Search results for "Ploidies"

showing 10 items of 37 documents

Patterns of DNA-ploidy in operable colorectal carcinoma: A prospective study of 100 cases

1991

A prospective study of cellular DNA content was made by means of flow cytometry in a nonconsecutive series of 100 patients undergoing surgery for primary colorectal adenocarcinoma. DNA-aneuploidy was present in 80% of cases (80/100); 39% of these were multiclonal (31/80). There was no significant correlation between DNA-ploidy and the clinical and pathological features examined, except for the primary tumor site (right colon vs. left colon vs. rectum: P less than 0.001). After a minimum follow-up of 30 months, out of 40 patients with no local invasion and/or distant metastases, 100% (9/9) of those with DNA-diploid neoplasias showed no signs of disease relapse, vs. 55% (17/31) of the DNA-ane…

AdultMaleOncologymedicine.medical_specialtyColorectal cancerAneuploidyRectumAdenocarcinomaGastroenterologyInternal medicinemedicineHumansProspective StudiesProspective cohort studyPathologicalDna ploidyAgedNeoplasm StagingAged 80 and overChi-Square DistributionPloidiesbusiness.industryDNA NeoplasmGeneral MedicineMiddle AgedPrognosismedicine.diseasePrimary tumorSurvival Ratemedicine.anatomical_structureOncologyAdenocarcinomaFemaleSurgeryColorectal NeoplasmsbusinessJournal of Surgical Oncology
researchProduct

DNA image cytometry

1992

In 68 patients the DNA content of tumor cells was measured by image cytometry after resection of the rectum because of cancer. In the DNA histogram a differentiation between diploid (n = 19), polyploid (n = 24), hypotriploid (n = 17), and hypertriploid (n = 8) tumors was possible. The best relapse-free survival time was found in patients with diploid tumors. The prognosis worsened from polyploid to hypotriploid and was worse in hypertriploid tumors. Testing for a prognostic advantage of diploid over aneuploid tumors without adjustment for additional factors simply by means of the log-rank statistic gave a (one-sided) P of 0.1013. In a multivariate analysis the degree of differentiation turn…

AdultMalePathologymedicine.medical_specialtyColorectal cancerRectumPolyploidRisk FactorsSurgical oncologymedicineHumansComputer SimulationDNA Image CytometryAgedAged 80 and overAnalysis of VariancePloidiesRectal Neoplasmsbusiness.industryGastroenterologyCancerDNA NeoplasmGeneral MedicineMiddle AgedPrognosismedicine.diseasemedicine.anatomical_structureCancer researchImage CytometryFemaleCytophotometryNeoplasm Recurrence LocalbusinessCytometryDiseases of the Colon & Rectum
researchProduct

Prognostic significance of DNA ploidy, S-phase fraction, and tissue levels of aspartic, cysteine, and serine proteases in operable gastric carcinoma

2000

A consecutive series of 63 untreated patients undergoing surgical resection for stage I-IV gastric adenocarcinomas (GCs) has been prospectively studied. Our purpose was to analyze the predictive relevance of DNA ploidy, S-phase fraction (SPF), and tissue levels of lysosomal proteinases cathepsin D (CD), cathepsin B (CB), cathepsin L (CL), and urokinase-type plasminogen activator (uPA) and that of the intracellular cysteine proteinase inhibitor stefin A on clinical outcome. All of the patients taking part in this study were followed up for a median of 73 months. DNA aneuploidy was present in 71% of the cases (45/63), whereas 9% of these (4/45) showed multiclonality. Both DNA ploidy and SPF w…

AdultMaleTime FactorsAdenocarcinomaS PhasePredictive Value of TestsStomach NeoplasmsBiomarkers TumorAspartic Acid EndopeptidasesHumansNeoplasm Invasivenesshumancell cycle S phase; disease association; female; histopathology; human; lymph node metastasisAgedProbabilityPloidieslymph node metastasisdisease associationSerine EndopeptidasesDNA NeoplasmMiddle AgedPrognosisSurvival AnalysisCysteine Endopeptidasescell cycle S phaseLymphatic MetastasishistopathologyFemaleFollow-Up Studies
researchProduct

Cytogenetic analysis of epithelial renal-cell tumors: Relationship with a new histopathological classification

1993

Renal-cell carcinomas (RCC) are clinically, histologically and cytogenetically very heterogeneous. The present histological WHO classification shows no clear correlation between histologic subtypes and specific chromosomal abnormalities. In 1986, a new classification was proposed by Thoenes and Storkel based on the cell type from which the tumor arises. They distinguish S cell types: clear-cell, chromophilic, chromophobic, ductus Bellini and oncocytic. Results of 105 primary tumors show that, in this new classification, there is a correlation between different subtypes of renal-cell tumor and specific chromosomal abnormalities at a microscopic and/or molecular level. The clear-cell compact …

