Search results for "Neovascularization"

showing 10 items of 351 documents

Exposure to radial extracorporeal shockwaves induces muscle regeneration after muscle injury in a surgical rat model

2019

The leading cause of training interruption in sport is a muscle injury, for which the standard treatment is nonsteroidal anti-inflammatory drugs (NSAIDs). To find alternative treatments, we investigated whether the radial extracorporeal shockwave application (rESWT) could stimulate muscle regeneration. A lesion with complete rupture (grade III muscle tear) was set in the musculus rectus femoris of 12-week-old Wistar rats, and the NSAID diclofenac, rESWT, or a combined therapy were applied on day 0, 3, and 5 directly following the surgery. Rats were euthanized at 2, 4, and 7 days after surgery and the area of muscle lesion was excised for histological and gene expression analysis to determin…

Extracorporeal Shockwave TherapyMalemedicine.medical_specialty0206 medical engineeringMuscle Fibers SkeletalUrologyH&E stain610 MedizinNeovascularization Physiologic02 engineering and technologyMyoDExtracorporealLesion03 medical and health sciences0302 clinical medicineDiclofenacDownregulation and upregulation610 Medical sciencesMyosinmedicineAnimalsPaired Box Transcription FactorsRegenerationOrthopedics and Sports MedicineRats WistarMuscle SkeletalMyoD Protein030203 arthritis & rheumatologyWound Healingbusiness.industryStandard treatmentAnti-Inflammatory Agents Non-Steroidal020601 biomedical engineeringRatsAthletic Injuriesmedicine.symptombusinessmedicine.drug
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Small-sized granules of biphasic bone substitutes support fast implant bed vascularization

2015

The present study investigated the influence of granule size of 2 biphasic bone substitutes (BoneCeramic® 400-700 μm and 500-1000 μm) on the induction of multinucleated giant cells (MNGCs) and implant bed vascularization in a subcutaneous implantation model in rats. Furthermore, degradation mechanisms and particle phagocytosis of both materials were examined by transmission electron microscopy (TEM). Both granule types induced tissue reactions involving primarily mononuclear cells and only small numbers of MNGCs. Higher numbers of MNGCs were detected in the group with small granules starting on day 30, while higher vascularization was observed only at day 10 in this group. TEM analysis reve…

Foreign-body giant cellPathologymedicine.medical_specialtyMaterials sciencebiphasic bone substitutePhagocytosisBiomedical EngineeringNeovascularization PhysiologicMedicine (miscellaneous)Giant CellsPeripheral blood mononuclear cellBone and BonesBiomaterialsMiceMultinucleatevascularizationMaterials TestingmedicineAnimalsParticle SizedegradationGranule (cell biology)granule sizephagocytosisGeneral MedicineRatsCell biologyDisease Models AnimalTransmission electron microscopyGiant cellBone SubstitutesLeukocytes MononuclearMicroscopy Electron ScanningBoneCeramicFemaleHydroxyapatitesImplantmultinucleated giant cellsResearch PaperBiomatter
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A BMP7 Variant Inhibits Tumor Angiogenesis In Vitro and In Vivo through Direct Modulation of Endothelial Cell Biology

2015

Bone morphogenetic proteins (BMPs), members of the TGF-β superfamily, have numerous biological activities including control of growth, differentiation, and vascular development. Using an in vitro co-culture endothelial cord formation assay, we investigated the role of a BMP7 variant (BMP7v) in VEGF, bFGF, and tumor-driven angiogenesis. BMP7v treatment led to disruption of neo-endothelial cord formation and regression of existing VEGF and bFGF cords in vitro. Using a series of tumor cell models capable of driving angiogenesis in vitro, BMP7v treatment completely blocked cord formation. Pre-treatment of endothelial cells with BMP7v significantly reduced their cord forming ability, indicating …

Genetics and Molecular Biology (all)MaleVascular Endothelial Growth Factor AFibroblast Growth FactorAngiogenesisBone Morphogenetic Protein 7Nudelcsh:MedicineSmad ProteinsFibroblast growth factorBiochemistryNeovascularizationMiceCell Movementlcsh:ScienceBMP7 Angiogenesis TumorTumorMultidisciplinaryCell DeathNeovascularization PathologicMedicine (all)Cell migrationCell biologyEndothelial stem cellSettore MED/26 - NEUROLOGIAVascular endothelial growth factor ADrug CombinationsAdipose TissueAdipose Tissue; Animals; Bone Morphogenetic Protein 7; Cell Death; Cell Line Tumor; Cell Movement; Cell Proliferation; Collagen; Drug Combinations; Endothelial Cells; Fibroblast Growth Factor 2; Glioblastoma; Human Umbilical Vein Endothelial Cells; Humans; Laminin; Male; Mice Nude; Neoplastic Stem Cells; Neovascularization Pathologic; Neovascularization Physiologic; Proteoglycans; Receptor Fibroblast Growth Factor Type 1; Signal Transduction; Smad Proteins; Vascular Endothelial Growth Factor A; Vascular Endothelial Growth Factor Receptor-2; Xenograft Model Antitumor Assays; Biochemistry Genetics and Molecular Biology (all); Agricultural and Biological Sciences (all)Neoplastic Stem CellsFibroblast Growth Factor 2ProteoglycansCollagenmedicine.symptomReceptorType 1Research ArticleSignal TransductionMice NudeNeovascularization PhysiologicBMP7BiologyCell LineSettore MED/04 - PATOLOGIA GENERALECell Line TumormedicineHuman Umbilical Vein Endothelial CellsAnimalsHumansAgricultural and Biological Sciences (all); Biochemistry Genetics and Molecular Biology (all); Medicine (all)Receptor Fibroblast Growth Factor Type 1PhysiologicNeovascularizationCell ProliferationPathologicMatrigelBiochemistry Genetics and Molecular Biology (all)lcsh:REndothelial CellsKinase insert domain receptorVascular Endothelial Growth Factor Receptor-2Xenograft Model Antitumor AssaysAgricultural and Biological Sciences (all)lcsh:QAngiogenesisLamininGlioblastomaPLoS ONE
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Mild Heat Stress Enhances Angiogenesis in a Co-culture System Consisting of Primary Human Osteoblasts and Outgrowth Endothelial Cells

