Search results for "NEUTROPHIL"

showing 10 items of 449 documents

Increased adhesion and activation of polymorphonuclear neutrophil granulocytes to endothelial cells under heavy metal exposure in vitro.

1994

Heavy metals have been implicated in the mechanisms of endothelial damage. Influences of heavy metal ions on diverse cell types have been studied using a variety of in vitro and in vivo methods. Polymorphonuclear neutrophil granulocytes (PMNs) have physiological and pathological functions, including the modulation of adhesion to and destruction of endothelial cells (ECs). PMNs were studied during interaction with human umbilical vein ECs under exposure to zinc, nickel and cobalt using an in vitro model. We studied adhesion processes with the help of a computer-controlled image-analyzing system and examined the activation of PMNs by quantification of leukotriene B4 (LTB4) release. The biphas…

Cell typeUmbilical VeinsLeukotriene B4NeutrophilsEnzyme-Linked Immunosorbent AssayPathology and Forensic MedicineMetalchemistry.chemical_compoundIn vivoNickelCell AdhesionImage Processing Computer-AssistedHumansMolecular BiologyCells CulturedPolymorphonuclear neutrophilChemistryHeavy metalsCell BiologyGeneral MedicineAdhesionCobaltIn vitroCell biologyZincBiochemistryvisual_artvisual_art.visual_art_mediumEndothelium VascularE-Selectinhuman activitiesCell Adhesion MoleculesPathobiology : journal of immunopathology, molecular and cellular biology
researchProduct

Skull and vertebral bone marrow are myeloid cell reservoirs for the meninges and CNS parenchyma.

2021

Getting around the blood–brain barrier The meninges comprise three membranes that surround and protect the central nervous system (CNS). Recent studies have noted the existence of myeloid cells resident there, but little is known about their ontogeny and function, and whether other meningeal immune cell populations have important roles remains unclear (see the Perspective by Nguyen and Kubes). Cugurra et al. found in mice that a large proportion of continuously replenished myeloid cells in the dura mater are not blood derived, but rather transit from cranial bone marrow through specialized channels. In models of CNS injury and neuroinflammation, the authors demonstrated that these myeloid c…

Central Nervous SystemPathologymedicine.medical_specialtyMyeloidEncephalomyelitis Autoimmune ExperimentalNeutrophilsCentral nervous systemBone Marrow CellsBiologyArticleMonocytesMiceImmune systemMeningesBone MarrowCell MovementCentral Nervous System DiseasesParenchymamedicineAnimalsHomeostasisMyeloid CellsNeuroinflammationSpinal Cord InjuriesMultidisciplinaryInnate immune systemSkullMeningesBrainSpinemedicine.anatomical_structureSpinal CordBone marrowDura MaterScience (New York, N.Y.)
researchProduct

Synthesis and pharmacological evaluation of 2'-hydroxychalcones and flavones as inhibitors of inflammatory mediators generation.

1995

2'-Hydroxy-3,4-dimethoxy-3',4'-dimethylchalcone (3a), 2'-hydroxy-3',4',3,4-tetramethoxychalcone (3b), and their corresponding flavones, 3',4'-dimethoxy-7,8-dimethylflavone (4a) and 3',4',7,8-tetramethoxyflavone (4b), were prepared from 3,4-dimethoxycinnamic acid and the respective phenol. The four compounds inhibited enzymic lipid peroxidation and showed weak peroxyl scavenging activity. They also reduced LTB 4 release from human neutrophils stimulated by A23187. The chalcone 3b was the only compound able to inhibit in a concentration-dependent way, synovial human recombinant phospholipase A 2 activity, human platelet TXB 2 generation, and human neutrophil degranulation. This chalcone exert…

ChalconeAntioxidantNeutrophilsmedicine.medical_treatmentFlavonoidChemical synthesisFlavonesCell DegranulationPhospholipases ALipid peroxidationchemistry.chemical_compoundMiceChalconeChalconesDrug DiscoverySynovial FluidmedicineAnimalsHumanschemistry.chemical_classificationFlavonoidsPhospholipase APancreatic ElastaseChemistryDegranulationFree Radical ScavengersPhospholipases A2BiochemistryMolecular MedicineEicosanoidsLipid PeroxidationInflammation MediatorsJournal of medicinal chemistry
researchProduct

Phenylsulphonyl urenyl chalcone derivatives as dual inhibitors of cyclo-oxygenase-2 and 5-lipoxygenase

