Search results for "Plasticity"

showing 10 items of 765 documents

Dysregulated Expression of Neuregulin-1 by Cortical Pyramidal Neurons Disrupts Synaptic Plasticity

2014

Summary Neuregulin-1 ( NRG1 ) gene variants are associated with increased genetic risk for schizophrenia. It is unclear whether risk haplotypes cause elevated or decreased expression of NRG1 in the brains of schizophrenia patients, given that both findings have been reported from autopsy studies. To study NRG1 functions in vivo, we generated mouse mutants with reduced and elevated NRG1 levels and analyzed the impact on cortical functions. Loss of NRG1 from cortical projection neurons resulted in increased inhibitory neurotransmission, reduced synaptic plasticity, and hypoactivity. Neuronal overexpression of cysteine-rich domain (CRD)-NRG1, the major brain isoform, caused unbalanced excitato…

MaleDendritic SpinesNeuregulin-1Nonsynaptic plasticityGene ExpressionMice TransgenicNeurotransmissionInhibitory postsynaptic potentialSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyCell MovementInterneuronsConditioning Psychologicalmental disordersAnimalsNeuregulin 1lcsh:QH301-705.5CA1 Region HippocampalNeuronal PlasticitybiologyPyramidal CellsAnatomyFearCortex (botany)Synaptic fatiguelcsh:Biology (General)Synaptic plasticitybiology.proteinExcitatory postsynaptic potentialFemaleNerve NetNeuroscience
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Hampered long-term depression and thin spine loss in the nucleus accumbens of ethanol-dependent rats.

2014

Alcoholism involves long-term cognitive deficits, including memory impairment, resulting in substantial cost to society. Neuronal refinement and stabilization are hypothesized to confer resilience to poor decision making and addictive-like behaviors, such as excessive ethanol drinking and dependence. Accordingly, structural abnormalities are likely to contribute to synaptic dysfunctions that occur from suddenly ceasing the use of alcohol after chronic ingestion. Here we show that ethanol-dependent rats display a loss of dendritic spines in medium spiny neurons of the nucleus accumbens (Nacc) shell, accompanied by a reduction of tyrosine hydroxylase immunostaining and postsynaptic density 95…

MaleDendritic spineDendritic SpinesGlutamic AcidNucleus accumbensNeurotransmissionMedium spiny neuronSynaptic TransmissionNucleus AccumbensOrgan Culture TechniquesAnimalsRats WistarLong-term depressionLong-Term Synaptic Depressiondopamine synaptic plasticity Golgi glutamateMultidisciplinaryNeuronal PlasticityEthanolDopaminergic NeuronsLong-Term Synaptic DepressionCentral Nervous System DepressantsRatsAlcoholismPNAS PlusSynaptic plasticitySettore BIO/14 - FarmacologiaPsychologyNeurosciencePostsynaptic densityProceedings of the National Academy of Sciences of the United States of America
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Chronic stress induces changes in the structure of interneurons and in the expression of molecules related to neuronal structural plasticity and inhi…

2011

Chronic stress in experimental animals, one of the most accepted models of chronic anxiety and depression, induces structural remodeling of principal neurons in the amygdala and increases its excitation by reducing inhibitory tone. These changes may be mediated by the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), a molecule related to neuronal structural plasticity and expressed by interneurons in the adult CNS, which is downregulated in the amygdala after chronic stress. We have analyzed the amygdala of adult mice after 21 days of restraint stress, studying with qRT-PCR the expression of genes related to general and inhibitory neurotransmission, and of PSA synthesizi…

MaleDendritic spineInterneuronDendritic SpinesSynaptophysinNeural Cell Adhesion Molecule L1BiologyNeurotransmissionSynaptic TransmissionAmygdalaImmobilizationMiceDevelopmental NeuroscienceInterneuronsmedicineAnimalsChronic stressNeuronal PlasticityGlutamate DecarboxylaseDendritesAmygdalaImmunohistochemistrySialyltransferasesDisease Models Animalmedicine.anatomical_structurenervous systemNeurologySialic AcidsSynaptophysinbiology.proteinNeural cell adhesion moleculeNeuroscienceStress PsychologicalBasolateral amygdalaExperimental Neurology
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Hippocampal GABAergic Synapses Possess the Molecular Machinery for Retrograde Nitric Oxide Signaling

