Search results for "Myelitis"

showing 10 items of 211 documents

Pre- and postsynaptic type-1 cannabinoid receptors control the alterations of glutamate transmission in experimental autoimmune encephalomyelitis

2013

Type-1 cannabinoid receptors (CB1R) are important regulators of the neurodegenerative damage in multiple sclerosis (MS) and in experimental autoimmune encephalomyelitis (EAE). In GABAergic striatal neurons, CB1R stimulation exerts protective effects by limiting inflammation-induced potentiation of glutamate-mediated spontaneous excitatory postsynaptic currents (sEPSCs). Here we show that CB1R located on GABAergic or on glutamatergic neurons are differentially involved in the pre- and postsynaptic alterations of sEPSCs caused by EAE in the striatum. After induction of EAE, mice selectively lacking CB1R on GABAergic neurons (GABA-CB1R-KO) showed exacerbated alterations of sEPSC duration in GA…

Encephalomyelitis Autoimmune ExperimentalTime FactorsPostsynaptic CurrentPresynaptic TerminalsExcitotoxicityGlutamic AcidIn Vitro TechniquesBiologyMedium spiny neuronmedicine.disease_causeSynaptic TransmissionMiceCellular and Molecular NeuroscienceGlutamatergicReceptor Cannabinoid CB1Postsynaptic potentialmedicineAnimalsgamma-Aminobutyric AcidMice KnockoutNeuronsPharmacologyExperimental autoimmune encephalomyelitisGlutamate receptorExcitatory Postsynaptic Potentialsmedicine.diseaseCorpus StriatumMice Inbred C57BLnervous systemDisease ProgressionExcitatory postsynaptic potentialFemaleSettore MED/26 - NeurologiaNeuroscience
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Cutting Edge: IL-23 Cross-Regulates IL-12 Production in T Cell-Dependent Experimental Colitis

2006

Abstract Although IL-12 and IL-23 share the common p40 subunit, IL-23, rather than IL-12, seems to drive the pathogenesis of experimental autoimmune encephalomyelitis and arthritis, because IL-23/p19 knockout mice are protected from disease. In contrast, we describe in this study that newly created LacZ knockin mice deficient for IL-23 p19 were highly susceptible for the development of experimental T cell-mediated TNBS colitis and showed even more severe colitis than wild-type mice by endoscopic and histologic criteria. Subsequent studies revealed that dendritic cells from p19-deficient mice produce elevated levels of IL-12, and that IL-23 down-regulates IL-12 expression upon TLR ligation. …

T-LymphocytesTransgeneT cellImmunologyDown-RegulationMice TransgenicInterleukin-23PathogenesisMiceInterleukin 23AnimalsImmunology and AllergyMedicineColitisCells Culturedbusiness.industryInterleukinsExperimental autoimmune encephalomyelitisColitismedicine.diseaseInterleukin-12Survival RateDisease Models AnimalProtein Subunitsmedicine.anatomical_structureImmunologyKnockout mouseInterleukin-23 Subunit p19Interleukin 12Disease SusceptibilitybusinessThe Journal of Immunology
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Osteomyelitis affecting mandible in tuberculosis patients

2012

Tuberculosis (TB) is a frequent health problem in developing nations. It has two forms pulmonary and secondary causing other kinds of TB, collectively denoted extra pulmonary tuberculosis. The prevalence of extra pulmonary TB has increased in the last couple of years. Maxillofacial manifestations of tuberculosis form nearly 10% of all extra pulmonary manifestations of the disease. Extra pulmonary TB involving maxillofacial region is our prime concern. Very few cases of TB of the temporomandibular joint (TMJ) and mandible have been reported in literature. The clinical appearance of TB infection of the TMJ has been described as unspecific, resembling arthritis, osteomyelitis, cancer or any ki…

medicine.medical_specialtyTuberculosisbusiness.industryOsteomyelitisMandibleArthritisClinical appearanceCase ReportOdontologíaDiseasemedicine.disease:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludSurgeryTemporomandibular jointLesionmedicine.anatomical_structurestomatognathic systemClinical and Experimental DentistryUNESCO::CIENCIAS MÉDICASmedicinemedicine.symptombusinessGeneral Dentistry
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Muscle versus Fasciocutaneous Flap in Lower Limb Reconstruction: Is There a Best Option?

