Search results for "RNAs"

showing 10 items of 710 documents

Vascular ageing and endothelial cell senescence: Molecular mechanisms of physiology and diseases

2016

Ageing leads to a progressive deterioration of structure and function of all organs over the time. During this process endothelial cells undergo senescence and manifest significant changes in their properties, resulting in impairment of the vascular functionality and neo-angiogenic capability. This ageing-dependent impairment of endothelial functions is considered a key factor contributing to vascular dysfunctions, which is responsible of several age-related diseases of the vascular system and other organs. Several mechanisms have been described to control ageing-related endothelial cell senescence including microRNAs, mitochondrial dysfunction and micro environmental stressors, such as hyp…

0301 basic medicineSenescenceAgingEndotheliump73Biologymedicine.disease_cause03 medical and health sciencesEndotheliocyte; Endothelium; Hypoxia; MicroRNAs; Mitochondrial dysfunction; Oxidative stress; P53 family; P73; Transglutaminase 2; VEGF; Aging; Developmental BiologymicroRNAmedicineAnimalsHumansSettore MED/05 - Patologia ClinicaEndotheliocyte; Endothelium; Hypoxia; Mitochondrial dysfunction; Oxidative stress; Transglutaminase 2; VEGF; microRNAs; p53 family; p73Vascular DiseasesEndotheliumHypoxiaCellular SenescenceEndothelial CellsMicroRNASettore MED/23 - Chirurgia CardiacaBECN1Hypoxia (medical)VEGFMitochondriamicroRNAsEndothelial stem cellTransglutaminase 2030104 developmental biologymedicine.anatomical_structureOxidative stressAgeingImmunologyOxidative stremedicine.symptomMitochondrial dysfunctionp53 familyEndotheliocyteNeuroscienceOxidative stressDevelopmental BiologyMechanisms of Ageing and Development
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Oxidative stress, autophagy, epigenetic changes and regulation by miRNAs as potential therapeutic targets in osteoarthritis

2015

Aging is a natural process characterized by the declining ability of the different organs and tissues to respond to stress, increasing homeostatic imbalance and risk of disease. Osteoarthritis (OA) is a multifactorial disease in which cartilage degradation is a central feature. Aging is the main risk factor for OA. In OA cartilage, a decrease in the number of chondrocytes and in their ability to regenerate the extracellular matrix and adequately respond to stress has been described. OA chondrocytes show a senescence secretory phenotype (SSP) consisting on the overproduction of cytokines (interleukins 1 and 6), growth factors (e.g., epidermal growth factor) and matrix metalloproteinases (MMP…

0301 basic medicineSenescenceMAPK/ERK pathwayAgingProgrammed cell deathDNA damageBiologymedicine.disease_causeBiochemistryChondrocyteEpigenesis Genetic03 medical and health sciencesChondrocytesOsteoarthritisAutophagymedicineAnimalsHumansMolecular Targeted TherapyEpigeneticsCellular SenescencePharmacologyAutophagyDNA MethylationCell biologyMicroRNAsOxidative Stress030104 developmental biologymedicine.anatomical_structureImmunologyReactive Oxygen SpeciesOxidative stressDNA DamageBiochemical Pharmacology
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Telomere Length Determines TERRA and R-Loop Regulation through the Cell Cycle

2017

Maintenance of a minimal telomere length is essential to prevent cellular senescence. When critically short telomeres arise in the absence of telomerase, they can be repaired by homology-directed repair (HDR) to prevent premature senescence onset. It is unclear why specifically the shortest telomeres are targeted for HDR. We demonstrate that the non-coding RNA TERRA accumulates as HDR-promoting RNA-DNA hybrids (R-loops) preferentially at very short telomeres. The increased level of TERRA and R-loops, exclusively at short telomeres, is due to a local defect in RNA degradation by the Rat1 and RNase H2 nucleases, respectively. Consequently, the coordination of TERRA degradation with telomere r…

