Search results for "RECEPTORS"

showing 10 items of 3254 documents

Proteostasis Deregulation in Neurodegeneration and Its Link with Stress Granules: Focus on the Scaffold and Ribosomal Protein RACK1.

2022

The role of protein misfolding, deposition, and clearance has been the dominant topic in the last decades of investigation in the field of neurodegeneration. The impairment of protein synthesis, along with RNA metabolism and RNA granules, however, are significantly emerging as novel potential targets for the comprehension of the molecular events leading to neuronal deficits. Indeed, defects in ribosome activity, ribosome stalling, and PQC—all ribosome-related processes required for proteostasis regulation—can contribute to triggering stress conditions and promoting the formation of stress granules (SGs) that could evolve in the formation of pathological granules, usually occurring during ne…

Ribosomal ProteinsRACK1neurodegenerationtranslationGeneral MedicineCytoplasmic GranulesReceptors for Activated C KinaseStress GranulesNeoplasm ProteinsSettore BIO/10 - BiochimicaFrontotemporal DementiaProteostasisRNAHumansCells
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A Candida albicans 37 kDa polypeptide with homology to the laminin receptor is a component of the translational machinery.

1998

A cDNA encoding a 37 kDa protein was isolated from an expression library using antibodies raised against mycelial cell walls fromCandida albicans.The 37 kDa protein has over 60% sequence identity with the 37 kDa laminin-binding protein (LBP) from humans and over 80% identity with the Yst proteins ofSaccharomyces cerevisiae. TheC. albicansprotein was named CaYst1. It was found in membrane and ribosome fractions but surprisingly, was not found in cell walls. Unlike the human LBP, CaYst1p does not bind laminin. These data indicate that CaYst1p is not a cell-surface receptor for laminin as has been proposed for the human LBP. Instead, like theS. cerevisiaeYst proteins, it appears to be a riboso…

Ribosomal ProteinsSaccharomyces cerevisiae ProteinsSaccharomyces cerevisiaeBlotting WesternMolecular Sequence DataMicrobiologyFungal ProteinsReceptors LamininRibosomal proteinComplementary DNACandida albicansAnimalsHumansCandida albicansAntibodies Fungalchemistry.chemical_classificationFungal proteinbiologyBase SequenceBinding proteinMembrane Proteinsbiology.organism_classificationBlotting NorthernMolecular biologyBlotting SouthernCytoskeletal ProteinsBiochemistrychemistryMembrane proteinProtein BiosynthesisRabbitsGlycoproteinSequence AlignmentMicrobiology (Reading, England)
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Synthesis and pharmacological evaluation of several ring-contracted amantadine analogs

2008

Graphical abstract Several bisnoradamantylamines and noradamantylamines have been synthesized and their antiviral, trypanocidal, NMDA receptor antagonist, and dopamine reuptake inhibitory activities have been studied.

RimantadineStereochemistryDopamineeducationClinical BiochemistryPharmaceutical Sciencemacromolecular substancesPharmacologymedicine.disease_causeAntiviral AgentsReceptors N-Methyl-D-AspartateBiochemistryChemical synthesisArticleInhibitory Concentration 50DogsPolycyclic compoundMemantineTrypanosomiasisDopamineDrug DiscoveryAmantadinemedicineInfluenza A virusAnimalsNMDA receptor antagonistMolecular BiologyCells Culturedchemistry.chemical_classificationChemistryOrganic ChemistryAmantadinePolycyclic cage compoundsBiological activityInfluenzanervous systemInfluenza A virusMolecular MedicineNMDA receptormedicine.drugBioorganic & Medicinal Chemistry
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Identification and purification of human erythroid progenitor cells by monoclonal antibody to the transferrin receptor (T� 67)

1988

Anti-TU 67 is a murine monoclonal antibody that recognizes the transferrin receptor. With respect to hematopoietic cells TU 67 is expressed by human multipotent colony-forming cells (CFU-Mix), erythroid progenitor cells (BFU-E and CFU-E) and a fraction of granulocyte/monocyte colony forming cells, but is not expressed by mature hematopoietic cells including erythrocytes, platelets, lymphocytes, and peripheral blood myeloid cells. The TU 67-positive fraction of normal bone marrow, separated by fluorescence-activated cell sorting (FACS) or immune rosettes, contained 87% of the erythroid progenitor cells. Erythroid progenitor cells were enriched up to 50-fold by using a combination of monoclon…

Rosette FormationErythroblastsmedicine.drug_classMonocyteAntibodies MonoclonalFluorescent Antibody TechniqueTransferrin receptorCell SeparationHematologyGeneral MedicineCell sortingBiologyFlow CytometryMonoclonal antibodyMolecular biologyHaematopoiesismedicine.anatomical_structurehemic and lymphatic diseasesReceptors TransferrinMonoclonalmedicinebiology.proteinAntibodyInterleukin 3Blut
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An Update on the Current and Emerging Use of Thiazolidinediones for Type 2 Diabetes.

