Search results for "Progenitor"

showing 10 items of 355 documents

Leukemic Colony-Forming Cells in Acute Myeloblastic Leukemia: Maturation Hierarchy and Growth Conditions

1987

Despite their primitive morphological appearance, the majority of leukemic blasts in acute myeloblastic leukemia (AML) are end-stage, nonproliferating cells. Only a small subset of AML blasts are capable of a sufficient number of divisions to form colonies in semisolid medium [1, 2]. It has been suggested that these leukemic colony-forming cells (L-CFC) may act in vivo as progenitor cells to maintain the rest of the leukemic cell population [3, 4]. L-CFC share several properties with normal myeloid progenitor cells, including self-renewal potential and high thymidine suicide index [2, 3]. As in the case of normal myeloid progenitor cells (NMPC), colony growth of L-CFC from most patients req…

education.field_of_studyAcute myeloblastic leukemiaPopulationCellPlacental tissueBiologymedicine.diseasechemistry.chemical_compoundmedicine.anatomical_structurechemistryIn vivoCancer researchmedicineProgenitor celleducationThymidineLeukemic Blasts
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Cell lineage and cell fate specification in the embryonic CNS of Drosophila.

1997

The Drosophila CNS derives from a population of neural stem cells, called neuroblasts (NBs), which delaminate individually from the neurogenic region of the ectoderm. In the embryonic ventral nerve cord each NB can be uniquely identified and gives rise to a specific lineage consisting of neurons and/or glial cells. This 'NB identity' is dependent on the position of the progenitor cells in the neuroectoderm before delamination. The positional information is provided by the products of segment polarity and dorsoventral (D/V) patterning genes. Subsequently, 'cell fate genes' like huckebein (hkb) and eagle (eg) contribute to the generation of specific NB lineages. These genes act downstream of …

education.field_of_studyNeuroectodermPopulationEctodermCell BiologyAnatomyBiologyCell fate determinationEmbryonic stem cellNeural stem cellCell biologymedicine.anatomical_structureNeuroblastmedicineProgenitor celleducationDevelopmental BiologySeminars in celldevelopmental biology
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Fracaso renal agudo en el trasplante de progenitores hematopoyéticos: análisis de la incidencia, factores de riesgo e implicaciones pronósticas. Valo…

2015

El trasplante de progenitores hematopoyéticos es el único tratamiento curativo de muchas enfermedades hematológicas, algunas alteraciones metabólicas y ciertas inmunodeficiencias. Persigue regenerar el tejido hematopoyético cuya función resulta insuficiente, bien por una enfermedad primaria de la médula ósea o a consecuencia de un tratamiento de quimioterapia y/o radioterapia. Precisa un rescate de la función hematopoyética con la obtención de células progenitoras que puedan regenerar la función medular tras ser trasplantadas a un paciente al que se ha suprimido su médula ósea. Actualmente existen otras dos finalidades: a) re-inicializar el sistema inmune para corregir la autorreactividad e…

fracaso renal agudotrasplante progenitores hematopoyeticoscistatina c
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Papel de IRS2 en la reparación del daño hepático y cáncer

2019

La resistencia a insulina es una característica típica de la diabetes tipo 2 y la obesidad, patologías vinculadas a un alto riesgo del desarrollo de enfermedades crónicas hepáticas y hepatocarcinoma. Sin embargo, se desconoce el papel que la resistencia a insulina pueda ejercer durante el daño hepático crónico y durante la hepatocarcinogénesis, por lo que se ha abordado esta cuestión a través del estudio del substrato receptor de insulina 2 (IRS2), principal promotor de la señalización de insulina en el hígado. La experimentación llevada a cabo durante este proyecto muestra que la deleción del gen Irs2 en el modelo murino Irs2-/- impide la inducción de Fgf7 en células estromales durante el …

hepatocarcinomacélulas estrelladas hepáticasUNESCO::CIENCIAS DE LA VIDAinsulinaprogresión del cáncercélulas progenitoras hepáticasdaño hepático:CIENCIAS DE LA VIDA [UNESCO]señalización FGF7-FGFR2-IIIb
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Qualitative and Quantitative Analysis of Cardiac Progenitor Cells in Cases of Myocarditis and Cardiomyopathy

2018

We aimed to identify and quantify CD117+ and CD90+ endogenous cardiac progenitor cells (CPC) in human healthy and diseased hearts. We hypothesize that these cells perform a locally acting, contributing function in overcoming medical conditions of the heart by endogenous means. Human myocardium biopsies were obtained from 23 patients with the following diagnoses: Dilatative cardiomyopathy (DCM), ischemic cardiomyopathy (ICM), myocarditis, and controls from healthy cardiac patients. High-resolution scanning microscopy of the whole slide enabled a computer-based immunohistochemical quantification of CD117 and CD90. Those signals were evaluated by Definiens Tissue Phenomics® Technology. Co-loca…

lcsh:Geneticshuman myocardium biopsylcsh:QH426-470c-Kit+ (CD117+) cellsembryonic structuresCD90+ cellslocal cardiac stem/progenitor cellsmyocarditiscardiomyopathydigestive system diseasesFrontiers in Genetics
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Cell therapy medicinal product regulatory framework in Europe and its application for MSC-based therapy development

