Search results for "Morphine"

showing 10 items of 145 documents

Morphine potentiates the impairing effects of neuroleptics on two-way active conditioned avoidance response in male mice

2004

The dopaminergic and opioid systems have effects on the conditioned avoidance response (CAR), although the possible interaction between these systems on this behaviour has not been studied. The effects of morphine (12.6 mg/kg), haloperidol (0.075 mg/kg), sulpiride (20 mg/kg) and risperidone (0.1 mg/kg) alone as well as morphine combined with these dopamine (DA) antagonists on the acquisition and performance of the CAR were explored in mice. Morphine increased avoidances but this seemed secondary to a rise in activity levels. All DA antagonists impaired CAR in the acquisition phase but only haloperidol disrupted performance. The combination of morphine plus neuroleptics impaired acquisition …

MaleNarcoticsConditioning ClassicalPharmacologyAvoidance responseMiceEscape ReactionDopamineAvoidance LearningmedicineHaloperidolAnimalsBiological PsychiatryPharmacologyAnalysis of VarianceMice Inbred BALB CRisperidoneBehavior AnimalMorphinebusiness.industryDopaminergicDrug SynergismOpioidMorphineDopamine AntagonistsSulpiridebusinesshuman activitiesAntipsychotic Agentsmedicine.drugProgress in Neuro-Psychopharmacology and Biological Psychiatry
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Involvement of nitric oxide synthesis in sensitization to the rewarding effects of morphine

2009

Abstract Knowledge about the specific brain changes and neural plasticity processes produced by repeated exposure to a drug is essential to progress in the field of neurobiology of addiction and the development of effective medication. In the present study, the influence of nitric oxide synthesis on sensitization to the rewarding effects of morphine has been evaluated. The effects of pre-treatment of mice with saline or 20 mg/kg of morphine plus the nitric oxide synthase inhibitor 7-nitroindazole (7NI) (12.5 or 25 mg/kg) on the place conditioning induced by a low dose of morphine (2 mg/kg) were assessed. The dose of 2 mg/kg of morphine was ineffective in animals pre-treated with saline but …

MaleNarcoticsIndazoles7-Nitroindazolemedia_common.quotation_subjectConditioning ClassicalPharmacologyNitric OxideNitric oxideMicechemistry.chemical_compoundRewardmedicineAnimalsSensitizationmedia_commonMorphinebiologybusiness.industryGeneral NeuroscienceAddictionConditioned place preferenceNitric oxide synthasemedicine.anatomical_structurechemistryNitric Oxide PathwayMorphinebiology.proteinNitric Oxide Synthasebusinessmedicine.drugNeuroscience Letters
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Effects of DA D1 and D2 antagonists on the sensitisation to the motor effects of morphine in mice

2002

Abstract Acute morphine administration produces hyperactivity in mice and repeated treatment induces an enhancement of this effect. In this experiment, we study the sensitisation to the hyperactivity induced by intermittent morphine administration (40 mg/kg) and the effects of dopamine (DA) antagonists on this phenomenon. Animals received three injections, separated by 48 h, and after each injection, their activity was registered between 30 and 60 min. In Experiment 1, animals were divided into two groups, which received saline and morphine (S–S–M) or only morphine (M–M–M). In Experiment 2, animals were divided into 12 groups. Half, which was designed to study the effects of DA antagonists …

MaleNarcoticsMotor ActivityPharmacologyMicechemistry.chemical_compoundDopamineAnimalsMedicineNeurotransmitterBiological PsychiatrySensitizationPharmacologyRacloprideSCH-23390Morphinebusiness.industryReceptors Dopamine D1AntagonistDopamine D2 Receptor Antagonistsmedicine.anatomical_structurechemistryToxicityMorphineDopamine Antagonistsbusinessmedicine.drugProgress in Neuro-Psychopharmacology and Biological Psychiatry
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Memantine blocks sensitization to the rewarding effects of morphine

