Search results for "Real-Time"

showing 10 items of 881 documents

Influence of Age on Cerebral Housekeeping Gene Expression for Normalization of Quantitative Polymerase Chain Reaction after Acute Brain Injury in Mice

2015

To prevent methodological errors of quantitative PCR (qPCR) normalization with reference genes is obligatory. Although known to influence gene expression, impact of age on housekeeping gene expression has not been determined after acute brain lesions such as traumatic brain injury (TBI). Therefore, expression of eight common control genes was investigated at 15 min, 24 h, and 72 h after experimental TBI in 2- and 21-month-old C57Bl6 mice. Expression of β2-microglobulin (B2M), β-actin (ActB), and porphobilinogen deaminase (PBGD) increased after TBI in both ages. β2M demonstrated age-dependent differences and highest inter- and intragroup variations. Expression of cyclophilin A, glyceraldehyd…

Brain ChemistryMaleAgingDNA ComplementaryGenes EssentialInterleukin-6Porphobilinogen deaminaseGene DosageBiologyPolymerase Chain ReactionMolecular biologyHousekeeping geneMice Inbred C57BLMiceCyclophilin AReal-time polymerase chain reactionGene Expression RegulationHypoxanthine-guanine phosphoribosyltransferaseBrain InjuriesReference genesGene expressionAnimalsRNANeurology (clinical)GeneJournal of Neurotrauma
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Cigarette smoke alters IL-33 expression and release in airway epithelial cells

2014

AbstractAirway epithelium is a regulator of innate immune responses to a variety of insults including cigarette smoke. Cigarette smoke alters the expression and the activation of Toll Like Receptor 4 (TLR4), an innate immunity receptor. IL-33, an alarmin, increases innate immunity Th2 responses. The aims of this study were to explore whether mini-bronchoalveolar lavage (mini-BAL) or sera from smokers have altered concentrations of IL-33 and whether cigarette smoke extracts (CSE) alter both intracellular expression (mRNA and protein) and release of IL-33 in bronchial epithelial cells. The role of TLR4 in the expression of IL-33 was also explored.Mini-BALs, but not sera, from smokers show red…

Bronchial epithelial cellLipopolysaccharidesBlotting WesternBronchiInflammationRespiratory MucosaBiologyReal-Time Polymerase Chain ReactionBronchoalveolar LavageImmunoenzyme TechniquesBronchial epithelial cell; COPD; Cigarette smoke; IL-33; InflammationSmokeacute lung injury cigarette smokeinterleukin 33medicineCOPDHumansRNA MessengerReceptorMolecular BiologyCells CulturedCell ProliferationInflammationToll-like receptorInnate immune systemReverse Transcriptase Polymerase Chain ReactionInterleukinsCigarette smokeFlow CytometryInterleukin-33Immunity Innaterespiratory tract diseasesCell biologyToll-Like Receptor 4Interleukin 33ImmunologyIL-33TLR4Molecular MedicineRespiratory epitheliummedicine.symptomIntracellularBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
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Detecting faulty wireless sensor nodes through Stochastic classification

2011

In many distributed systems, the possibility to adapt the behavior of the involved resources in response to unforeseen failures is an important requirement in order to significantly reduce the costs of management. Autonomous detection of faulty entities, however, is often a challenging task, especially when no direct human intervention is possible, as is the case for many scenarios involving Wireless Sensor Networks (WSNs), which usually operate in inaccessible and hostile environments. This paper presents an unsupervised approach for identifying faulty sensor nodes within a WSN. The proposed algorithm uses a probabilistic approach based on Markov Random Fields, requiring exclusively an ana…

Brooks–Iyengar algorithmComputer scienceDistributed computingReal-time computingProbabilistic logicMarkov processMarkov Random Fieldsymbols.namesakeKey distribution in wireless sensor networksWireless Sensor Networks.Autonomic ComputingSensor nodesymbolsOverhead (computing)Algorithm designWireless sensor network2011 IEEE International Conference on Pervasive Computing and Communications Workshops (PERCOM Workshops)
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Decreasing dietary linoleic acid promotes long chain omega-3 fatty acid incorporation into rat retina and modifies gene expression

