Search results for "OXIDATION"

showing 10 items of 1913 documents

Phenolic Composition of Hydrophilic Extract of Manna from Sicilian Fraxinus angustifolia Vahl and its Reducing, Antioxidant and Anti-Inflammatory Act…

2019

Manna, a very singular vegetable product derived from the spontaneous solidification of the sap of some Fraxinus species, has long been known for its mild laxative and emollient properties. In this work, a hydro-alcoholic extract of manna (HME) from Sicilian Fraxinus angustifolia Vahl was investigated using HPLC-DAD to find phenol components and using chemical and biological in vitro assays to determine its reducing, antioxidant and anti-inflammatory capacity. We identified elenolic acid, tyrosol, hydroxytyrosol, catechin, fraxetin, verbascoside, gallic acid, procyanidin-B1, and luteolin 3,7 glucoside, in order of abundance. Measurements of total antioxidant activity by Folin-Ciocalteu reac…

0301 basic medicinemanna bioactivityAntioxidantantioxidantPhysiologyDPPHmedicine.medical_treatmentClinical BiochemistryFraxinus angustifoliaPhytochemicalBiochemistry03 medical and health scienceschemistry.chemical_compound0302 clinical medicineLipid oxidation<i>Fraxinus</i>medicinereducing powerFraxinuFood scienceMolecular BiologyElenolic acidred blood cell oxidationbiologyintestinal bowel disease modellcsh:RM1-950Cell Biologybiology.organism_classificationphytochemicalsTyrosol030104 developmental biologylcsh:Therapeutics. Pharmacologychemistry030220 oncology & carcinogenesisHydroxytyrosolFraxetinmannaAntioxidants
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Effect of plant sterol and galactooligosaccharides enriched beverages on oxidative stress and longevity in Caenorhabditis elegans

2020

Abstract This study evaluates the impact of two plant sterol (PS) enriched fruit beverages (0.6 g /100 ml), without (MfB) or with GOS (MfB-G) (1.2 g/100 ml) on the resistance against oxidative stress induced by non-conventional (cholesterol oxidation products (COPs)) and conventional (H2O2) oxidant compounds, as well as their impact on C. elegans longevity. Nematodes fed with both beverages (0.005–0.25%, v/v) showed similar improved oxidative stress resistance against COPs and H2O2. This effect was dependent on daf-16 transcription factor, although GOS showed an additional beneficial effect independent to daf-16. In addition, both beverages extended nematode lifespan, independently of the p…

0301 basic medicinemedia_common.quotation_subjectMutantCholesterol oxidation productsMedicine (miscellaneous)Milk-based fruit beveragesmedicine.disease_causePlant sterols03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyIn vivomedicineTX341-641Caenorhabditis elegansTranscription factorCaenorhabditis elegansmedia_common030109 nutrition & dieteticsNutrition and DieteticsbiologyCholesterolNutrition. Foods and food supplyLongevity04 agricultural and veterinary sciencesbiology.organism_classification040401 food scienceNematodechemistryBiochemistryOxidative stressFood ScienceGalactooligosaccharidesJournal of Functional Foods
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Oxidative Stress in Non-Dialysis-Dependent Chronic Kidney Disease Patients

2021

Background: Cardiovascular complications are the leading cause of morbidity and mortality at any stage of chronic kidney disease (CKD). Moreover, the high rate of cardiovascular mortality observed in these patients is associated with an accelerated atherosclerosis process that likely starts at the early stages of CKD. Thus, traditional and non-traditional or uremic-related factors represent a link between CKD and cardiovascular risk. Among non-conventional risk factors, particular focus has been placed on anaemia, mineral and bone disorders, inflammation, malnutrition and oxidative stress and, in this regard, connections have been reported between oxidative stress and cardiovascular disease…

0301 basic medicinemedicine.medical_specialtyAntioxidantHealth Toxicology and Mutagenesismedicine.medical_treatment030232 urology & nephrologyRenal functionInflammationOxidative phosphorylationmedicine.disease_causeArticle03 medical and health scienceschemistry.chemical_compound0302 clinical medicinecardiovascular diseaseMalondialdehydeInternal medicineHumansMedicineoxidative stressRenal Insufficiency Chronicbusiness.industryPublic Health Environmental and Occupational HealthRGlutathionemedicine.diseaseMalondialdehyde030104 developmental biologyEndocrinologychemistry8-Hydroxy-2'-DeoxyguanosineChronic DiseaseMedicinemedicine.symptombusinessOxidation-ReductionOxidative stresschronic kidney diseaseKidney diseaseInternational Journal of Environmental Research and Public Health
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Impact of Plasma Oxidative Stress Markers on Post-race Recovery in Ultramarathon Runners: A Sex and Age Perspective Overview

