Search results for "Musculoskeletal"

showing 10 items of 1714 documents

The Drosophila Hox gene Ultrabithorax acts both in muscles and motoneurons to orchestrate formation of specific neuromuscular connections

2016

Hox genes are known to specify motoneuron pools in the developing vertebrate spinal cord and to control motoneuronal targeting in several species. However, the mechanisms controlling axial diversification of muscle innervation patterns are still largely unknown. We present data showing that the Drosophila Hox gene Ultrabithorax (Ubx) acts in the late embryo to establish target specificity of ventrally projecting RP motoneurons. In abdominal segments A2 to A7, RP motoneurons innervate the ventrolateral muscles VL1-4, with VL1 and VL2 being innervated in a Wnt4-dependent manner. In Ubx mutants, these motoneurons fail to make correct contacts with muscle VL1, a phenotype partially resembling t…

0301 basic medicineCell typeEmbryo Nonmammaliananimal structuresNeuromuscular JunctionGenes InsectMuscle DevelopmentNeuromuscular junctionAnimals Genetically ModifiedHox genes03 medical and health sciencesWNT4MorphogenesismedicineAnimalsDrosophila ProteinsHox geneWnt Signaling PathwayMolecular BiologyTranscription factorUltrabithoraxHomeodomain ProteinsMotor NeuronsGeneticsbiologyMusclesmusculoskeletal neural and ocular physiologyfungiGenes HomeoboxGene Expression Regulation Developmentalbiology.organism_classificationMuscle innervationSegmental patterningCell biologyMotoneuronsDrosophila melanogaster030104 developmental biologymedicine.anatomical_structurenervous system209embryonic structuresDrosophilaWnt signalling pathwayDrosophila melanogasterDrosophila ProteinTranscription FactorsResearch ArticleDevelopmental BiologyDevelopment
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The activation of NMDA receptors alters the structural dynamics of the spines of hippocampal interneurons

2017

N-Methyl-d-Aspartate receptors (NMDARs) are present in both pyramidal neurons and interneurons of the hippocampus. These receptors play a key role in the structural plasticity of excitatory neurons, but to date little is known about their influence on the remodeling of interneurons. Among hippocampal interneurons, the somatostatin expressing cells in the CA1 stratum oriens are of special interest because of their functional importance and structural characteristics: they display dendritic spines, which change their density in response to different stimuli. In order to understand the role of NMDAR activation on the structural dynamics of the spines of somatostatin expressing interneurons in …

0301 basic medicineDendritic spineDendritic SpinesHippocampusHippocampal formationBiologyHippocampusReceptors N-Methyl-D-Aspartate03 medical and health sciences0302 clinical medicineInterneuronsAnimalsReceptorCells CulturedMice KnockoutPyramidal Cellsmusculoskeletal neural and ocular physiologyGeneral NeuroscienceLong-term potentiationSpine030104 developmental biologySomatostatinnervous systemExcitatory postsynaptic potentialNMDA receptorSomatostatinNeuroscience030217 neurology & neurosurgeryNeuroscience Letters
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NMDA Receptors Regulate the Structural Plasticity of Spines and Axonal Boutons in Hippocampal Interneurons

2017

N-methyl-D-aspartate receptors (NMDARs) are present in both pyramidal neurons and interneurons of the hippocampus. These receptors play an important role in the adult structural plasticity of excitatory neurons, but their impact on the remodeling of interneurons is unknown. Among hippocampal interneurons, somatostatin-expressing cells located in the stratum oriens are of special interest because of their functional importance and structural characteristics: they display dendritic spines, which change density in response to different stimuli. In order to understand the role of NMDARs on the structural plasticity of these interneurons, we have injected acutely MK-801, an NMDAR antagonist, to …

