Search results for "Nuclear"

showing 10 items of 26799 documents

Retinal Thickness and Microvascular Pattern in Early Parkinson's Disease.

2020

A thinning of intraretinal layers has been previously described in Parkinson's disease (PD) patients compared to healthy controls (HCs). Few studies evaluated the possible correlation between retinal thickness and retinal microvascularization. Thus, here we assessed the thickness of retinal layers and microvascular pattern in early PD patients and HCs, using, respectively, spectral-domain optical coherence tomography (SD-OCT) and SD-OCT-angiography (SD-OCT-A), and more interestingly, we evaluated a possible correlation between retinal thickness and microvascular pattern. Patients fulfilling criteria for clinically established/clinically probable PD and HCs were enrolled. Exclusion criteria …

0301 basic medicineSystemic diseasemedicine.medical_specialtyretinaParkinson's diseasegenetic structuresNerve fiber layeroptical coherence tomography angiographylcsh:RC346-42903 medical and health scienceschemistry.chemical_compound0302 clinical medicinevascularizationOphthalmologyMedicinelcsh:Neurology. Diseases of the nervous systemOriginal ResearchRetinaoptical coherence tomographybusiness.industryMicrovascular DensityRetinalmedicine.diseaseInner plexiform layereye diseases030104 developmental biologymedicine.anatomical_structurechemistryNeurologyInner nuclear layerparkinson's diseaseNeurology (clinical)sense organsbusiness030217 neurology & neurosurgeryoptical coherence tomography; optical coherence tomography angiography; parkinson's disease; retina; vascularizationFrontiers in neurology
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Humanized mice in cutaneous leishmaniasis—Suitability analysis of human PBMC transfer into immunodeficient mice

2019

Humanized mice represent a suitable preclinical test system for example therapeutic interventions in various disease settings, including infections. Here, we intended to establish such system for cutaneous leishmaniasis by infecting T, B and NK cell-deficient mice adoptively transferred with human peripheral blood mononuclear cells (PBMC). L major infection led to the establishment of parasite lesions harbouring viable parasites and human T cells, but parasite elimination was not seen due to a species-specific activity of T cell-derived human IFNγ. In addition, up to 50% of infected mice succumbed to severe graft-versus-host disease. In summary, even though long-term disease outcome assessm…

0301 basic medicineT cellGraft vs Host DiseaseLeishmaniasis CutaneousDermatologyDiseaseBiochemistryPeripheral blood mononuclear cellLesionInterferon-gammaMice030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicineSpecies SpecificityCutaneous leishmaniasisT-Lymphocyte SubsetsIn vivoAnimalsHumansMedicineParasite hostingMolecular Biologybusiness.industryMacrophagesLeishmaniasismedicine.diseaseAdoptive Transfer030104 developmental biologymedicine.anatomical_structureModels AnimalImmunologyDisease ProgressionLeukocytes MononuclearHeterograftsmedicine.symptombusinessExperimental Dermatology
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The mRNA cap-binding protein Cbc1 is required for high and timely expression of genes by promoting the accumulation of gene-specific activators at pr…

2015

The highly conserved Saccharomyces cerevisiae cap-binding protein Cbc1/Sto1 binds mRNA co-transcriptionally and acts as a key coordinator of mRNA fate. Recently, Cbc1 has also been implicated in transcription elongation and pre-initiation complex (PIC) formation. Previously, we described Cbc1 to be required for cell growth under osmotic stress and to mediate osmostress-induced translation reprogramming. Here, we observe delayed global transcription kinetics in cbc1Δ during osmotic stress that correlates with delayed recruitment of TBP and RNA polymerase II to osmo-induced promoters. Interestingly, we detect an interaction between Cbc1 and the MAPK Hog1, which controls most gene expression c…

0301 basic medicineTBX1Saccharomyces cerevisiae ProteinsTranscription GeneticBiophysicsRNA polymerase IISaccharomyces cerevisiaeBiochemistry03 medical and health sciencesOsmotic PressureStructural BiologyTranscription (biology)Gene Expression Regulation FungalGene expressionGeneticsRNA MessengerMolecular BiologyTranscription factorTranscription Initiation GeneticbiologyActivator (genetics)Nuclear ProteinsPromoterMolecular biology030104 developmental biologyRNA Cap-Binding Proteinsbiology.proteinMitogen-Activated Protein KinasesCREB1Transcription FactorsBiochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms
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Activation and selective IL-17 response of human Vγ9Vδ2 T lymphocytes by TLR-activated plasmacytoid dendritic cells.