2017

AbstractSoft tissue defects of the lower extremity that expose underlying bones, joints, and tendons pose challenging problems and generally require free tissue transfer for a successful reconstruction. Historically, muscle flaps were the gold standard choice for lower limb reconstruction. To obviate the unpredictable appearance and high donor-site morbidity of muscle flaps, fasciocutaneous flaps were introduced. Recently, perforator flaps, such as the anterolateral thigh flap, gained a leading role in the reconstructive scenario. There is growing evidence in the literature supporting that fasciocutaneous and perforator flaps are comparable to muscle flaps in terms of flap survival, postope…

medicine.medical_specialtyfasciocutaneous flaps; free flaps; lower limb reconstruction; muscle flaps; SurgerySoft Tissue Injuriesfasciocutaneous flapSettore MED/19 - Chirurgia PlasticaLeg Injurie030230 surgeryLower limblower limb reconstruction03 medical and health sciences0302 clinical medicinemuscle flapsMedicineFlap survivalHumansReconstructive Surgical ProcedureFasciaMuscle Skeletalfree flapfasciocutaneous flapsbusiness.industryBone unionOsteomyelitisGraft SurvivalSoft tissueAnterolateral thighPlastic Surgery Proceduresmedicine.diseaseMyocutaneous Flapeye diseasesSurgerySoft Tissue InjurieFasciocutaneous flapTreatment Outcome030220 oncology & carcinogenesisSurgerybusinessfree flapsmuscle flapPerforator flapsLeg InjuriesHuman
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Modulation of Neurological Deficits and Expression of Glutamate Receptors during Experimental Autoimmune Encephalomyelitis after Treatment with Selec…

2013

The aim of our investigation was to characterize the role of group I mGluRs and NMDA receptors in pathomechanisms of experimental autoimmune encephalomyelitis (EAE), the rodent model of MS. We tested the effects of LY 367385 (S-2-methyl-4-carboxyphenylglycine, a competitive antagonist of mGluR1), MPEP (2-methyl-6-(phenylethynyl)-pyridine, an antagonist of mGluR5), and the uncompetitive NMDA receptor antagonists amantadine and memantine on modulation of neurological deficits observed in rats with EAE. The neurological symptoms of EAE started at 10-11 days post-injection (d.p.i.) and peaked after 12-13 d.p.i. The protein levels of mGluRs and NMDA did not increase in early phases of EAE (4 d.p…

Encephalomyelitis Autoimmune ExperimentalMultiple SclerosisArticle SubjectHydrolasesEncephalomyelitislcsh:MedicineBiologyPharmacologyReceptors N-Methyl-D-AspartateGeneral Biochemistry Genetics and Molecular Biologymental disordersmedicineAmantadineAnimalsHumansRNA MessengerGeneral Immunology and MicrobiologyMetabotropic glutamate receptor 5Experimental autoimmune encephalomyelitislcsh:RGlutamate receptorMemantineGeneral Medicinemedicine.diseaseRatsDisease Models AnimalGene Expression RegulationReceptors Glutamatenervous systemCompetitive antagonistImmunologyNMDA receptorMetabotropic glutamate receptor 1FemaleExcitatory Amino Acid Antagonistsmedicine.drugResearch ArticleBioMed Research International
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Astrocytic potassium and calcium channels as integrators of the inflammatory and ischemic CNS microenvironment.

2021

Abstract Astrocytes are key regulators of their surroundings by receiving and integrating stimuli from their local microenvironment, thereby regulating glial and neuronal homeostasis. Cumulating evidence supports a plethora of heterogenic astrocyte subpopulations that differ morphologically and in their expression patterns of receptors, transporters and ion channels, as well as in their functional specialisation. Astrocytic heterogeneity is especially relevant under pathological conditions. In experimental autoimmune encephalomyelitis (EAE), a mouse model of multiple sclerosis (MS), morphologically distinct astrocytic subtypes were identified and could be linked to transcriptome changes dur…

Voltage-gated ion channelVoltage-dependent calcium channelClinical BiochemistryExperimental autoimmune encephalomyelitisBiologymedicine.diseaseBiochemistryPotassium channelTransient receptor potential channelMicemedicine.anatomical_structureAstrocytesmedicinePotassiumAnimalsCalcium ChannelsMolecular BiologyNeuroscienceIon channelNeuroinflammationAstrocyteBiological chemistryReferences
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The iNOS Activity During an Immune Response Controls the CNS Pathology in Experimental Autoimmune Encephalomyelitis

2019

Inducible nitric oxide synthase (iNOS) plays a critical role in the regulation of multiple sclerosis (MS) and experimental autoimmune encephalomyelitis (EAE). Previous studies have shown that iNOS plays pathogenic as well as regulatory roles in MS and EAE. However, how does iNOS alters the pathophysiology of the central nervous system (CNS) in neuronal autoimmunity is not clearly understood. In the present work, we show that treatment of mice with L-NAME, an iNOS inhibitor, during the antigen-priming phase primarily alters brain pathology, while in the subsequent effector phase of the immune response, the spinal cord is involved. Inhibition of iNOS during the priming phase of the immune res…

