Search results for "RCI"

showing 10 items of 16009 documents

IL-17A mediated endothelial breach promotes metastasis formation

2015

Abstract The role of the IL23/IL17A axis in tumor–immune interactions is a matter of controversy. Although some suggest that IL17A-producing T cells (TH17) can suppress tumor growth, others report that IL17A and IL23 accelerate tumor growth. Here, we systematically assessed the impact of IL17A-secreting lymphocytes in several murine models of tumor lung metastasis. Genetic fate mapping revealed that IL17A was secreted within lung metastases predominantly by γδ T cells, whereas TH17 cells were virtually absent. Using different tumor models, we found Il17a−/− mice to consistently develop fewer pulmonary tumor colonies. IL17A specifically increased blood vessel permeability and the expression …

0301 basic medicineGenetically modified mouseCancer ResearchPathologymedicine.medical_specialtyLung NeoplasmsEndotheliumImmunologyMelanoma ExperimentalVascular permeability610 Medicine & healthBiology10263 Institute of Experimental ImmunologyCapillary Permeability03 medical and health sciencesCarcinoma Lewis LungCell Line TumormedicineCell AdhesionAnimals1306 Cancer ResearchCell adhesionMice Knockout2403 ImmunologyLungMelanomaInterleukin-17Transendothelial and Transepithelial MigrationEndothelial Cellsmedicine.diseaseMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureCell culture570 Life sciences; biologyInterleukin 17Endothelium VascularNeoplasm Transplantation
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2016

RNA 2'-O-methylation is one of the ubiquitous nucleotide modifications found in many RNA types from Bacteria, Archaea and Eukarya. RNAs bearing 2'-O-methylations show increased resistance to degradation and enhanced stability in helices. While the exact role of each 2'-O-Me residue remained elusive, the catalytic protein Fibrillarin (Nop1 in yeast) responsible for 2'-O-methylation in eukaryotes, is associated with human pathologies. Therefore, there is an urgent need to precisely map and quantify hundreds of 2'-O-Me residues in RNA using high-throughput technologies. Here, we develop a reliable protocol using alkaline fragmentation of total RNA coupled to a commonly used ligation approach, …

0301 basic medicineGenetics2'-O-methylation5.8S ribosomal RNARNAComputational biologyRNA integrity numberBiologyRibosomal RNA03 medical and health sciences030104 developmental biology0302 clinical medicineRNA editing030220 oncology & carcinogenesisGeneticsRNA extractionIllumina dye sequencingNucleic Acids Research
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Diagnostic implications of intrapatient genetic tumor heterogeneity

2015

The complex genetic composition of neuroblastoma emphasizes the importance of conscientious and meticulous diagnosis. Clones with amplification or segmental chromosomal aberrations sometimes remain hidden. Several determinations should be performed when sufficient tumor material is available to establish the final diagnosis by combining the results of different techniques on tumor fragments or liquid biopsies.

0301 basic medicineGeneticsCancer ResearchBiologymedicine.diseaseTumor heterogeneityIntratumoral Genetic Heterogeneity03 medical and health sciences030104 developmental biology0302 clinical medicine030220 oncology & carcinogenesisNeuroblastomamedicineMolecular MedicineAuthor's ViewGenetic composition
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Caretakers and Gatekeepers

2017

It has now been generally accepted that the genes responsible for familial cancer syndromes can be divided into two categories, known as caretakers and gatekeepers. Caretakers are genes that control the maintenance of the genetic information integrity in each cell while gatekeepers are those genes which directly regulate tumor growth, codifying for proteins which either stimulate or inhibit proliferation, differentiation or apoptosis. Keywords: gatekeeper genes; caretaker genes; tumor suppressor genes; cell cycle; hereditary syndromes

0301 basic medicineGeneticsCellCaretaker geneCell cycleBiologyInformation integritylaw.invention03 medical and health sciences030104 developmental biology0302 clinical medicinemedicine.anatomical_structureApoptosislaw030220 oncology & carcinogenesismedicineSuppressorTumor growthGeneeLS
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Epigenetic regulation of DNA repair genes and implications for tumor therapy

2017

DNA repair represents the first barrier against genotoxic stress causing metabolic changes, inflammation and cancer. Besides its role in preventing cancer, DNA repair needs also to be considered during cancer treatment with radiation and DNA damaging drugs as it impacts therapy outcome. The DNA repair capacity is mainly governed by the expression level of repair genes. Alterations in the expression of repair genes can occur due to mutations in their coding or promoter region, changes in the expression of transcription factors activating or repressing these genes, and/or epigenetic factors changing histone modifications and CpG promoter methylation or demethylation levels. In this review we …