2013

The repair and regeneration of large bone defects, including the formation of functional vasculature, represents a highly challenging task for tissue engineering and regenerative medicine. Recent studies have shown that vascularization and ossification can be stimulated by mild heat stress (MHS), which would offer the option to enhance the bone regeneration process by relatively simple means. However, the mechanisms of MHS-enhanced angiogenesis and osteogenesis, as well as potential risks for the treated cells are unclear. We have investigated the direct effect of MHS on angiogenesis and osteogenesis in a co-culture system of human outgrowth endothelial cells (OECs) and primary osteoblasts …

Hot TemperatureCell SurvivalAngiogenesisCellular differentiationBiomedical EngineeringNeovascularization PhysiologicMedicine (miscellaneous)ApoptosisBioengineeringBiologyRegenerative medicineArticleTissue engineeringOsteogenesisHeat shock proteinHumansRNA MessengerHeat shockBone regenerationCells CulturedCaspase 7OsteoblastsCaspase 3Regeneration (biology)Endothelial CellsCell DifferentiationCoculture TechniquesCapillariesUp-RegulationCell biologyImmunologyHeat-Shock ResponseTissue Engineering Part C: Methods
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Mast Cells Are Key Promoters of Contact Allergy that Mediate the Adjuvant Effects of Haptens

2011

SummaryA prominent feature of sensitizing environmental compounds that cause allergic contact dermatitis is the rapid induction of an innate inflammatory response that seems to provide danger signals for efficient T cell priming. We generated mouse models of mast cell deficiency, mast cell-specific gene inactivation, and mast cell reporter mice for intravital imaging and showed that these adjuvant effects of contact allergens are mediated by mast cells and histamine. Mast cell deficiency resulted in impaired emigration of skin DCs to the lymph node and contact hypersensitivity was dramatically reduced in the absence of mast cells. In addition, mast cell-specific inactivation of the Il10 gen…

ImmunologyMedizinPriming (immunology)BiologyMicechemistry.chemical_compoundImmune systemAdjuvants ImmunologicCell MovementmedicineAnimalsImmunology and AllergyMast CellsInterleukin 5Allergic contact dermatitisNeovascularization PathologicDendritic CellsHypertrophymedicine.diseaseMast cellImmunity InnateMice Inbred C57BLInterleukin 33Interleukin 10medicine.anatomical_structureInfectious DiseaseschemistryDermatitis Allergic ContactMutationImmunologyLymph NodesHaptensHistamineHistamineImmunity
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Cellular therapies for the endometrium: An update

2018

An update on the current state of endometrial cell therapies in terms of cell types, mechanisms of action, delivery, safety, regulatory frameworks and future perspectives. This review focuses on clinical trials using angiogenesis-promoting therapies and stromal therapies piloted in the last 10 years for alleviating Asherman's syndrome and long-term infertility. All studies present promising preliminary results, indicating increased endometrial thickness and resumed menstruation. Further characterization of individual cell products, their mode of action and larger clinical trials will be essential to establishing cell therapy as a viable option for the treatment of infertility and fertility …

Infertilitybusiness.industryFemale infertilityCell- and Tissue-Based TherapyNeovascularization PhysiologicObstetrics and GynecologyAsherman's syndromeGynatresiaGeneral MedicineEndometriummedicine.diseaseBioinformaticsClinical trialCell therapyEndometriummedicine.anatomical_structuremedicineHumansFemaleFertility preservationbusinessMode of actionInfertility FemaleActa Obstetricia et Gynecologica Scandinavica
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Perlecan Maintains microvessel integrity in vivo and modulates their formation in vitro

2012

Perlecan is a heparan sulfate proteoglycan assembled into the vascular basement membranes (BMs) during vasculogenesis. In the present study we have investigated vessel formation in mice, teratomas and embryoid bodies (EBs) in the absence of perlecan. We found that perlecan was dispensable for blood vessel formation and maturation until embryonic day (E) 12.5. At later stages of development 40% of mutant embryos showed dilated microvessels in brain and skin, which ruptured and led to severe bleedings. Surprisingly, teratomas derived from perlecan-null ES cells showed efficient contribution of perlecan-deficient endothelial cells to an apparently normal tumor vasculature. However, in perlecan…

