Search results for "Neurotransmitter"

showing 10 items of 275 documents

Pharmacological activation of CB2 receptors counteracts the deleterious effect of ethanol on cell proliferation in the main neurogenic zones of the a…

2015

Chronic alcohol exposure reduces endocannabinoid activity and disrupts adult neurogenesis in rodents, which results in structural and functional alterations. Cannabinoid receptor agonists promote adult neural progenitor cell (NPC) proliferation. We evaluated the protective effects of the selective CB1 receptor agonist ACEA, the selective CB2 receptor agonist JWH133 and the fatty-acid amide-hydrolase (FAAH) inhibitor URB597, which enhances endocannabinoid receptor activity, on NPC proliferation in rats with forced consumption of ethanol (10%) or sucrose liquid diets for 2 weeks. We performed immunohistochemical and stereological analyses of cells expressing the mitotic phosphorylation of his…

:Phenomena and Processes::Physiological Phenomena::Physiological Processes::Growth and Development::Morphogenesis::Embryonic and Fetal Development::Organogenesis::Neurogenesis [Medical Subject Headings]CB1 receptorTubulina (proteína)Cannabinoid receptorCarbamatosEtanol:Chemicals and Drugs::Amino Acids Peptides and Proteins::Proteins::Nuclear Proteins::Histones [Medical Subject Headings]Ventrículos lateralesSacarosaNeuronasSubgranular zone0302 clinical medicine:Chemicals and Drugs::Amino Acids Peptides and Proteins::Proteins::Membrane Proteins::Receptors Cell Surface::Receptors G-Protein-Coupled::Receptors Cannabinoid::Receptor Cannabinoid CB1 [Medical Subject Headings]Histonas:Chemicals and Drugs::Organic Chemicals::Carboxylic Acids::Acids Acyclic::Carbamates [Medical Subject Headings]Receptor cannabinoide CB1Cannabinoid receptor type 2:Organisms::Eukaryota::Animals [Medical Subject Headings]:Phenomena and Processes::Metabolic Phenomena::Metabolism::Phosphorylation [Medical Subject Headings]:Anatomy::Cells::Stem Cells::Neural Stem Cells [Medical Subject Headings]:Anatomy::Nervous System::Neurons [Medical Subject Headings]health care economics and organizations:Anatomy::Nervous System::Central Nervous System::Brain::Cerebral Ventricles::Lateral Ventricles [Medical Subject Headings]Original Research:Chemicals and Drugs::Nucleic Acids Nucleotides and Nucleosides::Nucleosides::Deoxyribonucleosides::Deoxyuridine::Bromodeoxyuridine [Medical Subject Headings]0303 health sciencesAlcoholismoalcoholConsumo de alcoholNeurogenesis:Phenomena and Processes::Genetic Phenomena::Phenotype::Genetic Markers [Medical Subject Headings]:Chemicals and Drugs::Chemical Actions and Uses::Pharmacologic Actions::Molecular Mechanisms of Pharmacological Action::Neurotransmitter Agents::Cannabinoid Receptor Modulators::Cannabinoid Receptor Agonists [Medical Subject Headings]Benzamidas:Chemicals and Drugs::Amino Acids Peptides and Proteins::Proteins::Membrane Proteins::Receptors Cell Surface::Receptors G-Protein-Coupled::Receptors Cannabinoid::Receptor Cannabinoid CB2 [Medical Subject Headings]Endocannabinoid system3. Good healthbromodesoxiuridinaneurogenesisEndocannabinoidesmedicine.anatomical_structure:Chemicals and Drugs::Enzymes and Coenzymes::Enzymes::Hydrolases [Medical Subject Headings]ACEADietaAlcoholFosforilaciónAgonistmedicine.medical_specialtyHidrolasasmedicine.drug_classNeurogenesiseducation:Psychiatry and Psychology::Mental Disorders::Substance-Related Disorders::Alcohol-Related Disorders::Alcoholism [Medical Subject Headings]Subventricular zoneBiology:Phenomena and Processes::Physiological Phenomena::Nutritional Physiological Phenomena::Diet [Medical Subject Headings]:Anatomy::Nervous System::Central Nervous System::Brain::Prosencephalon::Telencephalon::Cerebrum::Cerebral Cortex::Hippocampus::Dentate Gyrus [Medical Subject Headings]lcsh:RC321-57103 medical and health sciencesCellular and Molecular NeuroscienceRatasInternal medicine:Chemicals and Drugs::Amino Acids Peptides and Proteins::Proteins::Nerve Tissue Proteins::Tubulin [Medical Subject Headings]JWH133medicineGiro dentadolcsh:Neurosciences. Biological psychiatry. Neuropsychiatry030304 developmental biologyCélulas madre nerviosas:Chemicals and Drugs::Chemical Actions and Uses::Pharmacologic Actions::Molecular Mechanisms of Pharmacological Action::Neurotransmitter Agents::Endocannabinoids [Medical Subject Headings]Dentate gyrusmarcadores genéticosCB2 receptor:Chemicals and Drugs::Carbohydrates::Polysaccharides::Oligosaccharides::Disaccharides::Sucrose [Medical Subject Headings]:Anatomy::Nervous System::Central Nervous System::Brain::Prosencephalon::Diencephalon::Hypothalamus [Medical Subject Headings]:Chemicals and Drugs::Organic Chemicals::Alcohols::Ethanol [Medical Subject Headings]Endocrinology:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Rodentia::Muridae::Murinae::Rats [Medical Subject Headings]nervous system:Psychiatry and Psychology::Behavior and Behavior Mechanisms::Behavior::Drinking Behavior::Alcohol Drinking [Medical Subject Headings]:Chemicals and Drugs::Organic Chemicals::Amides::Benzamides [Medical Subject Headings]030217 neurology & neurosurgeryHipotálamoNeuroscience
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24. Aminooxyacetate, an inhibitor of H2S production, potentiates lindane-induced convulsions in rats