2007

Nitric oxide (NO) plays an important role in synaptic plasticity as a retrograde messenger at glutamatergic synapses. Here we describe that, in hippocampal pyramidal cells, neuronal nitric oxide synthase (nNOS) is also associated with the postsynaptic active zones of GABAergic symmetrical synapses terminating on their somata, dendrites, and axon initial segments in both mice and rats. The NO receptor nitric oxide-sensitive guanylyl cyclase (NOsGC) is present in the brain in two functional subunit compositions: α1β1and α2β1. The β1subunit is expressed in both pyramidal cells and interneurons in the hippocampus. Using immunohistochemistry andin situhybridization methods, we describe that the …

MaleInterneuronReceptors Cytoplasmic and NuclearNitric Oxide Synthase Type IBiologyNitric OxideHippocampusSynapseMiceSoluble Guanylyl CyclasemedicineAnimalsHumansRNA MessengerRats Wistargamma-Aminobutyric AcidMice Knockoutmusculoskeletal neural and ocular physiologyGeneral NeuroscienceArticlesAxon initial segmentRatsMice Inbred C57BLmedicine.anatomical_structurenervous systemGuanylate CyclaseSynaptic plasticitySynapsesbiology.proteinRetrograde signalingGABAergicSoluble guanylyl cyclaseNeuroscienceParvalbuminSignal Transduction
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A repetitive intracortical microstimulation pattern induces long-lasting synaptic depression in brain slices of the rat primary somatosensory cortex.

2000

Repetitive intracortical microstimulation (ICMS) applied to the rat primary somatosensory cortex (SI) in vivo was reported to induce reorganization of receptive fields and cortical maps. The present study was designed to exam- ine the effect of such an ICMS pattern applied to layer IV of brain slices containing SI on the efficacy of synaptic in- put to layer II/III. Effects of ICMS on the synaptic strength was quantified for the first synaptic component ( s1) of cor- tical field potentials (FPs) recorded from layer II/III of SI. FPs were evoked by stimulation in layer IV. The pattern of ICMS was identical to that used in vivo. However, stimula- tion intensity had to be raised to induce an a…

MaleLong-Term PotentiationNeurotransmissionIn Vitro TechniquesInhibitory postsynaptic potentialBicucullineReceptors N-Methyl-D-AspartateGABA AntagonistsRats Sprague-DawleymedicineAnimalsReceptors AMPASynaptic potentialNeuronal PlasticityChemistryGeneral NeuroscienceLong-term potentiationSomatosensory CortexBicucullineElectric StimulationRatsElectrophysiologyembryonic structuresSynaptic plasticitySynapsesExcitatory postsynaptic potentialNeuroscienceMicroelectrodesmedicine.drugExperimental brain research
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Role of the amygdala in antidepressant effects on hippocampal cell proliferation and survival and on depression-like behavior in the rat

2021

The stimulation of adult hippocampal neurogenesis by antidepressants has been associated with multiple molecular pathways, but the potential influence exerted by other brain areas has received much less attention. The basolateral complex of the amygdala (BLA), a region involved in anxiety and a site of action of antidepressants, has been implicated in both basal and stress-induced changes in neural plasticity in the dentate gyrus. We investigated here whether the BLA modulates the effects of the SSRI antidepressant fluoxetine on hippocampal cell proliferation and survival in relation to a behavioral index of depression-like behavior (forced swim test). We used a lesion approach targeting th…

MaleLong-Term Potentiationlcsh:MedicineHippocampal formationElement-Binding ProteinAmygdala/*drug effects/physiopathologyHippocampusMemory FormationRats Sprague-Dawleyddc:616.890302 clinical medicineMedial Prefrontal CortexElevated Plus-MazeSerotonin Uptake Inhibitors/*pharmacologylcsh:ScienceBasolateral Amygdala0303 health sciencesMultidisciplinaryNeuroscience/Behavioral NeuroscienceDepressionNeurogenesisBLAAmygdalaImmunohistochemistryChronic FluoxetineAdult-RatNeuroscience/Psychologymedicine.anatomical_structureFluoxetine/*pharmacologyDepression/*pathologyAntidepressantAntidepressive Agents Second-GenerationSelective Serotonin Reuptake InhibitorsResearch ArticleEstrèsElevated plus mazemedicine.medical_specialtyAnimal-ModelAntidepressive Agents Second-Generation/*pharmacologyCell SurvivalAmygdala03 medical and health sciencesFluoxetineNeuroplasticityHippocampus/cytology/*drug effectsmedicineAnimalsPsychiatryMaze Learning030304 developmental biologyCell Proliferationbusiness.industryDentate gyrusMental Health/Mood Disorderslcsh:RBasolateral complex of the amygdaleRatsCell Proliferation/*drug effectsDentate Gyruslcsh:QCell Survival/*drug effectsbusinessNeuroscience030217 neurology & neurosurgeryBasolateral amygdala
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Plasticity-related gene-1 inhibits lysophosphatidic acid-induced vascular smooth muscle cell migration and proliferation and prevents neointima forma…