2016

// Elena Lo Presti 1,2 , Nadia Caccamo 1,2 , Valentina Orlando 1,2 , Francesco Dieli 1,2 and Serena Meraviglia 1,2 1 Central Laboratory of Advanced Diagnosis and Biomedical Research (CLADIBIOR), University of Palermo, Palermo, Italy 2 Department of Biopathology and Medical Biotechnologies (DIBIMED), University of Palermo, Palermo, Italy Correspondence to: Serena Meraviglia, email: // Keywords : γδ T cells, plasmacytoid dendritic cells, IL-17, TLR activation, proliferation, Immunology and Microbiology Section, Immune response, Immunity Received : July 20, 2016 Accepted : August 02, 2016 Published :August 31, 2016 Abstract Vγ9Vδ2 T cells and plasmacytoid dendritic cells (pDCs) are two distinc…

0301 basic medicineTLR activationCellCell CommunicationLigandsLymphocyte Activation0302 clinical medicineT-Lymphocyte SubsetsCoculture TechniqueAntigen PresentationInterleukin-17Research Paper: Immunologyhemic and immune systemsIL-17medicine.anatomical_structurePhenotypeOncologyplasmacytoid dendritic cellsImmunology and Microbiology SectionInterleukin 17HumanCell typeproliferationCD40 LigandLigandBiologyDendritic Cellγδ T cells03 medical and health sciencesInducible T-Cell Co-Stimulator LigandInterferon-gammaImmune systemImmunityplasmacytoid dendritic cellmedicineHumansImmune responseCell Proliferationγδ T cellCD40Innate immune systemImmunityTLR9Dendritic CellsReceptors OX40Coculture TechniquesImmunity Innate030104 developmental biologyImmunologybiology.proteinLeukocytes MononuclearCpG IslandsCpG IslandImmunologic Memory030215 immunologyOncotarget
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The Stress-Inducible Protein DRR1 Exerts Distinct Effects on Actin Dynamics.

2018

Cytoskeletal dynamics are pivotal to memory, learning, and stress physiology, and thus psychiatric diseases. Downregulated in renal cell carcinoma 1 (DRR1) protein was characterized as the link between stress, actin dynamics, neuronal function, and cognition. To elucidate the underlying molecular mechanisms, we undertook a domain analysis of DRR1 and probed the effects on actin binding, polymerization, and bundling, as well as on actin-dependent cellular processes. Methods: DRR1 domains were cloned and expressed as recombinant proteins to perform in vitro analysis of actin dynamics (binding, bundling, polymerization, and nucleation). Cellular actin-dependent processes were analyzed in trans…

0301 basic medicineTU3ADRR1macromolecular substancesCatalysisArticleInorganic Chemistrylcsh:Chemistryactin dynamics03 medical and health sciencesSerum response factorCitosqueletProteïnes citosquelètiquesFAM107AHumansGenes Tumor SuppressorPhysical and Theoretical ChemistryCytoskeletonMolecular Biologylcsh:QH301-705.5SpectroscopyActinCytoskeletonstress physiologyMicroscopy ConfocalbiologyChemistryOrganic ChemistryFluorescence recovery after photobleachingNuclear ProteinscytoskeletonGeneral Medicinestress physiology ; cytoskeleton ; actin dynamics ; DRR1 ; TU3A ; FAM107AActinsComputer Science ApplicationsCell biologyddc:Cytoskeletal proteinsActinin alpha 1030104 developmental biologyTreadmillingProfilinlcsh:Biology (General)lcsh:QD1-999biology.proteinGelsolinFluorescence Recovery After PhotobleachingHeLa CellsInternational journal of molecular sciences
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Acute depletion of telomerase components DKC1 and NOP10 induces oxidative stress and disrupts ribosomal biogenesis via NPM1 and activation of the P53…