0301 basic medicineDendritic spineorganotypic culturesEn passantHippocampusHippocampal formationBiologyspine dynamicslcsh:RC321-57103 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineReceptorlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal ResearchMK-801interneuronsmusculoskeletal neural and ocular physiologyaxonal boutonsNMDARSpine (zoology)030104 developmental biologynervous systemExcitatory postsynaptic potentialNMDA receptorNeuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Cellular Neuroscience
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In vitro cytotoxic effects of DEHP-alternative plasticizers and their primary metabolites on a L929 cell line

2017

IF 4.208; International audience; Phthalic acid esters have been widely used to improve the plasticity of PVC medical devices. They carry a high exposure risk for both humans and the environment in clinical situations. Our study focuses on the cytotoxicity of alternative plasticizers. Postulated primary metabolites were synthesized, not being commercially available. Cytotoxicity assays were performed on L929 murine cells according to the ISO-EN 10993-5 standard design for the biocompatibility of medical devices. The tested concentrations of plasticizers (0.01, 0.05 and 0.1 mg/ml) covered the range likely to be found in biological fluids coming into direct contact with the medical devices. D…

0301 basic medicineEnvironmental EngineeringMetabolite synthesisBiocompatibilityCell SurvivalCytotoxicityHealth Toxicology and MutagenesisMetabolitePhthalic AcidsIn Vitro TechniquesDEHP-alternative plasticizers010501 environmental sciences01 natural sciences[ SDE ] Environmental SciencesMice03 medical and health scienceschemistry.chemical_compoundPhthalatesPlasticizersIn vivoDiethylhexyl PhthalateAnimalsEnvironmental ChemistryOrganic chemistryPolyvinyl ChlorideCytotoxicityCells Cultured0105 earth and related environmental sciences[SDV.MHEP.RSOA] Life Sciences [q-bio]/Human health and pathology/Rhumatology and musculoskeletal systemChromatography[SDV.MHEP.GEG] Life Sciences [q-bio]/Human health and pathology/Geriatry and gerontology[SDV.MHEP.GEG]Life Sciences [q-bio]/Human health and pathology/Geriatry and gerontologyPublic Health Environmental and Occupational HealthPlasticizerPrimary metaboliteEstersGeneral MedicineGeneral ChemistryFibroblastsPollutionIn vitro3. Good healthPhthalic acid030104 developmental biology[SDV.MHEP.RSOA]Life Sciences [q-bio]/Human health and pathology/Rhumatology and musculoskeletal systemchemistryChemosphere
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Low-energy extracorporeal shockwave therapy (ESWT) improves metaphyseal fracture healing in an osteoporotic rat model.

2017

Purpose As result of the current demographic changes, osteoporosis and osteoporotic fractures are becoming an increasing social and economic burden. In this experimental study, extracorporeal shock wave therapy (ESWT), was evaluated as a treatment option for the improvement of osteoporotic fracture healing. Methods A well-established fracture model in the metaphyseal tibia in the osteoporotic rat was used. 132 animals were divided into 11 groups, with 12 animals each, consisting of one sham-operated group and 10 ovariectomized (osteoporotic) groups, of which 9 received ESWT treatment. Different energy flux intensities (0.15 mJ/mm2, 0.35 mJ/mm2, or 0.55 mJ/mm2) as well as different numbers o…

0301 basic medicineExtracorporeal Shockwave TherapyCritical Care and Emergency Medicinemedicine.medical_treatmentOsteoporosisTest StatisticsDentistryGene Expressionlcsh:MedicineRats Sprague-Dawley0302 clinical medicineMathematical and Statistical TechniquesAnimal CellsMedicine and Health SciencesReproductive System ProceduresConnective Tissue Diseaseslcsh:ScienceMusculoskeletal SystemTrauma MedicineConnective Tissue CellsFracture Healing030222 orthopedicsMultidisciplinaryBiomechanicsBone FractureConnective TissueExtracorporeal shockwave therapyPhysical SciencesOvariectomized ratFemaleAnatomyCellular TypesTraumatic InjuryStatistics (Mathematics)Research ArticleOvariectomySurgical and Invasive Medical ProceduresBone healingResearch and Analysis Methods03 medical and health sciencesRheumatologymedicineGeneticsAnimalsTibiaStatistical MethodsSkeletonAnalysis of VarianceOsteoblastsSurgical ExcisionTibiabusiness.industrylcsh:RBiology and Life SciencesBone fractureCell Biologymedicine.diseaseRatsDisease Models Animal030104 developmental biologyBiological TissueAdjunctive treatmentOsteoporosislcsh:QbusinessOsteoporotic FracturesMathematicsPLoS ONE
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FGFR a promising druggable target in cancer: Molecular biology and new drugs.

