Search results for "Nociception"

showing 10 items of 110 documents

Coexpression of heat-evoked and capsaicin-evoked inward currents in acutely dissociated rat dorsal root ganglion neurons.

1997

Noxious heat is able to activate heat-sensitive nociceptors in the skin very rapidly, but little is known about the mechanisms by which heat is transduced. We used the whole-cell patch-clamp technique to study the effects of noxious heat and capsaicin on freshly dissociated rat dorsal root ganglion neurons in vitro. Using temperatures between 41 degrees C and 53 degrees C, 8 of 19 small neurons (phior = 30 microm) exhibited a heat-evoked inward current. All heat-sensitive neurons tested were also capsaicin-sensitive. Moreover, the heat response tended to be enhanced after capsaicin (360 +/- 150 pA versus 125 +/- 45 pA, P0.1, n = 7). Two of five heat-insensitive neurons were excited by capsa…

Hot TemperaturePatch-Clamp TechniquesCentral nervous systemRats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDorsal root ganglionGanglia SpinalmedicineAnimalsPatch clamp030304 developmental biologyCell SizeNeurons0303 health sciencesGeneral NeuroscienceReproducibility of ResultsSpinal cordSensory neuronRatsmedicine.anatomical_structureNociceptionnervous systemchemistryCapsaicinNociceptorBiophysicsCapsaicinNeuroscience030217 neurology & neurosurgeryNeuroscience letters
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DNA Methylation and Non-Coding RNAs during Tissue-Injury Associated Pain.

2022

While about half of the population experience persistent pain associated with tissue damages during their lifetime, current symptom-based approaches often fail to reduce such pain to a satisfactory level. To provide better patient care, mechanism-based analgesic approaches must be developed, which necessitates a comprehensive understanding of the nociceptive mechanism leading to tissue injury-associated persistent pain. Epigenetic events leading the altered transcription in the nervous system are pivotal in the maintenance of pain in tissue injury. However, the mechanisms through which those events contribute to the persistence of pain are not fully understood. This review provides a summar…

INFLAMMATORY PAINRNA UntranslatedChemistry MultidisciplinaryAdaptation BiologicalReviewUP-REGULATIONEpigenesis GeneticCpG islandsTranscripció genèticalncRNANeurociènciesnociceptionBiology (General)SpectroscopyGENE-EXPRESSIONGeneral MedicineComputer Science ApplicationsChemistryPhysical SciencesDisease SusceptibilityChronic PainLife Sciences & BiomedicineepigeneticALLEVIATES NEUROPATHIC PAINBiochemistry & Molecular Biologydorsal root ganglionQH301-705.50699 Other Biological SciencesCatalysisCONTRIBUTESInorganic ChemistryDiagnosis DifferentialCENTRAL SENSITIZATION0399 Other Chemical SciencesHumansPhysical and Theoretical ChemistryQD1-999Molecular Biologyspinal dorsal hornmiRNACHRONIC CONSTRICTION INJURYneuropathic pain0604 GeneticsScience & TechnologyChemical PhysicsNERVE INJURYMICRORNAGene Expression ProfilingOrganic ChemistryDNA MethylationCPG-BINDING PROTEIN-2gene transcriptionGene Expression RegulationsiRNARNAWounds and InjuriesBiomarkersInternational journal of molecular sciences
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Antihyperalgesic and analgesic properties of the N-methyl-d-aspartate (NMDA) receptor antagonist neramexane in a human surrogate model of neurogenic …

2006

Abstract NMDA-receptors are a major target in the prevention and treatment of hyperalgesic pain states in neuropathic pain. However, previous studies revealed equivocal results depending on study design and efficacy parameters. We tested the analgesic (generalized reduction of generation and processing of nociceptive signalling) and anti-hyperalgesic (prevention of central sensitization) properties of the NMDA-receptor antagonist neramexane and the potassium channel opener flupirtine in the intradermal capsaicin injection model. Furthermore, we tested the effect on pain summation (wind up). Eighteen healthy subjects received either a single dose of neramexane (40 mg p.o.), flupirtine (100 m…

Injections IntradermalAnalgesicAminopyridinesPainCyclopentanesPharmacologyPlaceboReceptors N-Methyl-D-AspartateDouble-Blind MethodPhysical StimulationHumansMedicineAnalgesicsCross-Over StudiesDose-Response Relationship Drugbusiness.industryNeramexaneTreatment OutcomeAnesthesiology and Pain MedicineAllodyniaNociceptionHyperalgesiaAnesthesiaNeuropathic painHyperalgesiaCapsaicinNervous System Diseasesmedicine.symptomFlupirtinebusinessmedicine.drugEuropean Journal of Pain
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Inactivation and tachyphylaxis of heat-evoked inward currents in nociceptive primary sensory neurones of rats.