2011

International audience; Age-related macular degeneration (AMD) may be partially prevented by dietary habits privileging the consumption of ω3 long chain polyunsaturated fatty acids (ω3s) while lowering linoleic acid (LA) intake. The present study aimed to document whether following these epidemiological guidelines would enrich the neurosensory retina and RPE with ω3s and modulate gene expression in the neurosensory retina. Rat progenitors and pups were fed with diets containing low or high LA, and low or high ω3s. After scotopic single flash and 8-Hz-Flicker electroretinography, rat pups were euthanized at adulthood. The fatty acid profile of the neurosensory retina, RPE, liver, adipose tis…

CD36 AntigensMaleMESH : RNA MessengerMESH: 5-Lipoxygenase-Activating ProteinsMESH : Receptors LDLMESH: Electroretinography0302 clinical medicineMESH: Fatty Acids Omega-3MESH: AnimalsMESH : Retinal Ganglion Cellschemistry.chemical_classification0303 health sciencesMESH : Gene Expression RegulationMESH : ElectroretinographyMESH: RetinaMESH: Chromatography GasMESH: Dietary Fats Unsaturateddocosahexaenoic acidpolyunsaturated fatty acidSensory Systems3. Good healthnutritionMESH: Photic StimulationAdipose TissueMESH: Adipose Tissuemedicine.medical_specialtyChromatography Gasmacular degenerationLinoleic acidMESH : Arachidonate 12-LipoxygenaseArachidonate 12-LipoxygenaseMESH : Adipose TissueMESH: Arachidonate 12-Lipoxygenasepufa03 medical and health sciencesMESH : Dietary Fats UnsaturatedlipidElectroretinographyRats Long-EvansRNA MessengerMESH: Linoleic AcidMESH: Antigens CD36MESH : RetinaFatty acidMESH: Retinal Ganglion Cellseye diseasesOphthalmologyEndocrinologychemistryMESH: Receptors LDL030221 ophthalmology & optometryATP-Binding Cassette Transportersn 3MESH: FemalePhotic StimulationMESH: LiverRetinal Ganglion CellsretinaMESH : 5-Lipoxygenase-Activating Proteinsgenetic structures[ SDV.AEN ] Life Sciences [q-bio]/Food and Nutritionretinal pigment epitheliumelectroretinogramMESH : Photic StimulationAdipose tissueangiogenesischemistry.chemical_compoundMESH : FemaleMESH : Rats Long-Evans2. Zero hungermedicine.diagnostic_testMESH : RatsMESH: Real-Time Polymerase Chain ReactionMESH: Gene Expression RegulationMESH : Antigens CD36medicine.anatomical_structureLiverALOX12BiochemistryMESH: ATP-Binding Cassette TransportersFemaleATP Binding Cassette Transporter 1Polyunsaturated fatty acidMESH : Fatty Acids Omega-3MESH: RatsbrainMESH : Male5-Lipoxygenase-Activating ProteinsMESH : Real-Time Polymerase Chain Reactionrhesus monkeyBiologyReal-Time Polymerase Chain ReactionMESH : Chromatography GasLinoleic AcidCellular and Molecular NeuroscienceDietary Fats UnsaturatedMESH : Linoleic AcidMESH: Rats Long-EvansInternal medicineFatty Acids Omega-3medicineAnimalsMESH : ATP-Binding Cassette TransportersOmega 3 fatty acidMESH: RNA Messenger030304 developmental biologydeficient dietRetinal pigment epitheliumMESH : LiverMESH: MaleRatsGene Expression RegulationReceptors LDLgene expressionMESH : Animalssense organs[SDV.AEN]Life Sciences [q-bio]/Food and NutritionElectroretinographyExperimental Eye Research
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The Lipid-Sensor Candidates CD36 and GPR120 Are Differentially Regulated by Dietary Lipids in Mouse Taste Buds: Impact on Spontaneous Fat Preference

2011

BACKGROUND: Recent studies in rodents and humans suggest that the chemoreception of long-chain fatty acids (LCFA) in oral cavity is involved in the spontaneous preference for fatty foods and might contribute to the obesity risk. CD36 and GPR120 are LCFA receptors identified in rodent taste bud cells. The fact that CD36 or GPR120 gene inactivation leads to a decrease in the preference for lipids raises the question of the respective role(s) played by these gustatory lipid-sensor candidates. METHODOLOGY/PRINCIPAL FINDINGS: Using a combination of biochemical, nutritional and behavioural studies in wild-type, CD36(+/-)and CD36(-/-) mice, it was found that: 1°) CD36 and GPR120 display different …

