Search results for "Mouse Models"

showing 10 items of 35 documents

Non-alcoholic fatty pancreas disease pathogenesis: a role for developmental programming and altered circadian rhythms.

2013

Objectives Emerging evidence suggests that maternal obesity (MO) predisposes offspring to obesity and the recently described non-alcoholic fatty pancreas disease (NAFPD) but involved mechanisms remain unclear. Using a pathophysiologically relevant murine model, we here investigated a role for the biological clock - molecular core circadian genes (CCG) in the generation of NAFPD. Design Female C57BL6 mice were fed an obesogenic diet (OD) or standard chow (SC) for 6 weeks, prior to pregnancy and throughout gestation and lactation: resulting offspring were subsequently weaned onto either OD (Ob_Ob and Con_Ob) or standard chow (Ob_Con and Con_Con) for 6 months. Biochemical, pro-inflammatory and…

HeredityPhysiologylcsh:MedicineCLOCK ProteinsGene ExpressionMouse ModelsGastroenterology and HepatologyResearch and Analysis MethodsModel OrganismsPregnancyGeneticsMedicine and Health SciencesAnimalsRNA MessengerObesitylcsh:ScienceNutritionAnalysis of Variancelcsh:RBody WeightGene Expression Regulation DevelopmentalPancreatic DiseasesBiology and Life SciencesAnimal ModelsCircadian RhythmMice Inbred C57BLPhysiological ParametersPrenatal Exposure Delayed Effectslcsh:QFemaleEpigeneticsAnatomyPhysiological ProcessesDigestive SystemChronobiologyResearch ArticlePloS one
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Blocking Activin Receptor Ligands Is Not Sufficient to Rescue Cancer-Associated Gut Microbiota—A Role for Gut Microbial Flagellin in Colorectal Cance…

2019

Colorectal cancer (CRC) and cachexia are associated with the gut microbiota and microbial surface molecules. We characterized the CRC-associated microbiota and investigated whether cachexia affects the microbiota composition. Further, we examined the possible relationship between the microbial surface molecule flagellin and CRC. CRC cells (C26) were inoculated into mice. Activin receptor (ACVR) ligands were blocked, either before tumor formation or before and after, to increase muscle mass and prevent muscle loss. The effects of flagellin on C26-cells were studied in vitro. The occurrence of similar phenomena were studied in murine and human tumors. Cancer modulated the gut microbiota witho…

INTERLEUKIN-6suolistomikrobisto3122 CancersmicrobiomeENTEROCOCCUS-FAECALISlcsh:RC254-282ArticlePATHWAYACTIVATIONMOUSE MODELSIL-6 EXPRESSIONpaksusuolisyöpätulehdusCOLON-CANCERactivinliganditlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensIL6inflammationmyostatinSKELETAL-MUSCLEproteiinitlihassurkastumasairaudetTUMOR MICROENVIRONMENTCCL2MCP-1Cancers
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Correction: Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

2018

[This corrects the article DOI: 10.1371/journal.pone.0163841.].

ImmunologyGene ExpressionMouse ModelsCell MigrationResearch and Analysis MethodsPathology and Laboratory MedicineCorpus CallosumDirected Cell MigrationModel OrganismsNerve FibersSigns and SymptomsAnimal CellsDiagnostic MedicineMedicine and Health SciencesGeneticsImmune ResponseNeuronsInflammationChemotaxisBiology and Life SciencesBrainCell DifferentiationAnimal ModelsCell BiologyAxonsCell MotilityCellular NeuroscienceCellular TypesAnatomyResearch ArticleDevelopmental BiologyNeurosciencePloS one
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Neuronal activity triggers uptake of hematopoietic extracellular vesicles in vivo

2019

Communication with the hematopoietic system is a vital component of regulating brain function in health and disease. Traditionally, the major routes considered for this neuroimmune communication are by individual molecules such as cytokines carried by blood, by neural transmission, or, in more severe pathologies, by the entry of peripheral immune cells into the brain. In addition, functional mRNA from peripheral blood can be directly transferred to neurons via extracellular vesicles (EVs), but the parameters that determine their uptake are unknown. Using varied animal models that stimulate neuronal activity by peripheral inflammation, optogenetics, and selective proteasome inhibition of dop…

