl-ammoniumiodide caused a concentration-dependent (0.1-30 microM) increase in evoked [3H]acetylcholine release from the phrenic nerve, whereby bell-shaped concentration-response curves were obtained. The rank order of decre…

medicine.medical_specialtyDiaphragmGuinea PigsMyenteric PlexusMotor nerveReceptors NicotinicMotor EndplateNicotineCytisinechemistry.chemical_compoundPostsynaptic potentialInternal medicineIntestine SmallDrug DiscoverymedicineAnimalsGenetics (clinical)Phrenic nerveDose-Response Relationship DrugMuscle SmoothGeneral MedicineAcetylcholineStimulation ChemicalRatsPhrenic NerveTracheaEndocrinologymedicine.anatomical_structureParasympathomimeticschemistryPeripheral nervous systemMolecular Medicinemedicine.symptomSecretory RateAcetylcholineMuscle Contractionmedicine.drugMuscle contractionThe Clinical Investigator
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Expressional down-regulation of neuronal-type nitric oxide synthase I by glucocorticoids in N1E-115 neuroblastoma cells.

1998

Neuronal-type nitric oxide synthase (NOS I) is involved in ischemia-induced brain damage, and glucocorticoids have been reported to protect from brain damage. This prompted us to investigate if the activity or expression of NOS I was influenced by glucocorticoids. We used the murine neuroblastoma cell line N1E-115 as our experimental model. Short-term incubation (30 min) of the N1E-115 cells with dexamethasone (10 nM to 1 microM) or hydrocortisone (100 nM to 10 microM) did not change the enzymatic activity of NOS I. However, the glucocorticoids inhibited NOS I mRNA expression in a concentration-dependent fashion (down to 53.3 +/- 2. 5% of control). In time-course experiments with 100 nM dex…

medicine.medical_specialtyDown-RegulationNitric Oxide Synthase Type IBiologyNitric OxideDexamethasonechemistry.chemical_compoundMiceNeuroblastomaInternal medicinemedicineTumor Cells CulturedAnimalsRNA MessengerGlucocorticoidsDexamethasonePharmacologyNeuronsMessenger RNAAntiglucocorticoidMifepristoneNitric oxide synthaseBlotEndocrinologychemistryCell culturebiology.proteinMolecular MedicineNitric Oxide SynthaseGlucocorticoidmedicine.drugMolecular pharmacology
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Unpredictable Performance of pH-Dependent Coatings Accentuates the Need for Improved Predictive in Vitro Test Systems.

2017

First introduced in the second half of the 19th century, enteric coatings are commonly used to protect acid-labile drugs, reduce the risk of gastric side effects due to irritating drugs, or for local drug delivery to the lower gastrointestinal (GI) tract. The currently available enteric-coatings are based on pH-sensitive weakly acidic polymers. Despite the long history of their use, the causes behind their performance often being unpredictable have not been properly investigated with most of the attention being focused only on the gastric emptying. However, little attention has been given to the postgastric emptying disintegration and dissolution of these dosage forms. This lack of attentio…

medicine.medical_specialtyDrug LiberationIn vitro testChemistry PharmaceuticalPharmaceutical SciencePh dependentBiological Availability02 engineering and technologyPharmacologyIn Vitro Techniques030226 pharmacology & pharmacyDosage formBiopharmaceuticsExcipients03 medical and health sciences0302 clinical medicineDrug DiscoveryIntestine SmallmedicineIntensive care medicineGastric emptyingbusiness.industryHydrogen-Ion Concentration021001 nanoscience & nanotechnologyEnteric coatingBioavailabilityDrug LiberationSolubilityDrug deliveryMolecular MedicineTablets Enteric-Coated0210 nano-technologybusinessmedicine.drugMolecular pharmaceutics
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Cocaine and MDMA Induce Cellular and Molecular Changes in Adult Neurogenic Systems: Functional Implications

