Search results for "Parasympathomimetics"

showing 10 items of 13 documents

Effects of several muscarinic agonists on cardiac performance and the release of noradrenaline from sympathetic nerves of the perfused rabbit heart

1972

Summary 1 The effects of several muscarinic agonists on atrial tension development, ventricular rate and noradrenaline release from terminal sympathetic fibres evoked by electrical nerve stimulation (SNS) and 1,1-dimethyl-4-phenylpiperazinium (DMPP) were measured in isolated perfused rabbit hearts. 2 Hexamethonium, in a concentration which almost abolished the release of noradrenaline by DMPP, had no effect on the release produced by SNS, confirming that the stimulation was postganglionic. 3 The order of potency for inhibition of atrial tension development was N-methyl-1,2,5,6, tetrahydro-nicotinic acid prop-2-yne ester (MH-1)>oxotremorine > acetylcholine > methacholine > carbachol > furtre…

AtropineMalemedicine.medical_specialtySympathetic Nervous SystemCarbacholAutopharmacologyHexamethonium CompoundsIn Vitro TechniquesPharmacologyNorepinephrinechemistry.chemical_compoundHeart RateInternal medicineMuscarinic acetylcholine receptormedicineOxotremorineAnimalsMethacholine CompoundsPharmacologyChemistryOxotremorinePilocarpineHeartAcetylcholineElectric StimulationPerfusionQuaternary Ammonium CompoundsAtropineEndocrinologyParasympathomimeticsPilocarpineCarbacholFemaleMethacholineHexamethoniumCarbamatesRabbitsDimethylphenylpiperazinium IodideAcetylcholinemedicine.drugBritish Journal of Pharmacology
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A muscarinic inhibition of the noradrenaline release evoked by postganglionic sympathetic nerve stimulation

1969

1. The noradrenaline output from isolated rabbit hearts perfused with Tyrode solution was estimated fluorimetrically. The postganglionic sympathetic nerves of the heart were stimulated (10 shocks/sec; 1 msec) for three 1 min periods with intervals of 10 min. 2. The noradrenaline output evoked by 3 consecutive stimulation periods decreased exponentially. 3. Acetylcholine (10−9–10−6 g/ml) administered continuously one min before to one min after the second stimulation caused a dose-dependent reduction of the noradrenaline output evoked by the second stimulation to as low as 19% of the normal value. Acetylcholine in the concentrations applied did not cause a noradrenaline output by itself. 4. …

AtropineMalemedicine.medical_specialtySympathetic Nervous SystemTyramineStimulationHexamethonium CompoundsIn Vitro TechniquesPiperazinesNorepinephrinechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsFluorometryReceptors CholinergicPharmacologyHeartAdrenergic nervous systemGeneral MedicineCoronary VesselsAcetylcholineElectric StimulationReceptors AdrenergicPerfusionQuaternary Ammonium CompoundsAtropineEndocrinologyParasympathomimeticschemistryDepression ChemicalAutonomic Fibers PostganglionicCholinergicFemaleHexamethoniumMethacholineRabbitsAcetylcholinemedicine.drugNaunyn-Schmiedebergs Archiv f�r Pharmakologie
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Absence of muscarinic modulation of vasopressin release from the isolated rat neurohypophysis

1975

1. Isolated rat neurohypophyses were incubated in Locke solution at 37°C and the vasopressin output into the medium determined by bioassay. 2. Potassium chloride 60 mM caused a 9-fold increase in the rate of vasopressin release that was abolished when calcium chloride was omitted from the Locke solution. 3. Acetylcholine 5.5×10−4 M neither alone nor in the presence of atropine 2.9×10−6 M changed the “resting” release of vasopressin. 4. Neither acetylcholine 5.5×10−4 M nor oxotremorine 10−4 and 3×10−4 M altered the vasopressin release evoked by potassium chloride 60 mM. 5. In contrast to the peripheral adrenergic nerve fibres, the secretory terminal fibres of the neurohypophysis do not appea…

Atropinemedicine.medical_specialtyVasopressinVasopressinschemistry.chemical_elementIn Vitro TechniquesCalciumInhibitory postsynaptic potentialPotassium ChloridePituitary Gland PosteriorInternal medicineMuscarinic acetylcholine receptormedicineOxotremorineAnimalsPharmacologyNeurosecretionChemistryOxotremorineGeneral MedicineAcetylcholineRatsAtropineEndocrinologyNicotinic agonistParasympathomimeticsCalciumFemaleAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Atropine-resistant effects of the muscarinic agonists McN-A-343 and AHR 602 on cardiac performance and the release of noradrenaline from sympathetic …

