Search results for "Predisposition"

showing 10 items of 771 documents

Impact of the AHI1 gene on the vulnerability to schizophrenia: a case-control association study.

2010

Background: The Abelson helper integration-1 (AHI1) gene is required for both cerebellar and cortical development in humans. While the accelerated evolution of AHI1 in the human lineage indicates a role in cognitive (dys)function, a linkage scan in large pedigrees identified AHI1 as a positional candidate for schizophrenia. To further investigate the contribution of AHI1 to the susceptibility of schizophrenia, we evaluated the effect of AHI1 variation on the vulnerability to psychosis in two samples from Spain and Germany. Methodology/Principal Findings: 29 single-nucleotide polymorphisms (SNPs) located in a genomic region including the AHI1 gene were genotyped in two samples from Spain (28…

AdultPsychosisLinkage disequilibriumAdolescentMental Health/Neuropsychiatric Disorderslcsh:MedicineSingle-nucleotide polymorphismPedigree chartBiologyGenetics and Genomics/Complex TraitsPolymorphism Single NucleotideLinkage DisequilibriumYoung AdultGenotypemedicineHumansGenetic Predisposition to Diseaseddc:610lcsh:ScienceGenetics and Genomics/Genetics of DiseaseAllelesAdaptor Proteins Signal TransducingAgedGeneticsMental Health/Schizophrenia and Other PsychosesMultidisciplinaryHaplotypelcsh:RCase-control studyMiddle AgedSchizophreniemedicine.diseaseAdaptor Proteins Vesicular TransportHaplotypesSchizophreniaCase-Control StudiesSchizophrenialcsh:QResearch ArticlePLoS ONE
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A polygenic approach to the association between smoking and schizophrenia.

2021

Smoking prevalence in schizophrenia is considerably larger than in general population, playing an important role in early mortality. We compared the polygenic contribution to smoking in schizophrenic patients and controls to assess if genetic factors may explain the different prevalence. Polygenic risk scores (PRSs) for smoking initiation and four genetically correlated traits were calculated in 1108 schizophrenic patients (64.4% smokers) and 1584 controls (31.1% smokers). PRSs for smoking initiation, educational attainment, body mass index and age at first birth were associated with smoking in patients and controls, explaining a similar percentage of variance in both groups. Attention-defi…

AdultPsychosisMultifactorial InheritanceSociodemographic FactorsPopulationMedicine (miscellaneous)Nerve Tissue ProteinsReceptors NicotinicGenetic correlationBody Mass IndexNicotineRisk Factorsmental disordersmedicineGenetic predispositionTobacco SmokingHumansGenetic Predisposition to DiseaseRisk factoreducationPharmacologyeducation.field_of_studybusiness.industryMiddle Agedmedicine.diseasePsychiatry and Mental healthPhenotypeSchizophreniaAttention Deficit Disorder with HyperactivitySchizophreniabusinessBody mass indexDemographymedicine.drugGenome-Wide Association StudyAddiction biologyREFERENCES
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Variable pulmonary manifestations in Chitayat syndrome: Six additional affected individuals

2020

Hand hyperphalangism leading to shortened index fingers with ulnar deviation, hallux valgus, mild facial dysmorphism and respiratory compromise requiring assisted ventilation are the key features of Chitayat syndrome. This condition results from the recurrent heterozygous missense variant NM_006494.2:c.266A>G; p.(Tyr89Cys) in ERF on chromosome 19q13.2, encoding the ETS2 repressor factor (ERF) protein. The pathomechanism of Chitayat syndrome is unknown. To date, seven individuals with Chitayat syndrome and the recurrent pathogenic ERF variant have been reported in the literature. Here, we describe six additional individuals, among them only one presenting with a history of assisted ventil…

Adultbronchomalacia2716 Genetics (clinical)hyperphalangismPediatricsmedicine.medical_specialtyAdolescent10039 Institute of Medical Genetics610610 Medicine & healthChitayat syndromeFingersYoung Adult03 medical and health sciences1311 Geneticsrespiratory distressExome SequencingGeneticsmedicineHumansMissense mutationGenetic Predisposition to DiseaseHallux ValgusRespiratory systemChildGenetics (clinical)030304 developmental biologyCHITAYAT SYNDROME0303 health sciencesPierre Robin SyndromebiologyRespiratory distressbusiness.industry030305 genetics & heredityFaciesmedicine.diseasebiology.organism_classificationPhenotype3. Good healthRepressor ProteinsValgusERFChild Preschoolulnar deviation570 Life sciences; biologyFemaleUlnar deviationBronchomalaciabusinessAmerican Journal of Medical Genetics Part A
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Major gender difference in association of FTO gene variant among severely obese children with obesity and obesity related phenotypes.