2011

The capacity of the brain to generate new adult neurons is a recent discovery that challenges the old theory of an immutable adult brain. A new and fascinating field of research now focuses on this regenerative process. The two brain systems that constantly produce new adult neurons, known as the adult neurogenic systems, are the dentate gyrus (DG) of the hippocampus and the lateral ventricules/olfactory bulb system. Both systems are involved in memory and learning processes. Different drugs of abuse, such as cocaine and MDMA, have been shown to produce cellular and molecular changes that affect adult neurogenesis. This review summarizes the effects that these drugs have on the adult neurog…

medicine.medical_specialtyDrugs of abuseMDMAlcsh:Medicinelcsh:RS1-441Pharmaceutical ScienceHippocampuscocaineReviewBiologylcsh:Pharmacy and materia medicamemoryDrug Discoverymedicinedentate gyrusPsychiatryDentate gyruslcsh:RNeurogenesisMDMAOlfactory bulbadult neurogenesisnervous systemMolecular MedicineNeurosciencemedicine.drugPharmaceuticals
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Effects of ovariectomy and steroid replacement on GABAA receptor binding in female rat brain.

1991

Abstract The specific binding of tritiated muscimol to γ-aminobutyric acid (GABA) receptor sites was studied in distinct brain areas of female rats during different endocrine states. In diestrous rats with intact ovaries the highest receptor densities were found in the cortex (10.24 pmol/mg protein) and the lowest concentrations in the mediobasal hypothalamus (3.29 pmol/mg protein). Four weeks after removal of the ovaries, the number of binding sites was enhanced up to 2.4-fold in all brain areas investigated: the preoptic brain area, mediobasal hypothalamus, corticomedial amygdala, and cerebral cortex. The affinity of the binding sites remained unchanged. Substitution of estradiol and prog…

medicine.medical_specialtyEndocrinology Diabetes and MetabolismOvariectomyClinical BiochemistryBiologyIn Vitro TechniquesTritiumBiochemistrySynaptic Transmissionchemistry.chemical_compoundEndocrinologyInternal medicineCortex (anatomy)medicineAnimalsReceptorMolecular BiologyProgesteroneEstradiolGABAA receptorMuscimolBrainRats Inbred StrainsCell BiologyLuteinizing HormoneReceptors GABA-ARatsPreoptic areamedicine.anatomical_structureEndocrinologynervous systemMuscimolchemistryCerebral cortexMolecular MedicineGABAergicFemaleHormoneThe Journal of steroid biochemistry and molecular biology
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Splenic respiratory gas exchange and glucose uptake in patients with splenomegaly in hypersplenism and Hodgkin's disease.

1977

Blood samples are taken from the splenic artery, vein and pulp of patients suffering from Hodgkin's disease (n=10) or hypersplenism (n=7) and undergoing splenectomy. In these samples, the relevant parameters of the respiratory gas exchange as well as glucose and lactate concentrations are determined. In hypersplenism (mean splenic wet weight: 543 g) the mean oxygen consumption of the splenic tissue amounts to 0.9 ml O2/100 g/min taking into account a mean splenic blood flow of 80 ml/100 g/min. The glucose uptake and the lactate release are 9 mg/100 g/min and 5.5 mg/100 g/min, respectively. These values are in close agreement with the results obtained in the normal and undisturbed spleen in …

medicine.medical_specialtyErythrocytesGlucose uptakeRespiratory gas exchangechemistry.chemical_elementSpleenSplenic arteryOxygenHypersplenismVeinsOxygen Consumptionmedicine.arteryInternal medicineDrug DiscoverymedicineHumansIn patientGenetics (clinical)business.industryGeneral MedicineVenous bloodHydrogen-Ion ConcentrationHodgkin DiseaseSurgerymedicine.anatomical_structureEndocrinologyGlucosechemistrySplenic TissueSplenomegalyLactatesMolecular MedicinebusinessSplenic ArterySpleenKlinische Wochenschrift
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Utilizing nutritional genomics to tailor diets for the prevention of cardiovascular disease: a guide for upcoming studies and implementations.