LipopolysaccharidesMaleGene ExpressionStimulationHippocampusBiochemistryStereotaxic Techniques0302 clinical medicineShort ReportsAnimal CellsMedicine and Health SciencesPremovement neuronal activityBiology (General)Routes of AdministrationNeurons0303 health sciencesBrain MappingKainic AcidBrainAnimal ModelsPeripheralCell biologyHaematopoiesisBioassays and Physiological AnalysisExperimental Organism SystemsHippocampus ; Yellow flourescent protein ; Intravenous injections ; Marker genes ; Gene expression ; Neurons ; Microglial cells ; OptogeneticsFemaleCellular TypesSignal TransductionProteasome Endopeptidase ComplexQH301-705.5Yellow Fluorescent ProteinMice TransgenicGlial CellsMouse ModelsStimulus (physiology)BiologyResearch and Analysis Methods03 medical and health sciencesExtracellular VesiclesImmune systemModel OrganismsIn vivoIntravenous InjectionsGeneticsAnimalsddc:610Molecular Biology TechniquesMolecular BiologyMicroglial Cells030304 developmental biologyInflammationPharmacologyMessenger RNABlood CellsUbiquitinDopaminergic NeuronsBiology and Life SciencesProteinsMarker GenesCell BiologyNeurophysiological AnalysisOptogeneticsLuminescent ProteinsCellular NeuroscienceAnimal Studies030217 neurology & neurosurgeryNeuroscience
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Coincident airway exposure to low-potency allergen and cytomegalovirus sensitizes for allergic airway disease by viral activation of migratory dendri…

2019

Despite a broad cell-type tropism, cytomegalovirus (CMV) is an evidentially pulmonary pathogen. Predilection for the lungs is of medical relevance in immunocompromised recipients of hematopoietic cell transplantation, in whom interstitial CMV pneumonia is a frequent and, if left untreated, fatal clinical manifestation of human CMV infection. A conceivable contribution of CMV to airway diseases of other etiology is an issue that so far attracted little medical attention. As the route of primary CMV infection upon host-to-host transmission in early childhood involves airway mucosa, coincidence of CMV airway infection and exposure to airborne environmental antigens is almost unavoidable. For i…

Lung DiseasesPulmonologyMedizinCytomegalovirusImmunoglobulin EPathology and Laboratory MedicineWhite Blood CellsMiceAnimal CellsMedicine and Health SciencesCytotoxic T cellBiology (General)Enzyme-Linked ImmunoassaysImmune ResponseLung0303 health sciencesAntigen PresentationbiologyT Cells030302 biochemistry & molecular biologyAnimal Modelsrespiratory systemExperimental Organism SystemsFemalemedicine.symptomCellular TypesResearch ArticleQH301-705.5OvalbuminImmune CellsAntigen presentationImmunologyInflammationCytotoxic T cellsMouse ModelsResearch and Analysis MethodsMicrobiology03 medical and health sciencesSigns and SymptomsModel OrganismsTh2 CellsAntigenDiagnostic MedicineVirologyGeneticsmedicineHypersensitivityAnimalsT Helper CellsMolecular Biology TechniquesImmunoassaysMolecular Biology030304 developmental biologyInflammationBlood Cellsbusiness.industryCD11 AntigensBiology and Life SciencesCell BiologyDendritic CellsRC581-607Allergensrespiratory tract diseasesTransplantationMice Inbred C57BLOvalbuminDisease Models AnimalImmunologyRespiratory Infectionsbiology.proteinAnimal StudiesImmunologic TechniquesParasitologyVirus ActivationImmunologic diseases. AllergybusinessCD8CloningPLoS Pathogens
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The Microbiota Promotes Arterial Thrombosis in Low-Density Lipoprotein Receptor-Deficient Mice

2019

Our results demonstrate a functional role for the commensal microbiota in atherothrombosis. In a ferric chloride injury model of the carotid artery, GF C57BL/6J mice had increased occlusion times compared to colonized controls. Interestingly, in late atherosclerosis, HFD-fed GF Ldlr−/− mice had reduced plaque rupture-induced thrombus growth in the carotid artery and diminished ex vivo thrombus formation under arterial flow conditions.