2008

Recent studies have shown that SNPs in the FTO gene predispose to childhood and adult obesity. In this study, we examined the association between variants in FTO and KIAA1005, a gene that maps closely to FTO, and obesity, as well as obesity related traits among 450 well characterised severely obese children and 512 normal weight controls. FTO showed significant association with several obesity related traits while SNPs in KIAA1005 did not. When stratified by gender, the FTO variant rs9939609 showed association with obesity and BMI among girls (P = 0.006 and 0.004, respectively) but not among boys. Gender differences were also found in the associations of the FTO rs9939609 with obesity relat…

Adultmedicine.medical_specialtyendocrine system diseasesAdolescentmedicine.medical_treatmentBiophysicsAlpha-Ketoglutarate-Dependent Dioxygenase FTOSingle-nucleotide polymorphismBiochemistryFTO genePolymorphism Single NucleotideRisk AssessmentBody Mass IndexInsulin resistanceSex FactorsRisk FactorsInternal medicineDiabetes mellitusmedicinePrevalenceSNPHumansGenetic Predisposition to DiseaseObesitySex DistributionChildMolecular BiologySwedenbusiness.industryInsulinnutritional and metabolic diseasesGenetic VariationProteinspathological conditions signs and symptomsCell Biologymedicine.diseaseObesityEndocrinologyPhenotypeInsulin ResistancebusinessBody mass indexBiochemical and biophysical research communications
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Major histocompatibility complex and sporadic Alzheimer's disease: a critical reappraisal

2003

Epidemiological data suggest that some genetic determinants of Alzheimer's disease (AD) might reside in those polymorphisms for the immune system genes that regulate immune inflammatory responses, such as the major histocompatibility complex (MHC). Therefore, MHC polymorphisms have been the focus of a large number of AD association studies. Class Ia, Ib (hemochromatosis gene (HFE)), class II and class III (complement, tumour necrosis factor and heat shock proteins) alleles have been studied. Nearly every positive result has been followed by several studies that have failed to replicate it or that have contradicted it. Several factors, including methodological biases, might explain these dis…

AgingGenes MHC Class IIGenes MHC Class ILocus (genetics)Human leukocyte antigenMajor histocompatibility complexBiochemistryMajor Histocompatibility ComplexEndocrinologyAlzheimer DiseaseMHC class IGeneticsHumansGenetic Predisposition to DiseaseAlleleMolecular BiologyAllelesAgedGenetic associationGeneticsbiologyTumor Necrosis Factor-alphaHaplotypeCell BiologyHistocompatibilitybiology.proteinExperimental Gerontology
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Meta-analysis of genome-wide association studies from the CHARGE consortium identifies common variants associated with carotid intima media thickness…

2011

Carotid intima media thickness (cIMT) and plaque determined by ultrasonography are established measures of subclinical atherosclerosis that each predicts future cardiovascular disease events. We conducted a meta-analysis of genome-wide association data in 31,211 participants of European ancestry from nine large studies in the setting of the Cohorts for Heart and Aging Research in Genomic Epidemiology (CHARGE) Consortium. We then sought additional evidence to support our findings among 11,273 individuals using data from seven additional studies. In the combined meta-analysis, we identified three genomic regions associated with common carotid intima media thickness and two different regions a…

AgingMultifunction cardiogramGenome-wide association studyDiseaseCoronary Artery Disease030204 cardiovascular system & hematologyCarotid Intima-Media ThicknessCoronary artery diseaseCohort Studies0302 clinical medicinecardiovascular diseaseRisk FactorsAging/geneticsgeneticsMyocardial infarctionEuropean Continental Ancestry Group/geneticsriskPlaque0303 health scienceseducation.field_of_studyGenomeCoronary Artery Disease/geneticsHeartSingle NucleotidePlaque Atherosclerotic/geneticsMiddle AgedPlaque Atherosclerotic3. Good healthPhenotypeHeart/physiopathologyCardiologyHumanAdultmedicine.medical_specialtygenetic epidemiologyGenotypesubclinical atherosclerosisPopulationSingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideWhite PeopleArticle03 medical and health sciencesSDG 3 - Good Health and Well-beingInternal medicinemedicinecohort studyHumanscarotid intima media thicknessGenetic Predisposition to DiseasePolymorphismeducation030304 developmental biologyAgedAtherosclerotic/geneticsGenome Humanmedicine.diseaseAtherosclerosismeta-analysisAtherosclerosis/geneticsIntima-media thicknessGenetic LociGenome-Wide Association Study
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Body Fat and Mobility Are Explained by Common Genetic and Environmental Influences in Older Women

2008

In older adults, mobility limitations often coexist with overweight or obesity, suggesting that similar factors may underlie both traits. This study examined the extent to which genetic and environmental influences explain the association between adiposity and mobility in older women. Body fat percentage (bioimpedance test), walking speed over 10 m, and distance walked in a 6-min test were evaluated in 92 monozygotic (MZ) and 104 dizygotic (DZ) pairs of twin sisters reared together, aged 63-76 years. Genetic and environmental influences on each trait were estimated using age-adjusted multivariate genetic modeling. The analyses showed that the means (and s.d.) for body fat percentage, walkin…