2017

Introduction: Personalized diets based on an individual’s genome to optimize the success of dietary intervention and reduce genetic cardiovascular disease (CVD) risk, is one of the challenges most frequently discussed in the scientific community. Areas covered: The authors gathered literature-based evidence on nutritional genomics and CVD phenotypes, our own results and research experience to provide a critical overview of the current situation of using nutritional genomics to tailor diets for CVD prevention and to propose guidelines for future studies and implementations. Expert commentary: Hundreds of studies on gene-diet interactions determining CVD intermediate (plasma lipids, hypertens…

medicine.medical_specialtyFuture studiesNutritional genomicsMediterranean dietDisease030204 cardiovascular system & hematologyBioinformaticsNutrigeneticsPathology and Forensic MedicineScientific evidence03 medical and health sciences0302 clinical medicineNutrigenomicsMediterranean dietPlasma lipidsGeneticsMedicineHumans030212 general & internal medicineDietary patternsIntensive care medicineMolecular BiologyNutrigeneticsbusiness.industryPrecision nutritionNutrientsCardiovascular diseaseLipidsDietNutrigenomicsCardiovascular DiseasesPersonalized dietsPractice Guidelines as TopicMolecular MedicineGenetic risk scoresbusinessDiet TherapyExpert review of molecular diagnostics
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The impact of alpha1-adrenoceptors up-regulation accompanied by the impairment of beta-adrenergic vasodilatation in hypertension

2008

9 pages, 7 figures, 3 tables.-- PMID: 19060223 [PubMed]

medicine.medical_specialtyG-Protein-Coupled Receptor Kinase 2Adrenergic receptorSystolemedia_common.quotation_subjectAdrenergicVasodilationModels BiologicalRats Inbred WKYDownregulation and upregulationHeart RateRats Inbred SHRReceptors Adrenergic alpha-1Internal medicineReceptors Adrenergic betamedicineAnimalsHumansRNA MessengerReceptorInternalizationAortamedia_commonPharmacologybiologyChemistryKinaseBeta adrenergic receptor kinaseRatsUp-RegulationVasodilationEndocrinologyHypertensionbiology.proteinMolecular Medicine
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Statin-Induced Liver Injury Involves Cross-Talk between Cholesterol and Selenoprotein Biosynthetic Pathways

2009

Statins have become the mainstay of hypercholesterolemia treatment. Despite a seemingly clear rationale behind their use, the inhibition of HMG-CoA reductase, these compounds have been shown to elicit a variety of unanticipated and elusive effects and side effects in vivo. Among the most frequently noted side effects of statin treatment are elevations in liver enzymes. Here, we report our finding that atorvastatin, cerivastatin, and lovastatin at clinically common concentrations induce a selective, differential loss of selenoprotein expression in cultured human HepG2 hepatocytes. The primarily affected selenoprotein was glutathione peroxidase (GPx), whose biosynthesis, steady-state expressi…

medicine.medical_specialtyGPX1Thioredoxin-Disulfide ReductaseStatinPyridinesmedicine.drug_classAtorvastatinBiologyGPX4tert-ButylhydroperoxideCell Line TumorInternal medicineAtorvastatinmedicineHumansPyrrolesLovastatinSelenoproteinsPharmacologychemistry.chemical_classificationGlutathione Peroxidaseintegumentary systemCytotoxinsGlutathione peroxidaseCerivastatinIsoenzymesCholesterolEndocrinologychemistryHeptanoic AcidsHepatocytesMolecular MedicineLovastatinSelenoproteinHydroxymethylglutaryl-CoA Reductase InhibitorsReactive Oxygen SpeciesSignal Transductionmedicine.drugMolecular Pharmacology
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�ber die unterschiedliche Wirkung von Follikelhormon und Stilbenen auf die Gonadotropinaussch�ttung der Rattenhypophyse

1955

medicine.medical_specialtyGeneral MedicineBiologyMolecular medicineHuman geneticsFollicular hormoneEndocrinologyInternal medicineDrug DiscoverymedicineMolecular MedicineSecretionGonadotropins pituitaryGenetics (clinical)Klinische Wochenschrift
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