IntegrinsAnatomy and PhysiologyGlycobiologylcsh:MedicineCardiovascularurologic and male genital diseasesCardiovascular SystemBiochemistryBiotecnologiaBasement MembraneMicePregnancyMolecular Cell BiologyMorphogenesisHistochemistrylcsh:ScienceSkinMice KnockoutPeripheral Vascular DiseasesExtracellular Matrix ProteinsNeovascularization PathologicTeratomaProteïnes de membranaBrainCell DifferentiationExtracellular MatrixConnective TissueCytochemistryMedicineFemaleFibroblast Growth Factor 2ProteoglycansResearch Articleendocrine systemMice 129 StrainCèl·lulesNeovascularization PhysiologicCell MigrationGrowth FactorsCell AdhesionAnimalsBirth DefectsBiologyExtracellular Matrix AdhesionsEmbryoid BodiesEmbryonic Stem Cellslcsh:RfungiProteinsExtracellular Matrix CompositionMice Inbred C57BLcarbohydrates (lipids)Cancer and OncologyMicrovesselsCardiovascular Anatomylcsh:QHeparan Sulfate ProteoglycansDevelopmental Biology
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Endothelial cell-matrix interactions.

2002

Dynamic interactions between endothelial cells and components of their surrounding extracellular matrix are necessary for the invasion, migration, and survival of endothelial cells during angiogenesis. These interactions are mediated by matrix receptors that initiate intracellular signaling cascades in response to binding to specific extracellular matrix molecules. The interactions between endothelial cells and their environment are also modulated by enzymes that degrade different matrix components and thus enable endothelial invasion. Recent reports on gene targeting in mice have confirmed the role of two classes of matrix receptors, integrins and cell surface heparan sulfate proteoglycans…

IntegrinsHistologybiologyNeovascularization PathologicAngiogenesisIntegrinProteolytic enzymesNeovascularization PhysiologicMatrix (biology)Matrix metalloproteinaseMatrix MetalloproteinasesCell biologyExtracellular MatrixExtracellular matrixEndothelial stem cellFibronectinMedical Laboratory TechnologyMicebiology.proteinAnimalsHumansEndothelium VascularAnatomyInstrumentationHeparan Sulfate ProteoglycansMicroscopy research and technique
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Use of a collagen/elastin-membrane for the tissue engineering of dermis.

1999

In an experimental model in rats, xenogeneic membranes consisting of processed native collagen and elastin were grafted to serve as a template for the formation of a neo-dermis, while in vitro-cultured autogeneic keratinocytes were applied on top of this to restore an epidermis. The process of tissue reconstruction and the digestion of the grafted membrane components were analysed by histological and immunohistochemical methods as well as electron microscopy. Approximately 3 weeks post grafting the membranes were completely vascularised and colonized by different types of cells. After 6 weeks, the collagenous fibres of the graft were mostly replaced by newly formed collagenous texture, wher…

KeratinocytesMaleDermatologic Surgical ProceduresNeovascularization PhysiologicHuman skinCritical Care and Intensive Care MedicineDermisTissue engineeringMedicineAnimalsCells CulturedSkinUltrasonographyBasement membraneSkin Artificialbiologybusiness.industryRats Inbred StrainsGeneral MedicineAnatomyEpitheliumElastinRatsMicroscopy Electronmedicine.anatomical_structureMembraneEmergency Medicinebiology.proteinBiophysicsMicroscopy Electron ScanningSurgeryEpidermisCollagenbusinessElastinBurns : journal of the International Society for Burn Injuries
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Granulocyte–Macrophage Colony-Stimulating Factor Is Essential for Normal Wound Healing

2006

Granulocyte-macrophage colony-stimulating factor (GM-CSF) is a multipotent growth factor, which plays an important role during the process of wound healing. In clinical settings it has occasionally been employed in the treatment of cutaneous wounds of diverse etiologies. In a previous study, we have shown the positive influence of GM-CSF on full thickness excisional wounds in transgenic mice overexpressing GM-CSF in the basal layer of the epidermis. Direct GM-CSF action as well as indirect processes through the induction of secondary cytokines were proposed to contribute towards the beneficial effects. In this study, we analyzed the process of wound healing in transgenic mice overexpressing…

KeratinocytesMaleGenetically modified mousePathologymedicine.medical_specialtyPulmonary Fibrosismedicine.medical_treatmentNeovascularization PhysiologicMice TransgenicDermatologyNeovascularizationMiceBasal (phylogenetics)FibrosismedicineAnimalsMolecular BiologyCell ProliferationWound HealingEpidermis (botany)business.industryGrowth factorGranulocyte-Macrophage Colony-Stimulating FactorCell BiologyGeneral Medicinemedicine.diseaseGranulocyte macrophage colony-stimulating factorGranulation TissueCancer researchFemalemedicine.symptomWound healingbusinessBiotechnologymedicine.drugJournal of Investigative Dermatology Symposium Proceedings
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