Search results for "OBESITY"

showing 10 items of 1833 documents

GLP2: An underestimated signal for improving glycaemic control and insulin sensitivity

2016

Glucagon-like peptide 2 (GLP2) is a proglucagon-derived peptide produced by intestinal enteroendocrine L-cells and by a discrete population of neurons in the brainstem, which projects mainly to the hypothalamus. The main biological actions of GLP2 are related to the regulation of energy absorption and maintenance of mucosal morphology, function and integrity of the intestine; however, recent experimental data suggest that GLP2 exerts beneficial effects on glucose metabolism, especially in conditions related to increased uptake of energy, such as obesity, at least in the animal model. Indeed, mice lacking GLP2 receptor selectively in hypothalamic neurons that express proopiomelanocortin show…

Blood Glucose0301 basic medicinemedicine.medical_specialtyEndocrinology Diabetes and MetabolismPopulation030209 endocrinology & metabolismEnteroendocrine cellType 2 diabetesBiologyCarbohydrate metabolismDiet High-FatModels BiologicalType 2 diabeteMice03 medical and health sciences0302 clinical medicineInsulin resistanceEndocrinologyInternal medicineGlucagon-Like Peptide 2medicineAnimalsHomeostasisHumansGlucose homeostasisObesityeducationeducation.field_of_studyGLP2Insulin resistanceGlucagon-like peptide-2medicine.disease030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2GLP2; Insulin resistance; Obesity; Type 2 diabetes; Endocrinology; Endocrinology Diabetes and MetabolismHomeostasisSignal Transduction
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Early Low-Fat Diet Enriched With Linolenic Acid Reduces Liver Endocannabinoid Tone and Improves Late Glycemic Control After a High-Fat Diet Challenge…

2016

International audience; Evidence suggests that alterations of glucose and lipid homeostasis induced by obesity are associated with the elevation of endocannabinoid tone. The biosynthesis of the two main endocannabinoids, N-arachidonoylethanolamine and 2-arachidonoyl-glycerol, which derive from arachidonic acid, is influenced by dietary fatty acids (FAs). We investigated whether exposure to n-3 FA at a young age may decrease tissue endocannabinoid levels and prevent metabolic disorders induced by a later high-fat diet (HFD) challenge. Three-week-old mice received a 5% lipid diet containing lard, lard plus safflower oil, or lard plus linseed oil for 10 weeks. Then, mice were challenged with a…

Blood Glucose0301 basic medicinemedicine.medical_specialty[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and MetabolismMice TransgenicCarbohydrate metabolismBiologyDiet High-FatMice03 medical and health scienceschemistry.chemical_compoundInternal medicineInternal MedicinemedicineAnimalsHomeostasisObesityDiet Fat-RestrictedGlycemic2. Zero hungerdiabetesalpha-Linolenic acidBody WeightFatty liveralpha-Linolenic AcidLipid metabolismLipid Metabolismmedicine.diseaseEndocannabinoid system3. Good healthFatty LiverMice Inbred C57BL[SDV.AEN] Life Sciences [q-bio]/Food and Nutrition030104 developmental biologyEndocrinologyLiverchemistryendocananbinoid systemCarbohydrate MetabolismArachidonic acidlipids (amino acids peptides and proteins)Metabolic syndrome[SDV.AEN]Life Sciences [q-bio]/Food and NutritionEndocannabinoids
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Evaluation of B-cell secretion and peripheral insulin resistance during pregnancy and after delivery in gestational diabetes mellitus with obesity

1988

Nine pregnant women with gestational diabetes mellitus (GDM) were studied. Six normal pregnant women and six normal nonpregnant women were evaluated as control groups. All the women underwent oral glucose tolerance test and glucose clamp during the third trimester of pregnancy and two months after delivery. During OGTT, glucose, C-peptide and insulin plasma levels were determined. C-peptide and insulin values in the late phase of OGTT were higher during pregnancy than after delivery in both groups. In gestational diabetic women, the M-value in the second steady-state during glucose clamp was lower than in controls, both during pregnancy and after delivery. Nevertheless, in both groups the M…