0301 basic medicineCD4-Positive T-LymphocytesPathologyexperimental autoimmune encephalomyelitisNitric Oxide Synthase Type IIApoptosismedicine.disease_causeAutoimmunityMice0302 clinical medicineImmunology and AllergyEnzyme InhibitorsOriginal ResearchMice KnockoutbiologyExperimental autoimmune encephalomyelitisautoimmunityCell DifferentiationNitric oxide synthaseOligodendrogliamedicine.anatomical_structureNG-Nitroarginine Methyl EsterIntegrin alpha Mlcsh:Immunologic diseases. Allergymedicine.medical_specialtyEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisLymphoid TissueCentral nervous systemImmunology03 medical and health sciencesInterferon-gammaImmune systemmedicineAnimalsHumansNOS2−/− neuroinflammationNeuroinflammationbusiness.industryMultiple sclerosisinducible nitric oxide synthaseDendritic Cellsmedicine.diseasecentral nervous systemMice Inbred C57BL030104 developmental biologybiology.proteinbusinesslcsh:RC581-607030215 immunologyGranulocytesFrontiers in Immunology
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Versatility of the Free Anterolateral Thigh Flap in the Reconstruction of Large Defects of the Weight-Bearing Foot: A Single-Center Experience with 2…

2016

Background  Reconstruction of the weight-bearing surface of the foot represents a challenging task. With very little scope to borrow glabrous tissue from adjacent areas means that achieving a “like for like” reconstruction is rarely possible. In this setting, alternative approaches need to be considered. In this article we present our experience with various differing designs of the anterolateral thigh flap (ALT) in the reconstruction of 20 large defects of the weight-bearing sole. Methods  Twenty patients with complex soft tissue defects of the weight-bearing sole underwent reconstruction over a 5-year period. Five cases were complicated by osteomyelitis resulting in significant calcaneal …

AdultMalemedicine.medical_specialtyFoot InjurieSoft Tissue InjuriesAdolescent030230 surgeryThighSingle Centermedicine.disease_causeFree Tissue FlapsWeight-bearinganterolateral thigh perforator flapWeight-Bearing03 medical and health sciencesYoung Adult0302 clinical medicinemedicineReconstructive Surgical ProcedureHumansReconstructive Surgical ProceduresFibulaFoot Injuriesanterolateral thigh perforator flap; calcaneal reconstruction; weight-bearing sole reconstruction; Adolescent; Adult; Aged; Female; Foot Injuries; Free Tissue Flaps; Humans; Male; Middle Aged; Recovery of Function; Retrospective Studies; Soft Tissue Injuries; Thigh; Treatment Outcome; Weight-Bearing; Young Adult; Reconstructive Surgical ProceduresFree Tissue FlapAgedRetrospective Studiesbusiness.industryOsteomyelitisSoft tissueRecovery of FunctionMiddle AgedPlastic Surgery Proceduresmedicine.diseaseGaitSurgerySoft Tissue Injuriemedicine.anatomical_structureTreatment OutcomeThigh030220 oncology & carcinogenesisweight-bearing sole reconstructionSurgeryFemaleFoot Injurybusinesscalcaneal reconstructionJournal of reconstructive microsurgery
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A case of sjögren's syndrome with severe anemia due to myelitis

1986

An unusual case of Sjögren's syndrome presenting with severe anemia as the predominant clinical feature is described. Histological examination of a bone marrow biopsy specimen demonstrated that the patient's anemia was caused by myelitis and vasculitis of the small intraosseous vessels. Our report might stimulate a more thorough investigation of bone marrow in patients with connective tissue diseases and anemia.

MalePathologymedicine.medical_specialtyAnemiaConnective tissueMyelitisSevere anemiaBone Marrowhemic and lymphatic diseasesDrug DiscoveryBiopsymedicineHumansGenetics (clinical)medicine.diagnostic_testbusiness.industryAnemiaOsteomyelitisArteriesGeneral MedicineMiddle AgedHematopoietic Stem Cellsmedicine.diseasePolyarteritis NodosaSjogren's Syndromemedicine.anatomical_structureMolecular MedicineBone marrowSjogren sbusinessVasculitisKlinische Wochenschrift
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A noninflammatory mRNA vaccine for treatment of experimental autoimmune encephalomyelitis.

2019

Precision therapy for immune tolerance Autoimmune diseases, such as multiple sclerosis (MS), result from a breach of immunological self-tolerance and tissue damage by autoreactive T lymphocytes. Current treatments can cause systemic immune suppression and side effects such as increased risk of infections. Krienke et al. designed a messenger RNA vaccine strategy that lacks adjuvant activity and delivers MS autoantigens into lymphoid dendritic cells. This approach expands a distinct type of antigen-specific effector regulatory T cell that suppresses autoreactivity against targeted autoantigens and promotes bystander suppression of autoreactive T cells against other myelin-specific autoantigen…

Encephalomyelitis Autoimmune ExperimentalMultiple SclerosisRegulatory T cellEncephalomyelitisAntigen presentationAntigen-Presenting CellsAutoantigensT-Lymphocytes RegulatoryMiceImmune systemAntigenmedicineAnimalsRNA MessengerAntigen-presenting cellImmunosuppression TherapyInflammationVaccines SyntheticMultidisciplinarybusiness.industryEffectorExperimental autoimmune encephalomyelitisBystander Effectmedicine.diseaseMice Inbred C57BLmedicine.anatomical_structureImmunologybusinessPseudouridineScience (New York, N.Y.)
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