1974

Abstract 1 The effects of 4-(m-chlorophenylcarbamoyloxy)-2-butynyltrimethylammonium chloride (McN-A-343) and N-benzyl-3-pyrrolidyl acetate methobromide (AHR 602) on cardiac performance and noradrenaline release from terminal sympathetic fibres were measured in isolated perfused hearts of rabbits. 2 In the presence of sufficient atropine to block muscarinic receptors, high concentrations of McN-A-343 and AHR 602 caused no cardiac stimulation and there was no increase in the resting output of noradrenaline into the perfusates. 3 McN-A-343 and AHR 602 increased both the mechanical responses and the transmitter overflow evoked by electrical stimulation of the sympathetic nerves (SNS) but inhibi…

ChronotropicAtropineMalemedicine.medical_specialtyPyrrolidinesSympathetic Nervous SystemStimulationAutopharmacologyHexamethonium CompoundsPharmacologyIn Vitro TechniquesPiperazinesHexamethonium compoundchemistry.chemical_compoundNorepinephrineCocaineInternal medicineDesipramineMuscarinic acetylcholine receptorBenzyl CompoundsmedicineAnimalsPharmacologyNeuronsHeartHydrogen-Ion ConcentrationAcetylcholineElectric StimulationPerfusionQuaternary Ammonium CompoundsAtropineEndocrinologychemistryParasympathomimeticsHexamethoniumFemaleCarbamatesRabbitsDimethylphenylpiperazinium IodideAcetylcholinemedicine.drug
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Nicotinic and muscarinic modulation of 5-hydroxytryptamine (5-HT) release from porcine and canine small intestine

1992

Strips of porcine and canine small intestine were incubated in vitro and the release of 5-hydroxytryptamine (5-HT) was determined by HPLC with electrochemical detection. The spontaneous outflow of 5-HT from the porcine and canine small intestine largely reflects calcium-dependent 5-HT secretion from enterochromaffin cells which are under a spontaneous neuronal, excitatory input as indicated by the inhibitory effect (30-40%) of tetrodotoxin. In both species, nicotine enhanced the release of 5-HT in a concentration-dependent manner by a maximum of about 50% at 100 microM. This effect was blocked by the nicotine receptor antagonist hexamethonium, but not by the subtype-selective nicotine recep…

MaleNicotineSerotoninmedicine.medical_specialtySwineScopolamineHexamethonium CompoundsTetrodotoxinReceptors NicotinicBiologyHexamethoniumNicotine03 medical and health scienceschemistry.chemical_compoundDogs0302 clinical medicineInternal medicineIntestine SmallDrug DiscoveryMuscarinic acetylcholine receptorEnterochromaffin CellsmedicineOxotremorineAnimalsGenetics (clinical)030304 developmental biology0303 health sciencesMuscarineOxotremorineParasympatholyticsGeneral MedicineHydroxyindoleacetic AcidBungarotoxinsReceptors MuscarinicAcetylcholine3. Good healthNicotinic agonistEndocrinologyParasympathomimeticschemistryEnterochromaffin cellMolecular MedicineCalciumFemaleHexamethoniumDimethylphenylpiperazinium Iodide030217 neurology & neurosurgeryAcetylcholinemedicine.drugThe Clinical Investigator
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A useful muscarinic parameter and the differential recording of atrial and ventricular tension in the perfused rabbit heart

1971

1. A simple method of recording right atrial tension from the Langendorff perfused rabbit has been described; it is based on the “transverse” method of recording cardiac contractions described by Beckett (1970). 2. Right atrial and ventricular tensions were recorded by transducers attached to threads stitched into the right atrium and right ventricle, the heart being retained and prevented from rotating by two threads stitched into the intraventricular septum as described by Beckett. 3. Right ventricular diastolic tension was adjusted to 7.5 g. Interference with the atrial record by ventricular systole was overcome by adjustment of diastolic right atrial tension to its optimal value between…

Malemedicine.medical_specialtyHeart VentriclesTransducersDiastoleBlood PressureHeart RateInternal medicineMuscarinic acetylcholine receptormedicineAnimalsVentricular FunctionHeart Atriacardiovascular diseasesSympathomimeticsPharmacologyCardiac cyclebusiness.industryP waveHeartVagus NerveGeneral MedicineAtrial FunctionStimulation ChemicalCardiovascular physiologyGanglionPerfusionmedicine.anatomical_structureParasympathomimeticsVentricleMuscle TonusAnesthesiacardiovascular systemCardiologyFemaleRabbitsbusinessPerfusionMuscle ContractionNaunyn-Schmiedebergs Archiv f�r Pharmakologie
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Distigmine bromide induced acute psychotic disorder in a patient with multiple sclerosis.

