Search results for "Neuron"

showing 10 items of 2611 documents

Polysialic Acid Acute Depletion Induces Structural Plasticity in Interneurons and Impairs the Excitation/Inhibition Balance in Medial Prefrontal Cort…

2016

The structure and function of the medial prefrontal cortex (mPFC) is affected in several neuropsychiatric disorders, including schizophrenia and major depression. Recent studies suggest that imbalances between excitatory and inhibitory activity (E/I) may be responsible for this cortical dysfunction and, therefore, may underlie the core symptoms of these diseases. This E/I imbalance seems to be correlated with alterations in the plasticity of interneurons but there is still scarce information on the mechanisms that may link these phenomena. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) is a good candidate, because it modulates the neuronal plasticity of interneurons…

0301 basic medicineGenetically modified mousePSA-NCAMneuronal structural plasticityInhibitory postsynaptic potential03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineE/I balanceNeuroplasticitymedicinePrefrontal cortexOriginal ResearchPolysialic acidmusculoskeletal neural and ocular physiologymedicine.diseaseschizophreniamPFC cultures030104 developmental biologynervous systemSchizophreniaExcitatory postsynaptic potentialNeural cell adhesion moleculemajor depressionPsychologyNeuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Cellular Neuroscience
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Piriform cortex alterations in the Ts65Dn model for down syndrome

2020

The piriform cortex is involved in olfactory information processing, that is altered in Down Syndrome. Moreover, piriform cortex has a crucial involvement in epilepsy generation and is one of the first regions affected in Alzheimer's Disease, both maladies being prevalent among Down Syndrome individuals. In this work, we studied the alterations in neuronal morphology, synaptology and structural plasticity in the piriform cortex of the Ts65Dn mouse model, which is the most used model for the study of this syndrome and mimics some of their alterations. We have observed that Ts65Dn piriform cortex displays: a reduction in dendritic arborisation, a higher density of inhibitory synapses (GAD67),…

0301 basic medicineGlutamate decarboxylasePresynaptic TerminalsMice TransgenicPiriform CortexInhibitory postsynaptic potentialMice03 medical and health sciences0302 clinical medicineAtrophyPostsynaptic potentialPiriform cortexmedicineNeuropilAnimalsMolecular BiologyNeuronsGephyrinbiologyGlutamate DecarboxylaseGeneral NeuroscienceMembrane Proteinsmedicine.disease030104 developmental biologymedicine.anatomical_structurenervous systemVesicular Glutamate Transport Protein 1biology.proteinExcitatory postsynaptic potentialNeurology (clinical)Down SyndromeNeuroscience030217 neurology & neurosurgeryDevelopmental BiologyBrain Research
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Comparative Performance of Linear Multielectrode Probes and Single-Tip Electrodes for Intracortical Microstimulation and Single-Neuron Recording in M…

2017

Intracortical microstimulation (ICMS) is one of the most widely employed techniques for providing causal evidence of the relationship between neuronal activity and specific motor, perceptual, or even cognitive functions. In recent years, several new types of linear multielectrode silicon probes have been developed, allowing researchers to sample neuronal activity at different depths along the same cortical site simultaneously and with high spatial precision. Nevertheless, silicon multielectrode probes have been rarely employed for ICMS studies and, more importantly, it is unknown whether and to what extent they can be used for combined recording and stimulation experiments. Here, we address…

0301 basic medicineHand regionMaterials scienceCognitive NeuroscienceNeuroscience (miscellaneous)Acute recording; Chronic recording; Electrical stimulation; Macaque monkey; Silicon probes; Neuroscience (miscellaneous); Developmental Neuroscience; Cognitive Neuroscience; Cellular and Molecular NeuroscienceMacaquechronic recording03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineDevelopmental Neurosciencebiology.animalmedicinePremovement neuronal activitysilicon probeselectrical stimulationOriginal Researchbiologytechnology industry and agricultureequipment and suppliesmacaque monkeyMicroelectrode030104 developmental biologyIntracortical microstimulationmedicine.anatomical_structureElectrodeacute recordingNeuron030217 neurology & neurosurgeryBiomedical engineeringMotor cortexNeuroscienceFrontiers in systems neuroscience
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Tetrahydrocarbazoles decrease elevated SOCE in medium spiny neurons from transgenic YAC128 mice, a model of Huntington's disease

2017

AbstractHuntington's disease (HD) is a hereditary neurodegenerative disease caused by a polyglutamine expansion within the huntingtin (HTT) gene. One of the cellular functions that is dysregulated in HD is store-operated calcium entry (SOCE), a process in which the depletion of Ca2+ from the endoplasmic reticulum (ER) induces Ca2+ influx from the extracellular space. We detected an enhanced activity of SOC channels in medium spiny neurons (MSNs) from YAC128 mice, a transgenic model of HD, and investigated whether this could be reverted by tetrahydrocarbazoles. The compound 6-bromo-N-(2-phenylethyl)-2,3,4,9-tetrahydro-1H-carbazol-1-amine hydrochloride was indeed able to restore the disturbed…