0301 basic medicineSenescenceTelomeraseSaccharomyces cerevisiae ProteinssenescenceDNA damageR-loopTelomere-Binding ProteinsSaccharomyces cerevisiaeBiologyDDRGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesRif2Cellular SenescenceTelomere-binding proteinRNA-DNA hybridtelomereBiochemistry Genetics and Molecular Biology (all)Telomere-Binding ProteinCell CycleRNANucleic Acid HybridizationRecombinational DNA RepairTERRARepressor ProteinMolecular biologyRat1ExoribonucleaseTelomereRepressor Proteins030104 developmental biologyCell AgingExoribonucleasesR-loopRNase H2Cell agingSaccharomyces cerevisiae ProteinDNA Damage
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Genetic 3’UTR variation is associated with human pigmentation characteristics and sensitivity to sunlight

2017

Sunlight exposure induces signalling pathways leading to the activation of melanin synthesis and tanning response. MicroRNAs (miRNAs) can regulate the expression of genes involved in pigmentation pathways by binding to the complementary sequence in their 3'untranslated regions (3'UTRs). Therefore, 3'UTR SNPs are predicted to modify the ability of miRNAs to target genes, resulting in differential gene expression. In this study, we investigated the role in pigmentation and sun-sensitivity traits, as well as in melanoma susceptibility, of 38 different 3'UTR SNPs from 38 pigmentation-related genes. A total of 869 individuals of Spanish origin (526 melanoma cases and 343 controls) were analysed.…

0301 basic medicineSkin NeoplasmsSNPSingle-nucleotide polymorphismSkin PigmentationDermatologyBiologyBiochemistryPolymorphism Single NucleotideWhite People03 medical and health sciencesGene FrequencyRisk FactorsWnt3A ProteinmicroRNAGene expressionGenotypeSNPHumansGenetic Predisposition to DiseasePhotosensitivity DisordersRNA MessengerHair ColorNaevusMolecular BiologyGene3' Untranslated RegionsMelanomaSolar lentiginesAdaptor Proteins Signal TransducingGeneticsLentigoBinding SitesEye ColorThree prime untranslated regionMicroRNAProtective Factors3' untranslated regionPhenotypeMicroRNAs030104 developmental biologyPhenotypeSpainCase-Control Studies
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Identification of microRNAS differentially regulated by water deficit in relation to mycorrhizal treatment in wheat.

2019

Arbuscular mycorrhizal fungi (AMF) are soil microrganisms that establish symbiosis with plants positively influencing their resistance to abiotic stresses. The aim of this work was to identify wheat miRNAs differentially regulated by water deficit conditions in presence or absence of AMF treatment. Small RNA libraries were constructed for both leaf and root tissues considering four conditions: control (irrigated) or water deficit in presence/absence of mycorrhizal (AMF) treatment. A total of 12 miRNAs were significantly regulated by water deficit in leaves: five in absence and seven in presence of AMF treatment. In roots, three miRNAs were water deficit-modulated in absence of mycorrhizal t…

0301 basic medicineSmall RNABiologyPlant Roots03 medical and health sciences0302 clinical medicineSymbiosisTranscription (biology)Gene Expression Regulation PlantStress PhysiologicalMycorrhizaeBotanymicroRNAGeneticsProtein biosynthesisTranscriptional regulationGene Regulatory NetworksMolecular BiologyDurum wheatWater deficitTriticummiRNAPlant ProteinsAbiotic componentGene Expression ProfilingfungiGene Expression Regulation DevelopmentalGeneral MedicineCell redox homeostasisDroughtsPlant LeavesMicroRNAs030104 developmental biologyRootRNA Plant030220 oncology & carcinogenesisWheatMolecular biology reports
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Temperature-dependent small RNA expression in Drosophila melanogaster

2018

Temperature has a major impact on gene expression in ectotherms. But until recently, it was not clear in which way, if any, small non-coding RNAs such as miRNAs or piRNAs contribute to thermosensitive gene regulation. We have recently shown that temperature-responsive miRNAs in Drosophila drive adaptation to different ambient temperatures on the transcriptome level. Moreover, we demonstrated that higher temperatures lead to a more efficient piRNA-dependent transposon silencing, possibly due to heat-induced unfolding of RNA secondary structures. In this commentary, we will dwell upon particular interesting aspects connected to our findings, hoping that our point of view may encourage other s…