CD36 AntigensMaleTasteChemoreceptorAnatomy and PhysiologyRodentCD36Blotting Westernlcsh:MedicineGene ExpressionReal-Time Polymerase Chain ReactionReceptors G-Protein-CoupledFood PreferencesMicebiology.animalIntegrative PhysiologyGene expressionAnimalsObesityReceptorlcsh:ScienceGeneBiologyNutritionMice KnockoutMultidisciplinarybiologylcsh:RGPR120Taste BudsDietary FatsImmunohistochemistrySensory SystemsCircadian RhythmBiochemistrybiology.proteinMedicinelipids (amino acids peptides and proteins)lcsh:QResearch ArticlePLoS ONE
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Lipid-mediated release of GLP-1 by mouse taste buds from circumvallate papillae: putative involvement of GPR120 and impact on taste sensitivity

2012

Glucagon-like peptide-1 (GLP-1) signaling modulates sweet-taste sensitivity in the mouse. Because circumvallate papillae (CVPs) express both GLP-1 and its receptor, a local regulation has been suggested. However, whether dietary lipids are involved in this regulation, as shown in the gut, is unknown. By using a combination of biochemical, immunohistochemical, and behavioral approaches, the present data i) confirm the role of GLP-1 signaling in the attraction for sucrose, ii) demonstrate that minute quantities of long-chain FAs (LCFAs) reinforce the attraction for sucrose in a GLP-1 receptor-dependent manner, iii) suggest an involvement of the LCFA receptor GPR120 expressed in taste buds in …

CD36 Antigensmedicine.medical_specialtyTasteendocrine systemCD36Blotting WesternQD415-436eating behaviorReal-Time Polymerase Chain ReactionBiochemistryGlucagon-Like Peptide-1 ReceptorReceptors G-Protein-CoupledMiceEndocrinologyTAS1R3TAS1R2Glucagon-Like Peptide 1Cell Line TumorInternal medicinelong-chain fatty acidReceptors GlucagonmedicineAnimalsHumansSecretionObesityReceptorLingual papillaResearch Articlesbiologydigestive oral and skin physiologyGPR120healthCell BiologyTaste BudsImmunohistochemistryMice Inbred C57BLEndocrinologyobesity riskbiology.proteinJournal of Lipid Research
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Soluble GARP has potent antiinflammatory and immunomodulatory impact on human CD4+ T cells

2013

Glycoprotein A repetitions predominant (GARP) is expressed on the surface of activated human regulatory T cells (Treg) and regulates the bioavailability of transforming growth factor-β (TGF-β). GARP has been assumed to require membrane anchoring. To investigate the function of GARP in more detail, we generated a soluble GARP protein (sGARP) and analyzed its impact on differentiation and activation of human CD4⁺ T cells. We demonstrate that sGARP efficiently represses proliferation and differentiation of naïve CD4⁺ T cells into T effector cells. Exposure to sGARP induces Foxp3, decreases proliferation and represses interleukin (IL)-2 and interferon-γ production, resulting in differentiation …

CD4-Positive T-LymphocytesCellular differentiationBlotting WesternTransplantation HeterologousImmunologyAnti-Inflammatory AgentsGraft vs Host DiseaseApoptosisBiologyReal-Time Polymerase Chain ReactionT-Lymphocytes RegulatoryBiochemistryProinflammatory cytokineInterferon-gammaMiceTransforming Growth Factor betamedicineAnimalsHumansRNA MessengerCells CulturedCell ProliferationInflammationMice KnockoutReverse Transcriptase Polymerase Chain ReactionEffectorInterleukinsMembrane ProteinsInterleukinPeripheral toleranceFOXP3Cell DifferentiationForkhead Transcription FactorsCell BiologyHematologyFlow Cytometrymedicine.diseaseCell biologyTransplant rejectionDNA-Binding ProteinsAnimals NewbornHumanized mouseImmunologyInterleukin-2FemaleSignal TransductionBlood
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Antibodies against tumor necrosis factor (TNF) induce T-cell apoptosis in patients with inflammatory bowel diseases via TNF receptor 2 and intestinal…

