Search results for "Phosphorylation"

showing 10 items of 975 documents

Light-Dependent Translocation of Arrestin in Rod Photoreceptors is Signaled through a Phospholipase C Cascade and Requires ATP

2009

Light adaptation of rod photoreceptors induces translocation of arrestin from inner segments (IS) to outer segments (OS). Our study suggests that components of the G-protein linked phosphoinositide pathway play a role in signaling the initiating events of arrestin translocation. We show that arrestin translocation can be stimulated by activators of phospholipase C (PLC) and protein kinase C (PKC) in the absence of light. Conversely, arrestin translocation to the OS is significantly slowed by inhibitors of PLC and PKC.In the second part of this study, we investigated the mechanism by which arrestin translocates in response to light. Other investigators have suggested that arrestin translocat…

Cholera ToxinLightgenetic structuresG proteinBiophysicsXenopusChromosomal translocationBiologyPhosphatidylinositolsArticleMiceXenopus laevisAdenosine TriphosphateRetinal Rod Photoreceptor CellsArrestinAnimalsEnzyme InhibitorsPotassium CyanideCells CulturedProtein Kinase CProtein kinase CArrestinPhosphoinositide PathwayPhospholipase CChemistryCell Biologybiology.organism_classificationeye diseasesCell biologyRhodopsinType C Phospholipasesbiology.proteinPhosphorylationArrestin beta 2Arrestin beta 1sense organsSignal transductionSignal TransductionBiophysical Journal
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Dissection of the elements of osmotic stress response transcription factor Hot1 involved in the interaction with MAPK Hog1 and in the activation of t…

2013

Abstract The response to hyperosmotic stress is mediated by the HOG pathway. The MAP kinase Hog1 activates several transcription factors, regulates chromatin-modifying enzymes and, through its interaction with RNA polymerase II, it directs this enzyme to osmotic stress-controlled genes. For such targeting, this kinase requires the interaction with transcription factors Hot1 and Sko1. However, phosphorylation of these proteins by Hog1 is not required for their functionality. In this study, we aim to identify the Hot1 elements involved in Hog1-binding and in the activation of transcription. Two-hybrid experiments demonstrated that the Hot1 sequence between amino acids 340 and 534 and the CD e…

Chromatin ImmunoprecipitationSaccharomyces cerevisiae ProteinsTranscription GeneticResponse elementBiophysicsRNA polymerase IIE-boxSaccharomyces cerevisiaeReal-Time Polymerase Chain ReactionResponse ElementsBiochemistryOsmoregulationStructural BiologyGene Expression Regulation FungalGeneticsImmunoprecipitationRNA MessengerPhosphorylationPromoter Regions GeneticMolecular BiologyTranscription factorRNA polymerase II holoenzymeGeneral transcription factorbiologyReverse Transcriptase Polymerase Chain ReactionChromatinBiochemistrybiology.proteinTranscription factor II DMitogen-Activated Protein KinasesTranscription factor II BProtein BindingTranscription FactorsBiochimica et biophysica acta
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Forever Young: Structural Stability of Telomeric Guanine Quadruplexes in the Presence of Oxidative DNA Lesions**

2021

International audience; Human telomeric DNA, in G-quadruplex (G4) conformation, is characterized by a remarkable structural stability that confers it the capacity to resist to oxidative stress producing one or even clustered 8-oxoguanine (8oxoG) lesions. We present a combined experimental/computational investigation, by using circular dichroism in aqueous solutions, cellular immunofluorescence assays and molecular dynamics simulations, that identifies the crucial role of the stability of G4s to oxidative lesions, related also to their biological role as inhibitors of telomerase, an enzyme overexpressed in most cancers associated to oxidative stress.

