Search results for "NEURODEGENERATIVE DISEASES"

showing 10 items of 155 documents

proNGF Measurement in Cerebrospinal Fluid Samples of a Large Cohort of Living Patients With Alzheimer's Disease by a New Automated Immunoassay

2021

The discovery of new biomarkers for Alzheimer's disease (AD) is essential for an accurate diagnosis, to conceive new strategies of treatments, and for monitoring the efficacy of potential disease-modifying therapies in clinical trials. proNGF levels in the cerebrospinal fluid (CSF) represent a promising diagnostic biomarker for AD, but its validation was hampered by the absence of a reliable immunoassay. In the literature, proNGF is currently measured in postmortem brain tissue by semiquantitative immunoblot. Here we describe the development and validation of a new method to measure proNGF in the CSF of living patients. This method, based on molecular size separation by capillary electropho…

OncologyAgingmedicine.medical_specialtydiagnosisCognitive NeuroscienceNeurosciences. Biological psychiatry. NeuropsychiatryDiseaseSettore BIO/09 - FisiologiaCerebrospinal fluidneurodegenerative diseaseInternal medicinemedicineDiagnostic biomarkerneurodegenerative diseasesproNGFimmunoassayOriginal Researchmedicine.diagnostic_testbusiness.industrySignificant differenceAlzheimer's diseaseLarge cohortdiagnosiImmunoassayBiomarker (medicine)biomarkerAutomated immunoassaybusinessRC321-571Neuroscience
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Focused Ultrasound in Neuroscience. State of the Art and Future Perspectives

2021

Transcranial MR-guided Focused ultrasound (tcMRgFUS) is a surgical procedure that adopts focused ultrasounds beam towards a specific therapeutic target through the intact skull. The convergence of focused ultrasound beams onto the target produces tissue effects through released energy. Regarding neurosurgical applications, tcMRgFUS has been successfully adopted as a non-invasive procedure for ablative purposes such as thalamotomy, pallidotomy, and subthalamotomy for movement disorders. Several studies confirmed the effectiveness of tcMRgFUS in the treatment of several neurological conditions, ranging from motor disorders to psychiatric disorders. Moreover, using low-frequencies tcMRgFUS sys…

OpinionComputer scienceNeurodegenerative diseaseFocused ultrasoundlcsh:RC321-57103 medical and health sciences0302 clinical medicineneurodegenerative diseaseslcsh:Neurosciences. Biological psychiatry. Neuropsychiatry030304 developmental biologyBlood-brain barriertcMRgFUSLIFU0303 health sciencesEpilepsySettore MED/27 - NeurochirurgiaGeneral NeuroscienceSettore MED/37 - NeuroradiologiaPsychiatric disorderHIFUpsychiatric disordersfocused ultrasoundSettore MED/26 - NeurologiaSettore MED/36 - Diagnostica Per Immagini E RadioterapiaNeuroscienceneuro-oncology030217 neurology & neurosurgeryBrain Sciences
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7-Nitroindazole protects striatal neurons against MPP+ -induced degeneration.

2006

The neuropathological hallmark of Parkinson's disease (PD) is the selective degeneration of dopaminergic (DAergic) neurons in the substantia nigra pars compacta (SNc). In this study, using a microdialysis technique, we investigated whether an inhibitor of neuronal nitric oxide synthase (nNOS), 7-nitrindazole (7-NI), could protect against DAergic neuronal damage induced by in vivo infusion of 1-methyl-4-phenylpiridinium iodide (MPP+) in freely moving rats. Experiments were performed over 2 days in three groups of rats: (a) nonlesioned, (b) MPP+-lesioned, and (c) 7-NI pretreated MPP+-lesioned rats. On day 1, control rats were perfused with an artificial CSF, while 1 mM MPP+ was infused into t…

PARKINSONS-DISEASERAT STRIATUMMONOAMINE-OXIDASENITRIC-OXIDE SYNTHASEBRAINNEURODEGENERATIVE DISEASESSODIUM-SALICYLATEMPTP NEUROTOXICITYTRANSPORTER ACTIVITYLIPID-PEROXIDATION
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Imaging of Substantia Nigra in Parkinson's Disease: A Narrative Review.