0301 basic medicineHuntingtinTransgeneCarbazolesBiophysicsMice TransgenicBiologyEndoplasmic ReticulumMedium spiny neuronYAC128BiochemistryMice03 medical and health sciences0302 clinical medicineHuntington's diseaseTetrahydrocarbazolesmedicineAnimalsHomeostasisHuntingtinMolecular BiologyCells CulturedMembrane Potential MitochondrialNeuronsSOC channelsMedium spiny neuronsIon TransportEndoplasmic reticulumHuntington's diseaseStore-operated calcium entryCell Biologymedicine.diseaseStore-operated calcium entryCulture MediaCell biology030104 developmental biologyBiochemistryCalcium030217 neurology & neurosurgeryHomeostasisBiochemical and Biophysical Research Communications
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Boosting Action Observation and Motor Imagery to Promote Plasticity and Learning

2018

Neural Plasticity, 2018

0301 basic medicineImagery PsychotherapyBoosting (machine learning)Article SubjectComputer scienceMovementMachine learningcomputer.software_genrestimulationlcsh:RC321-57103 medical and health sciences0302 clinical medicineMotor imageryHumansLearninglcsh:Neurosciences. Biological psychiatry. NeuropsychiatryComputingMilieux_MISCELLANEOUSNeuronal Plasticitybusiness.industryBraincortexEditorial030104 developmental biologyNeurologyAction observationImagination[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neurology (clinical)Artificial intelligencebusinesscomputer030217 neurology & neurosurgery
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TLR4 participates in the transmission of ethanol-induced neuroinflammation via astrocyte-derived extracellular vesicles

2019

Background Current evidence indicates that extracellular vesicles (EVs) participate in intercellular signaling, and in the regulation and amplification of neuroinflammation. We have previously shown that ethanol activates glial cells through Toll-like receptor 4 (TLR4) by triggering neuroinflammation. Here, we evaluate if ethanol and the TLR4 response change the release and inflammatory content of astrocyte-derived EVs, and whether these vesicles are capable of communicating with neurons by spreading neuroinflammation. Methods Cortical neurons and astrocytes in culture were used. EVs were isolated from the extracellular medium of the primary culture of the WT and TLR4-KO astrocytes treated …

0301 basic medicineImmunologyInflammationlcsh:RC346-42903 medical and health sciencesCellular and Molecular NeuroscienceMice0302 clinical medicineWestern blotNeuroinflammationGlial cellsExtracellularmedicineAnimalsProtein Interaction MapsReceptorNeuroinflammationCells Culturedlcsh:Neurology. Diseases of the nervous systemInflammationMice KnockoutNeuronsmedicine.diagnostic_testEthanolChemistryGeneral NeuroscienceResearchExtracellular vesiclesCell biologyMice Inbred C57BLToll-Like Receptor 4030104 developmental biologymedicine.anatomical_structureNeurologyAstrocytesTLR4medicine.symptom030217 neurology & neurosurgeryIntracellularAstrocyteJournal of Neuroinflammation
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In Situ, Light-Guided Axon Growth on Biomaterials via Photoactivatable Laminin Peptidomimetic IK(HANBP)VAV

2018

The ability to guide the growth of neurites is relevant for reconstructing neural networks and for nerve tissue regeneration. Here, a biofunctional hydrogel that allows light-based directional control of axon growth in situ is presented. The gel is covalently modified with a photoactivatable derivative of the short laminin peptidomimetic IKVAV. This adhesive peptide contains the photoremovable group 2-(4′-amino-4-nitro-[1,1′-biphenyl]-3-yl)propan-1-ol (HANBP) on the Lys rest that inhibits its activity. The modified peptide is highly soluble in water and can be simply conjugated to -COOH containing hydrogels via its terminal -NH 2 group. Light exposure allows presentation of the IKVAV adhesi…

0301 basic medicineIn situMaterials scienceNeuritePeptidomimeticNeuronal OutgrowthPeptideINGENIERÍAS Y TECNOLOGÍAS02 engineering and technologyBiotecnología Industrial03 medical and health sciencesMiceCoated Materials BiocompatibleNeural Stem CellsDIRECTIONAL NEURONAL GROWTHLamininIKVAVNeuritesAnimalsGeneral Materials Sciencechemistry.chemical_classificationbiologyPHOTO-TRIGGERED CELL ADHESIONBioproductos Biomateriales Bioplásticos Biocombustibles Bioderivados etc.Hydrogels021001 nanoscience & nanotechnologyNeural stem cellPeptide FragmentsLAMININ PEPTIDOMIMETICS030104 developmental biologychemistryCell cultureSelf-healing hydrogelsbiology.proteinBiophysicsLamininPeptidomimetics0210 nano-technologyACS Applied Materials & Interfaces
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Targeting RNA structure in SMN2 reverses spinal muscular atrophy molecular phenotypes