Circular dichroismTelomeraseOxidative phosphorylation010402 general chemistryImmunofluorescencemedicine.disease_cause01 natural scienceselectronic circular dichroismCatalysis[SPI.AUTO]Engineering Sciences [physics]/Automaticchemistry.chemical_compoundmedicineHumansimmunofluorescenceTelomerasechemistry.chemical_classificationmedicine.diagnostic_test010405 organic chemistryCircular DichroismOrganic ChemistryDNAGeneral ChemistryTelomeremolecular dynamics0104 chemical sciences3. Good healthG-QuadruplexesOxidative StressEnzymeBiochemistrychemistrySettore CHIM/03 - Chimica Generale E InorganicaGuanine quadruplexesNucleic Acid Conformationoxidative DNA lesionsGuanine-QuadruplexesDNAOxidative stress
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Inhibition of activated STAT5 in Bcr/Abl expressing leukemia cells with new pimozide derivatives.

2014

STATs are transcription factors acting as intracellular signaling after stimulation with cytokines, growth factors and hormones. STAT5 is also constitutively active in many forms of cancers, including chronic myelogenous leukemia, acute lymphoblastic leukemia and Hodgkin's lymphoma. Recently, literature reported that the neuroleptic drug pimozide inhibits STAT5 phosphorylation inducing apoptosis in CML cells. We undertook an investigation from pimozide structure, obtaining simple derivatives with cytotoxic and STAT5-inhibitory activity, two of them markedly more potent than pimozide.

Clinical BiochemistryFusion Proteins bcr-ablPharmaceutical ScienceApoptosisBiochemistrySettore MED/15 - Malattie Del SangueCell LineStructure-Activity RelationshipPimozideSettore BIO/13 - Biologia Applicatahemic and lymphatic diseasesDrug DiscoverymedicineSTAT5 Transcription FactorCytotoxic T cellHumansPhosphorylationMolecular BiologyTranscription factorSTAT5Cell ProliferationbiologyDose-Response Relationship DrugMolecular StructureChemistrySTAT5 inhibitorsPimozideBCR/ABL expressing leukemia ApoptosisCell growth inhibitionOrganic ChemistryCell CyclePimozidemedicine.diseaseSettore CHIM/08 - Chimica FarmaceuticaLeukemiaApoptosisCancer researchbiology.proteinSettore BIO/14 - FarmacologiaMolecular MedicinePhosphorylationK562 Cellsmedicine.drugChronic myelogenous leukemia
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Characterization of a Novel Type of Serine/Threonine Kinase That Specifically Phosphorylates the Human Goodpasture Antigen

1999

Goodpasture disease is an autoimmune disorder that occurs naturally only in humans. Also exclusive to humans is the phosphorylation process that targets the unique N-terminal region of the Goodpasture antigen. Here we report the molecular cloning of GPBP (Goodpasture antigen-binding protein), a previously unknown 624-residue polypeptide. Although the predicted sequence does not meet the conventional structural requirements for a protein kinase, its recombinant counterpart specifically binds to and phosphorylates the exclusive N-terminal region of the human Goodpasture antigen in vitro. This novel kinase is widely expressed in human tissues but shows preferential expression in the histologic…

Collagen Type IVMolecular Sequence DataSaccharomyces cerevisiaeProtein Serine-Threonine KinasesMolecular cloningBiologymedicine.disease_causeAutoantigensBiochemistryCell LineAutoimmunitymedicineHumansAmino Acid SequenceCloning MolecularPhosphorylationMolecular BiologyPeptide sequenceCeramide Transfer ProteinSerine/threonine-specific protein kinaseBase SequenceSequence Homology Amino AcidKinaseCell BiologyCeramide transportImmunohistochemistryCell biologyBiochemistryProtein BiosynthesisPhosphorylationCollagenJournal of Biological Chemistry
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The Divergent Effect of Insulin-Like Growth Factor Binding Protein (IGFBP) - 1 on IGF-induced Steroidogenesis in Bovine Adrenocortical Cells is not d…

2007

In previous studies we have shown that insulin-like growth factor (IGF)-II stimulates basal as well as ACTH-induced cortisol secretion from bovine adrenocortical cells more potently than IGF-I. The steroidogenic effect of both, IGF-I and IGF-II, is mediated through the IGF-I receptor and modified by locally produced IGF binding proteins. Further studies demonstrate that bovine adrenocortical cells do synthesize IGFBP-1 to -4 and that their secretion is regulated differentially by IGFs and ACTH. Since IGFBP-1 selectively is induced 3- to 4- fold by ACTH, the aim of the following studies was to evaluate the effect of IGFBP-1 on IGF-induced cortisol secretion from bovine adrenocortical cells i…