2020

Mutations in DKC1, NOP10, and TINF2 genes, coding for proteins in telomerase and shelterin complexes, are responsible for diverse diseases known as telomeropathies and ribosomopathies, including dyskeratosis congenita (DC, ORPHA 1775). These genes contribute to the DC phenotype through mechanisms that are not completely understood. We previously demonstrated in models of DC that oxidative stress is an early and independent event that occurs prior to telomere shortening. To clarify the mechanisms that induce oxidative stress, we silenced genes DKC1, NOP10, and TINF2 with siRNA technology. With RNA array hybridisation, we found several altered pathways for each siRNA model. Afterwards, we ide…

0301 basic medicineTelomeraseTelomere-Binding ProteinsCell Cycle ProteinsShelterin ComplexCell LineAdherens junction03 medical and health sciences0302 clinical medicineRibonucleoproteins Small NucleolarmedicineRNA Small InterferingMolecular BiologyTelomeraseTelomere ShorteningRibonucleoproteinChemistryRNANuclear ProteinsCell BiologyTelomereShelterinmedicine.diseaseCell biologyTelomereOxidative Stress030104 developmental biology030220 oncology & carcinogenesisMutationTumor Suppressor Protein p53NucleophosminRibosomesDyskeratosis congenitaBiogenesisBiochimica et biophysica acta. Molecular cell research
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Characterizing microstructural tissue properties in multiple sclerosis with diffusion MRI at 7 T and 3 T: The impact of the experimental design

2019

The recent introduction of advanced magnetic resonance (MR) imaging techniques to characterize focal and global degeneration in multiple sclerosis (MS), like the Composite Hindered and Restricted Model of Diffusion, or CHARMED, diffusional kurtosis imaging (DKI) and Neurite Orientation Dispersion and Density Imaging (NODDI) made available new tools to image axonal pathology non-invasively in vivo. These methods already showed greater sensitivity and specificity compared to conventional diffusion tensor-based metrics (e.g., fractional anisotropy), overcoming some of its limitations. While previous studies uncovered global and focal axonal degeneration in MS patients compared to healthy contr…

0301 basic medicineTime FactorsUltra-high field MRIAxonal pathologyCohort Studies0302 clinical medicineNuclear magnetic resonancemethods [Diffusion Magnetic Resonance Imaging]MicrostructureNODDImedicine.diagnostic_testGeneral NeuroscienceWATER DIFFUSIONmedicine.anatomical_structureResearch DesignKurtosisMulti-shell diffusion MRIAxonal degenerationWHITE-MATTERTENSORAdultMaterials sciencetherapy [Multiple Sclerosis]Sensitivity and SpecificityWhite matterMultiple sclerosis03 medical and health sciencesFractional anisotropyImage Interpretation Computer-Assistedmedicinediagnostic imaging [Nerve Degeneration]Journal ArticleHumansddc:610OPTIMIZATIONMultiple sclerosisinstrumentation [Diffusion Magnetic Resonance Imaging]diagnostic imaging [Multiple Sclerosis]Magnetic resonance imagingQUANTIFICATIONmedicine.diseaseMODELPATHOLOGYDiffusion Magnetic Resonance Imaging030104 developmental biologyRESOLUTIONDENSITYNerve Degeneration030217 neurology & neurosurgeryDiffusion MRI
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Hypoxia-Induced miR-675-5p Supports β-Catenin Nuclear Localization by Regulating GSK3-β  Activity in Colorectal Cancer Cell Lines

2020

The reduction of oxygen partial pressure in growing tumors triggers numerous survival strategies driven by the transcription factor complex HIF1 (Hypoxia Inducible Factor-1). Recent evidence revealed that HIF1 promotes rapid and effective phenotypic changes through the induction of non-coding RNAs, whose contribution has not yet been fully described. Here we investigated the role of the hypoxia-induced, long non-coding RNA H19 (lncH19) and its intragenic miRNA (miR-675-5p) into HIF1-Wnt crosstalk. During hypoxic stimulation, colorectal cancer cell lines up-regulated the levels of both the lncH19 and its intragenic miR-675-5p. Loss of expression experiments revealed that miR-675-5p inhibitio…