2000

In contrast to other sensory modalities, pain does not decrease when a noxious stimulus is applied at constant intensity (Greene & Hardy, 1962). From this lack of adaptation on the perceptive level it has traditionally been implied that primary nociceptive afferents also do not adapt upon constant stimulation. This is in contrast to the results of recordings from these afferents, which exhibit pronounced adaptation for physical as well as chemical stimuli (Meyer et al. 1994). Peripheral adaptation of nociceptive nerve endings is compensated by central summation (Mendell & Wall, 1965; Price et al. 1977); this slow summation process of small fibre input to the dorsal horn of the spinal cord i…

Intracellular FluidMaleHot TemperatureTime FactorsPhysiologyStimulationTachyphylaxisStimulus (physiology)Rats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicineGanglia SpinalNoxious stimulusAnimalsNeurons AfferentTachyphylaxisCells Cultured030304 developmental biology0303 health sciencesChemistryElectric ConductivityNociceptorsOriginal ArticlesRatsNociceptionNociceptorCalciumFemaleCapsazepineExtracellular SpaceNeuroscienceFree nerve ending030217 neurology & neurosurgeryThe Journal of physiology
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Immunohistochemical expression and distribution of orexin, orphanin and leptin in the major salivary glands of some mammals

2012

Abstract: The aim of the study was to assess the involvement of apoptotic factors, cytokeratins and metalloproteinase- 9 in the histogenesis of both Epithelialized Gingival Lesions (EGL) and Periapical Lesions (PAL). 55 consecutive patients, 30 with PAL and 25 with EGL, were selected for the study after clinical and radiological examinations. The PAL patients had severe periapical lesions and tooth decay with exposure of the pulp chamber. All PAL and EGL biopsies were surgically extracted, fixed in 10% buffered formalin, and processed for routine light microscopy. Ten biopsies of each category were processed for immunohistochemistry (IHC). Serial paraffin sections were stained by IHC with a…

LeptinKey words: cytokeratins MMP-9 caspase-3 caspase-9 perapical lesions epithelial gingival lesions apoptosis IHC PCNA TUNELSettore BIO/17 - Istologiamedicine.medical_specialtyHistologySubmandibular Glandcytokeratins MMP-9 caspase-3 caspase-9 perapical lesions epithelial gingival lesions apoptosis IHC PCNA TUNEL [Key words]major salivary glands orphanin FQ nociception orexin leptin IHC rat sheep cowBiologySalivary GlandsPathology and Forensic MedicineOrexin-ASublingual Glandstomatognathic systemInternal medicineMajor Salivary GlandOrexigenicmedicineEndocrine systemAnimalsParotid GlandMammalsOrexinsSheepSalivary glandNeuropeptidesConnective tissue stromaIntracellular Signaling Peptides and ProteinsGeneral MedicineImmunohistochemistryEpitheliumOrexinRatsmedicine.anatomical_structureEndocrinologyOpioid PeptidesCattlemedicine.drug
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Pain, Neural Basis of

2001

The term ‘pain’ refers on one hand to a subjective state of suffering, which is a conscious personal experience. On the other hand, pain is the percept that results from the activation of the nociceptive system. This specific sensory system is specialized for the detection of external stimuli and internal tissue states that threaten the integrity of the body. It is a part of the somatosensory system, but its peripheral and central neurons are separate from the tactile system. Functions such as the localization of painful stimuli or the discrimination of pain intensities are subserved by a projection pathway from thin peripheral nerve fibers, via processing centers in the spinal cord and lat…

Limbic systemmedicine.anatomical_structureNociceptionNeuroplasticityCentral nervous systemThalamusHyperalgesiamedicineSensory systemmedicine.symptomSomatosensory systemPsychologyNeuroscience
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Inhibition of rapid heat responses in nociceptive primary sensory neurons of rats by vanilloid receptor antagonists.

