Search results for "Nephrotoxicity"

showing 10 items of 29 documents

Impact of NADPH oxidase functional polymorphisms in acute myeloid leukemia induction chemotherapy.

2016

Efficacy and toxicity of anthracycline treatment in acute myeloid leukemia (AML) is mediated by reactive oxygen species (ROS). NADPH oxidase is the major endogenous source of ROS and a key mediator of oxidative cardiac damage. The impact of NADPH oxidase polymorphisms (CYBA:rs4673, NCF4:rs1883112, RAC2:rs13058338) was evaluated in 225 adult de novo AML patients. Variant alleles of NCF4 and RAC2 were related to higher complete remission (P=0.035, P=0.016), and CYBA homozygous variant showed lower overall survival with recessive model (P=0.045). Anthracycline-induced cardiotoxicity was associated to NCF4 homozygous variant (P=0.012) and CYBA heterozygous genotype (P=0.027). Novel associations…

0301 basic medicineMaleAnthracyclinePharmacologyBiologyPolymorphism Single NucleotideNephrotoxicity03 medical and health sciences0302 clinical medicineAntineoplastic Combined Chemotherapy ProtocolsGeneticsHumansAgedRetrospective StudiesPharmacologychemistry.chemical_classificationReactive oxygen speciesCardiotoxicityNADPH oxidaseRemission InductionMyeloid leukemiaNADPH OxidasesInduction ChemotherapyMiddle Agedrac GTP-Binding ProteinsRac GTP-Binding ProteinsLeukemia Myeloid Acute030104 developmental biologychemistry030220 oncology & carcinogenesisToxicitybiology.proteinMolecular MedicineFemaleReactive Oxygen SpeciesThe pharmacogenomics journal
researchProduct

Necrostatin-1 Attenuates Cisplatin-Induced Nephrotoxicity Through Suppression of Apoptosis and Oxidative Stress and Retains Klotho Expression

2018

Aim: Cisplatin is an effective chemotherapeutic drug, but the application in clinical is greatly limited by its nephrotoxicity. Necrostatin-1 (Nec-1), an inhibitor of RIP1 kinase, has been reported to inhibit RIP-mediated necroptosis. The aim of this study is to detect the protective effects of Nec-1 on the nephrotoxicity of cisplatin and to investigate its renoprotection mechanism.Methods: 8-week-old male C57BL/6 mice were randomly assigned into four groups: Control, Nec-1, Cisplatin, and Cisplatin+Nec-1. Mice were treated with cisplatin with or without Nec-1 pre-treatment. Renal function, histological changes, necroptosis, and apoptotic markers were investigated. NFκB pathway related prot…

0301 basic medicineNecroptosiscisplatinnecroptosisInflammationPharmacologymedicine.disease_causeKlothoNephrotoxicityProinflammatory cytokine03 medical and health sciences0302 clinical medicinemedicineoxidative stressPharmacology (medical)KlothoOriginal ResearchCisplatinPharmacologyKidneybusiness.industrylcsh:RM1-950apoptosisdigestive system diseases030104 developmental biologymedicine.anatomical_structurelcsh:Therapeutics. Pharmacologyinflammation030220 oncology & carcinogenesismedicine.symptomNecrostatin-1businessOxidative stressmedicine.drugFrontiers in Pharmacology
researchProduct

Cardiotoxicity and Cardioprotection by Artesunate in Larval Zebrafish

2019

Although artesunate (ART) is generally accepted as a safe and well-tolerated first-line treatment of severe malaria, cases of severe side effects and toxicity of this compound are also documented. This study applied larval zebrafishes to determine the acute toxicity and efficacy of ART and performed RNA-sequencing analyses to unravel the underlying signaling pathways contributing to ART’s activities. Results from acute toxicity assay showed that a single-dose intravenous injection of ART from 3.6 ng/fish (1/9 maximum nonlethal concentration) to 41.8 ng/fish (lethal dose 10%) obviously induced pericardial edema, circulation defects, yolk sac absorption delay, renal edema, and swim bladder l…

0301 basic medicinenatural productsHealth Toxicology and MutagenesisShort ReportmalariaDevelopmental toxicityPharmacologyToxicologyNephrotoxicity03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEdemamedicinecancernetwork pharmacologyCardiotoxicityChemical Health and Safetybusiness.industrylcsh:RM1-950Public Health Environmental and Occupational Healthmedicine.diseaseAcute toxicity030104 developmental biologylcsh:Therapeutics. PharmacologychemistryArtesunate030220 oncology & carcinogenesisHeart failureToxicitymedicine.symptombusinessDose-Response
researchProduct

Influence of polymorphisms in anthracyclines metabolism genes in the standard induction chemotherapy of acute myeloid leukemia