0301 basic medicineSmall RNAComputational biologyEpigenesis GeneticEvolution MolecularTranscriptome03 medical and health sciencesStress PhysiologicalmicroRNAGene expressionAnimalsDrosophila ProteinsGene silencingRNA Small InterferingPoint of ViewMolecular BiologyRegulation of gene expressionbiologyGene Expression ProfilingTemperatureRNACell Biologybiology.organism_classificationMicroRNAsDrosophila melanogaster030104 developmental biologyGene Expression RegulationDNA Transposable ElementsDrosophila melanogasterRNA Biology
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ERK1/2 activation in human taste bud cells regulates fatty acid signaling and gustatory perception of fat in mice and humans

2016

Obesity is a major public health problem. An in-depth knowledge of the molecular mechanisms of oro-sensory detection of dietary lipids may help fight it. Humans and rodents can detect fatty acids via lipido-receptors, such as CD36 and GPR120. We studied the implication of the MAPK pathways, in particular, ERK1/2, in the gustatory detection of fatty acids. Linoleic acid, a dietary fatty acid, induced via CD36 the phosphorylation of MEK1/2-ERK1/2-ETS-like transcription factor-1 cascade, which requires Fyn-Src kinase and lipid rafts in human taste bud cells (TBCs). ERK1/2 cascade was activated by Ca2+ signaling via opening of the calcium-homeostasis modulator-1 (CALHM1) channel. Furthermore, f…

0301 basic medicineSmall interfering RNAMouseCD36BiochemistryMapkObesechemistry.chemical_compound0302 clinical medicinegpr120Cd36Mice Knockoutchemistry.chemical_classificationGene knockdownbiologyKinaseFatty AcidsTaste PerceptionGPR120Taste BudsLipidsProtein-tyrosine kinases3. Good healthTasteBenzamidesBiotechnologymedicine.medical_specialtyMAP Kinase Signaling SystemLinoleic acid[SDV.BC]Life Sciences [q-bio]/Cellular BiologyPreferenceFood Preferences03 medical and health sciencesCalhm1Internal medicineDietary-fatGeneticsmedicineAnimalsHumans[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyCalcium SignalingObesityMolecular Biology[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular Biology[ SDV.BC ] Life Sciences [q-bio]/Cellular BiologyResearchDiphenylamineFatty acidDietary FatsMicroRNAs030104 developmental biologyEndocrinologychemistrybiology.proteinIon-channelCALHM1Src kinase030217 neurology & neurosurgery
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A Systematic Study of Dysregulated MicroRNA in Type 2 Diabetes Mellitus

2017

MicroRNAs (miRNAs) are small noncoding RNAs that modulate the cellular transcriptome at the post-transcriptional level. miRNA plays important roles in different disease manifestation, including type 2 diabetes mellitus (T2DM). Many studies have characterized the changes of miRNAs in T2DM, a complex systematic disease; however, few studies have integrated these findings and explored the functional effects of the dysregulated miRNAs identified. To investigate the involvement of miRNAs in T2DM, we obtained and analyzed all relevant studies published prior to 18 October 2016 from various literature databases. From 59 independent studies that met the inclusion criteria, we identified 158 dysregu…

0301 basic medicineSystematic surveytype 2 diabetes mellitussystematic study030209 endocrinology & metabolismDiseaseBioinformaticsCatalysisArticleInorganic ChemistryTranscriptomelcsh:Chemistry03 medical and health sciences0302 clinical medicineDiabetes mellitusmiRNA-mRNA interaction networkmicroRNAmedicineHumansGene Regulatory NetworksRNA MessengerPhysical and Theoretical Chemistry10. No inequalityMolecular Biologylcsh:QH301-705.5SpectroscopyAdipocytokine Signaling PathwaymicroRNA; type 2 diabetes mellitus; miRNA-mRNA interaction network; systematic studymicroRNAbusiness.industryGene Expression ProfilingOrganic ChemistryType 2 Diabetes MellitusGeneral Medicinemedicine.diseaseComputer Science ApplicationsMicroRNAs030104 developmental biologyDiabetes Mellitus Type 2Gene Expression Regulationlcsh:Biology (General)lcsh:QD1-999Organ SpecificityRNA InterferenceDisease manifestationbusinessTranscriptomeSignal TransductionInternational Journal of Molecular Sciences
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Fasting regulates EGR1 and protects from glucose- and dexamethasone-dependent sensitization to chemotherapy

