Search results for "NERVE"

showing 10 items of 1683 documents

Pancoast's syndrome caused by lymph node metastasis from breast cancer.

2018

Pancoast’s syndrome may be the result of neoplastic, inflammatory or infectious disease. We report an unusual case of Pancoast’s syndrome in a patient with metastatic breast cancer. A 54-year-old woman, affected by metastatic breast cancer, presented for severe shoulder pain, paraesthesia and numbness in the right arm. Despite further multiple lines of systemic chemotherapy, she developed a progressive enlargement of retropectoral, supraclavicular and infraclavicular lymph node metastases, which involved brachial plexus, apex of lung and anterior mediastinum. Physical examination revealed severe weakness of proximal muscles of the right arm. Neuropathic pain was managed with pharmacological…

medicine.medical_specialtypain (palliative care)Settore MED/06 - Oncologia MedicaPhysical examinationBreast Neoplasmspain (neurology)03 medical and health sciencesbreast cancer0302 clinical medicineBreast cancerFatal OutcomeShoulder PainmedicineHumansPain ManagementBrachial PlexusInfraclavicular Lymph NodeZiconotideLungUnusual Presentation of More Common Disease/Injurymedicine.diagnostic_testperipheral nerve diseasebusiness.industryPancoast SyndromeGeneral MedicineMiddle Agedmedicine.diseaseMetastatic breast cancermedicine.anatomical_structure030220 oncology & carcinogenesisLymphatic MetastasisNeuropathic painNeuralgiaSettore MED/26 - NeurologiaFemaleRadiologyLymph NodesbusinessBrachial plexus030217 neurology & neurosurgerymedicine.drugBMJ case reports
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Relevance of diagnostic investigations in chronic inflammatory demyelinating poliradiculoneuropathy: Data from the Italian CIDP database

2020

The objective of our work was to report the clinical features and the relevance of diagnostic investigations in patients with chronic inflammatory demyelinating polyradiculoneuropathy (CIDP). We retrospectively reviewed data from patients with a clinical diagnosis of CIDP included in a national database. Among the 500 included patients with a clinical diagnosis of CIDP, 437 patients (87%) fulfilled the European Federation of Neurological Societies and Peripheral Nerve Society criteria for CIDP (definite in 407, probable in 26, possible in four). In 352 patients (86%) motor nerve conduction abnormalities consistent with demyelination were sufficient for the diagnosis of definite CIDP. In 55 …

medicine.medical_specialtyperipheral neuropathyResponse to therapyMotor nerveCIDPSettore MED/26chronic inflammatory demyelinating neuropathy; CIDP; diagnostic criteria; EMG; peripheral neuropathy03 medical and health sciences0302 clinical medicineEMGInternal medicinemedicineIn patientNerve biopsymedicine.diagnostic_testbusiness.industryGeneral Neurosciencechronic inflammatory demyelinating neuropathyPolyradiculoneuropathymedicine.diseaseCIDP; Chronic inflammatory demyelinating neuropathy; Diagnostic criteria; EMG; Peripheral neuropathyPeripheral neuropathymedicine.anatomical_structure030220 oncology & carcinogenesisClinical diagnosisdiagnostic criteriaNeurology (clinical)business030217 neurology & neurosurgerySensory nerve
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Cholesterol and ocular pathologies: focus on the role of cholesterol-24S-hydroxylase in cholesterol homeostasis

2015

The retina is responsible for coding the light stimulus into a nervous signal that is transferred to the brain via the optic nerve. The retina is formed by the association of the neurosensory retina and the retinal pigment epithelium that is supported by Bruch’s membrane. Both the physical and metabolic associations between these partners are crucial for the functioning of the retina, by means of nutrient intake and removal of the cell and metabolic debris from the retina. Dysequilibrium are involved in the aging processes and pathologies such as age-related macular degeneration, the leading cause of visual loss after the age of 50 years inWestern countries. The retina is composed of severa…