Cancer ResearchPathologymedicine.medical_specialtyMonosomyCell typeCARCINOMAChromosome DisordersHistogenesisBiologyPolysomy 7Loss of heterozygositymedicineHumansCarcinoma Renal CellChromosome AberrationsChromosome 7 (human)PolysomyPloidiesABNORMALITIESCytogeneticsDNA Neoplasmmedicine.diseaseKidney NeoplasmsONCOCYTOMASOncologyTISSUEKaryotypingInternational Journal of Cancer
researchProduct

Molecular cytogenetics of childhood hematological malignancies

1998

Cytogenetic and molecular analyses are essential for the classification of childhood hematologic malignancies. Nearly all children with leukemia should have an adequate cytogenetic analysis which in 80-90% is expected to show clonal chromosomal abnormalities. Moreover, with the availability of appropriate gene probes and sophisticated molecular techniques, genetic rearrangements become detectable in the majority of leukemia patients. Genetic abnormalities often associate with particular clinical-biological characteristics of the disease. In ALL, for example, genetic alterations together with distinct immunologic and clinical features, define various subgroups. In AML, unique cytogenetic rea…

Cancer Researchmedicine.medical_specialtyDiseaseBioinformaticsMolecular cytogeneticsAcute lymphocytic leukemiamedicineHumansClinical significanceChildChromosome AberrationsGene RearrangementLeukemiaPloidiesbusiness.industryMyelodysplastic syndromesCytogeneticsHematologyGene rearrangementPrecursor Cell Lymphoblastic Leukemia-Lymphomamedicine.diseaseLeukemia Myeloid AcuteLeukemiaOncologyMyelodysplastic SyndromesImmunologybusinessLeukemia
researchProduct

Phylostratic Shift of Whole-Genome Duplications in Normal Mammalian Tissues towards Unicellularity Is Driven by Developmental Bivalent Genes and Reve…

2020

Tumours were recently revealed to undergo a phylostratic and phenotypic shift to unicellularity. As well, aggressive tumours are characterized by an increased proportion of polyploid cells. In order to investigate a possible shared causation of these two features, we performed a comparative phylostratigraphic analysis of ploidy-related genes, obtained from transcriptomic data for polyploid and diploid human and mouse tissues using pairwise cross-species transcriptome comparison and principal component analysis. Our results indicate that polyploidy shifts the evolutionary age balance of the expressed genes from the late metazoan phylostrata towards the upregulation of unicellular and early m…

CarcinogenesisCircadian clockAntineoplastic AgentsBiologyGenomeArticleCatalysisBivalent (genetics)Epigenesis Geneticlcsh:ChemistryProto-Oncogene Proteins c-mycInorganic ChemistryTranscriptomeMicePolyploidGene DuplicationNeoplasmsProtein Interaction MappingAnimalsHumanscancerEpigeneticsPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologyGenepolyploidybivalent genesSpectroscopyGeneticsGenomePloidiesCircadian Rhythm Signaling Peptides and ProteinsOrganic Chemistryearly multicellularityviral-origin oncogenesOncogenesGeneral MedicineembryonalityPhenotypeNeoplasm ProteinsunicellularityComputer Science ApplicationsGene Expression Regulation Neoplasticlcsh:Biology (General)lcsh:QD1-999Drug Resistance NeoplasmMetabolic Networks and PathwaysInternational Journal of Molecular Sciences
researchProduct

Reconstruction of the evolutionary history of Saccharomyces cerevisiae x S. kudriavzevii hybrids based on multilocus sequence analysis.

2012

In recent years, interspecific hybridization and introgression are increasingly recognized as significant events in the evolution of Saccharomyces yeasts. These mechanisms have probably been involved in the origin of novel yeast genotypes and phenotypes, which in due course were to colonize and predominate in the new fermentative environments created by human manipulation. The particular conditions in which hybrids arose are still unknown, as well as the number of possible hybridization events that generated the whole set of natural hybrids described in the literature during recent years. In this study, we could infer at least six different hybridization events that originated a set of 26 S…