2003

AbstractA female patient with multiple sclerosis (MS) suffered from an acute psychotic disorder after taking distigmine bromide for detrusor dysfunction. She showed a dramatic relief of her symptoms after the medication, distigmine bromide, was stopped. Distigmine is not supposed to penetrate the blood-brain barrier (BBB). However, in MS patients a leakage of the BBB could be hypothesized.

Malemedicine.medical_specialtyPsychosisMultiple Sclerosismedicine.medical_treatmentPyridinium CompoundsSuicide AttemptedBlood–brain barrierPsychoses Substance-InducedCentral nervous system diseasechemistry.chemical_compoundBromidemedicineHumansDistigminePsychiatric Status Rating ScalesChemotherapybusiness.industryMultiple sclerosisUrinary Retentionmedicine.diseasePathophysiologySurgeryPsychiatry and Mental healthmedicine.anatomical_structurechemistryParasympathomimeticsAnesthesiaFemalebusinessmedicine.drugEuropean psychiatry : the journal of the Association of European Psychiatrists
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Some pharmacological properties of the false cholinergic transmitter acetylpyrrolidinecholine and its precursor pyrrolidinecholine

1976

The acetylchline analogue acetylpyrrolidinecholine as well as the choline analogue pyrrolidinecholine were synthesized and the cholinergic properties of both substances were investigated on the guinea-pig ileum, rat blood pressure and frog rectus abdominis muscle. Acetylpyrrolidinecholine was 3-5 times less potent than acetylcholine on the three preparations tested. The dose-response curves to acetylpyrrolidinecholine were shifted to the right in a parallel manner by atropine and (+)-tubocurarine. The dissociation constants for atropine and (+)-tubocurarine obtained with acetylpyrrolidinecholine as agonist were not different from those obtained with acetylcholine. This indicates that acetyl…

Malemedicine.medical_specialtyPyrrolidinesGuinea PigsRana temporariaNeuromuscular JunctionNeuromuscular transmissionBlood PressureReceptors NicotinicSynaptic TransmissionCholinechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineAnimalsAcetylcholine receptorPharmacologyMuscarineMuscarinic acetylcholine receptor M3Muscle SmoothGeneral MedicineReceptors MuscarinicAcetylcholineRatsNicotinic agonistEndocrinologyParasympathomimeticschemistryCholinergicAcetylcholineMuscle Contractionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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EFFECTS OF VANADATE ON RESPONSES OF GUINEA-PIG ISOLATED TRACHEA TO SPASMOGENS

1993

Abstract The effects of vanadate on the contractility of the guinea-pig isolated trachea was examined. Vanadate (0·1 Mm) produced a sustained contraction that was abolished in Ca2+-free EGTA (0·1 Mm)-containing physiological salt solution but was resistant to verapamil (1 μm). Vanadate (0·1 Mm) depressed tracheal responses to CaCl2 (in Ca2+-free depolarizing solution), KCl, acetylcholine, histamine and 5-hydroxytryptamine. For vanadate (10 μm), the inhibition of spasmogenic responses only reached statistical significance for histamine and 5-hydroxytryptamine. Caffeine (1 Mm)-induced spasm (trachea at 20°C in the presence of indomethacin (2·8 μm)) was not affected by vanadate (10 μm-0·1 Mm).…

Malemedicine.medical_specialtySerotoninContraction (grammar)Guinea PigsPharmaceutical ScienceIn Vitro TechniquesPotassium ChlorideContractilitychemistry.chemical_compoundInternal medicineCaffeinemedicineAnimalsVanadatePharmacologyChemistryCell MembraneMuscle SmoothAcetylcholineTracheaEGTAEndocrinologyParasympathomimeticsVerapamilVerapamilCalciumFemalemedicine.symptomVanadatesAcetylcholineHistaminemedicine.drugMuscle contractionHistamineMuscle Contraction
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A muscarinic mechanism inhibiting the release of noradrenaline from peripheral adrenergic nerve fibres by nicotinic agents.

1968

SympathomimeticsMalemedicine.medical_specialtySympathetic nervous systemSympathetic Nervous SystemReceptors DrugGuinea PigsAdrenergicParasympathomimeticsPharmacologyPiperazinesNorepinephrine (medication)NorepinephrineHeart Conduction SystemInternal medicineMuscarinic acetylcholine receptormedicineAnimalsHeart AtriaSympathomimeticsDrug AntagonismChemistryGeneral MedicinePeripheralPerfusionmedicine.anatomical_structureEndocrinologyParasympathomimeticsFemaleRabbitsDrug Antagonismmedicine.drugResearch Article
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