Agingmedicine.medical_specialtyEndocrinology Diabetes and MetabolismMedicine (miscellaneous)030209 endocrinology & metabolismWalkingEnvironmentOverweightBiologyBody fat percentageCorrelation03 medical and health sciences0302 clinical medicineEndocrinologyRisk FactorsInternal medicineActivities of Daily LivingElectric ImpedanceTwins DizygoticmedicineHumansGenetic Predisposition to DiseaseObesity030212 general & internal medicineMobility Limitation10. No inequalityFinlandAdiposityAgedNutrition and DieteticsModels GeneticAge FactorsTwins MonozygoticMiddle Agedmedicine.diseaseTwin studyObesityPreferred walking speedEndocrinologyMobility LimitationPhysical EnduranceTraitFemalemedicine.symptomLocomotionDemographyObesity
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Association between the PNPLA3 (rs738409 C>G) variant and hepatocellular carcinoma: Evidence from a meta-analysis of individual participant data

2014

The incidence of hepatocellular carcinoma (HCC) is increasing in Western countries. Although several clinical factors have been identified, many individuals never develop HCC, suggesting a genetic susceptibility. However, to date, only a few single-nucleotide polymorphisms have been reproducibly shown to be linked to HCC onset. A variant (rs738409 C>G, encoding for p.I148M) in the PNPLA3 gene is associated with liver damage in chronic liver diseases. Interestingly, several studies have reported that the minor rs738409[G] allele is more represented in HCC cases in chronic hepatitis C (CHC) and alcoholic liver disease (ALD). However, a significant association with HCC related to CHC has not b…

Alcoholic liver diseasemedicine.medical_specialtyCirrhosisCarcinoma HepatocellularBioinformaticsGastroenterologyPolymorphism Single NucleotideWhite PeopleLiver Cirrhosis AlcoholicInternal medicinemedicineGenetic predispositionHumansHepatologyModels Geneticbusiness.industryLiver NeoplasmsMembrane ProteinsOdds ratioLipaseHepatologyHepatitis C Chronicmedicine.diseasedigestive system diseasesHepatocellular carcinomabusinessBody mass indexTM6SF2
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Interaction between filaggrin mutations and neonatal cat exposure in atopic dermatitis

2020

Atopic dermatitis (AD) is a prevalent inflammatory skin disease. Loss-of-function mutations in filaggrin gene (FLG) represent the strongest genetic risk factors for AD, being strongly associated with early disease onset and persistence into adulthood.1 The epidermis of individuals with mutations in FLG is fundamentally different from normal skin being characterized by increased penetration of allergens.2 Recent birth cohort studies showed a significant interaction between cat ownership at birth and mutations in FLG (R501X, 2282del4) on the development of early-onset AD.3 This finding was replicated for the 2282del4 FLG mutation in a Dutch cohort study, and extended to further associate with…

AllergyAllergyImmunologyFilaggrin ProteinsDermatitis Atopic03 medical and health sciences0302 clinical medicineCAT EXPOSUREIntermediate Filament ProteinsmedicineImmunology and AllergyAnimalsHumansGenetic Predisposition to Disease030304 developmental biologyRISK0303 health sciencesScience & TechnologyCATSbusiness.industryInfant NewbornAtopic dermatitismedicine.disease030228 respiratory system1107 ImmunologyMutation (genetic algorithm)ImmunologyMutationCatsbusinessLife Sciences & BiomedicineFilaggrinAllergy
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Endothelial Leptin Receptor Deletion Promotes Cardiac Autophagy and Angiogenesis Following Pressure Overload by Suppressing Akt/mTOR Signaling.

2019

Background: Cardiac remodeling is modulated by overnutrition or starvation. The adipokine leptin mediates energy balance between adipose tissue and brain. Leptin and its receptors are expressed in the heart. Methods and Results: To examine the importance of endothelial leptin signaling in cardiac hypertrophy, transverse aortic constriction was used in mice with inducible endothelium-specific deletion of leptin receptors (End.LepR-KO) or littermate controls (End.LepR-WT). End.LepR-KO was associated with improved left ventricular function (fractional shortening, 28.4% versus 18.8%; P =0.0114), reduced left ventricular dilation (end-systolic inner left ventricular diameter, 3.59 versus 4.08 m…

AngiogenesisAdipose tissueAdipokineCardiomegaly030204 cardiovascular system & hematologyVentricular Function Left03 medical and health sciences0302 clinical medicineAutophagyMedicineAnimalsHumansGenetic Predisposition to Disease030212 general & internal medicineProtein kinase BCells Cultured2. Zero hungerPressure overloadHeart FailureMice KnockoutLeptin receptorNeovascularization Pathologicbusiness.industryLeptinMyocardiumTOR Serine-Threonine KinasesAutophagyEndothelial CellsFibrosisCell biologyDisease Models AnimalPhenotypeReceptors LeptinFemaleCardiology and Cardiovascular MedicinebusinessProto-Oncogene Proteins c-aktGene DeletionSignal TransductionCirculation. Heart failure
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