2005

Two series of phenylsulphonyl urenyl chalcone derivatives (UCH) with various patterns of substitution were tested for their effects on nitric oxide (NO) and prostaglandin E2 (PGE2) overproduction in RAW 264.7 macrophages. None of the tested compounds reduced NO production more than 50% at 10 microM but most of them inhibited the generation of PGE2 with IC50 values under the micromolar range. Me-UCH 1, Me-UCH 5, Me-UCH 9, Cl-UCH 1, and Cl-UCH 9 were selected to evaluate their influence on human leukocyte functions and eicosanoids generation. These derivatives selectively inhibited cyclo-oxygenase-2 (COX-2) activity in human monocytes being Me-UCH 5 the most potent (IC50 0.06 microM). Selecte…

ChalconeNeutrophilsNitric OxideLeukotriene B4DinoprostoneGeneral Biochemistry Genetics and Molecular BiologyCell LineNitric oxideMiceStructure-Activity Relationshipchemistry.chemical_compoundChalconesmedicineAnimalsHumansCyclooxygenase InhibitorsLipoxygenase InhibitorsGeneral Pharmacology Toxicology and PharmaceuticsProstaglandin E2IC50Molecular StructurebiologySuperoxideMacrophagesElastaseGeneral MedicinechemistryBiochemistryCyclooxygenase 2MyeloperoxidaseArachidonate 5-lipoxygenasebiology.proteinmedicine.drugLife Sciences
researchProduct

Synthesis and anti-inflammatory activity of chalcone derivatives

1998

Chalcones and their derivatives were synthesized and evaluated for their anti-inflammatory activity. In vitro, chalcones 2, 4, 8, 10 and 13 inhibited degranulation and 5-lipoxygenase in human neutrophils, whereas 11 behaved as scavenger of superoxide. Only four compounds (4-7) inhibited cyclo-oxygenase-2 activity. The majority of these samples showed anti-inflammatory effects in the mouse air pouch model.

ChalconeNeutrophilsmedicine.drug_classLeukotriene B4Clinical BiochemistryPharmaceutical ScienceLeukotriene B4BiochemistryChemical synthesisAnti-inflammatoryMiceStructure-Activity Relationshipchemistry.chemical_compoundChalconeIn vivoDrug DiscoverymedicineAnimalsHumansCyclooxygenase InhibitorsLipoxygenase InhibitorsMolecular BiologyPropiophenonesArachidonate 5-LipoxygenaseCyclooxygenase 2 InhibitorsMolecular StructureChemistrySuperoxideAnti-Inflammatory Agents Non-SteroidalOrganic ChemistryDegranulationMembrane ProteinsIn vitroIsoenzymesBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesDrug DesignMolecular MedicineBioorganic & Medicinal Chemistry Letters
researchProduct

Studies on the mechanism of PMN activation. I. By dextran sulfates.

1982

Evidence is presented that enhanced reduction of the dye nitroblue-tetrazolium (NBT) by polymorphonuclear leukocytes (PMN) which are stimulated by dextran sulfates (DS) is not exclusively due to the phagocytosis of particles formed by NBT and DS. Not only the size of phagocytizable particles but the degree of substitution determines the acceleration of NBT-reduction. A likely cause of this acceleration is the triggering of the alternative pathway of the complement activation.

ChemistryNeutrophilsPhagocytosisNitroblue TetrazoliumComplement Pathway Alternativechemical and pharmacologic phenomenaDextransHematologyGeneral MedicineComplement systemchemistry.chemical_compoundDegree of substitutionDextranBiochemistryPhagocytosisAlternative complement pathwayBiophysicsHumansBlut
researchProduct

An Alternative Pathway of Imiquimod-Induced Psoriasis-Like Skin Inflammation in the Absence of Interleukin-17 Receptor A Signaling

2013

Topical application of imiquimod (IMQ) on the skin of mice induces inflammation with common features found in psoriatic skin. Recently, it was postulated that IL-17 has an important role both in psoriasis and in the IMQ model. To further investigate the impact of IL-17RA signaling in psoriasis, we generated IL-17 receptor A (IL-17RA)-deficient mice (IL-17RA(del)) and challenged these mice with IMQ. Interestingly, the disease was only partially reduced and delayed but not abolished when compared with controls. In the absence of IL-17RA, we found persisting signs of inflammation such as neutrophil and macrophage infiltration within the skin. Surprisingly, already in the naive state, the skin …

ChemokineInflammationImiquimodDermatologyInterleukin-17 receptorBiochemistryMiceAdjuvants ImmunologicPsoriasismedicineAnimalsPsoriasisMacrophageMolecular BiologySkinMice KnockoutImiquimodReceptors Interleukin-17biologyInterleukin-6InterleukinsMacrophagesInterleukin-17Cell Biologymedicine.diseaseDisease Models AnimalCXCL2Neutrophil InfiltrationImmunologyAminoquinolinesbiology.proteinFemaleTumor necrosis factor alphamedicine.symptomSignal Transductionmedicine.drugJournal of Investigative Dermatology
researchProduct

Thy-1 (CD90) regulates the extravasation of leukocytes during inflammation.