2021

Oxidative stress has been widely studied in association to ultra-endurance sports. Although it is clearly demonstrated the increase in reactive oxygen species and free radicals after these extreme endurance exercises, the effects on the antioxidant defenses and the oxidative damage to macromolecules, remain to be fully clarified. Therefore, the aim of this study was to elucidate the impact of an ultramarathon race on the plasma markers of oxidative stress of 32 runners and their post-race recovery, with especial focused on sex and age effect. For this purpose, the antioxidant enzymes glutathione peroxidase (GPx) and glutathione reductase (GR) activity, as well as the lipid peroxidation prod…

0301 basic medicinemedicine.medical_specialtyAntioxidantPhysiologymedicine.medical_treatmentClinical BiochemistryGlutathione reductaseBiologymedicine.disease_causeBiochemistryArticleLipid peroxidation03 medical and health sciencesBasal (phylogenetics)chemistry.chemical_compound0302 clinical medicineultraendurance exerciseInternal medicinemedicineoxidative stressmuscle injuryMolecular Biologychemistry.chemical_classificationReactive oxygen speciesGlutathione peroxidaselcsh:RM1-950030229 sport sciencesCell BiologyMalondialdehydelcsh:Therapeutics. Pharmacologyantioxidants030104 developmental biologyEndocrinologychemistryOxidative stressAntioxidants
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Oxidative stress biomarkers in Fabry disease: is there a room for them?

2020

Abstract Background Fabry disease (FD) is an X-linked lysosomal storage disorder, caused by deficient activity of the alpha-galactosidase A enzyme leading to progressive and multisystemic accumulation of globotriaosylceramide. Recent data point toward oxidative stress signalling which could play an important role in both pathophysiology and disease progression. Methods We have examined oxidative stress biomarkers [Advanced Oxidation Protein Products (AOPP), Ferric Reducing Antioxidant Power (FRAP), thiolic groups] in blood samples from 60 patients and 77 healthy controls. Results AOPP levels were higher in patients than in controls (p &lt; 0.00001) and patients presented decreased levels of…

0301 basic medicinemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentGlobotriaosylceramideOxidative phosphorylationDiseasemedicine.disease_cause03 medical and health scienceschemistry.chemical_compound0302 clinical medicinelysoGb3Internal medicinemedicineHumansFabry diseaseOriginal Communicationbusiness.industryBiomarkermedicine.diseaseFabry diseasePathophysiologyOxidative Stress030104 developmental biologyEndocrinologyNeurologychemistryAdvanced oxidation protein productsalpha-GalactosidaseMutationNeurology (clinical)businessBiomarkers030217 neurology & neurosurgeryOxidative stressJournal of Neurology
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Loss of c-Met signaling sensitizes hepatocytes to lipotoxicity and induces cholestatic liver damage by aggravating oxidative stress.

2016

Recent studies confirmed a critical importance of c-Met signaling for liver regeneration by modulating redox balance. Here we used liver-specific conditional knockout mice (MetKO) and a nutritional model of hepatic steatosis to address the role of c-Met in cholesterol-mediated liver toxicity. Liver injury was assessed by histopathology and plasma enzymes levels. Global transcriptomic changes were examined by gene expression microarray, and key molecules involved in liver damage and lipid homeostasis were evaluated by Western blotting. Loss of c-Met signaling amplified the extent of liver injury in MetKO mice fed with high-cholesterol diet for 30days as evidenced by upregulation of liver enz…

0301 basic medicinemedicine.medical_specialtyCell SurvivalCholestasis IntrahepaticBiologyToxicologymedicine.disease_causeArticleCholesterol Dietary03 medical and health sciencesMice0302 clinical medicineLiver Function TestsInternal medicinemedicineAnimalsLiver X receptorLiver injuryMice Knockoutmedicine.diagnostic_testLipid metabolismProto-Oncogene Proteins c-metmedicine.diseaseLipid MetabolismGlutathioneLipidsLiver regenerationOxidative Stress030104 developmental biologyEndocrinologyLipotoxicity030220 oncology & carcinogenesisHepatocytesLipid PeroxidationSteatosisLiver function testsOxidative stressSignal TransductionToxicology
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Influence of mental stress and environmental toxins on circadian clocks : implications for redox regulation of the heart and cardioprotection

2020

Risk factors in the environment such as air pollution and mental stress contribute to the development of chronic non-communicable disease. Air pollution was identified as the leading health risk factor in the physical environment, followed by water pollution, soil pollution/heavy metals/chemicals and occupational exposures, however neglecting the non-chemical environmental health risk factors (e.g. mental stress and noise). Epidemiological data suggest that environmental risk factors are associated with higher risk for cardiovascular, metabolic and mental diseases, including hypertension, heart failure, myocardial infarction, diabetes, arrhythmia, stroke, depression and anxiety disorders. W…