2021

Objectives Genetic variability in anthracycline metabolism could modify the response and safety of acute myeloid leukemia (AML) induction. Methods Polymorphisms in genes that encodes enzymes of anthracyclines metabolic pathway (CBR3: rs1056892, rs8133052, NQO1: rs1800566, NQO2: rs1143684, NOS3: rs1799983, rs2070744) were evaluated in 225 adult de novo AML patients. Results The variant CBR3 rs8133052 was associated with lower hepatotoxicity (P = 0.028). Wild-type genotype of NQO2 rs1143684 was related to higher complete remission (P = 0.014), and the variant allele with greater gastrointestinal toxicity (P = 0.024). However, the variant genotype of NQO1 rs1800566 was associated with mucositi…

Adult0301 basic medicineAnthracycline030226 pharmacology & pharmacyNephrotoxicity03 medical and health sciences0302 clinical medicineGenotypeGeneticsmedicineMucositisHumansIdarubicinAnthracyclinesGenetic variabilityGeneral Pharmacology Toxicology and PharmaceuticsMolecular BiologyAllelesGenetics (clinical)Polymorphism Geneticbusiness.industryInduction chemotherapyMyeloid leukemiaInduction Chemotherapymedicine.diseaseLeukemia Myeloid Acute030104 developmental biologyCancer researchMolecular Medicinebusinessmedicine.drugPharmacogenetics and Genomics
researchProduct

Prospective study of amphotericin B formulations in immunocompromised patients in 4 European countries

2005

Background. Amphotericin B is a widely used broad-spectrum antifungal agent, despite being associated with significant adverse events, including nephrotoxicity. Methods. The present prospective study collected data on outcomes for 418 adult patients treated consecutively with polyenes in hematology and oncology wards in 20 hospitals in Europe. Results. Patients initially received amphotericin B deoxycholate (62% of patients), liposomal amphotericin B (27%), or other lipid formulations of amphotericin B (11%). Of the patients initially treated with amphotericin B deoxycholate, 36% had therapy switched to lipid formulations of amphotericin B, primarily because of increased serum creatinine le…

AdultMaleMicrobiology (medical)medicine.medical_specialtyAntifungal AgentsRenal functionPolyenesGastroenterologyNephrotoxicityImmunocompromised HostAmphotericin BAmphotericin B deoxycholateInternal medicineAmphotericin BmedicineHumansProspective StudiesAdverse effectProspective cohort studyAgedHematologybusiness.industryMortality rateLength of StayMiddle AgedSurgeryEuropeInfectious DiseasesMycosesFemaleKidney Diseasesbusinessmedicine.drug
researchProduct

Two yr mycophenolate mofetil plus low-dose calcineurin inhibitor for renal dysfunction after liver transplant

2009

We assessed the efficacy and outcome of low through level of calcineurin inhibitors (CNI) and introducing mycophenolate mofetil (MMF) in liver transplant (LT) patients with CNI-related renal dysfunction. Thirty LT patients were converted to combined therapy and compared with 30 patients used as a contemporary control group receiving CNI only. The two groups were matched for sex, age, months after LT, immunosuppressive treatment, creatinine level, presence of diabetes and calculated glomerular filtration rate (GFR) via Cockroft-Gault method. After two years, in the MMF serum creatinine decreased from 1.65 mg/dL (range 1.33-3.5) to 1.4 mg/dL (range 0.9-4.7) (p = 0.002) and GFR increased from …

AdultMalemedicine.medical_specialtyUrinary systemmedicine.medical_treatmentCalcineurin InhibitorsUrologyRENAL DYSFUNCTIONRenal functionCALCINEURIN INHIBITORS; IMMUNOSUPPRESSION; LIVER TRANSPLANTATION; MYCOPHENOLATE MOFETIL; RENAL DYSFUNCTIONLiver transplantationKidney Function TestsTacrolimusMycophenolic acidNephrotoxicitychemistry.chemical_compoundmedicineHumansMYCOPHENOLATE MOFETILAgedTransplantationCreatinineIMMUNOSUPPRESSIONDose-Response Relationship Drugbusiness.industryGraft SurvivalMiddle AgedMycophenolic AcidTacrolimusLiver TransplantationCalcineurinliver transplantTreatment OutcomechemistryCreatinineImmunologyKidney Failure ChronicDrug Therapy CombinationFemalebusinessImmunosuppressive AgentsGlomerular Filtration Ratemedicine.drug
researchProduct