0301 basic medicineTranscription factor complexKaplan-Meier Estimatelcsh:Chemistry0302 clinical medicineGSK-3poxiahylcsh:QH301-705.5long non-coding H19Spectroscopybeta CateninKinaseChemistryGeneral MedicineCell HypoxiaComputer Science ApplicationsCell biologyGene Expression Regulation Neoplastic030220 oncology & carcinogenesisColorectal NeoplasmsProtein BindingActive Transport Cell Nucleuscolorectal cancermiR-675TransfectionCatalysisArticleInorganic Chemistry03 medical and health sciencesCell Line TumormicroRNAGene silencingHumansPhysical and Theoretical ChemistryMolecular BiologyGlycogen Synthase Kinase 3 betahypoxiaOrganic ChemistryRNAComputational Biologyβ-cateninHCT116 CellsMicroRNAs030104 developmental biologylcsh:Biology (General)lcsh:QD1-999Microscopy FluorescenceCateninMutationNuclear localization sequenceInternational Journal of Molecular Sciences
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ISWI ATP-dependent remodeling of nucleoplasmic ω-speckles in the brain of Drosophila melanogaster.

2017

Heterogeneous nuclear ribonucleoproteins (hnRNPs) belong to the RNA-binding proteins family. They are involved in processing heterogeneous nuclear RNAs (hnRNAs) into mature mRNAs. These proteins participate in every step of mRNA cycle, such as mRNA export, localization, translation, stability and alternative splicing. At least 14 major hnRNPs, which have structural and functional homologues in mammals, are expressed in Drosophila melanogaster. Until now, six of these hnRNPs are known to be nucleus-localized and associated with the long non-coding RNA (lncRNA) heat shock responsive ω (hsrω) in the omega speckle compartments (ω-speckles). The chromatin remodeler ISWI is the catalytic subunit …

0301 basic medicineTranscription GeneticBiologyHeterogeneous ribonucleoprotein particleHeterogeneous-Nuclear RibonucleoproteinsNuclear body03 medical and health scienceslncRNAAdenosine TriphosphateChromatin remodelersGene expressionGeneticsOmega speckleAnimalsMolecular BiologyGeneticsAdenosine TriphosphatasesCell NucleusAlternative splicingChromatin remodelers; hnRNPs; lncRNA; Nuclear body; Omega speckles; Molecular Biology; GeneticsRNABrainTranslation (biology)biology.organism_classificationChromatin Assembly and DisassemblyhnRNPsChromatinCell biology030104 developmental biologyDrosophila melanogasterGene Expression RegulationOmega specklesDrosophila melanogasterTranscription FactorsJournal of genetics and genomics = Yi chuan xue bao
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An in vitro investigation on the cytotoxic and nuclear receptor transcriptional activity of the mycotoxins fumonisin B1 and beauvericin.

2016

Fumonisin B1 (FB1) and beauvericin (BEA) are secondary metabolites of filamentous fungi, which under appropriate temperature and humidity conditions may develop on various foods and feeds. To date few studies have been performed to evaluate the toxicological and endocrine disrupting effects of FB1 and BEA. The present study makes use of various in vitro bioassays including; oestrogen, androgen, progestagen and glucocorticoid reporter gene assays (RGAs) for the study of nuclear receptor transcriptional activity, the thiazolyl blue tetrazolium bromide (MTT) assay to monitor cytotoxicity and high content analysis (HCA) for the detection of pre-lethal toxicity in the RGA and Caco-2 human colon …

0301 basic medicineTranscription GeneticCell SurvivalBiologyAdenocarcinomaEndocrine DisruptorsToxicologyFumonisins03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyGlucocorticoid receptorReceptors GlucocorticoidGenes ReporterDepsipeptidesmedicineHumansCytotoxicityReceptorCell NucleusFumonisin B1Dose-Response Relationship Drug04 agricultural and veterinary sciencesGeneral Medicine040401 food scienceBeauvericin030104 developmental biologychemistryNuclear receptorBiochemistryReceptors AndrogenToxicityColonic NeoplasmsCaco-2 CellsReceptors ProgesteroneGlucocorticoidmedicine.drugToxicology letters
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