1999

Recent studies demonstrated that heat-sensitive nociceptive primary sensory neurons respond to the vanilloid receptor (VR) agonist capsaicin, and the first cloned VR is a heat-sensitive ion channel. Therefore we studied to what extent heat-evoked currents in nociceptive dorsal root ganglion (DRG) neurons can be attributed to the activation of native vanilloid receptors. Heat-evoked currents were investigated in 89 neurons acutely dissociated from adult rat DRGs as models for their own terminals using the whole cell patch-clamp technique. Locally applied heated extracellular solution (effective temperature ∼53°C) rapidly activated reversible and reproducible inward currents in 80% (62/80) o…

MaleAgonistHot TemperaturePatch-Clamp TechniquesPhysiologymedicine.drug_classReceptors DrugRats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDorsal root ganglionGanglia SpinalmedicineAnimalsNeurons AfferentPatch clamp030304 developmental biology0303 health sciencesDose-Response Relationship DrugChemistryGeneral NeuroscienceNociceptorsRuthenium RedRatsElectrophysiologySolutionsElectrophysiologymedicine.anatomical_structureNociceptionCapsaicinBiophysicsNociceptorFemaleCapsaicinCapsazepineNeuroscience030217 neurology & neurosurgerySignal Transduction
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Therapeutic Potential of Inhibitors of Endocannabinoid Degradation for the Treatment of Stress-Related Hyperalgesia in an Animal Model of Chronic Pain

2014

The occurrence of chronic stress, depression, and anxiety can increase nociception in humans and may facilitate the transition from localized to chronic widespread pain. The mechanisms underlying chronic widespread pain are still unknown, hindering the development of effective pharmacological therapies. Here, we exposed C57BL/6J mice to chronic unpredictable stress (CUS) to investigate how persistent stress affects nociception. Next, mice were treated with multiple intramuscular nerve growth factor (NGF) injections, which induced chronic widespread nociception. Thus, combination of CUS and NGF served as a model where psychophysiological impairment coexists with long-lasting hyperalgesia. We…

MaleAnxietyPharmacologyAmidohydrolaseschemistry.chemical_compoundPiperidinesFatty acid amide hydrolaseNerve Growth FactorAnimalsMedicineChronic stressBenzodioxolesEnzyme InhibitorsJZL184PharmacologyDepressionbusiness.industryUncertaintyChronic painBrainAnalgesics Non-NarcoticURB597medicine.diseaseEndocannabinoid systemMonoacylglycerol LipasesMice Inbred C57BLDisease Models AnimalPsychiatry and Mental healthNociceptionchemistryHyperalgesiaAnesthesiaBenzamidesHyperalgesiaOriginal ArticleCarbamatesChronic Painmedicine.symptombusinessStress PsychologicalEndocannabinoidsNeuropsychopharmacology
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2-sulfonyliminodihydropyrimidines: a novel class of analgesic compounds.

2008

A series of 2-sulfonyliminodihydropyrimidine derivatives have been synthesized and evaluated in vivo for their antinociceptive and anti-inflammatory activities. The results were compared with that of acetyl salicylic acid. Compounds 6Ab-d and 6Be displayed an interesting analgesic profile in the acetic acid-induced abdominal contractions test. Based on the results of the carrageenan-hind paw edema test, compound 6Af showed potential anti-inflammatory activity.

MaleMagnetic Resonance SpectroscopyStereochemistryAnalgesicAnti-Inflammatory AgentsDrug Evaluation PreclinicalPharmaceutical SciencePainPharmacologychemistry.chemical_compoundMiceStructure-Activity RelationshipIn vivoDrug DiscoveryStructure–activity relationshipAnimalsEdemaAnalgesicsMolecular StructureChemistryRatsNociceptionPyrimidinesFemaleSalicylic acidPaw edemaArchiv der Pharmazie
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Antinociceptive and anti-inflammatory activities of Acacia pennata wild (Mimosaceae)

2005

The butanolic fraction of dried leaves of Acacia pennata (Mimosaceae) was tested for analgesic and anti-inflammatory activities in animal models. It showed significant protective effects against chemical stimuli (acetic acid and formalin) in the mouse. It also produced a significant increase of the threshold of sensitivity to pressure-induced pain in the rats. The extract revealed an inhibitory effect in carrageenin-induced rat paw oedema in the late phase. The results suggested that a peripheral mechanism is involved in the analgesic, associated to anti-inflammatory effect (NSAIDs-like). Among the class of compounds characterized in this fraction, flavonoids may be mainly responsible for t…

MaleMimosamedicine.drug_classButanolsAnalgesicDrug Evaluation PreclinicalAdministration OralPainAcacia pennataAcaciaPharmacognosyCarrageenanAnti-inflammatoryMiceAcetic acidchemistry.chemical_compoundFormaldehydeDrug DiscoveryPressuremedicineAnimalsEdemaRats WistarTramadol5-HT receptorAcetic AcidFlavonoidsPharmacologyDose-Response Relationship DrugbiologyTraditional medicinePlant ExtractsAnti-Inflammatory Agents Non-SteroidalAnalgesics Non-Narcoticbiology.organism_classificationStimulation ChemicalHindlimbRatsPlant LeavesDisease Models AnimalNociceptionchemistryFemaleJournal of Ethnopharmacology
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