Search results for "Neurogenesi"

showing 10 items of 336 documents

Malformations of Cortical Development and Neocortical Focus

2014

Abstract Developmental neocortical malformations resulting from abnormal neurogenesis, disturbances in programmed cell death, or neuronal migration disorders may cause a long-term hyperexcitability. Early generated Cajal–Retzius and subplate neurons play important roles in transient cortical circuits, and structural/functional disorders in early cortical development may induce persistent network disturbances and epileptic disorders. In particular, depolarizing GABAergic responses are important for the regulation of neurodevelopmental events, like neurogenesis or migration, while pathophysiological alterations in chloride homeostasis may cause epileptic activity. Although modern imaging tech…

LesionElectrophysiologymedicine.anatomical_structureFocus (geometry)Cerebral cortexSubplateNeurogenesismedicineGABAergicEpilepsy surgeryBiologymedicine.symptomNeuroscience
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On the roles of Notch, Delta, kuzbanian, and inscuteable during the development of Drosophila embryonic neuroblast lineages

2009

AbstractThe generation of cellular diversity in the nervous system involves the mechanism of asymmetric cell division. Besides an array of molecules, including the Par protein cassette, a heterotrimeric G protein signalling complex, Inscuteable plays a major role in controlling asymmetric cell division, which ultimately leads to differential activation of the Notch signalling pathway and correct specification of the two daughter cells. In this context, Notch is required to be active in one sibling and inactive in the other. Here, we investigated the requirement of genes previously known to play key roles in sibling cell fate specification such as members of the Notch signalling pathway, e.g…

Lineage (genetic)Embryo NonmammalianNotchCell divisionCell fate specificationDisintegrinsNeurogenesisContext (language use)BiologyCell fate determinationPolymerase Chain Reaction03 medical and health sciences0302 clinical medicineNeuroblastAsymmetric cell divisionAnimalsDrosophila ProteinsCell LineageMolecular Biology030304 developmental biologyDNA PrimersGeneticsNeurons0303 health sciencesBase SequenceReceptors NotchNeurogenesisIntracellular Signaling Peptides and ProteinsMembrane ProteinsMetalloendopeptidasesCell BiologyEmbryonic stem cellImmunohistochemistryCytoskeletal ProteinsAsymmetric cell divisionDrosophilakuzbanian030217 neurology & neurosurgerySignal TransductionDevelopmental BiologyDevelopmental Biology
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Subventricular zone neural progenitors protect striatal neurons from glutamatergic excitotoxicity.

2012

The functional significance of adult neural stem and progenitor cells in hippocampal-dependent learning and memory has been well documented. Although adult neural stem and progenitor cells in the subventricular zone are known to migrate to, maintain and reorganize the olfactory bulb, it is less clear whether they are functionally required for other processes. Using a conditional transgenic mouse model, selective ablation of adult neural stem and progenitor cells in the subventricular zone induced a dramatic increase in morbidity and mortality of central nervous system disorders characterized by excitotoxicity-induced cell death accompanied by reactive inflammation, such as 4-aminopyridine-i…

LipopolysaccharidesPolyunsaturated AlkamidesSubventricular zoneGlutamic AcidMice TransgenicArachidonic AcidsBiologyAmidohydrolasesGlutamatergicMiceNeural Stem CellsLateral VentriclesmedicineAnimalsDronabinolProgenitor cell4-Aminopyridineneurogenesis; ischaemia; neural stem cells; excitotoxicity; endocannabinoidsGanciclovirEpilepsyStem CellsNeurogenesisExcitatory Postsynaptic PotentialsNeural stem cellCorpus StriatumNeuroepithelial cellMice Inbred C57BLStrokeneurogenesisDisease Models Animalmedicine.anatomical_structureNeuroprotective AgentsBenzamidesSettore MED/26 - NeurologiaNeurology (clinical)ischaemiaCarbamatesStem cellNeuroscienceexcitotoxicityExcitatory Amino Acid AntagonistsAdult stem cellEndocannabinoidsBrain : a journal of neurology
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Neural stem cell lineage-specific cannabinoid type-1 receptor regulates neurogenesis and plasticity in the adult mouse hippocampus

2018

Abstract Neural stem cells (NSCs) in the adult mouse hippocampus occur in a specific neurogenic niche, where a multitude of extracellular signaling molecules converges to regulate NSC proliferation as well as fate and functional integration. However, the underlying mechanisms how NSCs react to extrinsic signals and convert them to intracellular responses still remains elusive. NSCs contain a functional endocannabinoid system, including the cannabinoid type-1 receptor (CB1). To decipher whether CB1 regulates adult neurogenesis directly or indirectly in vivo, we performed NSC-specific conditional inactivation of CB1 by using triple-transgenic mice. Here, we show that lack of CB1 in NSCs is su…