Blood GlucoseAdultmedicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentPregnancy in DiabeticsObesity; Glucose Tolerance Test; Reference Values; Humans; Diabetes Mellitus; Islets of Langerhans; Insulin; Blood Glucose; Pregnancy; Puerperal Disorders; Adult; Insulin Resistance; Female; Pregnancy in DiabeticsIslets of LangerhansEndocrinologyInsulin resistanceReference ValuesPregnancyDiabetes mellitusInternal medicineInternal MedicinemedicineDiabetes MellitusHumansInsulinObesityPregnancyGlucose tolerance testSettore MED/12 - Gastroenterologiamedicine.diagnostic_testbusiness.industryInsulinGeneral MedicinePuerperal DisordersGlucose clamp techniqueGlucose Tolerance Testmedicine.diseaseGestational diabetesEndocrinologyGestationFemaleInsulin Resistancebusiness
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Health benefits of pistachios consumption

2017

The health benefits of nuts, mainly in relation to the improvement of dysmetabolic conditions such as obesity, type 2 diabetes mellitus and the related cardiovascular diseases, have been widely demonstrated. Compared to other nuts, pistachios have a lower fat and caloric content, and contain the highest levels of unsaturated fatty acids, potassium, γ-tocopherol, phytosterols and xanthophyll carotenoids, all substances that are well known for their antioxidant and anti-inflammatory actions. This variety of nutrients contributes to the growing body of evidence that the consumption of pistachios improves health, leading to a greater potential of healthy antioxidant and anti-inflammatory activi…

Blood GlucoseAntioxidantmedicine.medical_treatmentPhytochemicalsPlant ScienceHealth benefits01 natural sciencesBiochemistrymetabolic syndromeAnalytical ChemistryNutrientRisk FactorsmedicineHumansNutsObesityFood sciencePistachioanti-inflammatory activityGlycemicInflammationConsumption (economics)010405 organic chemistrybusiness.industryBody WeightOrganic ChemistryType 2 Diabetes Mellitusmedicine.diseaseLipidsObesityDiet0104 chemical sciencesOxidative Stress010404 medicinal & biomolecular chemistrypolyphenolDiabetes Mellitus Type 2Cardiovascular DiseasesPistaciaMetabolic syndromebusinessNutritive Valueunsaturated fatty acids
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A Randomized, Controlled Trial of 3.0 mg of Liraglutide in Weight Management.

2015

BACKGROUND: Obesity is a chronic disease with serious health consequences, but weight loss is difficult to maintain through lifestyle intervention alone. Liraglutide, a glucagon-like peptide-1 analogue, has been shown to have potential benefit for weight management at a once-daily dose of 3.0 mg, injected subcutaneously. METHODS: We conducted a 56-week, double-blind trial involving 3731 patients who did not have type 2 diabetes and who had a body-mass index (BMI; the weight in kilograms divided by the square of the height in meters) of at least 30 or a BMI of at least 27 if they had treated or untreated dyslipidemia or hypertension. We randomly assigned patients in a 2:1 ratio to receive on…

Blood GlucoseCounselingMaleType 2 diabeteslaw.inventionBody Mass IndexRandomized controlled trialWeight losslawGlucagon-Like Peptide 1Weight managementSubcutaneousMedicine (all)ReducingNauseaGeneral MedicineMiddle AgedCombined Modality Therapy3. Good healthFemaletype 2 diabetesmedicine.symptomHumanmedicine.drugAdultDiarrheamedicine.medical_specialtyDiet ReducingInjections SubcutaneousInjections SubcutaneouPlaceboInjectionsDouble-Blind MethodInternal medicineWeight LossmedicineHumansHypoglycemic AgentsObesityExerciseHypoglycemic AgentLiraglutidebusiness.industryLiraglutidemedicine.diseaseWeight LoDietEndocrinologybusiness[SDV.AEN]Life Sciences [q-bio]/Food and NutritionBody mass index[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyDyslipidemiaAdult; Blood Glucose; Body Mass Index; Combined Modality Therapy; Counseling; Diarrhea; Diet Reducing; Double-Blind Method; Exercise; Female; Glucagon-Like Peptide 1; Humans; Hypoglycemic Agents; Injections Subcutaneous; Liraglutide; Male; Middle Aged; Nausea; Obesity; Weight Loss; Medicine (all)The New England journal of medicine
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Chronic exposure of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) induces an obesogenic effect in C57BL/6J mice fed a high fat diet