2012

International audience; Plasticity-related gene-1 (PRG-1) protects neuronal cells from lysophosphatidic acid (LPA) effects. In vascular smooth muscle cells (VSMCs), LPA was shown to induce phenotypic modulation in vitro and vascular remodeling in vivo. Thus we explored the role of PRG-1 in modulating VSMC response to LPA. PCR, Western blot, and immunofluorescence experiments showed that PRG-1 is expressed in rat and human vascular media. PRG-1 expression was strongly inhibited in proliferating compared with quiescent VSMCs both in vitro and in vivo (medial vs. neointimal VSMCs), suggesting that PRG-1 expression is dependent on the cell phenotype. In vitro, adenovirus-mediated overexpression…

MaleMAPK/ERK pathwayNeointimaVascular smooth musclePhysiologyPhenotypic modulation[SDV]Life Sciences [q-bio]Genetic VectorsBiologyPlasticityMuscle Smooth VascularAdenoviridaechemistry.chemical_compoundCell MovementNeointimaLysophosphatidic acidAnimalsHumansRats WistarCells CulturedCell ProliferationCell BiologyLipid-phosphate phosphatasePhosphoric Monoester HydrolasesIn vitroRatsCell biologyGene Expression RegulationchemistryBiochemistryCalmodulin-Binding ProteinsLysophospholipidsAmerican Journal of Physiology-Cell Physiology
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Food Intake Adaptation to Dietary Fat Involves PSA-Dependent Rewiring of the Arcuate Melanocortin System in Mice

2012

International audience; Hormones such as leptin and ghrelin can rapidly rewire hypothalamic feeding circuits when injected into rodent brains. These experimental manipulations suggest that the hypothalamus might reorganize continually in adulthood to integrate the metabolic status of the whole body. In this study, we examined whether hypothalamic plasticity occurs in naive animals according to their nutritional conditions. For this purpose, we fed mice with a short-term high-fat diet (HFD) and assessed brain remodeling through its molecular and functional signature. We found that HFD for 3 d rewired the hypothalamic arcuate nucleus, increasing the anorexigenic tone due to activated pro-opio…

MaleMESH: Signal TransductionPro-Opiomelanocortin[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionSYNAPTIC INPUT ORGANIZATIONMESH: Energy IntakeWeight GainMESH: Mice KnockoutMice0302 clinical medicineMESH : Sialic AcidsNPY/AGRP NEURONSMESH: Pro-OpiomelanocortinMESH: AnimalsMESH : Neuronal PlasticityMESH: Neuronal PlasticityPLASTICITYMESH : Pro-OpiomelanocortinMESH : Adaptation PhysiologicalMice KnockoutFEEDING CIRCUITSMESH : Organ Culture TechniquesINSULIN-RESISTANCE0303 health sciencesNeuronal PlasticityPOLYSIALIC ACIDGeneral NeuroscienceLeptinMESH: Energy Metabolismdigestive oral and skin physiologyINDUCED OBESITYMESH : SialyltransferasesMESH : Weight GainArticlesAdaptation PhysiologicalMESH : Mice TransgenicBODY-WEIGHTMESH: Dietary FatsHypothalamusCELL-ADHESION MOLECULEMESH: Weight GainGhrelinENERGY-BALANCEMelanocortinhormones hormone substitutes and hormone antagonistsSignal Transductionmedicine.medical_specialtyMESH: Mice TransgenicMESH : MaleMESH: SialyltransferasesMESH: Arcuate NucleusMice TransgenicMESH : Mice Inbred C57BLBiologyMESH : Arcuate NucleusMESH: Sialic Acids03 medical and health sciencesOrgan Culture TechniquesInsulin resistanceMESH: Mice Inbred C57BLArcuate nucleusInternal medicineMESH : MicemedicineAnimalsMESH: Mice030304 developmental biologyMESH : Signal TransductionArcuate Nucleus of HypothalamusMESH : Energy Intakemedicine.diseaseDietary FatsMESH: Adaptation PhysiologicalSialyltransferasesMESH: Organ Culture TechniquesMESH: MaleMice Inbred C57BLMESH : Energy MetabolismEndocrinologyMESH: Nerve NetSialic AcidsMESH : Nerve NetMESH : Mice KnockoutMESH : AnimalsNerve NetEnergy IntakeEnergy Metabolism[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH : Dietary Fats030217 neurology & neurosurgeryHomeostasisHormoneThe Journal of Neuroscience
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Analysis of synaptic bodies in the Sprague–Dawley rat pineal gland under extreme photoperiods