Male0209 industrial biotechnologyVery low-density lipoproteinChemokine CXCL102 engineering and technology030204 cardiovascular system & hematologyarterial thrombosisApplied Microbiology and BiotechnologyACTIVATIONMicechemistry.chemical_compound020901 industrial engineering & automation0302 clinical medicinegermfree0202 electrical engineering electronic engineering information engineeringMedicinevascular inflammationPlateletChemokine CCL7lcsh:QH301-705.5platelet0303 health sciencesatherosclerosis mouse modelsfood and beveragesThrombosisPlaque AtheroscleroticQR1-502late atherosclerosis3. Good healthHolobiontlow-density lipoprotein receptorgerm-freeplateletscardiovascular systemFemalelipids (amino acids peptides and proteins)GLYCOPROTEIN-VIBlood streamResearch ArticleRECRUITMENTmedicine.medical_specialtyNutritional compositionCOAGULATION610 Medicine & healthBiologyMETABOLISMBiochemistry Genetics and Molecular Biology (miscellaneous)MicrobiologyMicrobiologyHost-Microbe BiologyProinflammatory cytokinePLATELET HYPERREACTIVITY03 medical and health sciencesINFLAMMATIONVirologyInternal medicineatherothrombosisGeneticsmicrobiotaAnimalsInterleukin 9Platelet activationcardiovascular diseasesThrombusMolecular Biology030304 developmental biologygut microbiotabusiness.industryCholesterolcarotid artery020208 electrical & electronic engineeringcholesterolnutritional and metabolic diseasesCell Biologymedicine.diseaseMicroreviewCHLAMYDIA-PNEUMONIAEMice Mutant StrainsGastrointestinal MicrobiomeEndocrinologyReceptors LDLlcsh:Biology (General)chemistryArterial thrombusLDL receptorParasitologyatherosclerosisbusinessEx vivoLipoproteinmBio
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Identification of periplakin as a major regulator of lung injury and repair in mice

2018

IF 12.784 (2016); International audience; Periplakin is a component of the desmosomes that acts as a cytolinker between intermediate filament scaffolding and the desmosomal plaque. Periplakin is strongly expressed by epithelial cells in the lung and is a target antigen for autoimmunity in idiopathic pulmonary fibrosis. The aim of this study was to determine the role of periplakin during lung injury and remodeling in a mouse model of lung fibrosis induced by bleomycin. We found that periplakin expression was downregulated in the whole lung and in alveolar epithelial cells following bleomycin-induced injury. Deletion of the Ppl gene in mice improved survival and reduced lung fibrosis developm…

Male0301 basic medicinePulmonologylcsh:MedicineMouse modelsMiceIdiopathic pulmonary fibrosischemistry.chemical_compoundFibrosisPeriplakinMice KnockoutLung InjuryGeneral Medicinerespiratory system3. Good healthmedicine.anatomical_structureCytokinesmedicine.symptomSignal TransductionResearch ArticleCell signalingDown-RegulationInflammationRespiratory MucosaLung injuryBleomycinBleomycin03 medical and health sciencesmedicineAnimalsHumansInflammationLung030102 biochemistry & molecular biologybusiness.industryMacrophagesPlakinslcsh:Rmedicine.diseaseFibrosisIdiopathic Pulmonary Fibrosisrespiratory tract diseasesMice Inbred C57BLDisease Models Animal030104 developmental biologychemistryAlveolar Epithelial CellsCancer researchbusiness[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyJCI Insight
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Comparison of Diffusion MRI Acquisition Protocols for the In Vivo Characterization of the Mouse Spinal Cord: Variability Analysis and Application to …

2016

Diffusion-weighted Magnetic Resonance Imaging (dMRI) has relevant applications in the microstructural characterization of the spinal cord, especially in neurodegenerative diseases. Animal models have a pivotal role in the study of such diseases; however, in vivo spinal dMRI of small animals entails additional challenges that require a systematical investigation of acquisition parameters. The purpose of this study is to compare three acquisition protocols and identify the scanning parameters allowing a robust estimation of the main diffusion quantities and a good sensitivity to neurodegeneration in the mouse spinal cord. For all the protocols, the signal-to-noise and contrast-to noise ratios…

MaleDTI-MRI spinal cord ALSPathologylcsh:MedicineSignal-To-Noise RatioNervous System030218 nuclear medicine & medical imagingDiagnostic RadiologyDiffusionMice0302 clinical medicineSuperoxide Dismutase-1Materials PhysicsMedicine and Health SciencesImage Processing Computer-AssistedAmyotrophic lateral sclerosisDiffusion (business)lcsh:ScienceMicrostructureMusculoskeletal SystemBrain MappingMultidisciplinarymedicine.diagnostic_testRadiology and ImagingPhysicsAnimal ModelsCondensed Matter PhysicsMagnetic Resonance Imagingmedicine.anatomical_structureDiffusion Tensor ImagingSpinal CordPhysical SciencesAnatomyResearch Articlemedicine.medical_specialtyImaging TechniquesBrain MorphometryMaterials ScienceMaterial PropertiesNeuroimagingMouse ModelsMice TransgenicResearch and Analysis Methods03 medical and health sciencesModel OrganismsDiagnostic MedicineFractional anisotropymedicineAnimalsSensitivity (control systems)AllelesProtocol (science)business.industryAmyotrophic Lateral Sclerosislcsh:RBiology and Life SciencesReproducibility of ResultsMagnetic resonance imagingmedicine.diseaseSpinal cordSpineNeuroanatomyDisease Models AnimalDiffusion Magnetic Resonance ImagingMutationAnisotropylcsh:Qbusiness030217 neurology & neurosurgeryBiomedical engineeringDiffusion MRINeurosciencePLoS ONE
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Delivery of epirubicin via slow infusion as a strategy to mitigate chemotherapy-induced cardiotoxicity