2017

IF 3.582; International audience; Contaminant involvement in the pathophysiology of obesity is widely recognized. It has been shown that low dose and chronic exposure to endocrine disruptor compounds (EDCs) potentiated diet- induced obesity. High and acute exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD), a persistent organic pollutant (POP) and an EDC with anti-estrogenic property, causes wasting syndrome . However at lower doses, the TCDD metabolic effects remain poorly understood. We investigated the obesogenic effect during chronic exposure of TCDD at 1μg/kg body weight (bw)/week in adult C57BL/6J mice fed with a high fat diet (HFD) and exposed from 10 to 42 weeks old to TCDD or e…

Blood GlucoseLeptinMale0301 basic medicineTCDDPolychlorinated DibenzodioxinsTime FactorsAdipose tissue010501 environmental sciencesToxicology01 natural sciencesBasic Helix-Loop-Helix Transcription FactorsInsulinAdiposity2. Zero hunger[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism3. Good healthLiverEndocrine disruptorReceptors AndrogenCytokinesEnvironmental PollutantsFemaleInflammation Mediatorsmedicine.symptomStearoyl-CoA Desaturasemedicine.medical_specialtyLipolysisInflammationchronic exposureIntra-Abdominal FatDiet High-FatRisk Assessment03 medical and health sciencesSex FactorsobesogenInternal medicinemedicineAnimalsEndocrine systemObesityRNA MessengerWasting SyndromeTriglycerides0105 earth and related environmental sciencesbusiness.industrymedicine.diseaseObesityMice Inbred C57BL030104 developmental biologyEndocrinologyReceptors Aryl HydrocarbonInsulin ResistancebusinessBiomarkersObesogenDrug metabolism
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Asperuloside Enhances Taste Perception and Prevents Weight Gain in High-Fat Fed Mice

2021

Asperuloside is an iridoid glycoside found in many medicinal plants that has produced promising anti-obesity results in animal models. In previous studies, three months of asperuloside administration reduced food intake, body weight, and adipose masses in rats consuming a high fat diet (HFD). However, the mechanisms by which asperuloside exerts its anti-obesity properties were not clarified. Here, we investigated homeostatic and nutrient-sensing mechanisms regulating food intake in mice consuming HFD. We confirmed the anti-obesity properties of asperuloside and, importantly, we identified some mechanisms that could be responsible for its therapeutic effect. Asperuloside reduced body weight …

Blood GlucoseLeptinMalecannabinoid (CB) receptor 10301 basic medicineTastePro-Opiomelanocortinfood intakeEndocrinology Diabetes and MetabolismAdipose tissueWeight Gainnutrient-sensing mechanismslcsh:Diseases of the endocrine glands. Clinical endocrinologyCyclopentane MonoterpenesEnergy homeostasisMiceEndocrinology0302 clinical medicineGlucosidesWeight lossInsulinasperuloside; cannabinoid (CB) receptor 1; CD36; FFAR1-4; food intake; nutrient-sensing mechanisms; TAS1R2-3; weight lossReceptorOriginal ResearchLeptindigestive oral and skin physiologyTaste PerceptionGhrelinTAS1R2-3Ghrelinmedicine.symptommedicine.medical_specialtyHypothalamusBiologyDiet High-Fatasperuloside03 medical and health sciencesInternal medicinemedicineAnimalsPyranslcsh:RC648-665Body WeightFFAR1-4030104 developmental biologyEndocrinologyAnti-Obesity Agentsweight lossEnergy IntakeCD36Weight gain030217 neurology & neurosurgery
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Hepatic and very low-density lipoprotein fatty acids in obese offspring of overfed dams.

2010

The combined effects of developmental programming and high-fat feeding at weaning on fatty acid metabolism of the offspring are not well known. In the present study, we aim at characterizing the influence of maternal and offspring's own diets on liver and very low-density lipoprotein (VLDL) lipids; fatty acid profiles of VLDL and liver phospholipids, triglycerides, and cholesteryl esters; and hepatic enzyme activities. Twenty obese male rats born to cafeteria diet-fed dams and 20 control rats born to control diet-fed dams were selected. At weaning, 10 rats of each group were fed control or cafeteria diet. Obese rats had a significant increase in serum glucose, insulin, leptin, VLDL apolipop…