2022

Guidelines have increasingly stressed the concept that adequate glycemic control is required to prevent or decrease the macro- and microvascular complications of type 2 diabetes mellitus (T2DM). PPAR-gamma agonists (“glitazones”) are no longer prioritized due to their effects on heart failure. However, the association between these drugs and innovative therapies could be a valuable tool to attenuate the risk factors of the metabolic syndrome. Glitazones are used for the treatment of diabetes and associated comorbidities. There is substantial scientific evidence demonstrating the effect of glitazones at a cardiometabolic level, as well as on hematological and neurological pathologies that po…

RosiglitazoneDiabetes Mellitus Type 2PioglitazonePeroxisome Proliferator-Activated ReceptorsHumansHypoglycemic AgentsThiazolidinedionescardiovascular risk metabolic syndrome pioglitazone type 2 diabetes mellitusGeneral MedicineMedicina (Kaunas, Lithuania)
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Distribution and phenotype of rotavirus-specific B cells induced during the antigen-driven primary response to 2/6 virus-like particles administered …

2007

AbstractSelection of mucosal sites is an important step in mucosal vaccine development. The intrarectal (IR) route represents an alternative to the oral route of immunization; nevertheless, immune responses induced by this route are not well defined. Here, we studied the early primary B cell response (induction, homing, and phenotype) induced by IR immunization with rotavirus (RV)-2/6 virus-like particles (VLP). Using flow cytometry, we traced RV-specific B cells in different lymphoid tissues and analyzed the expression of α4β7 and CCR9, which are important receptors for homing to the gut, as well as CD5, a marker expressed by B1-a cells, which are a major source of natural antibodies. We o…

RotavirusAntibodies ViralMicePeyer's Patches0302 clinical medicineCell MovementImmunology and AllergyMesenteric lymph nodes[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyMesenteryAntigens ViralmucosaB-LymphocytesMice Inbred BALB C0303 health sciencesmedicine.diagnostic_testrodent3. Good healthIntestinesPhenotypemedicine.anatomical_structure[SDV.IMM]Life Sciences [q-bio]/ImmunologyFemaleAntibodyImmunologyReceptors Lymphocyte HomingBiologyCD5 AntigensFlow cytometryReceptors CCR03 medical and health sciencesImmune systemAntigenmedicineAnimalsImmunity MucosalAdministration IntranasalB cell030304 developmental biologyLumbosacral RegionRotavirus VaccinesCell BiologyvaccinationB-1 cellB-1a cellsImmunologybiology.proteinImmunizationLymph Nodescell traffickingCD5030215 immunology
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Protein kinase CK2 governs the molecular decision between encephalitogenic T H 17 cell and T reg cell development

2016

T helper 17 (TH17) cells represent a discrete TH cell subset instrumental in the immune response to extracellular bacteria and fungi. However, TH17 cells are considered to be detrimentally involved in autoimmune diseases like multiple sclerosis (MS). In contrast to TH17 cells, regulatory T (Treg) cells were shown to be pivotal in the maintenance of peripheral tolerance. Thus, the balance between Treg cells and TH17 cells determines the severity of a TH17 cell-driven disease and therefore is a promising target for treating autoimmune diseases. However, the molecular mechanisms controlling this balance are still unclear. Here, we report that pharmacological inhibition as well as genetic ablat…

STAT3 Transcription Factor0301 basic medicineEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisCellMice Transgenicchemical and pharmacologic phenomenaBiologySeverity of Illness IndexT-Lymphocytes RegulatoryMice03 medical and health sciences0302 clinical medicineImmune systemmedicineAnimalsHumansIL-2 receptorPhosphorylationCasein Kinase IISTAT3MultidisciplinaryCell growthInterleukin-17Experimental autoimmune encephalomyelitisGranulocyte-Macrophage Colony-Stimulating FactorFOXP3Peripheral toleranceForkhead Transcription Factorshemic and immune systemsReceptors Interleukinmedicine.diseasePeptide FragmentsMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureGene Expression RegulationImmunologybiology.proteinCancer researchTh17 CellsMyelin-Oligodendrocyte GlycoproteinSignal Transduction030215 immunologyProceedings of the National Academy of Sciences
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Parthenolide sensitizes hepatocellular carcinoma cells to trail by inducing the expression of death receptors through inhibition of STAT3 activation