2018

Modification of SMN2 exon 7 (E7) splicing is a validated therapeutic strategy against spinal muscular atrophy (SMA). However, a target-based approach to identify small-molecule E7 splicing modifiers has not been attempted, which could reveal novel therapies with improved mechanistic insight. Here, we chose as a target the stem-loop RNA structure TSL2, which overlaps with the 5′ splicing site of E7. A small-molecule TSL2-binding compound, homocarbonyltopsentin (PK4C9), was identified that increases E7 splicing to therapeutic levels and rescues downstream molecular alterations in SMA cells. High-resolution NMR combined with molecular modelling revealed that PK4C9 binds to pentaloop conformati…

0301 basic medicineIndolesCOMPOUND LIBRARIESDrug Evaluation PreclinicalGeneral Physics and AstronomyBiotecnologiaAnimals Genetically ModifiedExonMolecular Targeted TherapyRegulatory Elements Transcriptionallcsh:ScienceHUMAN-DISEASE GENESBIOACTIVE SMALL MOLECULESMultidisciplinaryChemistryDrug discovery[CHIM.ORGA]Chemical Sciences/Organic chemistryQImidazolesMUTATION PATTERNExonsSMA*3. Good healthCell biologySurvival of Motor Neuron 2 ProteinPhenotypeCribratgeRNA splicingNUCLEOTIDE STRUCTUREDrosophilaMESSENGER-RNACOMPUTATIONAL TOOLSMedical screeningMYOTONIC-DYSTROPHYScienceMuscular atrophyArticleGeneral Biochemistry Genetics and Molecular BiologyGenètica molecularMuscular Atrophy Spinal03 medical and health sciencesddc:570SPLICING MODIFIERSmedicineAnimalsHumansHIV-1 TARRNA MessengerAtròfia muscularMessenger RNAAlternative splicingRNAGeneral ChemistrySpinal muscular atrophymedicine.diseaseAlternative Splicing030104 developmental biologyRNAlcsh:QRNA Splice SitesHeLa CellsNature Communications
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Chronic Stress Modulates Interneuronal Plasticity: Effects on PSA-NCAM and Perineuronal Nets in Cortical and Extracortical Regions.

2018

Chronic stress has an important impact on the adult brain. However, most of the knowledge on its effects is focused on principal neurons and less on inhibitory neurons. Consequently, recent reports have begun to describe stress-induced alterations in the structure, connectivity and neurochemistry of interneurons. Some of these changes appear to be mediated by certain molecules particularly associated to interneurons, such as the polysialylated form of the neural cell adhesion molecule (PSA-NCAM) and components of the perineuronal nets (PNN), specialized regions of the extracellular matrix. These plasticity-related molecules modulate interneuronal structure and connectivity, particularly of …

0301 basic medicineInterneuronPSA-NCAMhippocampusHippocampuslcsh:RC321-57103 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicinemedicineChronic stresslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal Researchchronic stressreticular thalamic nucleusThalamic reticular nucleusbiologyhabenulaPerineuronal netmusculoskeletal neural and ocular physiology030104 developmental biologymedicine.anatomical_structureHabenulanervous systembiology.proteinperineuronal netNeuroscience030217 neurology & neurosurgeryParvalbuminmedial prefrontal cortexbasolateral amygdalaBasolateral amygdalaNeuroscienceFrontiers in cellular neuroscience
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Phenotypic characterization of MCP-1 expressing neurons in the rat cerebral cortex.

2020

Chemokines are small, secreted molecules that mediate inflammatory reactions. Neurons and astrocytes constitutively express chemokines implicated in the process of neuroinflammation associated with neurodegenerative diseases. The monocyte chemoattractant protein-1 (MCP-1) has been widely related to this process. However, the constitutive expression of this molecule by neurons has not been elucidated so far. In this study, we set out to characterize the neurochemical phenotype of MCP-1-expressing neurons in the rat neocortex to infer its role in basal conditions. We observed the presence of two populations of neurons expressing MCP-1: One population of cells with weak expression of MCP-1 cor…

0301 basic medicineInterneuronPopulationBiologyInhibitory postsynaptic potential03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineNeurochemicalInterneuronsmedicineAnimalseducationNeuroinflammationChemokine CCL2Cerebral CortexNeuronseducation.field_of_studyNeocortexPyramidal CellsChemotaxisCell biologyRats030104 developmental biologymedicine.anatomical_structurePhenotypenervous systemCerebral cortex030217 neurology & neurosurgeryJournal of chemical neuroanatomy
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