Blood GlucoseLeptinMalemedicine.medical_specialtyVery low-density lipoproteinOffspringEndocrinology Diabetes and MetabolismLinoleic acidFatty Acids NonesterifiedLipoproteins VLDLchemistry.chemical_compoundEndocrinologyPregnancyInternal medicinemedicineAnimalsInsulinObesityRats Wistarchemistry.chemical_classificationbiologyFatty acid metabolismBody WeightFatty acidOrgan Sizemedicine.diseaseRatsFatty acid synthaseEndocrinologychemistryLiverMaternal Exposurebiology.proteinlipids (amino acids peptides and proteins)FemaleSteatosisEnergy IntakeEnergy MetabolismPolyunsaturated fatty acidMetabolism: clinical and experimental
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Bile acid receptor TGR5 is critically involved in preference for dietary lipids and obesity

2020

International audience; We investigated the implication of Takeda G protein-coupled receptor 5 (TGR5) in fat preference and fat sensing in taste bud cells (TBC) in C57BL/6 wild-type (WT) and TGR5 knock out (TGR5-/-) male mice maintained for 20 weeks on a high-fat diet (HFD). We also assessed the implication of TGR5 single nucleotide polymorphism (SNP) in young obese humans. The high-fat diet (HFD)-fed TGR5-/- mice were more obese, marked with higher liver weight, lipidemia and steatosis than WT obese mice. The TGR5-/- obese mice exhibited high daily food/energy intake, fat mass and inflammatory status. WT obese mice lost the preference for dietary fat, but the TGR5-/- obese mice exhibited n…

Blood GlucoseLipopolysaccharidesMale0301 basic medicine[SDV]Life Sciences [q-bio]Endocrinology Diabetes and MetabolismClinical BiochemistryBiochemistryReceptors G-Protein-CoupledMice0302 clinical medicineInsulinReceptorMice Knockout2. Zero hungerchemistry.chemical_classificationNutrition and DieteticsLipidsG protein-coupled bile acid receptor[SDV] Life Sciences [q-bio]medicine.anatomical_structuremedicine.medical_specialtyMice Transgenic030209 endocrinology & metabolismSingle-nucleotide polymorphismDiet High-FatPolymorphism Single NucleotideBile Acids and SaltsFood Preferences03 medical and health sciencesInternal medicineTaste budmedicineAnimalsObesityMolecular BiologyInflammationbusiness.industryTaste budFatty acidFatty acidmedicine.diseaseDietary FatsObesityIn vitroFatty LiverMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinologychemistryFatCalciumSteatosisbusinessThe Journal of Nutritional Biochemistry
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Development and characterization of an experimental model of diet-induced metabolic syndrome in rabbit

2017

Metabolic syndrome (MetS) has become one of the main concerns for public health because of its link to cardiovascular disease. Murine models have been used to study the effect of MetS on the cardiovascular system, but they have limitations for studying cardiac electrophysiology. In contrast, the rabbit cardiac electrophysiology is similar to human, but a detailed characterization of the different components of MetS in this animal is still needed. Our objective was to develop and characterize a diet-induced experimental model of MetS that allows the study of cardiovascular remodeling and arrhythmogenesis. Male NZW rabbits were assigned to control (n = 15) or MetS group (n = 16), fed during 2…

Blood GlucoseMale0301 basic medicinePhysiologylcsh:MedicineBlood Pressure030204 cardiovascular system & hematologyVascular MedicineBiochemistryEatingchemistry.chemical_compound0302 clinical medicineGlucose MetabolismDietary SucroseBlood plasmaMedicine and Health Scienceslcsh:ScienceMammalsMetabolic SyndromeMultidisciplinaryLiver DiseasesFatty liverAnimal ModelsBody FluidsBloodExperimental Organism SystemsPhysiological ParametersLiverVertebratesHypertensionMetabolomeCarbohydrate MetabolismRabbitsAnatomyResearch Articlemedicine.medical_specialtyMean arterial pressureBilirubinDiastoleGastroenterology and HepatologyBiologyResearch and Analysis MethodsDiet High-FatBlood Plasma03 medical and health sciencesInternal medicineGlucose IntolerancemedicineAnimalsMetabolomicsObesityNuclear Magnetic Resonance BiomolecularNutritionAnalysis of VarianceBody Weightlcsh:ROrganismsBiology and Life Sciencesmedicine.diseaseDietFatty LiverDisease Models AnimalMetabolism030104 developmental biologyEndocrinologyBlood pressurechemistryAmnioteslcsh:QMetabolic syndromeSteatosisPLOS ONE
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