Male0301 basic medicineCell signalingCannabinoid receptorNeurogenesisCognitive NeuroscienceLong-Term PotentiationMice Transgenicmouse hippocampus ; neural stem cells ; neurogenesis-dependent behavior ; CB1 ; adult neurogenesisHippocampal formationBiologyHippocampus03 medical and health sciencesCellular and Molecular Neurosciencemouse hippocampus0302 clinical medicineNeural Stem CellsReceptor Cannabinoid CB1Animalsreproductive and urinary physiologySpatial MemoryBehavior AnimalNeurogenesisLong-term potentiationOriginal ArticlesCB1Endocannabinoid systemneurogenesis-dependent behaviorNeural stem cellCell biologyadult neurogenesisMice Inbred C57BL030104 developmental biologynervous systemlipids (amino acids peptides and proteins)biological phenomena cell phenomena and immunityStem cell030217 neurology & neurosurgeryCerebral Cortex
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Neurovascular EGFL7 regulates adult neurogenesis in the subventricular zone and thereby affects olfactory perception

2016

Adult neural stem cells reside in a specialized niche in the subventricular zone (SVZ). Throughout life they give rise to adult-born neurons in the olfactory bulb (OB), thus contributing to neural plasticity and pattern discrimination. Here, we show that the neurovascular protein EGFL7 is secreted by endothelial cells and neural stem cells (NSCs) of the SVZ to shape the vascular stem-cell niche. Loss of EGFL7 causes an accumulation of activated NSCs, which display enhanced activity and re-entry into the cell cycle. EGFL7 pushes activated NSCs towards quiescence and neuronal progeny towards differentiation. This is achieved by promoting Dll4-induced Notch signalling at the blood vessel-stem …

Male0301 basic medicineGeneral Physics and AstronomyNEURAL STEM-CELLSMOUSEMiceSUBEPENDYMAL ZONENeural Stem CellsLateral VentriclesLINEAGE PROGRESSIONBRAININ-VIVOMice KnockoutNeuronal PlasticityMultidisciplinaryCell CycleQNeurogenesisNICHEAnatomyNeural stem cellCell biologyAdult Stem Cellsmedicine.anatomical_structureSignal TransductionSTIMULATES NEUROGENESISEGF Family of ProteinsNeurogenesisScienceNotch signaling pathwaySubventricular zoneBiologyInhibitory postsynaptic potentialArticleGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesNeuroplasticitymedicineBiological neural networkAnimalsCalcium-Binding ProteinsProteinsGeneral ChemistryOlfactory PerceptionENDOTHELIAL-CELLSnervous system diseasesOlfactory bulbMice Inbred C57BLSELF-RENEWAL030104 developmental biologynervous system
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Protective effects of mirtazapine in mice lacking the Mbnl2 gene in forebrain glutamatergic neurons: Relevance for myotonic dystrophy 1

2019

Myotonic dystrophy type 1 (DM1) is a multisystemic disorder characterized by muscle weakness and wasting and by important central nervous system-related symptoms including impairments in executive functions, spatial abilities and increased anxiety and depression. The Mbnl2 gene has been implicated in several phenotypes consistent with DM1 neuropathology. In this study, we developed a tissue-specific knockout mouse model lacking the Mbnl2 gene in forebrain glutamatergic neurons to examine its specific contribution to the neurobiological perturbations related to DM1. We found that these mice exhibit long-term cognitive deficits and a depressive-like state associated with neuronal loss, increa…

Male0301 basic medicineMirtazapineGlutamic AcidHippocampusMice TransgenicMirtazapineMyotonic dystrophyAnimals Genetically ModifiedMice03 medical and health sciencesCellular and Molecular NeuroscienceGlutamatergicProsencephalon0302 clinical medicinemedicineAnimalsMyotonic DystrophyDentate gyrusInflammationMice KnockoutNeuronsPharmacologyDepressionbusiness.industryCognitive deficitsDentate gyrusNeurogenesisRNA-Binding Proteinsmedicine.disease3. Good healthMice Inbred C57BLNeuroprotective Agents030104 developmental biologynervous systemKnockout mouseForebrainNeuronal lossDrosophilaFemaleDM1businessNeuroscience030217 neurology & neurosurgerymedicine.drugNeuropharmacology
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Amelioration of the abnormal phenotype of a new L1 syndrome mouse mutation with L1 mimetics

2021

L1 syndrome is a rare developmental disorder characterized by hydrocephalus of varying severity, intellectual deficits, spasticity of the legs, and adducted thumbs. Therapy is limited to symptomatic relief. Numerous gene mutations in the L1 cell adhesion molecule (L1CAM, hereafter abbreviated L1) were identified in L1 syndrome patients, and those affecting the extracellular domain of this transmembrane type 1 glycoprotein show the most severe phenotypes. Previously analyzed rodent models of the L1 syndrome focused on L1-deficient animals or mouse mutants with abrogated cell surface expression of L1, making it difficult to test L1 function-triggering mimetic compounds with potential therapeu…

Male0301 basic medicineToluidinesL1NeurogenesisCellNeural Cell Adhesion Molecule L1Gene mutationBiologyDuloxetine Hydrochloridemedicine.disease_causeBiochemistryCerebral VentriclesCorpus CallosumMice03 medical and health sciences0302 clinical medicineCerebellumIntellectual DisabilityGeneticsmedicineExtracellularAnimalsL1 syndromeMolecular BiologyCells CulturedNeuronsMutationSpastic Paraplegia HereditaryTrimebutineGenetic Diseases X-LinkedCell migrationSymptomatic reliefMice Inbred C57BLPhenotype030104 developmental biologymedicine.anatomical_structureMutationCancer researchPeptidomimeticsLocomotion030217 neurology & neurosurgeryBiotechnologyThe FASEB Journal
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New neurons use Slit-Robo signaling to migrate through the glial meshwork and approach a lesion for functional regeneration

2018

Appropriate positioning of new neurons in the brain promotes post-stroke functional recovery.