2009

Knowledge regarding the specific brain changes and neural plasticity processes produced by repeated drug exposure may be used to advance the understanding of the neurobiology of addiction in order to design appropriate medications. In the present study, the influence of N-methyl-d-aspartate (NMDA) glutamatergic receptors on sensitization to the motor and rewarding effects of morphine was evaluated. The effects of pre-exposure to saline or 20 mg/kg morphine plus the NMDA receptor antagonist memantine (10 or 20 mg/kg) on motor activity and place conditioning induced by a low dose of morphine (2 mg/kg) were assessed. The dose of 2 mg/kg of morphine was ineffective in mice pre-exposed to saline…

MaleNarcoticsMotor ActivityPharmacologyReceptors N-Methyl-D-AspartateMiceGlutamatergicRewardMemantineConditioning PsychologicalNeuroplasticityAnimalsMedicineMolecular BiologySensitizationAnalysis of VarianceMotivationDose-Response Relationship DrugMorphinebusiness.industryGeneral NeuroscienceGlutamate receptorMemantineConditioned place preferencemedicine.anatomical_structureMorphineNMDA receptorNeurology (clinical)businessExcitatory Amino Acid AntagonistsDevelopmental Biologymedicine.drugBrain Research
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Effect of adolescent exposure to MDMA and cocaine on acquisition and reinstatement of morphine-induce CPP

2007

It is well known that an elevated percentage of ecstasy users also consume cocaine. Recently, it has been reported that a high frequency of heroin smokers first consumed heroin under the effects of ecstasy with the hope of reducing the stimulant effects of the latter drug. The aim of the present study was to evaluate the effect of exposure to MDMA and cocaine during adolescence on morphine-induced conditioned place preference (CPP) and reinstatement in adulthood. In the first experiment, adolescent mice were exposed to six injections of MDMA and three weeks later their response to the reinforcing properties of 40 mg/kg of morphine was evaluated using the CPP paradigm. All the treatment grou…

MaleNarcoticsN-Methyl-34-methylenedioxyamphetaminemedicine.medical_treatmentEcstasyPharmacologyExtinction PsychologicalHeroinMiceCocaineDopamine Uptake InhibitorsmedicineAnimalsDrug InteractionsBiological PsychiatryPharmacologyAnalysis of VarianceGateway drugAdrenergic Uptake InhibitorsBehavior AnimalDose-Response Relationship DrugMorphineMDMAExtinction (psychology)Conditioned place preferenceStimulantAnimals NewbornMorphineConditioning OperantPsychologyReinforcement Psychologypsychological phenomena and processesmedicine.drugProgress in Neuro-Psychopharmacology and Biological Psychiatry
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The dopamine release inhibitor CGS 10746B blocks conditioned physical signs of morphine withdrawal

2003

Environment previously paired with morphine withdrawal leads to conditioned physical signs of withdrawal, this effect being modulated by additional exposition to morphine administration. In this study, the putative role of dopamine in conditioned withdrawal signs is evaluated by administering the dopamine release inhibitor CGS 10746B prior to suffering two naloxone-induced withdrawals in a distinctive environment associated or not with morphine administration. The results show that dopamine seems to be necessary for the development of conditioned somatic signs of morphine withdrawal, as animals which received CGS 10746B do not present paw tremor or body shakes when they are placed in the en…

MaleNarcoticsPharmacologyBehavior AnimalMorphineNaloxoneThiazepinesNarcotic AntagonistsMedicine (miscellaneous)PharmacologySubstance Withdrawal SyndromeMicePsychiatry and Mental healthMorphine withdrawalDopamineConditioning PsychologicalMorphinemedicineAnimalsConditioningSocial BehaviorPsychologymedicine.drugAddiction Biology
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Reinstatement of Morphine-Induced Conditioned Place Preference in Mice by Priming Injections

2004

To construct a model of relapse of drug abuse in mice, the induction, we evaluated the extinction and reinstatement of morphine-induced place preference. In Experiment 1, we examined the effects of morphine (0, 2, 3, 5, 10, 20 and 40 mg/kg) in the conditioned place preference (CPP) paradigm. Mice showed CPP with 5, 10, 20 and 40 mg/kg. In Experiment 2, we evaluated the effects of two different extinction procedures. After conditioning with 40 mg/kg of morphine, the mice underwent daily extinction sessions of 60 or 15 min of duration. CPP was extinguished after seven and nine sessions, respectively. In Experiment 3, we tested the reinstating effects of several priming doses of morphine. Mice…

MaleNarcoticsReinforcement SchedulePharmacologyArticleExtinction Psychologicallcsh:RC321-571MiceRewardmedicineAnimalslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryDose-Response Relationship DrugMorphineExtinction (psychology)Conditioned place preferenceDose–response relationshipNeurologyAnesthesiaMorphineConditioning OperantConditioningNeurology (clinical)PsychologyReinforcement PsychologyPriming (psychology)Injections Intraperitonealmedicine.drugNeural Plasticity
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Dose-dependent impairing effects of morphine on avoidance acquisition and performance in male mice.

