Search results for "Receptor tyrosine kinase"

showing 10 items of 82 documents

Design of antitumor drugs targeting c-kit receptor by a new mixed ligand-structure based method

2020

An important challenge, in the medicinal chemistry field, is the research of novel forceful drugs to overcome tumor-acquired resistance. The c-Kit tyrosine kinase receptor (TKR) represents a suitable target for the carcinogenesis control of gastro-intestinal stromal (GIST), leukemia, and mastocytosis tumors; nevertheless, several hotspot mutations of the protein limit the efficacy of a few clinical administered TKRs inhibitors. In this study, a new in silico protocol based on ligand and structure-based combined method is proposed, with the aim to identify a set of new c-Kit inhibitors able to complex c-Kit mutated proteins. A recent and freely available web-server DRUDIT is used for the lig…

Gastrointestinal Stromal TumorsIn silicoAntineoplastic AgentsComputational biologyDrug resistanceIn silico protocolsmedicine.disease_causeLigandsReceptor tyrosine kinase03 medical and health sciences0302 clinical medicineDRUDIT web-serverc-KitMaterials ChemistrymedicineHumansPhysical and Theoretical ChemistryProtein Kinase InhibitorsSpectroscopy030304 developmental biology0303 health sciencesbiologyChemistryLigandMixed ligandmedicine.diseaseComputer Graphics and Computer-Aided DesignLeukemiaProto-Oncogene Proteins c-kitDocking (molecular)Drug Resistance Neoplasm030220 oncology & carcinogenesisDrug resistanceMutationMolecular dockingbiology.proteinCarcinogenesis
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Inhibition of the HER2 pathway by n-3 polyunsaturated fatty acids prevents breast cancer in fat-1 transgenic mice

2013

Overexpression of the tyrosine kinase receptor, ErbB2/HER2/Neu, occurs in 25–30% of invasive breast cancer (BC) with poor patient prognosis. Due to confounding factors, inconsistencies still remain regarding the protective effects of n-3 polyunsaturated fatty acids (PUFAs) on BC. We therefore evaluated whether fat-1 transgenic mice, endogenously synthesizing n-3 PUFAs from n-6 PUFAs, were protected against BC development, and we then aimed to study in vivo a mechanism potentially involved in such protection. E0771 BC cells were implanted into fat-1 and wild-type (WT) mice. After tumorigenesis examination, we analyzed the expression of proteins involved in the HER2 signaling pathway and lipi…

Genetically modified mouseFatty Acid DesaturasesDocosahexaenoic AcidsReceptor ErbB-3Receptor ErbB-2Breast NeoplasmsMice TransgenicQD415-436Biologymedicine.disease_causexenograft preventionBiochemistryReceptor tyrosine kinaseProto-Oncogene Proteins c-mycMiceEndocrinologyDownregulation and upregulationCell Line TumorFatty Acids Omega-3medicineAnimalsHumansCaenorhabditis elegans ProteinsResearch ArticlesCell Proliferationchemistry.chemical_classificationCell growthCell BiologyXenograft Model Antitumor AssaysGene Expression Regulation NeoplasticMice Inbred C57BLchemistryDocosahexaenoic acidImmunologyn-3 tissue enrichmentbiology.proteinCancer researchFemaleSignal transductionCarcinogenesispolyunsaturated fatty acid-derived mediatorsPolyunsaturated fatty acidSignal TransductionJournal of Lipid Research
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n-3 polyunsaturated fatty acids and HER2-positive breast cancer: interest of the fat-1 transgenic mouse model over conventional dietary supplementati…

2013

Overexpression of the tyrosine kinase receptor ErbB2/HER2/Neu, occurs in 25%-30% of invasive breast cancer (BC) with poor patient prognosis. Even if numerous studies have shown prevention of breast cancer by n-3 fatty acid intake, the experimental conditions under which n-3 fatty acids exert their protective effect have been variable from study to study, preventing unifying conclusions. Due to confounding factors, inconsistencies still remain regarding protective effects of n-3 polyunsaturated fatty acids (PUFA) on BC. When animals are fed with dietary supplementation in n-3 fatty acids (the traditional approach to modify tissue content and decrease the n-6/n-3 ratio) complex dietary intera…

