Search results for "Neurofilament"

showing 9 items of 59 documents

Implications of extreme serum neurofilament light chain levels for the management of patients with relapsing multiple sclerosis

2021

Background: Serum neurofilament light chain (sNfL) is a promising biomarker to complement the decision-making process in multiple sclerosis (MS) patients. However, although sNfL levels are able to detect disease activity and to predict future disability, the growing evidence has not yet been translated into practicable recommendations for an implementation into clinical routine. Methods: The observation of a patient with extensive inflammatory activity in magnetic resonance imaging (MRI) along with an extremely high sNfL level in the absence of any clinical symptoms prompted us to investigate common characteristics of our MS patients with the highest sNfL levels in a retrospective cohort st…

Pharmacologybusiness.industrysubclinical disease activityNeurofilament lightMultiple sclerosismultiple sclerosismedicine.diseaseextreme levels03 medical and health sciencesneurofilament light chain0302 clinical medicineNeurologyImmunologymedicinebiomarkerBiomarker (medicine)Neurology. Diseases of the nervous system030212 general & internal medicineNeurology (clinical)RC346-429business030217 neurology & neurosurgeryOriginal ResearchTherapeutic Advances in Neurological Disorders
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Neurons and ECM regulate occludin localization in brain endothelial cells

2000

We report that extracellular matrix and neurons modulate the expression of occludin, one of the main components of tight junctions, by rat brain endothelial cells (RBE4.B). Of the three extracellular matrix proteins which we tested (collagen I, collagen IV, and laminin), collagen IV stimulated at the best the expression of occludin mRNA. The corresponding protein, however, was not synthesized. Significant amounts of occludin accumulated only when RBE4.B cells were cultured on collagen IV-coated inserts, in the presence of cortical neurons, plated on laminin-coated companion wells. Finally, occludin segregated at the cell periphery, only when endothelial cells were co- cultured with neurons …

Time FactorsEndothelial cellsCellOccludinTight JunctionsExtracellular matrixRats Sprague-DawleyFetusLamininNeurofilament ProteinsOccludinSettore BIO/10 - BiochimicaGlial Fibrillary Acidic ProteinmedicineAnimalsRNA MessengerCells CulturedBlood-brain barrierNeuronsbiologyTight junctionGeneral NeuroscienceBrainMembrane ProteinsCortical NeuronsExtracellular matrixImmunohistochemistryCell biologyRatsEndothelial stem cellmedicine.anatomical_structureMembrane proteinCell cultureSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoCerebrovascular Circulationbiology.proteinSettore MED/26 - NeurologiaCollagenEndothelium VascularLamininNeuroscience
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De novo formation of cytokeratin filament networks originates from the cell cortex in A-431 cells

2001

Of the three major cytoskeletal filament systems, the intermediate filaments are the least understood. Since they differ fundamentally from the actin- and microtubulebased networks by their lack of polarity, it has remained a mystery how and where these principally endless filaments are formed. Using a recently established epithelial cell system in which fluorescently labeled intermediate filaments of the cytokeratin type can be monitored in living cells, we address these issues. By multidimensional time-lapse fluorescence microscopy, we examine de novo intermediate filament network formation from non-filamentous material at the end of mitosis and show that it mirrors disassembly. It is dem…

Time FactorsNeurofilamentGreen Fluorescent ProteinsMitosisArp2/3 complexmacromolecular substancesModels BiologicalCell LineProtein filamentStructural BiologyCell cortexTumor Cells CulturedHumansPhosphorylationCytoskeletonIntermediate filamentMicroscopy VideoDose-Response Relationship DrugbiologyCell BiologyCell biologyLuminescent ProteinsTreadmillingMicroscopy Fluorescencebiology.proteinKeratinsCell DivisionCytokinesisProtein BindingCell Motility and the Cytoskeleton
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NEURONS PRODUCE FGF-2 AND VEGF SECRETE THEM AT LEST IN PART BY SHEDDING EXTRACELLULAR VESCICLES

2007

Abstract We previously found that neurons are able to affect the ability of brain capillary endothelial cells to form in vitro a monolayer with properties resembling the blood-brain barrier. We then looked, by immunofluorescence and western analysis, for factors, produced by neurons, with the potential to influence growth and differentiation of endothelial cells. In the present paper, we report that neurons produce both vascular endothelial growth factor and fibroblast growth factor 2, two well-known angiogenic factors. More interestingly, we gained evidence that both factors are released by neurons, at least in part, by shedding of extracellular vesicles, that contain β1 integrin, a membra…

Vascular Endothelial Growth Factor AFGF-2BiologyFibroblast growth factorchemistry.chemical_compoundsheddingNeurofilament ProteinsGlial Fibrillary Acidic ProteinExtracellularAnimalsSecretionRats WistarCells CulturedNeuronsVesicleIntegrin beta1Secretory VesiclesCell BiologyArticlesVEGFTransport proteinCell biologyRatsVascular endothelial growth factorVascular endothelial growth factor AProtein TransportMembrane proteinchemistryAstrocytesMolecular Medicineneurons vesicles fibroblastic growth factor-2 vascular endothelial growth factorCamptothecinFibroblast Growth Factor 2Extracellular Spaceextracellular vesicles
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Transgenic overexpression of corticotropin releasing hormone provides partial protection against neurodegeneration in an in vivo model of acute excit…