medicine.medical_specialtyretinaOrganes des sensgenetic structuresrétineCellSensory OrgansMédecine humaine et pathologielcsh:TP670-699BiologyBiochemistrydégénérescence maculaire liée à l'âgechemistry.chemical_compoundlipid[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyInternal medicinemedicine[SDV.MHEP.OS]Life Sciences [q-bio]/Human health and pathology/Sensory Organsage-related macular degenerationlipideRetinaRetinal pigment epitheliumCholesterolagingcholesterolcholestérolMetabolismMacular degenerationmedicine.diseaseSteroleye diseasesmedicine.anatomical_structureEndocrinologyglaucomachemistry[ SDV.MHEP.OS ] Life Sciences [q-bio]/Human health and pathology/Sensory OrgansglaucomeOptic nerveHuman health and pathologysense organslcsh:Oils fats and waxesretina;lipid;cholesterol;glaucoma;age-related macular degeneration;aging;rétine;lipide;cholestérol;glaucome;dégénérescence maculaire liée à l'âgeAgronomy and Crop Science[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyFood Science
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EFFICACY OF REPETITIVE TRANSCRANIAL MAGNETIC STIMULATION/TRANSCRANIAL DIRECT CURRENT STIMULATION IN COGNITIVE NEUROREHABILITATION

2008

Summary: Cognitive deficits are a common consequence of neurologic disease, in particular, of traumatic brain injury, stroke, and neurodegenerative disorders, and there is evidence that specific cognitive training may be effective in cognitive rehabilitation. Several investigations emphasize the fact that interacting with cortical activity, by means of cortical stimulation, can positively affect the short-term cognitive performance and improve the rehabilitation potential of neurologic patients. In this respect, preliminary evidence suggests that cortical stimulation may play a role in treating aphasia, unilateral neglect, and other cognitive disorders. Several possible mechanisms can accou…

medicine.medical_treatmentBiophysicsLANGUAGE050105 experimental psychologylcsh:RC321-57103 medical and health sciences0302 clinical medicinemedicineHumans0501 psychology and cognitive sciencesCognitive rehabilitation therapylcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNeurorehabilitationCerebral CortexRehabilitationTranscranial direct-current stimulationSettore M-PSI/02 - Psicobiologia E Psicologia FisiologicaGeneral NeuroscienceTRANSCRANIAL DIRECT CURRENT STIMULATION05 social sciencesMEMORYATTENTIONCognitionCOGNITIVE REHABILITATIONTranscranial Magnetic StimulationCognitive trainingTranscranial magnetic stimulationTreatment OutcomeCOGNITIVE DEFICITSBrain stimulationREPETITIVE TRANSCRANIAL MAGNETIC STIMULATIONTranscutaneous Electric Nerve StimulationNeurology (clinical)PsychologyCognition DisordersNeuroscience030217 neurology & neurosurgery
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The rodent tibia fracture model: A critical review and comparison with the complex regional pain syndrome literature

2018

Abstract Distal limb fracture is the most common cause of complex regional pain syndrome (CRPS), thus the rodent tibia fracture model (TFM) was developed to study CRPS pathogenesis. This comprehensive review summarizes the published TFM research and compares these experimental results with the CRPS literature. The TFM generated spontaneous and evoked pain behaviors, inflammatory symptoms (edema, warmth), and trophic changes (skin thickening, osteoporosis) resembling symptoms in early CRPS. Neuropeptides, inflammatory cytokines, and nerve growth factor (NGF) have been linked to pain behaviors, inflammation, and trophic changes in the TFM model and proliferating keratinocytes were identified …

medicine.medical_treatmentOsteoporosisTibia FractureInflammationBioinformaticsArticleProinflammatory cytokine03 medical and health sciencesMice0302 clinical medicine030202 anesthesiologymedicineAnimalsbusiness.industrymedicine.diseasePathophysiologyRatsTibial FracturesDisease Models AnimalAnesthesiology and Pain MedicineCytokineNerve growth factorComplex regional pain syndromeNeurologyNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgeryComplex Regional Pain Syndromes
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A single mutation in the recombinant light chain of tetanus toxin abolishes its proteolytic activity and removes the toxicity seen after reconstituti…