Cortisol secretionmedicine.medical_specialtyHydrocortisoneEndocrinology Diabetes and Metabolismmedicine.medical_treatmentBiologyCulture Media Serum-FreeInsulin-like growth factor-binding proteinInsulin-like growth factorEndocrinologyAdrenocorticotropic HormoneSomatomedinsInternal medicineInternal MedicinemedicineAnimalsPhosphorylationReceptorCells CulturedAdrenal cortexGrowth factorGeneral MedicineLigand (biochemistry)Insulin-Like Growth Factor Binding Protein 1KineticsEndocrinologymedicine.anatomical_structureInsulin-like growth factor 2Adrenal Cortexbiology.proteinCattlehormones hormone substitutes and hormone antagonistsExperimental and Clinical Endocrinology & Diabetes
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ChemInform Abstract: Enantioselective Palladium-Catalyzed Oxidative β,β-Fluoroarylation of α,β-Unsaturated Carbonyl Derivatives.

2016

The site-selective palladium-catalyzed three-component coupling of deactivated alkenes, arylboronic acids, and N-fluorobenzenesulfonimide is disclosed herein. The developed methodology establishes a general, modular, and step-economical approach to the stereoselective β-fluorination of α,β-unsaturated systems.

Coupling (electronics)ChemistryEnantioselective synthesisCarbonyl derivativesHalogenationchemistry.chemical_elementStereoselectivityGeneral MedicineOxidative phosphorylationCombinatorial chemistryCatalysisPalladiumChemInform
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ChemInform Abstract: Versatile Oxidative Approach to Carbazoles and Related Compounds Using MoCl5.

2014

Oxidative intramolecular coupling of tertiary amines with at least two electron-rich arenes gives carbazoles in good to excellent yields.

Coupling (electronics)Computational chemistryChemistryIntramolecular forceeducationGeneral MedicineOxidative phosphorylationhumanitiesChemInform
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ChemInform Abstract: Powerful Fluoroalkoxy Molybdenum(V) Reagent for Selective Oxidative Arene Coupling Reaction.

2014

A novel dinuclear fluoroalkoxy Mo(V)-complex is efficient as reagent for the oxidative arene coupling of electron-rich arenes with superior reactivity compared to MoCl5 and MoCl5/TiCl4.

Coupling (electronics)chemistryMolybdenumReagentPolymer chemistrychemistry.chemical_elementReactivity (chemistry)General MedicineOxidative phosphorylationCoupling reactionChemInform
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Calmodulin binds to p21(Cip1) and is involved in the regulation of its nuclear localization.

1999

p21(Cip1), first described as an inhibitor of cyclin-dependent kinases, has recently been shown to have a function in the formation of cyclin D-Cdk4 complexes and in their nuclear translocation. The dual behavior of p21(Cip1) may be due to its association with other proteins. Different evidence presented here indicate an in vitro and in vivo interaction of p21(Cip1) with calmodulin: 1) purified p21(Cip1) is able to bind to calmodulin-Sepharose in a Ca(2+)-dependent manner, and this binding is inhibited by the calmodulin-binding domain of calmodulin-dependent kinase II; 2) both molecules coimmunoprecipitate when extracted from cellular lysates; and 3) colocalization of calmodulin and p21(Cip…

Cyclin-Dependent Kinase Inhibitor p21CalmodulinMolecular Sequence DataBiologyBiochemistryCell LineCalmodulinIn vivoCyclinsProto-Oncogene ProteinsmedicineAnimalsCyclin D1Amino Acid SequencePhosphorylationMolecular BiologyCyclinCell NucleusSulfonamidesKinaseColocalizationCyclin-Dependent Kinase 4Cell BiologyImmunogold labellingPrecipitin TestsCyclin-Dependent KinasesCell biologyRatsEnzyme ActivationCell nucleusMicroscopy Electronmedicine.anatomical_structurebiology.proteinNuclear localization sequenceProtein BindingThe Journal of biological chemistry
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