Search results for "Peroxidation"

showing 10 items of 308 documents

Hypertensive status and lipoprotein oxidation in an elderly population at high cardiovascular risk.

2008

BACKGROUND: In elderly individuals, hypertension is a main risk factor for cardiovascular disease and oxidative damage is increased. Our aim was to assess the relationship between the degree of in vivo low-density lipoprotein (oxLDL) oxidation and the hypertensive status in a elderly population at high cardiovascular risk. METHODS: Cross-sectional study with baseline data from the PREDIMED (PREvención con DIeta MEDiterránea) trial, an intervention study directed at testing the efficacy of the Mediterranean diet on the primary prevention of cardiovascular disease. Participants were 1,130 subjects at high cardiovascular risk aged 55-80. At baseline, in vivo circulating oxLDL was measured, and…

MaleCoronary DiseaseDiet MediterraneanSeverity of Illness IndexRisk groupsRisk FactorsMedicineAged 80 and overCoronary diseaseBioquímica y tecnologíaIncidence0895-7061Follow up studiesAge FactorsMiddle AgedPrognosisBiochemistry and technologyHypertensionChristian ministrylipids (amino acids peptides and proteins)HipertensióFemaleHypertensive StatusLypoproteinsmedicine.medical_specialtyLipoproteinsLipid peroxidationEnzyme-Linked Immunosorbent AssayBioquímica i biotecnologiaEnvironmental healthElderly populationInternal medicineInternal MedicineHumansLipoprotein oxidationLipoprotein OxidationAgedRetrospective Studiesbusiness.industryLipoproteïnes -- OxidacióUnited StatesPersones grans -- NutricióElderly PopulationOxidative StressEndocrinologyCross-Sectional StudiesMulticenter studyOxidative stressHypertension complicationsLipid PeroxidationbusinessOlive oilFollow-Up StudiesAmerican journal of hypertension
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Stock-specific variation of trophic position, diet and environmental stress markers in Atlantic salmon Salmo salar during feeding migrations in the B…

2012

This study investigated stock-specific variation in selected ecophysiological variables during the feeding migrations of Atlantic salmon Salmo salar in the Baltic Sea. Oxidative stress biomarkers and EROD (ethoxyresorufin-O-deethylase, Cyp1A enzyme) activity were used as indicators of possible environmental stress and stable isotopes as determinants of diet and trophic position. Latvian S. salar stocks Daugava and Gauja had distinct stable-isotope signatures compared to the other stocks, indicating differences in migration patterns, residency or arrival times, or dietary specialization among stocks. Salmo salar originating from Daugava and Gauja also had lower catalase enzyme activity than …

MaleForagingSalmo salarAquatic ScienceBiologyEnvironmentEnvironmental stressPredationCytochrome P-450 CYP1A1AnimalsSalmoEcology Evolution Behavior and SystematicsStock (geology)Trophic levelCarbon IsotopesNitrogen IsotopesEcologySequence Analysis DNAbiology.organism_classificationGlutathioneDietOxidative StressBaltic seaAnimal MigrationFemaleLipid PeroxidationBayBiomarkersMicrosatellite RepeatsJournal of fish biology
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Increased Oxidative Damage Associated with Unfavorable Cytogenetic Subgroups in Chronic Lymphocytic Leukemia

2014

Oxidative stress contributes to genomic instability in chronic lymphocytic leukemia (CLL), but its relationship with the acquisition of specific chromosomal abnormalities is unknown. We recruited 55 untreated CLL patients and assessed 8-oxo-2′-deoxyguanosine (8-oxo-dG), glutathione, and malondialdehyde (MDA) levels, and we compared them among the cytogenetic subgroups established using fluorescence in situ hybridization (FISH). Significant increases in 8-oxo-dG and/or MDA were observed in patients with unfavorable cytogenetic aberrations (17p and 11q deletions) compared to the 13q deletion group.TP53deletion patients exhibited a diminished DNA repair efficiency. Finally, cases with normal F…

MaleGenome instabilityArticle SubjectDNA RepairDNA damageDNA repairChronic lymphocytic leukemialcsh:MedicineBiologymedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyCohort Studieschemistry.chemical_compoundMalondialdehydemedicineHumansLymphocytesIn Situ Hybridization FluorescenceAgedAged 80 and overChromosome AberrationsGeneral Immunology and Microbiologymedicine.diagnostic_testlcsh:RDeoxyguanosineGeneral MedicineGlutathioneMiddle AgedMalondialdehydemedicine.diseaseGlutathioneLeukemia Lymphocytic Chronic B-CellOxidative Stresschemistry8-Hydroxy-2'-DeoxyguanosineImmunologyFemaleLipid PeroxidationReactive Oxygen SpeciesGene DeletionOxidative stressDNA DamageResearch ArticleFluorescence in situ hybridizationBioMed Research International
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Protein oxidation in chronic kidney disease.