2017

Abstract: Introduction: The Fibroblast Growth Factor Receptor (FGFR) family consists of Tyrosine Kinase Receptors (TKR) involved in several biological functions. Recently, alterations of FGFR have been reported to be important for progression and development of several cancers. In this setting, different studies are trying to evaluate the efficacy of different therapies targeting FGFR. Areas Covered: This review summarizes the current status of treatments targeting FGFR, focusing on the trials that are evaluating the FGFR profile as inclusion criteria: Multi-Target, Pan-FGFR Inhibitors and anti-FGF (Fibroblast Growth Factor)/FGFR Monoclonal Antibodies. Expert opinion: Most of the TKR share …

0301 basic medicineFibroblast Growth FactorDruggabilityFibroblast growth factorTyrosine-kinase inhibitorReceptor tyrosine kinase0302 clinical medicineNeoplasmsFGFR inhibitorsFGFMolecular Targeted TherapyCancerCancer; FGF; FGFR; FGFR inhibitors; Drug Resistance Neoplasm; Fibroblast Growth Factors; Gene Fusion; Humans; Molecular Targeted Therapy; Mutation; Neoplasms; Protein Kinase Inhibitors; Receptors Fibroblast Growth Factor; Signal Transduction; Hematology; Oncology; Geriatrics and GerontologybiologyFGFRHematologyFGFR inhibitorOncologyFibroblast growth factor receptor030220 oncology & carcinogenesisembryonic structuresSignal transductionbiological phenomena cell phenomena and immunityGene FusionHumanSignal Transductionmusculoskeletal diseasesanimal structuresmedicine.drug_classProtein Kinase Inhibitor03 medical and health sciencesmedicineHumansProtein Kinase InhibitorsCancer; FGF; FGFR; FGFR inhibitorsbusiness.industryCancermedicine.diseaseMolecular biologyReceptors Fibroblast Growth FactorFibroblast Growth Factors030104 developmental biologyDrug Resistance NeoplasmCancer cellMutationbiology.proteinNeoplasmHuman medicineGeriatrics and GerontologybusinessCritical reviews in oncology/hematology
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Polysialic Acid Acute Depletion Induces Structural Plasticity in Interneurons and Impairs the Excitation/Inhibition Balance in Medial Prefrontal Cort…

2016

The structure and function of the medial prefrontal cortex (mPFC) is affected in several neuropsychiatric disorders, including schizophrenia and major depression. Recent studies suggest that imbalances between excitatory and inhibitory activity (E/I) may be responsible for this cortical dysfunction and, therefore, may underlie the core symptoms of these diseases. This E/I imbalance seems to be correlated with alterations in the plasticity of interneurons but there is still scarce information on the mechanisms that may link these phenomena. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) is a good candidate, because it modulates the neuronal plasticity of interneurons…

0301 basic medicineGenetically modified mousePSA-NCAMneuronal structural plasticityInhibitory postsynaptic potential03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineE/I balanceNeuroplasticitymedicinePrefrontal cortexOriginal ResearchPolysialic acidmusculoskeletal neural and ocular physiologymedicine.diseaseschizophreniamPFC cultures030104 developmental biologynervous systemSchizophreniaExcitatory postsynaptic potentialNeural cell adhesion moleculemajor depressionPsychologyNeuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Cellular Neuroscience
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FANCD2 modulates the mitochondrial stress response to prevent common fragile site instability