2013

Purpose: H 2 S is a gaseous molecule recently recognized as endogenously produced neurotransmitter with different, still not well known, physiological and pathological roles. Cystathionine- β -synthase (CBS) is a major enzyme responsible for H 2 S production in the brain. The aim of this study was to investigate the effects of aminooxyacetate, potent CBS inhibitor, on convulsions induced by lindane in rats. Methods: Adult male Wistar albino rats were intraperitoneally (i.p.) treated with lindane 4 mg/kg and observed for convulsive behavioral manifestations during next 30 min. Aminooxyacetate (0.25 mmol/kg) or saline were injected 30 min prior to lindane administration. Seizure behavior was …

Adult malemedicine.medical_treatmentPharmacology050105 experimental psychology03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePhysiology (medical)Medicine0501 psychology and cognitive sciencesNeurotransmitterSalinechemistry.chemical_classificationbiologybusiness.industry05 social sciencesCystathionine beta synthaseSensory Systems3. Good healthFirst seizureEnzymeNeurologychemistryAnesthesiabiology.proteinNeurology (clinical)businessLindane030217 neurology & neurosurgeryClinical Neurophysiology
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Disentangling common and specific neural subprocesses of response inhibition.

2012

article i nfo Response inhibition is disturbed in several disorders sharing impulse control deficits as a core symptom. Since response inhibition is a cognitively and neurally multifaceted function which has been shown to rely on differing neural subprocesses and neurotransmitter systems, further differentiation to define neurophys- iological endophenotypes is essential. Response inhibition may involve at least three separable cognitive sub- components, i.e. interference inhibition, action withholding, and action cancelation. Here, we introduce a novel paradigm - the Hybrid Response Inhibition task - to disentangle interference inhibition, action withholding and action cancelation and their…

AdultMaleCognitive NeuroscienceDecision MakingInferior frontal gyrusNeurotransmitter systemsYoung AdultmedicineHumansResponse inhibitionCerebral CortexCommunicationMotor areaArtificial neural networkmedicine.diagnostic_testbusiness.industryCognitionNeural InhibitionMagnetic Resonance ImagingInhibition PsychologicalNeurologyEndophenotypeFemaleNerve NetFunctional magnetic resonance imagingPsychologybusinessNeuroscienceNeuroImage
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Reduced Cerebral Fluoro-l-Dopamine Uptake in Adult Patients Suffering from Phenylketonuria

2007

Deficiency of phenylalanine hydroxylase activity in phenylketonuria (PKU) causes an excess of phenylalanine (Phe) throughout the body, predicting impaired synthesis of catecholamines in the brain. To test this hypothesis, we used positron emission tomography (PET) to measure the utilization of 6-[18F]fluoro-l-dopamine (FDOPA) in the brain of adult patients suffering from PKU and in healthy controls. Dynamic 2-h long FDOPA emission recordings were obtained in seven adult PKU patients (five females, two males; age: 21 to 27 years) with elevated serum Phe levels, but lacking neurologic deficits. Seven age-matched, healthy volunteers were imaged under identical conditions. The utilization of F…

AdultMaleFluorine Radioisotopesmedicine.medical_specialtyPhenylalanineCentral nervous system diseasechemistry.chemical_compoundDopaminePhenylketonuriasInternal medicinemedicineHumansNeurotransmitterAdult patientsmedicine.diagnostic_testbusiness.industrymedicine.diseaseCorpus StriatumDihydroxyphenylalanineEndocrinologyNeurologychemistryPositron emission tomographyPositron-Emission TomographyCatecholamineFemaleNeurology (clinical)Cardiology and Cardiovascular MedicinebusinessPhenylalanine hydroxylase activitymedicine.drugJournal of Cerebral Blood Flow & Metabolism
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Influence of St John's wort on catecholamine turnover and cardiovascular regulation in humans