2010

Human Thy-1 (CD90) has been shown to mediate adhesion of inflammatory cells to activated microvascular endothelial cells via interaction with Mac-1 (CD11b/CD18) in vitro. Since there are no data showing the physiological relevance of Thy-1 for the recruitment of inflammatory cells in vivo, different inflammation models were investigated in Thy-1-deficient mice and littermate controls. In thioglycollate-induced peritonitis, the number of neutrophils and monocytes was significantly diminished in Thy-1-deficient mice. During acute lung inflammation, the extravasation of eosinophils and monocytes into the lung was significantly reduced in Thy-1-deficient mice. Moreover, during chronic lung infl…

ChemokineMice 129 StrainNeutrophilsmedicine.medical_treatmentT-LymphocytesImmunologyInflammationCD18In Vitro TechniquesPeritonitisMonocytesMiceCell MovementCell AdhesionLeukocytesImmunology and AllergyMedicineAnimalsHumansCD90Thy-1InflammationMice KnockoutTransplantation Chimerabiologybusiness.industryInterleukinsEndothelial CellsPneumoniaExtravasationTransplantationEosinophilsMice Inbred C57BLCytokineIntegrin alpha MImmunologybiology.proteinThy-1 Antigensmedicine.symptomChemokinesbusinessextravasationPeptide HydrolasesEuropean journal of immunology
researchProduct

Effects of SCA40 on human isolated bronchus and human polymorphonuclear leukocytes: comparison with rolipram, SKF94120 and levcromakalim

1996

1. SCA40 (0.1 nM-0.1 mM) produced concentration-dependent suppression of the spontaneous tone of human isolated bronchus (-log EC50 = 6.85 +/- 0.09; n = 10) and reached a maximal relaxation similar to that of theophylline (3 mM). The potency (-log EC50 values) of SCA40 compared to other relaxants was rolipram (7.44 +/- 0.12; n = 9) > SCA40 > or = levcromakalim (6.49 +/- 0.04; n = 6) > SKF94120 (5.87 +/- 0.10; n = 9). 2. When tested against the activity of the isoenzymes of cyclic nucleotide phosphodiesterase (PDE) isolated from human bronchus, SCA40 proved highly potent against PDE III (-log IC50 = 6.47 +/- 0.16; n = 4). It was markedly less potent against PDE IV (4.82 +/- 0.18; n = 4) and …

Cromakalimmedicine.medical_specialtyCardiotonic AgentsNeutrophilsLeukotriene B4Muscle Relaxationchemistry.chemical_elementBronchiIn Vitro TechniquesCalciumPharmacologyLeukotriene B4chemistry.chemical_compound3'5'-Cyclic-GMP PhosphodiesterasesSuperoxidesInternal medicinemedicineHumansBenzopyransPyrrolesRolipramCyclic Nucleotide Phosphodiesterases Type 5PharmacologyCyclic nucleotide phosphodiesterasePhosphoric Diester HydrolasesSuperoxideAnti-Inflammatory Agents Non-SteroidalElastaseImidazolesN-Formylmethionine leucyl-phenylalanineCyclic Nucleotide Phosphodiesterases Type 3PyrrolidinonesBronchodilator AgentsCyclic Nucleotide Phosphodiesterases Type 4N-Formylmethionine Leucyl-PhenylalanineEndocrinologychemistry3'5'-Cyclic-AMP PhosphodiesterasesPyrazinesCalciumLeukocyte ElastaseRolipramCromakalimResearch Articlemedicine.drugBritish Journal of Pharmacology
researchProduct

Circulating cell-free DNA: an up-coming molecular marker in exercise physiology.

2012

The phenomenon of circulating cell-free DNA (cfDNA) concentrations is of importance for many biomedical disciplines including the field of exercise physiology. Increases of cfDNA due to exercise are described to be a potential hallmark for the overtraining syndrome and might be related to, or trigger adaptations of, immune function induced by strenuous exercise. At the same time, exercise provides a practicable model for studying the phenomenon of cfDNA that is described to be of pathophysiological relevance for different topics in clinical medicine like autoimmune diseases and cancer. In this review, we are summarizing the current knowledge of exercise-based acute and chronic alterations i…

Cumulative Trauma DisordersPhysiologyPhysical Therapy Sports Therapy and RehabilitationInflammationApoptosisRunningEndurance trainingmedicineHumansOrthopedics and Sports MedicineExercise physiologyExercisebiologyOvertrainingbusiness.industryResistance TrainingNeutrophil extracellular trapsDNAmedicine.diseaseCirculating Cell-Free DNAbiology.proteinExercise TestPhysical EnduranceCreatine kinasemedicine.symptombusinessBiomarkersBlood samplingSports medicine (Auckland, N.Z.)
researchProduct