2021

Common fragile sites (CFSs) are genomic regions frequently involved in cancer-associated rearrangements. Most CFSs lie within large genes, and their instability involves transcription- and replication-dependent mechanisms. Here, we uncover a role for the mitochondrial stress response pathway in the regulation of CFS stability in human cells. We show that FANCD2, a master regulator of CFS stability, dampens the activation of the mitochondrial stress response and prevents mitochondrial dysfunction. Genetic or pharmacological activation of mitochondrial stress signaling induces CFS gene expression and concomitant relocalization to CFSs of FANCD2. FANCD2 attenuates CFS gene transcription and pr…

0301 basic medicineGenome instabilitymusculoskeletal diseasesTranscription GeneticQH301-705.5RegulatorMedicine (miscellaneous)MitochondrionBiology[SDV.BBM.BM] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyGeneral Biochemistry Genetics and Molecular BiologyOxidative PhosphorylationArticle03 medical and health sciences0302 clinical medicineTranscription (biology)Stress Physiologicalhemic and lymphatic diseasesGene expressionFANCD2HumansBiology (General)GeneUbiquitinsChromosomal fragile siteChromosome Fragile SitesChromosome FragilityFanconi Anemia Complementation Group D2 ProteinDNA damage and repair[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyHCT116 CellsCell biologyMitochondriaSettore BIO/18 - Genetica030104 developmental biologyGene Expression Regulation030220 oncology & carcinogenesisUnfolded Protein ResponseGeneral Agricultural and Biological SciencesDNA Damage
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Sarcopenia, frailty and their prevention by exercise.

2019

Sarcopenia is a major component of the frailty syndrome, both being considered as strong predictors of morbidity, disability, and death in older people. In this review, we explore the definitions of sarcopenia and frailty and summarize the current knowledge on their relationship with oxidative stress and the possible therapeutic interventions to prevent or treat them, including exercise-based interventions and multimodal strategies. We highlight the relevance of the impairment of the nervous system and of the anabolic response (protein synthesis) in muscle aging leading to frailty and sarcopenia. We also discuss the importance of malnutrition and physical inactivity in these geriatric syndr…

0301 basic medicineGerontologySarcopeniaNutritional SupplementationFrailty syndromePsychological interventionBiochemistry03 medical and health sciences0302 clinical medicineAtrophyPhysiology (medical)medicineAnimalsHumansExerciseAgedFrailtybusiness.industryMalnutritionmusculoskeletal systemmedicine.diseaseMalnutritionOxidative Stress030104 developmental biologySarcopeniaDietary SupplementsOlder peoplebusinesshuman activities030217 neurology & neurosurgeryFree radical biologymedicine
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Can the transverse foramen/vertebral artery ratio of double transverse foramen subjects be a risk for vertebrobasilar transient ischemic attacks?

2018

The C6 is the cervical vertebra into which the vertebral artery enters the passage of the transverse foramen and it is the vertebra most affected by double transverse foramina. There is currently little information about the relation between the vertebral artery and the double transverse foramen in C6. We aimed to test whether subjects with a double transverse foramen in C6 have a reduced transverse foramen/vertebral artery ratio when compared with normal anatomy subjects who possess a single transverse foramen which may be a risk for transient vertebral artery stenosis. We measured the area of the transverse foramen and the vertebral artery in 27 double transverse and 56 normal anatomy sub…

0301 basic medicineHistologyVertebral artery03 medical and health sciences0302 clinical medicinestomatognathic systemmedicine.arterymedicineForamenVertebrobasilar insufficiencyMolecular BiologyEcology Evolution Behavior and SystematicsComputed tomography angiographymedicine.diagnostic_testNormal anatomybusiness.industryCell BiologyAnatomyOriginal Articlesmedicine.diseasemusculoskeletal systemVertebraTransverse planemedicine.anatomical_structure030101 anatomy & morphologyAnatomybusiness030217 neurology & neurosurgeryDevelopmental BiologyCervical vertebraeJournal of anatomy
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