2021

Parkinson’s disease (PD) is a progressive neurodegenerative disorder, characterized by motor and non-motor symptoms due to the degeneration of the pars compacta of the substantia nigra (SNc) with dopaminergic denervation of the striatum. Although the diagnosis of PD is principally based on a clinical assessment, great efforts have been expended over the past two decades to evaluate reliable biomarkers for PD. Among these biomarkers, magnetic resonance imaging (MRI)-based biomarkers may play a key role. Conventional MRI sequences are considered by many in the field to have low sensitivity, while advanced pulse sequences and ultra-high-field MRI techniques have brought many advantages, partic…

Parkinson's diseaseSettore MED/50 - Scienze Tecniche Mediche ApplicateSubstantia nigraNeurosciences. Biological psychiatry. NeuropsychiatryDiseaseStriatumReview030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineironneurodegenerative diseasemedicinemagnetic resonance imagingneurodegenerative diseasesmedicine.diagnostic_testPars compactabusiness.industryGeneral NeuroscienceradiomicSettore MED/37 - NeuroradiologiabiomarkersMagnetic resonance imagingmedicine.diseaseparkinsonian disordersnigrosome-1radiomicsParkinson’s diseasebiomarkerSettore MED/26 - NeurologiaDifferential diagnosisneuromelaninbusinessSettore MED/36 - Diagnostica Per Immagini E RadioterapiaNeuroscience030217 neurology & neurosurgeryDiffusion MRIRC321-571Brain sciences
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The Blood–Brain Barrier as a Target in Traumatic Brain Injury Treatment

2014

Traumatic brain injury (TBI) is one of the most frequent causes of death in the young population. Several clinical trials have unsuccessfully focused on direct neuroprotective therapies. Recently immunotherapeutic strategies shifted into focus of translational research in acute CNS diseases. Cross-talk between activated microglia and blood–brain barrier (BBB) could initiate opening of the BBB and subsequent recruitment of systemic immune cells and mediators into the brain. Stabilization of the BBB after TBI could be a promising strategy to limit neuronal inflammation, secondary brain damage and acute neurodegeneration. This review provides an overview on the pathophysiology of TBI and brain…

Pathologymedicine.medical_specialtyTraumatic brain injuryPeroxisome Proliferator-Activated ReceptorsBrain EdemaInflammationBrain damageBlood–brain barrierNeuroprotectionRosiglitazoneReceptors GlucocorticoidmedicineHumansHypoglycemic AgentsMyosin-Light-Chain KinaseNeuroinflammationInflammationPioglitazoneMicrogliabusiness.industryNeurodegenerationNeurodegenerative DiseasesGeneral Medicinemedicine.diseaseCell HypoxiaNeuroprotective Agentsmedicine.anatomical_structurenervous systemBlood-Brain BarrierBrain InjuriesThiazolidinedionesmedicine.symptombusinessNeuroscienceArchives of Medical Research
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The close link between brain vascular pathological conditions and neurodegenerative diseases: Focus on some examples and potential treatments

2022

A close relationship is emerging among the age-related neurodegenerative decline, and the age-related typical alterations, dysfunctions, and related diseases of the cerobro-and/or cardiovascular system, which contributes in a significative manner to the triggering and progressing of neurodegenerative diseases (NeuroDegD). Specifically, macroinfarcts, microinfarcts, micro-hemorrhages (and particularly their number), atherosclerosis, arteriolosclerosis and cerebral amyloid angiopathy have been documented to be significantly associated with the onset of the cognitive impairment. In addition, vascular alterations and dysfunctions resulting in a reduced cerebral blood flow, and anomalies in the …

PharmacologyAlterations dysfunctions and related diseases of the cerobro-and/or cardiovascular systemBlood-Brain BarrierPotential treatmentsPhysiologyBrain blood barrier (BBB)Neurodegenerative diseasesHumansSettore MED/05 - Patologia ClinicaMolecular MedicineNeurovascular unit (NVU)Vascular Pharmacology
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Antioxidants as treatment for neurodegenerative disorders.