2012

Advanced therapy medicinal products (ATMPs), including cell therapy products, form a new class of medicines in the European Union. Since the ATMPs are at the forefront of scientific innovation in medicine, specific regulatory framework has been developed for these medicines and implemented from 2009. The Committee for Advanced Therapies (CAT) has been established at the European Medicines Agency (EMA) for centralized classification, certification and evaluation procedures, and other ATMP-related tasks. Guidance documents, initiatives, and interaction platforms are available to make the new framework more accessible for small- and medium-sized enterprises, academia, hospitals, and foundation…

lcsh:Immunologic diseases. Allergymesenchymal stem/progenitor cellMesenchymal stem cells (MSC)ImmunologyReview ArticleCertificationBest interestsAgency (sociology)Hospital Exemption authorisationImmunology and AllergyMedicinemedia_common.cataloged_instanceProduct (category theory)European unionHospital Exemptionmedia_commonbusiness.industrynational competent authorityCommittee for Advanced TherapiesCell therapy medicinal product (CTMP)AuthorizationGuidance documentsCommittee for Advanced Therapies (CAT)Engineering managementNew product developmentadvanced therapy medicinal productlcsh:RC581-607businesscell therapy medicinal productAdvanced therapy medicinal product (ATMP)Frontiers in Immunology
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Neural Stem Cells in the Adult Brain: From Benchside to Clinic

2012

Increasing evidence indicates that neural stem cells (NSCs) play an important role in sustaining cellular homeostasis and brain tissue restoration. The study of all mechanisms that control and modulate the function of NSC is a crucial step for the design of therapies against chronic neurodegenerative processes. In this special issue of the journal, we had the pleasure to edit the topic entitled “Neural Stem Cells in the Adult Brain: From Benchside to Clinic.” This special compilation of paper was aimed to provide a global forum for publications of original peer-reviewed manuscripts that reported original research findings in the field of adult neural stem cell, including short communication…

lcsh:Internal medicineArticle SubjectNeurogenesisSubventricular zoneCellular homeostasisCell BiologyNestinBiologyBioinformaticsNeural stem cellmedicine.anatomical_structureEditorialmedicineStem cellProgenitor celllcsh:RC31-1245Molecular BiologyNeurosciencePI3K/AKT/mTOR pathwayStem Cells International
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Progenitor death drives retinal dysplasia and neuronal degeneration in a mouse model of Atrip-Seckel syndrome

2020

ABSTRACT Seckel syndrome is a type of microcephalic primordial dwarfism (MPD) that is characterized by growth retardation and neurodevelopmental defects, including reports of retinopathy. Mutations in key mediators of the replication stress response, the mutually dependent partners ATR and ATRIP, are among the known causes of Seckel syndrome. However, it remains unclear how their deficiency disrupts the development and function of the central nervous system (CNS). Here, we investigated the cellular and molecular consequences of ATRIP deficiency in different cell populations of the developing murine neural retina. We discovered that conditional inactivation of Atrip in photoreceptor neurons …

lcsh:MedicineMedicine (miscellaneous)315BlindnessMicechemistry.chemical_compoundImmunology and Microbiology (miscellaneous)Cell DeathneurodevelopmentStem CellsNeurodegenerationapoptosisneurodegenerationSyndromeCell biologyDNA-Binding Proteinsdna damage responsemedicine.anatomical_structurePhotoreceptor Cells VertebrateResearch Articlelcsh:RB1-214NeurogenesisNeuroscience (miscellaneous)Embryonic DevelopmentBiologyRetinaGeneral Biochemistry Genetics and Molecular Biologylcsh:PathologymedicineAnimalsAbnormalities MultipleProgenitor cellVision OcularAdaptor Proteins Signal TransducingCell ProliferationProgenitorRetinalcsh:RRetinalEmbryo Mammalianmedicine.diseasephotoreceptorDisease Models AnimalSeckel syndromechemistryvisual system developmentNerve DegenerationRetinal dysplasiaRetinal DysplasiaTumor Suppressor Protein p53Primordial dwarfismDNA DamageDisease Models & Mechanisms
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Stem cell populations in the heart and the role of Isl1 positive cells

2013

Cardiac progenitor cells are multipotent stem cells isolated from both embryonic and adult hearts in several species and are able to differentiate at least into smooth muscle cells, endothelial cells and cardiomyocytes. The embryonic origin of these cells has not yet been demonstrated, but it has been suggested that these cells may derive from the first and secondary heart fields and from the neural crest. In the last decade, two diffe-rent populations of cardiac progenitor or stem cells have been identified and isolated, i.e., the Islet1 positive (Isl1+) and c-Kit positive (c-Kit+)/Stem Cell Antigen-1 positive (Sca-1+) cells. Until 2012, these two populations have been considered two separ…

lineagesHistologymuscleLIM-Homeodomain ProteinsBiophysicscardiac progenitor cellsheartBiologycardiac progenitor cells stem cells heart lineages biology muscle.stem cellsHumansProgenitor celllcsh:QH301-705.5Induced stem cellsViews and CommentsbiologySettore BIO/16 - Anatomia UmanaMyocardiumCell BiologyCell biologyEndothelial stem cellP19 celllcsh:Biology (General)Gene Expression RegulationMultipotent Stem CellAmniotic epithelial cellsImmunologycardiac progenitor cells stem-cells heart lineages biology muscle.Stem cellAdult stem cellTranscription Factors
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Author response: PTEN negatively regulates the cell lineage progression from NG2+ glial progenitor to oligodendrocyte via mTOR-independent signaling

2018

medicine.anatomical_structurebiology.proteinmedicinePTENCell lineageBiologyPI3K/AKT/mTOR pathwayOligodendrocyteCell biologyProgenitor
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