0301 basic medicinemedicine.medical_specialtyExposomeCircadian clock610 MedizinReview ArticleDiseaseCardiovascular System03 medical and health sciences0302 clinical medicineCircadian ClocksDiabetes mellitusEnvironmental health610 Medical sciencesEpidemiologymedicineHumansReview ArticlesStrokeDepression (differential diagnoses)Pharmacologybusiness.industryHeartmedicine.disease3. Good health030104 developmental biology13. Climate actionHypertensionAnxietymedicine.symptombusinessOxidation-Reduction030217 neurology & neurosurgery
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Long-Term Aspartame Administration Leads to Fibrosis, Inflammasome Activation, and Gluconeogenesis Impairment in the Liver of Mice

2021

Background: Aspartame is an artificial sweetener used in foods and beverages worldwide. However, it is linked to oxidative stress, inflammation, and liver damage through mechanisms that are not fully elucidated yet. This work aimed to investigate the effects of long-term administration of aspartame on the oxidative and inflammatory mechanisms associated with liver fibrosis progression in mice. Methods: Mice were divided into two groups with six animals each: control and aspartame. Aspartame (80 mg/kg, via oral) or vehicle was administrated for 12 weeks. Results: Aspartame caused liver damage and elevated serum transaminase levels. Aspartame also generated liver fibrosis, as evidenced by his…

0301 basic medicinemedicine.medical_specialtyPGC-1αInflammationBiologymedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyArticleaspartameNrf2Lipid peroxidation03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDownregulation and upregulationFibrosislipidinflammasomeInternal medicinemedicinelcsh:QH301-705.5liver fibrosisGeneral Immunology and MicrobiologyAspartameInflammasomelipid peroxidationmedicine.diseaseCollagen type I alpha 1030104 developmental biologyEndocrinologyhypoglycemiagluconeogenesischemistrylcsh:Biology (General)030211 gastroenterology & hepatologymedicine.symptomGeneral Agricultural and Biological SciencesOxidative stressmedicine.drugBiology
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Circulating inflammation-related factors are correlated with systemic redox status in IgA nephropathy; a case-control study.

2020

Abstract Background IgA nephropathy (IGAN) is characterized by oxidative stress and inflammation. In the present study, we explored the relationship of redox status vs. that of circulating inflammation-related factors with other biomarkers in patients with IGAN. Methods This is a case-control study comparing patients with IGAN (Stage 1–4) to healthy controls. Forty patients and 40 controls were matched for age and sex. Two circulating dynamic redox parameters were analysed: oxidized free cysteine (Cys) and nitrate. Thirty-seven inflammation-related factors were measured in serum. Results The patients had elevated levels of oxidized free Cys and nitrate, indicating the presence of oxidative …

0301 basic medicinemedicine.medical_specialtyParathyroid hormoneRenal functionInflammationmedicine.disease_causeBiochemistryNephropathy03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePhysiology (medical)Internal medicinemedicineHumansOsteopontinInflammationCreatininebiologybusiness.industryCase-control studyGlomerulonephritis IGAmedicine.disease030104 developmental biologyEndocrinologychemistryCase-Control Studiesbiology.proteinmedicine.symptombusinessOxidation-Reduction030217 neurology & neurosurgeryOxidative stressBiomarkersGlomerular Filtration RateFree radical biologymedicine
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Olesoxime improves cerebral mitochondrial dysfunction and enhances Aβ levels in preclinical models of Alzheimer's disease.

2019

Abstract Background Approved drugs for Alzheimer's disease (AD) only have a symptomatic effects and do not intervene causally in the course of the disease. Olesoxime (TRO19622) has been tested in AD disease models characterized by improved amyloid precursor protein processing (AβPP) and mitochondrial dysfunction. Methods Three months old Thy-1-AβPPSL (tg) and wild type mice (wt) received TRO19622 (100 mg/kg b.w.) in supplemented food pellets for 15 weeks (tg TRO19622). Mitochondrial membrane potential (MMP) and adenosine triphosphate (ATP) levels were determined in dissociated brain cells (DBC). Respiration was analyzed in mitochondria isolated from brain tissue. Citrate synthase (CS) activ…

0301 basic medicinemedicine.medical_specialtyRespiratory chainMice TransgenicMitochondrionLipid peroxidation03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicineDevelopmental NeuroscienceAlzheimer DiseaseInternal medicineMembrane fluidityAmyloid precursor proteinmedicineCitrate synthaseAnimalsHumansCholestenonesAmyloid beta-PeptidesbiologyBrainRotenoneMitochondriaMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinologyHEK293 CellsNeurologychemistrybiology.proteinOlesoximeFemale030217 neurology & neurosurgeryExperimental neurology
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