2002

Oxidative stress is a ubiquitously observed hallmark of neurodegenerative disorders. Neuronal cell dysfunction and cell death due to oxidative stress may causally contribute to the pathogenesis of progressive neurodegenerative disorders, such as Alzheimer's disease and Parkinson's disease, as well as acute syndromes of neurodegeneration, such as ischaemic and haemorrhagic stroke. Neuroprotective antioxidants are considered a promising approach to slowing the progression and limiting the extent of neuronal cell loss in these disorders. The clinical evidence demonstrating that antioxidant compounds can act as protective drugs in neurodegenerative disease, however, is still relatively scarce. …

PharmacologyPathologymedicine.medical_specialtyParkinson's diseaseFree Radicalsbusiness.industryNeurodegenerationNeurodegenerative DiseasesGeneral MedicineDiseasemedicine.diseaseBioinformaticsmedicine.disease_causeNeuroprotectionAntioxidantsDrug developmentHuntington's diseaseMedicineAnimalsHumansPharmacology (medical)Amyotrophic lateral sclerosisbusinessOxidative stressExpert opinion on investigational drugs
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Membrane breakdown in acute and chronic neurodegeneration: focus on choline-containing phospholipids.

2000

Breakdown of cellular membranes is a characteristic feature of neuronal degeneration in acute (stroke) and chronic (senile dementia) neurological disorders. The present review summarizes recent experimental and clinical work which concentrated on changes of choline-containing phospholipids as indicators of neuronal membrane breakdown. Experimental studies identified glutamate release, calcium influx, and activation of cellular phospholipase A2 (PLA2) as important steps initiating membrane breakdown in cultured neurons or brain slices under hypoxic or ischemic conditions. Proton NMR studies have shown an elevation of choline-containing compounds in the brain of Alzheimer patients while neuro…

PhospholipidPhospholipasechemistry.chemical_compoundNeurochemicalPhospholipase A2PhosphatidylcholinemedicineCholineAnimalsHumansBiological PsychiatrybiologyChemistryNeurodegenerationCell MembraneGlutamate receptorNeurodegenerative Diseasesmedicine.diseasePsychiatry and Mental healthNeurologyBiochemistryAcute DiseaseChronic Diseasebiology.proteinPhosphatidylcholinesNeurology (clinical)Journal of neural transmission (Vienna, Austria : 1996)
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Therapeutic potential of dietary polyphenols against brain ageing and neurodegenerative disorders

2010

In recent years there has been a growing interest, supported by a large number of experimental and epidemiological studies, in the beneficial effects of some commonly used food-derived products in preventing various age-related pathologic conditions, ranging from cancer to neurodegenerative diseases. Spices and herbs often contain active phenolic substances endowed with potent antioxidative and chemopreventive properties. Curcumin is a phytochemical compound extracted from the rhizome of Curcuma Longa. It is the pigment responsible for the characteristic yellow color of Indian curry. Data from our and other laboratories demonstrated that curcumin, as well as some other polyphenols, strongly…

PolyphenolAgingCurcuminNeuroprotective AgentDiseasePharmacologyAntioxidantsCatechinchemistry.chemical_compoundCaffeic AcidsPhenolsCellular stress responseMedicineCurcumaAntioxidants; Caffeic Acids; Catechin; Curcumin; Flavonoids; Neuroprotective Agents; Phenols; Phenylethyl Alcohol; Polyphenols; Aging; Brain; Diet; Neurodegenerative DiseasesFlavonoidsbiologyPhenolbusiness.industryPolyphenolsBrainNeurodegenerative DiseasesPhenylethyl Alcoholbiology.organism_classificationDietHeme oxygenaseNeuroprotective AgentschemistryPhytochemicalCaffeic AcidAgeingPhase II DetoxificationCurcuminFlavonoidAntioxidantbusiness
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The unsolved relationship of brain aging and late-onset Alzheimer disease.

2009

Late-onset Alzheimer disease is the most common form of dementia and is strongly associated with age. Today, around 24 million people suffer from dementia and with aging of industrial populations this number will significantly increase throughout the next decades. An effective therapy that successfully decelerates or prevents the progressive neurodegeneration does not exist. Histopathologically Alzheimer disease is characterized by extensive extracellular amyloid beta (Abeta) plaques, intracellular neurofibrillary tangles (NFTs), synaptic loss and neuronal cell death in distinct brain regions. The molecular correlation of Abeta or NFTs and development of late-onset Alzheimer disease needs f…

Programmed cell deathAgingAmyloid βFree RadicalsBiophysicsmedicine.disease_causeBiochemistryModels BiologicalAtrophyAlzheimer DiseasemedicineExtracellularDementiaAnimalsHumansMolecular BiologyAmyloid beta-Peptidesbusiness.industryBrainNeurodegenerative Diseasesmedicine.diseaseDementiaAlzheimer's diseasebusinessNeuroscienceOxidative stressIntracellularBiochimica et biophysica acta
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