2017

Background Continuous infusion of doxorubicin has been a strategy to reduce cardiotoxicity. Epirubicin is another anthracycline in common clinical use. However, evidence is lacking regarding whether this strategy can reduce cardiotoxicity of epirubicin without compromising antineoplastic efficacy. Design and methods Healthy rats were randomized into groups: epirubicin (8 mg/kg) delivered intraperitoneally via micro osmotic pumps (MOP), epirubicin (8 mg/kg) by intraperitoneal (IP) bolus injection, and placebo control. Blood samples were collected for analyzing biomarkers of myocardial injury and pharmacokinetics. At chosen times, sub-groups of animals were sacrificed for histopathology. A mo…

MalePhysiologyCancer Treatmentlcsh:Medicine030204 cardiovascular system & hematologyPharmacologyBiochemistryRats Sprague-DawleyMice0302 clinical medicineBolus (medicine)Intraperitoneal InjectionsBreast TumorsMedicine and Health Scienceslcsh:Scienceskin and connective tissue diseasesInfusions IntravenousRoutes of AdministrationMultidisciplinaryAntibiotics AntineoplasticArea under the curveHeartAnimal ModelsBody FluidsBloodExperimental Organism SystemsOncology030220 oncology & carcinogenesisAnatomyEpirubicinmedicine.drugResearch ArticleAnthracyclineMouse ModelsResearch and Analysis MethodsBlood Plasma03 medical and health sciencesModel OrganismsPharmacokineticsIn vivoCell Line TumorBreast CancermedicineAnimalsDoxorubicinPharmacokineticsAnimal Models of DiseaseEpirubicinPharmacologyCardiotoxicitybusiness.industrylcsh:RCancers and NeoplasmsBiology and Life SciencesRatsAnimal Studieslcsh:QbusinessBiomarkersPLoS ONE
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Molecular topology as novel strategy for discovery of drugs with aβ lowering and anti-aggregation dual activities for Alzheimer's disease.

2014

Background and Purpose: In this study, we demonstrate the use of Molecular topology (MT) in an Alzheimer's disease (AD) drug discovery program. MT uses and expands upon the principles governing the molecular connectivity theory of numerically characterizing molecular structures, in the present case, active anti-AD drugs/agents, using topological descriptors to build models. Topological characterization has been shown to embody sufficient molecular information to provide strong correlation to therapeutic efficacy. Experimental Approach: We used MT to include multiple bioactive properties that allows for the identification of multifunctional single agent compounds, in this case, the dual func…

Models MolecularDrug Evaluation Preclinicallcsh:MedicineDiseaseProtein aggregationBioinformaticsBiochemistryMechanical Treatment of SpecimensAnimal CellsMolecular Cell BiologyDrug DiscoveryMedicine and Health Scienceslcsh:ScienceTopology (chemistry)NeuronsMultidisciplinaryDrug discoveryMedicine (all)Anti aggregationNeurodegenerative DiseasesAnimal ModelsElectroporationTreatment OutcomeNeurologySpecimen DisruptionDatabases as TopicFemaleMolecular topologyAlzheimer's diseaseCellular TypesResearch ArticleDrug Research and DevelopmentMouse ModelsMice TransgenicComputational biologyBiologyResearch and Analysis MethodsProtein AggregatesModel OrganismsAlzheimer DiseaseMental Health and PsychiatrymedicineAnimalsHumansPharmacologyAmyloid beta-PeptidesBiochemistry Genetics and Molecular Biology (all)lcsh:RBiology and Life SciencesProteinsComputational BiologyCell BiologyDUAL (cognitive architecture)medicine.diseaseDisease Models AnimalAgricultural and Biological Sciences (all)Specimen Preparation and TreatmentFeasibility StudiesDementialcsh:QClinical MedicineProtein MultimerizationPLoS ONE
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