1994

Specific proteolysis by the tetanus toxin light chain of a vesicle-associated membrane protein (VAMP) involved in exocytosis is thought to underlie its intracellular blockade of neurotransmitter release. To substantiate this mechanism, recombinant light chain was expressed as a maltose binding protein-light chain fusion product in Escherichia coli. After purification of affinity chromatography and cleavage with factor Xa, the resultant light chain was isolated and its identity confirmed by Western blotting and N-terminal sequencing. It exhibited activity similar to that of the native light chain in proteolyzing its target in isolated bovine small synaptic vesicles and in hydrolyzing a 62-re…

medicine.medical_treatmentRecombinant Fusion ProteinsMolecular Sequence DataNeurotoxinsGlutamic AcidMaltose bindingNerve Tissue ProteinsIn Vitro TechniquesImmunoglobulin light chainBiochemistrySynaptic vesicleExocytosislaw.inventionR-SNARE ProteinsMiceStructure-Activity RelationshipAffinity chromatographyGlutamatesTetanus ToxinlawThermolysinEndopeptidasesmedicineEscherichia coliAnimalsAmino Acid SequenceProteaseBase SequenceChemistryMembrane ProteinsMolecular biologyPeptide FragmentsRecombinant DNAMutagenesis Site-DirectedCattleBiochemistry
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Translation of HTT mRNA with expanded CAG repeats is regulated by the MID1-PP2A protein complex.

2012

Expansion of CAG repeats is a common feature of various neurodegenerative disorders, including Huntington's disease. Here we show that expanded CAG repeats bind to a translation regulatory protein complex containing MID1, protein phosphatase 2A and 40S ribosomal S6 kinase. Binding of the MID1-protein phosphatase 2A protein complex increases with CAG repeat size and stimulates translation of the CAG repeat expansion containing messenger RNA in a MID1-, protein phosphatase 2A- and mammalian target of rapamycin-dependent manner. Our data indicate that pathological CAG repeat expansions upregulate protein translation leading to an overproduction of aberrant protein and suggest that the MID1-com…

metabolism [Microtubule Proteins]General Physics and AstronomyHTT protein humanRibosomal s6 kinaseMice0302 clinical medicinemetabolism [Transcription Factors]Protein Phosphatase 2Luciferasesgenetics [Nerve Tissue Proteins]genetics [Protein Biosynthesis]0303 health sciencesHuntingtin ProteinMultidisciplinarybiologyTOR Serine-Threonine KinasesNuclear ProteinsTranslation (biology)3. Good healthmetabolism [Luciferases]Microtubule Proteinsddc:500metabolism [Nuclear Proteins]genetics [Trinucleotide Repeat Expansion]Protein Bindingcongenital hereditary and neonatal diseases and abnormalitiesMTOR protein humanUbiquitin-Protein LigasesBlotting WesternNerve Tissue Proteinsmetabolism [TOR Serine-Threonine Kinases]metabolism [RNA Messenger]General Biochemistry Genetics and Molecular Biology03 medical and health sciencesgenetics [RNA Messenger]mental disordersHuntingtin ProteinAnimalsHumansEukaryotic Small Ribosomal SubunitRNA MessengerNucleotide Motifs030304 developmental biologyMessenger RNAmetabolism [Nerve Tissue Proteins]RNAmetabolism [Protein Phosphatase 2]General ChemistryProtein phosphatase 2Molecular biologynervous system diseasesProtein Biosynthesisbiology.proteinTrinucleotide repeat expansionTrinucleotide Repeat Expansion030217 neurology & neurosurgeryMid1 protein humanHeLa CellsTranscription FactorsNature communications
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The E3 Ubiquitin Ligase MID1 Catalyzes Ubiquitination and Cleavage of Fu

2014

Sonic Hedgehog (SHH)-GLI signalling plays an important role during embryogenesis and in tumorigenesis. The survival and growth of several types of cancer depend on autonomously activated SHH-GLI signalling. A protein complex containing the ubiquitin-ligase MID1 and protein phosphatase 2A (PP2A) regulates the nuclear localization and transcriptional activity of GLI3, a transcriptional effector molecule of SHH, in cancer cell lines with autonomously activated SHH signalling. However, the exact molecular mechanisms that mediate the interaction between MID1 and GLI3 remained unknown. Here, we show that MID1 catalyses the ubiquitination and proteasomal cleavage of the GLI3-regulator Fu. Our data…