2011

This article shows that HepG2, Hep3B, and SK-Hep1 cells, three lines of human hepatocellular carcinoma (HCC) cells, are resistant to apoptosis induced by tumor necrosis factor-related apoptosis-inducing ligand (TRAIL). Parthenolide, a sesquiterpene lactone found in European feverfew, has been shown to exert both anti-inflammatory and anti-cancer activities. This article demonstrates that co-treatment with parthenolide and TRAIL-induced apoptosis with synergistic interactions in the three lines of HCC cells. In order to explain these effects we ascertained that parthenolide increased either at protein or mRNA level the total content of death receptors TRAIL-R1 and -R2 as well as their surfac…

STAT3 Transcription FactorCarcinoma HepatocellularPhysiologyClinical BiochemistryCellDown-RegulationTRAILApoptosisPharmacologyParthenolideSTAT3TNF-Related Apoptosis-Inducing Ligandchemistry.chemical_compoundSettore BIO/10 - BiochimicamedicineHumansParthenolidePhosphorylationReceptorSTAT3CaspaseJanus KinasesbiologyLiver NeoplasmsProto-Oncogene Proteins c-mdm2Hep G2 CellsReceptors Death DomainCell BiologyapoptosiEnzyme ActivationGene Expression Regulation Neoplasticmedicine.anatomical_structurechemistryCell cultureApoptosisCaspasesbiology.proteinSTAT proteinDrug Screening Assays AntitumorTumor Suppressor Protein p53SesquiterpenesJournal of Cellular Physiology
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Combined interleukin 6 and soluble interleukin 6 receptor accelerates murine liver regeneration.

2000

Abstract Background & Aims: Liver regeneration after loss of hepatic tissue leads to hepatocyte and nonparenchymal cell proliferation and rapid restoration of liver parenchyma. Interleukin (IL)-6 is a key inducer of transcription factors involved in liver regeneration. Whenever IL-6 activates target cells, it binds to a specific IL-6 receptor (IL-6R). The IL-6/IL-6R complex then associates with the signal transducer gp130, leading to activation of intracellular signaling. Methods: We have recently constructed the designer cytokine Hyper-IL-6 consisting of soluble IL-6R covalently linked to IL-6, which directly stimulates gp130 even in the absence of membrane-bound IL-6R. We compared the inf…

STAT3 Transcription FactorTime Factorsmedicine.medical_treatmentMicemedicineAnimalsHepatectomyHumansPostoperative PeriodPhosphorylationInterleukin 6HepatologybiologyInterleukin-6Regeneration (biology)GastroenterologyInterleukinOrgan SizeGlycoprotein 130Receptors Interleukin-6Liver regenerationLiver RegenerationDNA-Binding ProteinsMice Inbred C57BLmedicine.anatomical_structureGene Expression RegulationLiverSolubilityHepatocyteInterleukin-6 receptorImmunologybiology.proteinCancer researchTrans-ActivatorsHepatectomyCell DivisionGastroenterology
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Induction of tolerogenic lung CD4+ T cells by local treatment with a pSTAT-3 and pSTAT-5 inhibitor ameliorated experimental allergic asthma.

2010

Signal transducer and activator of transcription (STAT)-3 inhibitors play an important role in regulating immune responses. Galiellalactone (GL) is a fungal secondary metabolite known to interfere with the binding of phosphorylated signal transducer and activator of transcription (pSTAT)-3 as well of pSTAT-6 dimers to their target DNA in vitro. Intra nasal delivery of 50 μg GL into the lung of naive Balb/c mice induced FoxP3 expression locally and IL-10 production and IL-12p40 in RNA expression in the airways in vivo. In a murine model of allergic asthma, GL significantly suppressed the cardinal features of asthma, such as airway hyperresponsiveness, eosinophilia and mucus production, after…

STAT3 Transcription Factormedicine.medical_treatmentImmunologyCD11cSuppressor of Cytokine Signaling ProteinsT-Lymphocytes RegulatoryLactonesMiceImmune systemIn vivomedicineSTAT5 Transcription FactorImmunology and AllergyAnimalsIndoleamine-Pyrrole 23-DioxygenaseAnti-Asthmatic AgentsLungAdministration IntranasalCells CulturedMice Inbred BALB CbiologyChemistryFOXP3General MedicineDendritic CellsT-Lymphocytes Helper-Inducerrespiratory systemAsthmaReceptors Interleukin-3CD11c Antigenrespiratory tract diseasesOvalbuminInterleukin 10CytokineSuppressor of Cytokine Signaling 3 ProteinImmunologySTAT proteinCancer researchbiology.proteinFemaleInterleukin-4T-Box Domain Proteins
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