2004

BACKGROUND: St John's wort (Hypericum perforatum) is a popular over-the-counter antidepressant. Its antidepressive effect has been attributed in part to inhibition of monoamine transporters and monoamine oxidase, on the basis of in vitro studies. METHODS: In a double-blind, randomized, placebo-controlled, crossover study, 16 healthy subjects (11 men and 5 women; mean age, 31 +/- 5 years) ingested either St John's wort (300 mg three times daily) or placebo for 7 days. Imipramine treatment (50 mg three times daily) in 7 subjects served as a positive control. After treatment, physiologic and biochemical tests included cardiovascular reflex testing, graded head-up tilt testing, and plasma catec…

AdultMaleNitroprussideImipraminemedicine.medical_specialtyPosturePharmacologyAutonomic Nervous SystemPlaceboMethoxyhydroxyphenylglycolNorepinephrine uptakeCardiovascular Physiological PhenomenaNorepinephrineCatecholaminesDouble-Blind MethodInternal medicineHeart rateSupine PositionmedicineHumansNitric Oxide DonorsPharmacology (medical)PeryleneAnthracenesPharmacologyCross-Over StudiesAdrenergic Uptake Inhibitorsbusiness.industryHemodynamicsHypericum perforatumEndocrinologyBlood pressureMonoamine neurotransmitterCatecholamine34-Dihydroxyphenylacetic AcidAntidepressantFemalebusinessHypericummedicine.drugClinical Pharmacology & Therapeutics
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Effects of antiepileptic drugs on cortical excitability in humans: A TMS-EMG and TMS-EEG study.

2018

Brain responses to transcranial magnetic stimulation (TMS) recorded by electroencephalography (EEG) are emergent noninvasive markers of neuronal excitability and effective connectivity in humans. However, the underlying physiology of these TMS-evoked EEG potentials (TEPs) is still heavily underexplored, impeding a broad application of TEPs to study pathology in neuropsychiatric disorders. Here we tested the effects of a single oral dose of three antiepileptic drugs with specific modes of action (carbamazepine, a voltage-gated sodium channel (VGSC) blocker; brivaracetam, a ligand to the presynaptic vesicle protein VSA2; tiagabine, a gamma-aminobutyric acid (GABA) reuptake inhibitor) on TEP a…

AdultMaleTiagabinemedicine.medical_treatmentElectroencephalographyBrivaracetam050105 experimental psychology03 medical and health scienceschemistry.chemical_compoundYoung Adult0302 clinical medicineDouble-Blind MethodMedicineHumans0501 psychology and cognitive sciencesRadiology Nuclear Medicine and imagingNeurotransmitterTiagabineEvoked PotentialsResearch ArticlesCerebral CortexN100Cross-Over StudiesRadiological and Ultrasound Technologymedicine.diagnostic_testbusiness.industryElectromyography05 social sciencesElectroencephalographyCarbamazepineTranscranial Magnetic StimulationHealthy VolunteersPyrrolidinonesTranscranial magnetic stimulationCarbamazepineNeurologychemistryAnticonvulsantsNeurology (clinical)AnatomybusinessReuptake inhibitorNeuroscience030217 neurology & neurosurgerymedicine.drugHuman brain mapping
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Age-dependent decline of steady state dopamine storage capacity of human brain: an FDOPA PET study.

2010

Conventional indices of the utilization of FDOPA in living human brain have not consistently revealed important declines in dopamine function with normal aging. However, most methods of kinetic analysis have assumed irreversible trapping of decarboxylated FDOPA metabolites in brain, an assumption that is violated even in PET recordings of short duration. Therefore, we have developed methods for the calculation of steady-state storage of FDOPA together with its decarboxylated metabolites (V(d), mlg(-1)), based upon improved kinetic analysis of 120-min emission recordings. In a group of 28 normal male subjects, of age ranging from 23 to 73 years, the magnitude of V(d) in the striatum and in e…

AdultMalemedicine.medical_specialtyAgingMonoamine oxidaseDopamineModels NeurologicalStriatumchemistry.chemical_compoundYoung AdultDopamineInternal medicinemedicineHumansNeurotransmitterAgedCerebral CortexAromatic L-amino acid decarboxylaseChemistryGeneral NeuroscienceBrainHuman brainMiddle AgedCorpus StriatumKineticsmedicine.anatomical_structureEndocrinologyCerebral cortexPositron-Emission TomographyCatecholamineDopa DecarboxylaseNeurology (clinical)Geriatrics and GerontologyDevelopmental Biologymedicine.drugNeurobiology of aging
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Relationship between dopamine D2 receptor occupancy, clinical response, and drug and monoamine metabolites levels in plasma and cerebrospinal fluid. …