1998

The effects of morphine (6.3, 12.6, and 25.2 mg/kg) on active avoidance behavior of BALB/C mice are explored in three acquisition sessions and in two subsequent performance sessions. Morphine-treated animals showed an increase in avoidance acquisition with respect to control group without differences in performance. However, a dramatical, concomitant rise in the locomotor activity of the animals (increase in the number of crossings during the intertrial intervals) prompted us to transform the data employing a formula with which a measure of actual learning was obtained. Applying this formula, we have observed that morphine administration impairs, dose-dependently, acquisition and performanc…

MaleNarcoticsTime FactorsCognitive Neurosciencemedicine.medical_treatmentDose dependenceMale miceExperimental and Cognitive PsychologyPharmacologyLocomotor activityDevelopmental psychologyBehavioral NeuroscienceMicemedicineAnimal activityAvoidance LearningAnimalsMice Inbred BALB CBehavior AnimalDose-Response Relationship DrugMorphineStimulantDose–response relationshipMorphinePsychologyNeurosciencemedicine.drugNeurobiology of learning and memory
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Memantine presents different effects from MK-801 in motivational and physical signs of morphine withdrawal

2003

Adaptive changes in neural systems due to chronic opiate exposure are related to the neural plasticity phenomenon, NMDA receptors being implicated in these processes, e.g. tolerance, dependence or withdrawal. In this work, we investigated the effect of two non-competitive NMDA antagonists, memantine and MK-801, in motivational (Conditioned Place Aversion paradigm, CPA) and physical aspects of morphine withdrawal. After the induction of morphine dependence, animals in which the CPA was studied, received memantine (5 and 10 mg/kg) or MK-801 (0.3-0.006 mg/kg) either during the acquisition (conditioning) or expression (test) phase of this procedure. Both drugs were capable of inhibiting conditi…

MaleNarcoticsTime FactorsNarcotic AntagonistsMotor ActivityPharmacologyMiceBehavioral NeuroscienceMemantineConditioning PsychologicalNeuroplasticitymedicineAnimalsDrug InteractionsBehavior AnimalDose-Response Relationship DrugMorphineNaloxoneKindlingMemantineSubstance Withdrawal SyndromeBlockadeMorphineNMDA receptorConditioningDizocilpine MaleateOpiatePsychologyExcitatory Amino Acid AntagonistsMorphine Dependencemedicine.drugBehavioural Brain Research
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GHB differentially affects morphine actions on motor activity and social behaviours in male mice

2003

There are several reports suggesting that gamma-hydroxybutyric acid (GHB) influences the endogenous opioid system. The present study aimed to investigate the effects of GHB on motor and social activities and to examine its influence on morphine's actions on these behaviours. In a first experiment, several doses of GHB were studied but only the highest (200 and 400 mg/kg) produced a decrease in spontaneous motor activity measured in an actimeter cage. When hyperactivity induced by injecting 50 mg/kg of morphine was evaluated, all the GHB doses efficiently counteracted this morphine action. Using the paradigm of isolation-induced aggression, administration of 200 mg/kg of GHB significantly de…

MaleNarcoticsmedicine.drug_classClinical BiochemistryMale miceMotor ActivityPharmacologyToxicologyBiochemistryMiceBehavioral NeuroscienceOpioid receptormedicineAnimalsDrug InteractionsMotor activitySocial BehaviorBiological PsychiatryEndogenous opioidPharmacologyMorphineAggressionBiological activityAggressionOpioidExploratory BehaviorMorphinemedicine.symptomSodium OxybatePsychologyAnesthetics Intravenousmedicine.drugPharmacology Biochemistry and Behavior
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