2007

Synaptic bodies (SBs) are small, prominent organelles in pinealocytes, most probably involved in signal transduction processes. To check the influence of the photoperiod on their shape plasticity and number we chose two extreme lighting conditions, i.e. 20 h of illumination followed by 4 h of darkness (LD 20:4) versus (LD 4:20). Pineal glands were assessed at 0, 4 and 13 h after dark onset. Under both conditions reconstructed SBs were plates or ribbons but never spheres and there were no obvious differences in morphology. Photoperiodic changes in SB profile size and number were investigated: application of the established method for SB quantification based on single section profile counts (…

MaleMorphology (linguistics)LightPhotoperiodGeneral Physics and AstronomyBiologyPlasticityPineal GlandPinealocytelaw.inventionRats Sprague-DawleyRat Pineal GlandMicroscopy Electron TransmissionStructural BiologylawImage Processing Computer-AssistedAnimalsGeneral Materials ScienceOrganellesphotoperiodismCell BiologyAnatomyRatsSprague dawleyDarknessBiophysicsFemaleElectron microscopeMicron
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Long-term effects on motor cortical excitability induced by repeated muscle vibration during contraction in healthy subjects

2008

article i nfo Objective: The effects of a novel repeated muscle vibration intervention (rMV; 100 Hz, 90 min over 3 consecutive days) on corticomotor excitability were studied in healthy subjects. Methods: rMV was applied over the flexor carpi radialis (FCR) during voluntary contraction (experiment 1), during relaxation and during contraction without vibration (experiment 2). Focal transcranial magnetic stimulation (TMS) was applied before rMV and one hour, and one, two and three weeks after the last muscle vibration intervention. At each of these time points, we assessed the motor map area and volume in the FCR, extensor digitorum communis (EDC) and abductor digiti minimi (ADM). Short-inter…

MaleMotor disorderpaired-pulse tmsTime FactorsContraction (grammar)H-Reflex; Analysis of Variance; Humans; Electromyography; Neural Inhibition; Electric Stimulation; Muscle Skeletal; Motor Cortex; Brain Mapping; Vibration; Evoked Potentials Motor; Adult; Middle Aged; Muscle Contraction; Transcranial Magnetic Stimulation; Time Factors; Female; Malemedicine.medical_treatmentH-ReflextmsEvoked PotentialsBrain Mappingcortical plasticitySkeletalMiddle AgedTranscranial Magnetic Stimulationmedicine.anatomical_structureMotorNeurologyAnesthesiaMuscleFemaleSettore MED/26 - NeurologiaPrimary motor cortexmedicine.symptomPsychologyMuscle ContractionMotor cortexMuscle contractionAdultintracortical inhibitionVibrationNOmuscle vibrationmotor cortexNeuroplasticitymedicineHumansMuscle SkeletalAnalysis of VarianceneurorehabilitationElectromyographytms; muscle vibration; paired-pulse tms; neurorehabilitation; motor cortex; cortical plasticity; intracortical inhibitionNeural InhibitionEvoked Potentials Motormedicine.diseaseElectric Stimulationbody regionsTranscranial magnetic stimulationNeurology (clinical)NeuroscienceExtensor Digitorum Communis
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