Effectiveness and safety of once-daily aminoglycosides: A meta-analysis

1996

The effectiveness and safety of once-daily versus several-times-daily aminoglycosides were studied in a meta-analysis. MEDLINE for 1988 to 1995 was searched, and additional studies were identified from review articles and references in retrieved articles. Studies selected for meta-analysis were randomized controlled clinical trials in nonneutropenic adult patients comparing the clinical effectiveness or nephrotoxicity or ototoxicity of once-daily with several-times-daily aminoglycosides. Differences between groups were expressed as odds ratios. The results were combined by the procedure of Mantel and Haenszel, and 95% confidence intervals and exact confidence intervals were computed. An odd…

Adultmedicine.medical_specialtyMEDLINEInfectionsDrug Administration ScheduleNephrotoxicityOtotoxicityInternal medicinemedicineHumansNetilmicinEar DiseasesAmikacinAgedAntibacterial agentPharmacologybusiness.industryHealth PolicyOdds ratioMiddle Agedmedicine.diseaseConfidence intervalAnti-Bacterial AgentsSurgeryClinical trialMeta-analysisKidney DiseasesControlled Clinical Trials as TopicGentamicinsbusinessAmerican Journal of Health-System Pharmacy
researchProduct

Nephrotoxicity in the setting of invasive fungal diseases

2008

Amphotericin B, a broad spectrum antifungal agent, is widely used despite significant adverse events including nephrotoxicity. Nephrotoxicity occurs frequently in patients receiving amphotericin B. Different definitions for nephrotoxicity are reviewed in the context of outcome in patients with invasive fungal diseases. In most publications, mortality was higher in patients experiencing nephrotoxicity and mean hospital length of stay was prolonged. As a consequence, the use of less nephrotoxic antifungal agents could improve treatment outcomes.

Antifungalmedicine.medical_specialtyAntifungal Agentsmedicine.drug_classTreatment outcomeLength of hospitalizationContext (language use)DermatologyNephrotoxicityImmunocompromised HostAmphotericin BAmphotericin BmedicineHumansIn patientIntensive care medicineAdverse effectbusiness.industryGeneral MedicineLength of StayTreatment OutcomeInfectious DiseasesMycosesKidney Diseasesbusinessmedicine.drugMycoses
researchProduct

Regression Modeling of the Antioxidant-to-Nephroprotective Relation Shows the Pivotal Role of Oxidative Stress in Cisplatin Nephrotoxicity

2021

The clinical utility of the chemotherapeutic drug cisplatin is significantly limited by its nephrotoxicity, which is characterized by electrolytic disorders, glomerular filtration rate decline, and azotemia. These alterations are consequences of a primary tubulopathy causing injury to proximal and distal epithelial cells, and thus tubular dysfunction. Oxidative stress plays a role in cisplatin nephrotoxicity and cytotoxicity, but its relative contribution to overall toxicity remains unknown. We studied the relation between the degree of oxidative reduction (provided by antioxidant treatment) and the extent of nephrotoxicity amelioration (i.e., nephroprotection) by means of a regression anal…

AntioxidantPhysiologymedicine.medical_treatmentClinical BiochemistrycisplatinOxidative phosphorylationRM1-950Pharmacologymedicine.disease_causeBiochemistryArticleNephrotoxicitypreventionpreclinicalMedicineMolecular BiologyCisplatinlinear fitbusiness.industrynephrotoxicityCell Biologymedicine.diseaseantioxidantsErythropoietinToxicityAzotemiaTherapeutics. PharmacologybusinessOxidative stressmedicine.drugAntioxidants
researchProduct

Nephrotoxic and hepatotoxic effects of chromium compounds in rats

1986

The nephrotoxic, hepatotoxic and cardiotoxic actions of hexavalent chromium compounds, as well as their effects on lung, blood and circulation may contribute to the fatal outcome of chromium intoxication. Although trivalent chromium have been regarded as relatively biologically inert, there are a few salts of chromium III that have been found to be carcinogenic when inhaled, ingested or brought in contact with the tissues. Sensitive persons and industry workers have been subjects of dermatitis, respiratory tract injuries and digestive ulcers due to chromium compounds. In this work, the authors have studied the effect of trivalent and hexavalent chromium compounds on rats measuring the trans…

Chromiuminorganic chemicalsTime FactorsChromium CompoundsHealth Toxicology and Mutagenesischemistry.chemical_elementPharmacologyToxicologyNephrotoxicitychemistry.chemical_compoundChromiumotorhinolaryngologic diseasesAnimalsUreaAspartate AminotransferasesHexavalent chromiumCarcinogenCreatininetechnology industry and agricultureAlanine TransaminaseRats Inbred StrainsGeneral MedicinePollutionRatschemistryBiochemistryBlood chemistryCreatinineKidney DiseasesChromium toxicityChemical and Drug Induced Liver InjuryBulletin of Environmental Contamination and Toxicology
researchProduct