2013

An imbalance between oxidative processes and antioxidant systems has been widely demonstrated in chronic kidney diseases (CKD). In this study we enrolled 26 healthy subjects, 27 patients with CKD on conservative treatment (CT-CKD) with various degrees of renal failure, and 31 CKD subjects in haemodialysis treatment (HD-CKD), evaluated before and after a standard haemodialysis session. In each group we measured protein carbonyl groups (PC) as an index of protein oxidation, lipid peroxidation (TBARS) and two plasma markers of leukocyte activation, elastase and myeloperoxidase (MPO). In CT-CKD subjects the PC level was significantly higher than in normal controls, and it was negatively correla…

MaleHEMODIALYSISmedicine.medical_specialtySettore MED/09 - Medicina InternaPhysiologyBIOMARKERSRenal functionurologic and male genital diseasesProtein oxidationThiobarbituric Acid Reactive SubstancesLipid peroxidationDiabetes Complicationschemistry.chemical_compoundCARBONYL STRESSMARKERSINFLAMMATIONGlycationRenal DialysisPhysiology (medical)Internal medicinemedicineTBARSHumansRenal Insufficiency ChronicPeroxidasebiologyPancreatic Elastasebusiness.industryNITRIC-OXIDE METABOLITESElastaseHematologyMiddle Agedmedicine.diseasefemale genital diseases and pregnancy complicationsOxidative StressEndocrinologychemistryMyeloperoxidaseNITRIC-OXIDE METABOLITES; CHRONIC-RENAL-FAILURE; CARBONYL STRESS; HEMODIALYSIS; BIOMARKERS; MARKERS; INFLAMMATIONImmunologybiology.proteinFemaleCHRONIC-RENAL-FAILURELipid PeroxidationCardiology and Cardiovascular MedicinebusinessOxidation-ReductionKidney diseaseClinical hemorheology and microcirculation
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Quantitative evaluation of ethane and n-pentane as indicators of lipid peroxidation in vivo.

1983

The use of exhalation of ethane and n-pentane in experimental animals as parameters of lipid peroxidation led to an examination of pharmacokinetics of both compounds in rats. When rats were exposed, in a closed desiccator jar chamber, to a wide range of ethane concentrations, linear elimination pharmacokinetics were observed. n-Pentane, when concentrations higher than 100 ppm were applied, displayed saturation kinetics. These were formally explained by action of two competing metabolizing pathways or enzymes. Application of preexisting models could describe exhalation of both ethane and n-pentane by untreated control rats. Stimulation of lipid peroxidation by ferrous ions or by carbon tetra…

MaleLipid PeroxidesChromatography GasHealth Toxicology and MutagenesisIronKineticsInorganic chemistryPentanesToxicologyLipid peroxidationchemistry.chemical_compoundPharmacokineticsPentanesAnimalsCarbon TetrachlorideEthaneChromatographyExhalationRats Inbred StrainsGeneral MedicineMetabolismRatsPentaneKineticschemistryBreath TestsCarbon tetrachlorideArchives of toxicology
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4-Hydroxynonenal, a lipid peroxidation product, induces relaxation of human cerebral arteries.

1994

The relaxant effect of 4-hydroxynonenal (4-HNE), a lipid peroxidation product, on human cerebral arteries was studied. Addition of 4-HNE to artery rings promoted no contraction, and after stimulation with prostaglandin F2α (PFG2α; 10−7-3 × 10−6 M), 100% relaxation was obtained with 3 × 10−5 M 4-HNE. Inhibition of nitric oxide formation with NG-nitro-l-arginine methyl ester hydrochloride (l-NAME; (10−4 M), as well as prostaglandin synthesis with indomethacin (3 × 10−6 M), partially prevented 4-HNE-induced relaxation, but each of these substances separately failed to inhibit complete relaxation. Addition of both inhibitors together reduced 4-HNE-induced relaxation to ≈50%, but relaxation cou…

MaleLipid PeroxidesContraction (grammar)EndotheliumIndomethacinCerebral arteriesStimulationVasodilationArginineDinoprostNitric Oxide4-HydroxynonenalNitric oxideLipid peroxidationchemistry.chemical_compoundCadavermedicineHumansAgedAged 80 and overAldehydesDose-Response Relationship DrugChemistryOsmolar ConcentrationCerebral ArteriesMiddle AgedVasodilationNG-Nitroarginine Methyl Estermedicine.anatomical_structureNeurologyBiochemistryBiophysicsEndothelium VascularNeurology (clinical)Cardiology and Cardiovascular Medicine
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Effect of acidosis and anoxia on iron delocalization from brain homogenates.