2011

Background & Aims The anti–tumor necrosis factor (TNF) antibodies infliximab, adalimumab, and certolizumab pegol have proven clinical efficacy in Crohn's disease. Here, we assessed the effects of anti-TNF antibodies on apoptosis in inflammatory bowel disease (IBD). Methods CD14 + macrophages and CD4 + T cells were isolated from peripheral blood and lamina propria mononuclear cells from patients with IBD and control patients. Cell surface markers and apoptosis were assessed by immunohistology and fluorescence-activated cell sorting techniques. Results Lamina propria CD14 + macrophages showed significantly more frequent and higher membrane-bound TNF (mTNF) expression than CD4 + T cells in IBD…

CD4-Positive T-LymphocytesMaleNecrosisCD14Anti-Inflammatory AgentsLipopolysaccharide ReceptorsApoptosisEnzyme-Linked Immunosorbent AssayBiologyAntibodies Monoclonal HumanizedReal-Time Polymerase Chain ReactionPeripheral blood mononuclear cellPolyethylene GlycolsImmunoglobulin Fab FragmentsYoung AdultmedicineHumansReceptors Tumor Necrosis Factor Type IIAntigen-presenting cellAgedLamina propriaHepatologyCluster of differentiationTumor Necrosis Factor-alphaMacrophagesGastroenterologyAdalimumabAntibodies MonoclonalMiddle AgedFlow CytometryInflammatory Bowel DiseasesInfliximabmedicine.anatomical_structureApoptosisCase-Control StudiesImmunologyCertolizumab PegolTumor necrosis factor alphaFemalemedicine.symptomGastroenterology
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IL-28A Is a Key Regulator of T-Cell–Mediated Liver Injury via the T-Box Transcription Factor T-Bet

2006

Background & Aims: T-cell–mediated fulminant hepatitis is a potentially life-threatening event for which the underlying pathogenic mechanisms are not fully understood. Here, we demonstrate a key regulatory role of IL-28A in T-cell–mediated hepatitis. Methods: We cloned the murine IL-28A gene by reverse-transcription polymerase chain reaction, assessed the effects of recombinant IL-28A, and generated IL-28A–transgenic mice. Results: IL-28A induced TH1 cytokine production by CD4+ T lymphocytes in a T-bet–dependent manner and was up-regulated in a murine model of T-cell–mediated hepatitis upon Con A administration. In vivo, CD4+ T cells from newly created IL-28A–transgenic animals revealed an …

CD4-Positive T-Lymphocytesmedicine.medical_treatmentT cellCodon InitiatorMice TransgenicBiologyAntibodiesProinflammatory cytokineInterferon-gammaMiceT-Lymphocyte SubsetsInterferonConcanavalin AmedicineAnimalsCloning MolecularReceptors CytokineFulminant hepatitisLiver injuryHepatitisHepatologyInterleukinsGastroenterologyLiver Failure AcuteOligonucleotides Antisensemedicine.diseaseMolecular biologyMice Inbred C57BLSTAT1 Transcription FactorReal-time polymerase chain reactionCytokinemedicine.anatomical_structureInterleukin-2Interleukin-4MitogensT-Box Domain ProteinsCell DivisionSignal Transductionmedicine.drugGastroenterology
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Embedded neural network system for microorganisms growth analysis

2020

This study presents autonomous system for microorganisms’ growth analysis in laboratory environment. As shown in previous research, laser speckle analysis allows detecting submicron changes of substrate with growing bacteria. By using neural networks for speckle analysis, it is possible to develop autonomous system, that can evaluate microorganisms’ growth by using cheap optics and electronics elements. System includes embedded processing module, CMOS camera, 670nm laser diode and optionally WiFi module for connecting to external image storage system. Due to small size, system could be fully placed in laboratory incubator with constant humidity and temperature. By using laser diode, Petri d…

CMOS sensorLaser diodeArtificial neural networkComputer sciencebusiness.industryReal-time computingENCODElaw.inventionSpeckle patternlawComputer data storageElectronicsAutonomous system (mathematics)businessSaratov Fall Meeting 2019: Optical and Nano-Technologies for Biology and Medicine
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