Genetically modified mouseFatty Acid Desaturasesmedicine.medical_specialtyReceptor ErbB-2Breast NeoplasmsMice TransgenicBiologyBiochemistryReceptor tyrosine kinaseMiceBreast cancerInternal medicineFatty Acids Omega-3medicineAnimalsAnticarcinogenic AgentsHumansDietary supplementationCaenorhabditis elegans Proteinschemistry.chemical_classificationConfoundingFatty acidGeneral Medicinemedicine.disease3. Good healthDisease Models AnimalEndocrinologychemistryDietary Supplementsbiology.proteinFemaleSignal transductionPolyunsaturated fatty acidBiochimie
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Gene structure and function of tyrosine kinases in the marine sponge Geodia cydonium: Autapomorphic characters of Metazoa

1999

Abstract Porifera (sponges) represent the most ancient, extant metazoan phylum. They existed already prior to the ‘Cambrian Explosion’. Based on the analysis of aa sequences of informative proteins, it is highly likely that all metazoan phyla evolved from only one common ancestor (monophyletic origin). As ‘autapomorphic’ proteins which are restricted to Metazoa only, integrin receptors, receptors with scavenger receptor cysteine-rich repeats, neuronal-like receptors and protein–tyrosine kinases (PTKs) have been identified in Porifera. From the marine sponge Geodia cydonium , a receptor tyrosine kinase (RTK) has been cloned that comprises the characteristic structural topology known from oth…

GeneticsDNA ComplementarySequence Homology Amino AcidMolecular Sequence DataIntronReceptor Protein-Tyrosine KinasesGeneral MedicineBiologyExon shufflingAntibodiesIntronsReceptor tyrosine kinasePoriferaTransmembrane domainGeneticsbiology.proteinAnimalsCoding regionCalciumAmino Acid SequenceReceptorTyrosine kinaseGeneGene
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A novel mutation in FGFR-3 disrupts a putative N-glycosylation site and results in hypochondroplasia

2000

Winterpacht, Andreas, Katja Hilbert, Christiane Stelzer, Thorsten Schweikardt, Heinz Decker, Hugo Segerer, Jürgen Spranger, and Bernhard Zabel. A novel mutation in FGFR-3 disrupts a putative N-glycosylation site and results in hypochondroplasia. Physiol. Genomics 2: 9–12, 2000.—Fibroblast growth factor receptor 3 (FGFR3) is a glycoprotein that belongs to the family of tyrosine kinase receptors. Specific mutations in the FGFR3 gene are associated with autosomal dominant human skeletal disorders such as hypochondroplasia, achondroplasia, and thanatophoric dysplasia. Hypochondroplasia (HCH), the mildest form of this group of short-limbed dwarfism disorders, results in ∼60% of cases from a mut…

GlycosylationGlycosylationPhysiologyDNA Mutational AnalysisHypochondroplasiaOsteochondrodysplasiasReceptor tyrosine kinaseMicechemistry.chemical_compoundGeneticsmedicineAnimalsHumansPoint MutationReceptor Fibroblast Growth Factor Type 3N-Glycosylation SiteGeneticschemistry.chemical_classificationBinding SitesBase SequencebiologyInfantDNAProtein-Tyrosine Kinasesmedicine.diseaseReceptors Fibroblast Growth FactorMolecular biologyProtein Structure TertiaryMice Inbred C57BLAmino Acid SubstitutionchemistryFibroblast growth factor receptorMutationbiology.proteinFemaleGlycoproteinNovel mutationPhysiological Genomics
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Polymorphism in the immunoglobulin-like domains of the receptor tyrosine kinase from the sponge Geodia cydonium.