0301 basic medicineGeneticsDNA RepairDNA repairHealth Toxicology and MutagenesisDNA MethylationBiologyEpigenesis Genetic03 medical and health sciences030104 developmental biology0302 clinical medicineEpigenetics of physical exerciseNeoplasms030220 oncology & carcinogenesisDNA Repair ProteinDNA methylationGeneticsCancer researchAnimalsHumansCpG IslandsDNA mismatch repairEpigeneticsCancer epigeneticsEpigenomicsMutation Research/Reviews in Mutation Research
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5' and 3' RACE Method to Obtain Full-Length 5' and 3' Ends of Ciona robusta Macrophage Migration Inhibitory Factors Mif1 and Mif2 cDNA

2019

The 5' and 3' RACE is a method to obtain full-length 5' and 3' ends of cDNA using known cDNA sequences from expressed sequence tags (ESTs), subtracted cDNA, differential display, or library screening. Here is described the identification of full-length 5' and 3' ends of Ciona robusta Mif1 and Mif2 cDNA by using 5' and 3' RACE method.

0301 basic medicineGeneticsExpressed sequence tagCiona robustaDifferential displayBiologyGenome03 medical and health sciencesRace (biology)030104 developmental biology0302 clinical medicine030220 oncology & carcinogenesisComplementary DNA5' and 3' RACE MIF Ciona robustaMacrophage migration inhibitory factor
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Water: The First Archaic Mutagen of Evolution, the Adoptive Mother of Currently Nucleic Acids

2016

It would be intuitive to think that for the evolutionary onset of nucleic acids, and even before of their monomeric constituents, the water was crucial. Within the series of evolutions which start from the origin of the universe and still cannot, by definition, be considered concluded, certainly among the geothermal evolution end and the beginning of the chemical one, on Earth, the moment was favorable for the arrival of the first proto-nucleotides: from underground deposits of methanehydrate [1] and phosphate, with the support of all known pre-biotic physical-chemical conditions, were made the monomeric components of nucleic acids. The cradle of nucleic acids does not seem to have been so …

0301 basic medicineGeneticsHuman evolutionary geneticsDNA damageMutagenesisMutagenBiologymedicine.disease_causeEvolutionary geneticsSettore BIO/18 - Genetica03 medical and health sciences030104 developmental biologymedicineNucleic acidEvolutionary mutagenesiDuctal adenocarcinomaAdoptive motherCarcinogenesisJournal of Carcinogenesis & Mutagenesis
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2021

Familial pancreatic cancer (FPC) is an established but rare inherited tumor syndrome that accounts for approximately 5% of pancreatic ductal adenocarcinoma (PDAC) cases. No major causative gene defect has yet been identified, but germline mutations in predisposition genes BRCA1/2, CDKN2A and PALB2 could be detected in 10–15% of analyzed families. Thus, the genetic basis of disease susceptibility in the majority of FPC families remains unknown. In an attempt to identify new candidate genes, we performed whole-genome sequencing on affected patients from 15 FPC families, without detecting BRCA1/2, CDKN2A or PALB2 mutations, using an Illumina based platform. Annotations from CADD, PolyPhen-2, S…

0301 basic medicineGeneticsMutationCandidate genePALB2Medicine (miscellaneous)Gene mutationBiologymedicine.disease_causePenetranceGermline03 medical and health sciences030104 developmental biology0302 clinical medicineGermline mutationCDKN2A030220 oncology & carcinogenesismedicineJournal of Personalized Medicine
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2017

Purpose BRCA1/2 mutations increase the risk of breast and prostate cancer in men. Common genetic variants modify cancer risks for female carriers of BRCA1/2 mutations. We investigated—for the first time to our knowledge—associations of common genetic variants with breast and prostate cancer risks for male carriers of BRCA1/ 2 mutations and implications for cancer risk prediction. Materials and Methods We genotyped 1,802 male carriers of BRCA1/2 mutations from the Consortium of Investigators of Modifiers of BRCA1/2 by using the custom Illumina OncoArray. We investigated the combined effects of established breast and prostate cancer susceptibility variants on cancer risks for male carriers of…

0301 basic medicineGeneticsOncologyCancer Researchmedicine.medical_specialtymedicine.diagnostic_testbusiness.industryCase-control studyGenome-wide association studyOdds ratiomedicine.disease3. Good health03 medical and health sciencesProstate cancer030104 developmental biology0302 clinical medicineBreast cancerOncology030220 oncology & carcinogenesisInternal medicinemedicineOverdiagnosisbusinessRisk assessmentGenetic testingJournal of Clinical Oncology
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Telomeric length heterogeneity influences spontaneous regression of malignant melanoma

2016

0301 basic medicineGeneticsbusiness.industryMelanomaDermatologyTelomeremedicine.diseaseRegressionTelomere03 medical and health sciences030104 developmental biology0302 clinical medicineInfectious DiseasesNeoplasm Regression Spontaneous030220 oncology & carcinogenesisCancer researchHumansMedicinebusinessMelanomaIn Situ Hybridization FluorescenceJournal of the European Academy of Dermatology and Venereology
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