metabolism [Microtubule Proteins]Ubiquitin-conjugating enzymeBiochemistrymetabolism [Protein Serine-Threonine Kinases]Ubiquitinmetabolism [Transcription Factors]Nuclear proteinSonic hedgehogbiologymetabolism [Protein-Serine-Threonine Kinases]Nuclear Proteinsrespiratory systemProtein-Serine-Threonine KinasesUbiquitin ligaseGene Expression Regulation NeoplasticGLI3 protein humanBiochemistryddc:540embryonic structuresMicrotubule Proteinsmetabolism [Hedgehog Proteins]Function and Dysfunction of the Nervous Systemmetabolism [Nuclear Proteins]Signal Transductionmetabolism [Kruppel-Like Transcription Factors]Proteasome Endopeptidase Complexanimal structuresSTK36 protein humanUbiquitin-Protein LigasesKruppel-Like Transcription FactorsNerve Tissue ProteinsProtein Serine-Threonine Kinaseschemistry [Ubiquitin-Protein Ligases]CatalysisZinc Finger Protein Gli3Cell Line TumorGLI3HumansHedgehog Proteinsmetabolism [Proteasome Endopeptidase Complex]metabolism [Cell Nucleus]Molecular Biologychemistry [Lysine]DNA PrimersCell Nucleusmetabolism [Nerve Tissue Proteins]UbiquitinLysineUbiquitinationCell BiologyProtein phosphatase 2chemistry [Ubiquitin]Proteasomebiology.proteinSHH protein humanhuman activitiesMid1 protein humanHeLa CellsTranscription FactorsJournal of Biological Chemistry
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Posttraumatic Propofol Neurotoxicity Is Mediated via the Pro–Brain-Derived Neurotrophic Factor-p75 Neurotrophin Receptor Pathway in Adult Mice*

2016

Objectives:The gamma-aminobutyric acid modulator propofol induces neuronal cell death in healthy immature brains by unbalancing neurotrophin homeostasis via p75 neurotrophin receptor signaling. In adulthood, p75 neurotrophin receptor becomes down-regulated and propofol loses its neurotoxic effect. H

musculoskeletal diseases0301 basic medicineBrain-derived neurotrophic factorProgrammed cell deathbiologybusiness.industryNeurotoxicityCaspase 3PharmacologyCritical Care and Intensive Care Medicinemedicine.disease03 medical and health sciences030104 developmental biology0302 clinical medicinenervous systemAnesthesiamedicinebiology.proteinLow-affinity nerve growth factor receptorReceptorbusiness030217 neurology & neurosurgeryHomeostasisNeurotrophinCritical Care Medicine
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Surgical repair of acute Achilles tendon rupture with an end-to-end tendon suture and tendon flap.

2015

Abstract Background Achilles tendon ruptures are becoming more common. Complications after open or minimally invasive surgery are: recurrent rupture (2–8%), wound breakdown, deep infections, granuloma, and fistulas. The authors expose their experience with a personal technique. Materials In 8 patients with acute rupture of Achilles tendon the surgery was performed at least 25 days after trauma. Clinical exam and MR demonstrated in all case a total lesion of tendon. After a posterolateral skin incision the tendon stumps were debrided and suture in end-to-end fashion. A tendon flap was harvested from the proximal part of the tendon, in order to protect and reinforce the suture itself. A plast…

musculoskeletal diseasesAdultMalemedicine.medical_specialtyFistulaSettore MED/19 - Chirurgia PlasticaSural nerveAchilles TendonSurgical FlapsWeight-BearingVascularityPostoperative ComplicationsTendon InjuriesmedicineHumansSurgical Wound InfectionOrthopedic ProceduresRange of Motion ArticularGeneral Environmental ScienceSurgical repairFibrous jointAchilles tendon ruptureRuptureAchilles tendonGranulomabusiness.industrySuture TechniquesSoft tissueSurgical treatment of tendon rupture Achilles tendon injuryRecovery of FunctionMiddle Agedmusculoskeletal systemSurgeryTendonmedicine.anatomical_structureTreatment OutcomeGeneral Earth and Planetary SciencesFemaleAchilles tendon rupturemedicine.symptombusinessFollow-Up StudiesInjury
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