2009

Combining measurements of the monoamine metabolites in the cerebrospinal fluid (CSF) and neuroimaging can increase efficiency of drug discovery for treatment of brain disorders. To address this question, we examined five drug-naive patients suffering from schizophrenic disorder. Patients were assessed clinically, using the Positive and Negative Syndrome Scale (PANSS): at baseline and then at weekly intervals. Plasma and CSF levels of quetiapine and norquetiapine as well CSF 3,4-dihydroxyphenylacetic acid (DOPAC), homovanillic acid (HVA), 5-hydroxyindole-acetic acid (5-HIAA) and 3-methoxy-4-hydroxyphenylglycol (MHPG) were obtained at baseline and again after at least a 4 week medication trai…

AdultMalemedicine.medical_specialtyDibenzothiazepinesFluorine RadioisotopesPyrrolidinesmedicine.drug_classCaudate nucleusAtypical antipsychoticPilot ProjectsTritiumMethoxyhydroxyphenylglycolchemistry.chemical_compoundQuetiapine FumarateYoung AdultInternal medicinemedicineHumansBiogenic MonoaminesBiological PsychiatryTemporal cortexFirst episodeBrain Mappingbusiness.industryReceptors Dopamine D2Homovanillic acidHomovanillic AcidMiddle AgedMagnetic Resonance ImagingPsychiatry and Mental healthMonoamine neurotransmitterEndocrinologyFallypridechemistryPositron-Emission TomographyBenzamidesSchizophreniaQuetiapine34-Dihydroxyphenylacetic Acidbusinessmedicine.drugAntipsychotic AgentsProtein BindingJournal of psychiatric research
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Norepinephrine transporter gene polymorphism is not associated with susceptibility to alcohol dependence

2002

Abnormalities in monoamine neurotransmission have been implicated in the pathogenesis of alcoholism, mood disorders and schizophrenia. Murine norepinephrine transporter gene (NET) has been mapped to a region on chromosome 8 where a quantitative trait locus for ethanol sensitivity. Therefore we tested whether norepinephrine transporter (NET) gene variants confer susceptibility to either alcohol dependence or severe alcohol withdrawal symptoms. There is a highly polymorphic silent G1287A mutation in the NET gene. In our study 157 alcoholics and 185 healthy unrelated matched control subjects were analyzed for a silent G1287A mutation. No significant differences in allele and genotype distribut…

AdultMalemedicine.medical_specialtyGenotypeDNA Mutational AnalysisMolecular Sequence DataAlcohol Withdrawal DeliriumGene FrequencyPolymorphism (computer science)Internal medicineGenotypemedicineHumansGenetic Predisposition to DiseaseRNA MessengerAlleleAllelesBiological PsychiatryGeneticsNorepinephrine Plasma Membrane Transport ProteinsPolymorphism GeneticSymportersbiologybusiness.industryAlcohol dependenceExonsMiddle Agedmedicine.diseaseAlcoholismPsychiatry and Mental healthMonoamine neurotransmitterEndocrinologyMood disordersNorepinephrine transporterbiology.proteinFemaleGene polymorphismbusinessPolymorphism Restriction Fragment LengthPsychiatry Research
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Does habituation depend on cortical inhibition? Results of a rTMS study in healthy subjects

2010

Habituation, i.e. the decremental response to repeated sensorial stimulation, is studied in humans through evoked potential stimulation. Mechanisms underlying habituation are not yet cleared, even if inhibitory circuits are supposed to play an important role. Light deprivation (LD) increases visual cortical excitability likely through down-regulation of GABA circuits. We previously found that high-frequency repetitive transcranial magnetic stimulation (hf-rTMS) can revert these facilitatory effects likely restoring the activity of inhibitory circuits. Here, we studied the effects of LD and rTMS on habituation of visual evoked potentials (VEPs). The hypothesis was that if the inhibitory circ…

AdultMalemedicine.medical_specialtyNeurologymedicine.medical_treatmentStimulationInhibitory postsynaptic potentialbehavioral disciplines and activitieschemistry.chemical_compoundmedicineHumansHabituationEvoked potentialHabituation PsychophysiologicNeurotransmitterVisual Cortexmusculoskeletal neural and ocular physiologyGeneral NeuroscienceNeural InhibitionDarknesshabituation cortical inhibition rTMSTranscranial Magnetic StimulationTranscranial magnetic stimulationElectrophysiologynervous systemchemistryEvoked Potentials VisualFemalePsychologyNeuroscienceExperimental Brain Research
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