2008

Abstract Corticotropin releasing hormone (CRH) is the central modulator of the mammalian hypothalamic–pituitary–adrenal (HPA) axis. In addition, CRH affects other processes in the brain including learning, memory, and synaptic plasticity. Moreover, CRH has been shown to play a role in nerve cell survival under apoptotic conditions and to serve as an endogenous neuroprotectant in vitro . Employing mice overexpressing murine CRH in the CNS, we observed a differential response of CRH-overexpressing mice (CRH-COE hom -Nes) to acute excitotoxic stress induced by kainate compared with controls (CRH-COE con -Nes). Interestingly, CRH-overexpression reduced the duration of epileptic seizures and pre…

endocrine systemmedicine.medical_specialtyIndolesRNA UntranslatedCorticotropin-Releasing HormoneExcitotoxicityMice TransgenicNerve Tissue ProteinsBiologymedicine.disease_causeNeuroprotectionHippocampusNestinCorticotropin-releasing hormoneMiceIntermediate Filament ProteinsNeurotrophic factorsNeurofilament ProteinsSeizuresInternal medicineGlial Fibrillary Acidic Proteinpolycyclic compoundsmedicineExcitatory Amino Acid AgonistsReaction TimeAnimalsNeuroinflammationBrain-derived neurotrophic factorAnalysis of VarianceKainic AcidCell DeathGeneral NeuroscienceBrain-Derived Neurotrophic FactorNeurodegenerationProteinsLong-term potentiationmedicine.diseaseDisease Models AnimalEndocrinologynervous systemGene Expression RegulationNerve DegenerationNeurotoxicity SyndromesPlant Lectinshormones hormone substitutes and hormone antagonistsNeuroscience
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Association of serum neurofilament light chain levels and neuropsychiatric manifestations in systemic lupus erythematosus

2021

Background: The aim was to evaluate the diagnostic potential of serum neurofilament light chain (sNfL) measurements in patients with neuropsychiatric systemic lupus erythematosus (NPSLE). Methods: sNfL levels were determined by single molecule array assay in a retrospective cross-sectional cohort of 144 patients with systemic lupus erythematosus (SLE). After log-transformation of sNfL levels, mean sNfL levels were compared between NPSLE patients and SLE patients without neuropsychiatric disease using Student’s t test. Furthermore, the association of different neuropsychiatric manifestations with sNfL levels was assessed using a one-way analysis of variance (ANOVA) with post hoc analysis. As…

medicine.medical_specialtyNeurofilament lightCentral nervous systemGastroenterologychemistry.chemical_compoundInternal medicinePost-hoc analysismedicineRC346-429Original ResearchPharmacologyCreatininebusiness.industrymedicine.anatomical_structureneurofilament light chainNeurologychemistryCohortBiomarker (medicine)biomarkerMultiple linear regression analysisNeurology (clinical)Neurology. Diseases of the nervous systemCNSbusinessNeuropsychiatric diseaseneurolupusNPSLETherapeutic Advances in Neurological Disorders
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Molecular anatomy of the neuro-immune connection.

1991

Light microscopic immunohistochemistry was employed to elucidate and compare the presence, distribution, and coexistence of various peptides, neuroendocrine markers and enzymes of the catecholamine pathway in nerves supplying lymphoid tissues in a variety of mammalian species. All lymphoid organs and tissues receive innervation by fibers containing dopamine-beta-hydroxylase and/or tyrosine hydroxylase, neural markers like protein gene product 9.5, synaptophysin and neurofilament and a varied spectrum of peptides. The prominent peptides were tachykinins (substance P, neurokinin A), calcitonin gene-related peptide (CGRP), neuropeptide Y (NPY), and vasoactive intestinal polypeptide/peptide his…

medicine.medical_specialtyNeurofilamentLymphoid TissueNeuroimmunomodulationSwineVasoactive intestinal peptideGuinea PigsPalatine TonsilFluorescent Antibody TechniqueSubstance PThymus GlandCalcitonin gene-related peptidechemistry.chemical_compoundMiceDogsBone MarrowInternal medicineNeural PathwaysmedicineAnimalsHumansNeuronsSheepTyrosine hydroxylasebiologyChemistryGeneral NeuroscienceGeneral MedicineAnatomyNeuropeptide Y receptorImmunohistochemistryRatsEndocrinologySynaptophysinbiology.proteinCatsNeurokinin ALymph NodesPeptidesSpleenThe International journal of neuroscience
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Combined pre- and postnatal ethanol exposure in rats disturbs the myelination of optic axons.

2011

Aims: To analyse myelination and outgrowth of the optic axons in relation to the neuro-ophthalmological manifestations of ethanol (EtOH) abuse during pregnancy. Methods: An experimental model of chronic EtOH exposure was developed in rats and their offspring by subjecting the dams to a liquid diet (35% of the daily total calories as either EtOH or maltose-dextrose nutritional controls (Con). Eyeballs and optic nerves were obtained at key developmental stages and processed for morphologic, immunocytochemical and immunoblotting procedures, using alternatively antibodies against myelin basic protein (MBP) or neurofilament (NF) protein, and image analysing. Results: A significant delay in onset…

medicine.medical_specialtyNeurofilamentTime Factorsgenetic structuresOffspringEyeRetinaMyelinchemistry.chemical_compoundNeurofilament ProteinsPregnancyInternal medicinemedicineAnimalsAxonRats WistarMyelin SheathRetinabiologyEthanolChemistryBody WeightCentral Nervous System DepressantsRetinalMyelin Basic ProteinOptic NerveGeneral MedicineAnatomyeye diseasesAxonsMyelin basic proteinRatsDisease Models Animalmedicine.anatomical_structureEndocrinologynervous systemAnimals NewbornPrenatal Exposure Delayed Effectsbiology.proteinOptic nerveFemaleAlcohol and alcoholism (Oxford, Oxfordshire)
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Aging-associated changes in NF-kappa Β signaling

2000

neurofilamenttiproteiinitcell agingtranscription factorsagingapoptosisNF-kappa ΒvanheneminenI-kappa Βgenessolubiologia
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