Male0301 basic medicineanimal structuresNeurogenesisBiologyLesionMice03 medical and health sciences0302 clinical medicineDevelopmental NeuroscienceNeuroblastCell MovementNeuroblast migrationmental disordersmedicineAnimalsRegenerationcardiovascular diseasesReceptors ImmunologicProgenitor cellcdc42 GTP-Binding ProteinResearch Articlesreproductive and urinary physiologyMice KnockoutNeuronsMultidisciplinaryRegeneration (biology)fungiNeurogenesisSciAdv r-articlesBrainActin cytoskeletonSlit-RoboActin Cytoskeleton030104 developmental biologynervous systemCellular NeuroscienceAstrocytesembryonic structuresIntercellular Signaling Peptides and ProteinsProtein Multimerizationmedicine.symptomNeurogliaNeurosciencepsychological phenomena and processes030217 neurology & neurosurgeryResearch ArticleProtein BindingSignal TransductionScience Advances
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A Periodic Diet that Mimics Fasting Promotes Multi-System Regeneration, Enhanced Cognitive Performance, and Healthspan

2015

SummaryProlonged fasting (PF) promotes stress resistance, but its effects on longevity are poorly understood. We show that alternating PF and nutrient-rich medium extended yeast lifespan independently of established pro-longevity genes. In mice, 4 days of a diet that mimics fasting (FMD), developed to minimize the burden of PF, decreased the size of multiple organs/systems, an effect followed upon re-feeding by an elevated number of progenitor and stem cells and regeneration. Bi-monthly FMD cycles started at middle age extended longevity, lowered visceral fat, reduced cancer incidence and skin lesions, rejuvenated the immune system, and retarded bone mineral density loss. In old mice, FMD c…

MaleAbdominal Fat; Adult; Aged; Aging; Animals; Body Weight; Cardiovascular Diseases; Diet; Female; Humans; Male; Mice; Mice Inbred C57BL; Middle Aged; Neoplasms; Neurogenesis; Pilot Projects; Psychomotor Performance; Regeneration; Saccharomyces cerevisiae; Young Adult; Cognition; Fasting; LongevityAgingPhysiologyPilot ProjectsMiceCognitionNeoplasmsCardiovascular DiseaseSettore MED/49 - Scienze Tecniche Dietetiche Applicatemedia_common2. Zero hungerNeurogenesisLongevityFastingMiddle Aged3. Good healthCardiovascular DiseasesFemaleStem cellHumanAdultmedicine.medical_specialtyNeurogenesismedia_common.quotation_subjectLongevityAbdominal FatSaccharomyces cerevisiaeBiologyArticleYoung AdultImmune systemInternal medicineDiabetes mellitusmedicineAnimalsHumansRegenerationPilot ProjectAdverse effectCell Biology; Molecular Biology; PhysiologyMolecular BiologyAgedAnimalBody WeightCell Biologymedicine.diseaseMiddle ageDietMice Inbred C57BLEndocrinologyCancer cellNeoplasmNeurogenesiPsychomotor Performance
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Influences of prenatal and postnatal stress on adult hippocampal neurogenesis: The double neurogenic niche hypothesis

2015

International audience; Adult hippocampal neurogenesis (AHN) is involved in learning, memory, and stress, and plays a significant role in neurodegenerative and psychiatric disorders. As an age-dependent process, AHN is largely influenced by changes that occur during the pre- and postnatal stages of brain development, and constitutes an important field of research. This review examines the current knowledge regarding the regulators of AHN and the influence of prenatal and postnatal stress on later AHN. In addition, a hypothesis is presented suggesting that each kind of stress influences a specific neurogenic pool, developmental or postnatal, that later becomes a precursor with important repe…

MaleAgingBrain developmentprogenitor cellNeurogenesisNicheAdult hipocampal neurogenesis (AHN)neural stem-cellHippocampal formationgrowth-factorHippocampusHypothalamic-pituitary-adrenal (HPA)03 medical and health sciencesBehavioral Neuroscience0302 clinical medicinePregnancyRisk FactorsPrecursor cellPostnatal stressAnimalsHumanspattern separation030304 developmental biologyCell Proliferationrat dentate gyrus0303 health sciencesMental DisordersNeurogenesisStressorsubventricular zoneCell DifferentiationPrecursor cellsPostnatal stress (PTS)Neurogenic poolgenetic influencePrenatal Exposure Delayed Effectsolfactory-bulbPrenatal stress (PS)Female[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]decreases neurogenesisPsychologyNeuroscience030217 neurology & neurosurgeryStress Psychologicalbrain neurogenesis
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