1992

Abstract Cortical homogenates were prepared from rat brain in Krebs-Ringer phosphate media adjusted to pH 7, 6 or 5 and incubated for 1 hr under aerotic or anaerobic conditions in the presence of dipyridyl, an iron chelator. Low molecular weight species (LMWS) iron was measured spectrophotometrically after passing of the homogenates through a 10,000- M , ultrafiltration membrane. Following aerobic incubation, LMWS iron reached 1.24 μg/g tissue at pH 7, and increased 1.7-fold at pH 6 and 3.1-fold at pH 5. Anoxia enhanced significantly the amount of ultrafiltrable iron at the three pH values, the LMWS iron level being increased by 190% at pH 7, by 113% at pH 6, and by 77% at pH 5. Addition of…

MaleLipid PeroxidesThiobarbituric acidIronAscorbic AcidBiochemistryLipid peroxidationchemistry.chemical_compound22'-DipyridylmedicineAnimalsChelationFerrous CompoundsHypoxia BrainAcidosisPharmacologyBrain ChemistryRats Inbred StrainsHydrogen-Ion ConcentrationPhosphateRatsOxygenUltrafiltration (renal)chemistryBiochemistryLipid Peroxidationmedicine.symptomAcidosisAnaerobic exerciseIntracellularBiochemical pharmacology
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Endurance training reduces the susceptibility of mouse skeletal muscle to lipid peroxidation in vitro

1983

Selected estimates of the lipid peroxidative capacity were assayed in the red and white skeletal muscles of control and endurance-trained mice. Endurance training decreased the lipid peroxidation rate in vitro in both muscle types. The concentration of lipids susceptible to Fe2+-induced lipid peroxidation was greater in the red than in the white skeletal muscle and increased after endurance training in the red muscle. Endurance training, however, decreased highly significantly the sensitivity of red muscle to in vitro stimulated lipid peroxidation. The activity of catalase and the concentration of vitamin E were considerably higher in the red muscle, whereas the activity of glutathione pero…

MaleLipid Peroxidesmedicine.medical_specialtyPhysiologymedicine.medical_treatmentMice Inbred StrainsLipid peroxidationMicechemistry.chemical_compoundEndurance trainingInternal medicinemedicineAnimalschemistry.chemical_classificationbiologyChemistryMusclesVitamin EGlutathione peroxidaseSkeletal muscleGlutathioneLipid MetabolismIn vitroEndocrinologymedicine.anatomical_structureCatalasePhysical Endurancebiology.proteinOxidation-ReductionActa Physiologica Scandinavica
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Increased toxicity of cocaine on human hepatocytes induced by ethanol: role of GSH.

1999

Increased toxicity of cocaine to human hepatocytes is observed when cells are simultaneously incubated with ethanol. Ethanol might exacerbate cocaine hepatocyte toxicity by three different pathways: a) by increasing the oxidative metabolism of cocaine and hence the oxidative damage; b) by the formation of a more toxic metabolite, namely cocaethylene; or c) by decreasing the defence mechanisms of the cell (i.e. GSH). In the present study, experiments were conducted to investigate the feasibility of these hypotheses. In hepatocytes preincubated for 48 hr with ethanol, neither significant changes in cocaine metabolism nor cytotoxicity were found despite differences in hepatocyte p-nitrophenol …

MaleLiver cytologyCell SurvivalPharmacologymedicine.disease_causeBiochemistryLipid peroxidationchemistry.chemical_compoundCocaethyleneCocaineDopamine Uptake InhibitorsmedicineHumansCells CulturedAgedGlutathione TransferasePharmacologyEthanolDrug SynergismGlutathioneCYP2E1Middle AgedOxidative Stressmedicine.anatomical_structurechemistryBiochemistryLiverHepatocyteToxicityFemaleOxidative stressmedicine.drugBiochemical pharmacology
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AZT induces oxidative damage to cardiac mitochondria: Protective effect of vitamins C and E

2004

Abstract AZT (zidovudine) is a potent inhibitor of HIV replication and a major antiretroviral drug used for AIDS treatment. A major limitation in the use of AZT is the occurrence of severe side effects. The aim of this work was to test whether AZT causes oxidative damage to heart mitochondria and whether this can be prevented by supranutritional doses of antioxidant vitamins. An experimental animal model was used in which mice were treated with AZT for 35 days (10 mg/kg/day) in drinking water. Animals treated with antioxidant vitamins were fed the same diet as controls but supplemented with vitamins C (ascorbic acid, 10 g/ kg diet) and E (α-dl-tocopherol, 0.6 g/kg diet) for 65 days before s…

MaleMitochondrial Diseasesmedicine.medical_treatmentAscorbic AcidOxidative phosphorylationMitochondrionPharmacologyBiologymedicine.disease_causeDNA MitochondrialMitochondria HeartGeneral Biochemistry Genetics and Molecular BiologyLipid peroxidationMiceZidovudinechemistry.chemical_compoundmedicineAnimalsVitamin Eheterocyclic compoundsGeneral Pharmacology Toxicology and PharmaceuticsVitamin CVitamin EDeoxyguanosineGeneral MedicineAscorbic acidGlutathioneBiochemistrychemistry8-Hydroxy-2'-DeoxyguanosineLipid PeroxidationZidovudineOxidative stressmedicine.drugLife Sciences
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