2017

Fasting reduces glucose levels and protects mice against chemotoxicity, yet drugs that promote hyperglycemia are widely used in cancer treatment. Here, we show that dexamethasone (Dexa) and rapamycin (Rapa), commonly administered to cancer patients, elevate glucose and sensitize cardiomyocytes and mice to the cancer drug doxorubicin (DXR). Such toxicity can be reversed by reducing circulating glucose levels by fasting or insulin. Furthermore, glucose injections alone reversed the fasting-dependent protection against DXR in mice, indicating that elevated glucose mediates, at least in part, the sensitizing effects of rapamycin and dexamethasone. In yeast, glucose activates protein kinase A (P…

0301 basic medicineTime FactorsImmunology and Microbiology (all)Peptide Hormonesmedicine.medical_treatmentAMP-Activated Protein KinasesToxicologyPathology and Laboratory MedicineBiochemistryDexamethasoneMiceEndocrinologyAMP-activated protein kinaseAtrial natriuretic peptideNatriuretic Peptide BrainMedicine and Health SciencesNatriuretic peptideInsulinSmall interfering RNAsBiology (General)Statistical DatabiologyOrganic CompoundsGeneral NeuroscienceMonosaccharidesHeartFastingMetformin3. Good healthMetforminNucleic acidsChemistryPhysical SciencesFemaleAnatomyGeneral Agricultural and Biological SciencesStatistics (Mathematics)Atrial Natriuretic FactorResearch Articlemedicine.drugmedicine.medical_specialtyQH301-705.5medicine.drug_classCarbohydratesEGR1Antineoplastic AgentsCardiotoxinsGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesNatriuretic PeptideStress PhysiologicalInternal medicineGeneticsmedicineAnimalsNon-coding RNAProtein kinase AEarly Growth Response Protein 1Diabetic EndocrinologyNeuroscience (all)Biochemistry Genetics and Molecular Biology (all)Biology and life sciencesToxicityGeneral Immunology and MicrobiologyInsulinOrganic ChemistryChemical CompoundsCorrectionAMPKCyclic AMP-Dependent Protein KinasesHormonesGene regulationDietAtrial Natriuretic PeptideMice Inbred C57BLNeuroscience (all); Immunology and Microbiology (all); Biochemistry Genetics and Molecular Biology (all); Agricultural and Biological Sciences (all)Glucose030104 developmental biologyEndocrinologyAgricultural and Biological Sciences (all)CytoprotectionMetabolic DisordersHyperglycemiaCardiovascular Anatomybiology.proteinRNAGene expressionMathematicsPLOS Biology
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Transient postnatal over nutrition induces long-term alterations in cardiac NLRP3-inflammasome pathway.

2018

International audience; Background and aims: The prevalence of obesity is increasing worldwide at an alarming rate. Altered early nutrition, in particular postnatal overfeeding (PNOF), is a risk factor for impaired cardiac function in adulthood. In the understanding of the initiation or progression of heart diseases, NLRP3 inflammasome and non-coding RNAs have been proposed as key players. In this context, the aim of this study was to decipher the role of NLRP3 inflammasome and its post transcriptional control by micro-RNAs in the regulation of cardiac metabolic function induced by PNOF in mice. Methods and results: Based on a model of mice exposed to PNOF through litter size reduction, we …

0301 basic medicineTime FactorsLitter SizeInflammasomesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentMedicine (miscellaneous)InflammasomeOvernutritionInsulinNutrition and Dieteticsintegumentary systembiologyInflammasomeMicro-RNAsTransfection[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemAnimal Nutritional Physiological PhenomenaSignal transductionCardiology and Cardiovascular Medicinemedicine.drugSignal TransductionCardiac function curvemedicine.medical_specialtyHeart DiseasesCardiac dysfunctionsNutritional StatusContext (language use)Cell LineProto-Oncogene Protein c-ets-103 medical and health sciences[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicineNLR Family Pyrin Domain-Containing 3 ProteinmedicineAnimalsPost-transcriptional regulationNutritionbusiness.industryInsulinMyocardiumRatsMice Inbred C57BLInsulin receptorDisease Models AnimalMicroRNAs030104 developmental biologyEndocrinologyAnimals Newbornbiology.proteinbusinessNutrition, metabolism, and cardiovascular diseases : NMCD
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