Evolutionary Geneticslcsh:MedicineYeast and Fungal ModelsWineSaccharomycesGenomeSouth AfricaNatural SelectionFungal EvolutionDNA FungalMycological Typing Techniqueslcsh:ScienceGenome EvolutionPhylogenyRecombination GeneticGeneticsMultidisciplinarybiologyfood and beveragesGenomicsBiological EvolutionEuropePhylogeographyPloidyResearch ArticleGenome evolutionEvolutionary ProcessesGenotypeGenes FungalIntrogressionGenomicsMycologySaccharomyces cerevisiaeMicrobiologySaccharomycesModel OrganismsPhylogeneticsGeneticsHumansAdaptationBiologyHybridizationHybridEvolutionary BiologyPloidiesChimeralcsh:RComparative GenomicsSouth Americabiology.organism_classificationYeastGenetic Polymorphismlcsh:QPopulation GeneticsMultilocus Sequence TypingPLoS ONE
researchProduct

Past climate changes facilitated homoploid speciation in three mountain spiny fescues (Festuca, Poaceae)

2016

Apart from the overwhelming cases of allopolyploidization, the impact of speciation through homoploid hybridization is becoming more relevant than previously thought. Much less is known, however, about the impact of climate changes as a driven factor of speciation. To investigate these issues, we selected Festuca picoeuropeana, an hypothetical natural hybrid between the diploid species F. eskia and F. gautieri that occurs in two different mountain ranges (Cantabrian Mountains and Pyrenees) separated by more than 400 km. To unravel the outcomes of this mode of speciation and the impact of climate during speciation we used a multidisciplinary approach combining genome size and chromosome coun…

Festuca0106 biological sciences0301 basic medicineReproductive IsolationFestucaGenetic SpeciationClimate ChangeNicheIntrogressionBiology010603 evolutionary biology01 natural sciencesArticlePolyploidy03 medical and health sciencesSpecies SpecificityGenetic algorithmPhylogenyHybridEcological nichePloidiesMultidisciplinaryGeographyGenetic VariationReproductive isolation15. Life on landbiology.organism_classificationDiploidy030104 developmental biologyGenetic SpeciationSpain13. Climate actionEvolutionary biologyHybridization GeneticGenome PlantScientific Reports
researchProduct

Impact of oxygenation status and patient age on DNA content in cancers of the uterine cervix.

2003

Abstract Purpose In carcinomas of the uterine cervix, the tumor oxygenation status has been shown to be a prognostic indicator that is independent of treatment modality. In vitro studies suggest gene amplification and polyploidization to be among the major consequences of hypoxia (with or without consecutive reoxygenation) and to be associated with treatment resistance and tumor progression. This study analyzed whether hypoxia alters net DNA content in uterine cervix cancer cells to the extent that it is identifiable by DNA image cytometry. Methods and materials In 64 patients with primary cervical cancer, tumor oxygenation was assessed polarographically and correlated with cell DNA content…

Genome instabilityAdultCancer ResearchPathologymedicine.medical_specialtyGene duplicationMedicineHumansRadiology Nuclear Medicine and imagingDNA Image CytometryAgedImage CytometryCervical cancerAged 80 and overRadiationPloidiesTumor hypoxiabusiness.industryAge FactorsDNA NeoplasmTumor OxygenationHypoxia (medical)Middle Agedmedicine.diseaseCell HypoxiaOxygenOncologyTumor progressionUterine NeoplasmsFemalemedicine.symptombusinessInternational journal of radiation oncology, biology, physics
researchProduct

Simultaneous Aurora-A/STK15 overexpression and centrosome amplification induce chromosomal instability in tumour cells with a MIN phenotype

2007

Abstract Background Genetic instability is a hallmark of tumours and preneoplastic lesions. The predominant form of genome instability in human cancer is chromosome instability (CIN). CIN is characterized by chromosomal aberrations, gains or losses of whole chromosomes (aneuploidy), and it is often associated with centrosome amplification. Centrosomes control cell division by forming a bipolar mitotic spindle and play an essential role in the maintenance of chromosomal stability. However, whether centrosome amplification could directly cause aneuploidy is not fully established. Also, alterations in genes required for mitotic progression could be involved in CIN. A major candidate is represe…

Genome instabilityCancer ResearchCellular differentiationAneuploidyApoptosisCell CommunicationSpindle ApparatusBiologyProtein Serine-Threonine Kinaseslcsh:RC254-282Aurora KinasesChromosome instabilityChromosomal InstabilitymedicineTumor Cells CulturedGeneticsHumansRNA Small InterferingMitosisIn Situ Hybridization FluorescenceAurora Kinase ACentrosomePloidiesReverse Transcriptase Polymerase Chain ReactionAurora-A centrosomes amplification aneuploidyCell Differentiationlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseAneuploidyCell biologySpindle apparatusUp-RegulationSettore BIO/18 - GeneticaCell Transformation NeoplasticPhenotypeMicroscopy FluorescenceOncologyCentrosomeColonic NeoplasmsEctopic expressionMicrosatellite InstabilityResearch ArticleBMC Cancer
researchProduct