1996

Sponges [Porifera] are the phylogenetically oldest phylum of the Metazoa. They are provided with both cellular and humoral allorecognition systems. The underlying molecules are not yet known. To study allorecognition in sponges we first determined the frequency of graft rejection in a natural population of the marine sponge Geodia cydonium. We then determined, for the first time at the molecular level, the degree of sequence polymorphism in segments of one molecule which may be related to sponge allorecognition and host defense: the Ig-like domains from the receptor tyrosine kinase [RTK]. Thirty six pairs of auto- and allografts were assayed, either by parabiotic attachment or insertion of …

Graft RejectionDNA ComplementaryGeodia cydoniumMolecular Sequence DataImmunoglobulinsPolymerase Chain ReactionReceptor tyrosine kinaseMolecular levelSequence Homology Nucleic AcidAnimalsGeodiaAmino Acid SequenceAllorecognitionGene LibraryPolymorphism GeneticGraft rejectionbiologyBase SequenceSequence Homology Amino AcidReceptor Protein-Tyrosine KinasesGeneral MedicineAnatomySequence Analysis DNAbiology.organism_classificationCell biologyPoriferaSpongesurgical procedures operativebiology.proteinAntibodyCell adhesion and communication
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Origin of metazoan adhesion molecules and adhesion receptors as deduced from cDNA analyses in the marine sponge Geodia cydonium: a review.

1997

The phylogenetic relationships of the kingdom Animalia (Metazoa) have long been questioned. Whether the lowest eukaryotic multicellular organisms, the metazoan phylum Porifera (sponges), independently evolved multicellularity from a separate protist lineage (polyphyly of animals) or whether they were derived from the same protist group as the other animal phyla (monophyly) remains unclear. Analyses of the genes that are typical for multicellularity, e.g. those coding for adhesion molecules (galectin) and adhesion receptors (receptor tyrosine kinase, integrin receptor, receptors featuring scavenger receptor cysteine-rich domains) or elements involved in signal transduction pathways (G-protei…

HistologyDNA ComplementaryMembrane GlycoproteinsbiologyCell adhesion moleculeProtistMembrane ProteinsCell Biologybiology.organism_classificationmedicine.disease_causeReceptor tyrosine kinasePathology and Forensic MedicineCell biologyPoriferaSuberites domunculaSpongeBiochemistryPlatelet Glycoprotein GPIb-IX Complexbiology.proteinmedicineAnimalsSignal transductionReceptorCell Adhesion MoleculesGalectinCell and tissue research
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Towards an understanding of the molecular basis of immune responses in sponges: The marine demospongeGeodia cydonium as a model

1999

The phylogenetic position of the phylum Porifera (sponges) is near the base of the kingdom Metazoa. During the last few years, not only rRNA sequences but, more importantly, cDNA/genes that code for proteins have been isolated and characterized from sponges, in particular from the marine demosponge Geodia cydonium. The analysis of the deduced amino acid sequences of these proteins allowed a molecular biological approach to the question of the monophyly of the Metazoa. Molecules of the extracellular matrix/basal lamina, with the integrin receptor, fibronectin, and galectin as prominent examples, and of cell-surface receptors (tyrosine kinase receptor), elements of sensory systems (crystallin…

HistologybiologyCell adhesion moleculeIntegrinbiology.organism_classificationReceptor tyrosine kinaseFibronectinMedical Laboratory TechnologySpongeDemospongeBiochemistrybiology.proteinAnatomySignal transductionInstrumentationGalectinMicroscopy Research and Technique
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Not all KIT 557/558 codons mutations have the same prognostic influence on recurrence-free survival: breaking the exon 11 mutations in gastrointestin…

2021

Background: Although the gastrointestinal stromal tumor (GIST) genotype is not currently included in risk-stratification systems, a growing body of evidence shows that the pathogenic variant (PV) type and codon location hold a strong prognostic influence on recurrence-free survival (RFS). This information has particular relevance in the adjuvant setting, where an accurate prognostication could help to better identify high-risk tumors and guide clinical decision-making. Materials and Methods: Between January 2005 and December 2020, 96 patients with completely resected GISTs harboring a KIT proto-oncogene receptor tyrosine kinase ( KIT) exon 11 PV were included in the study. We analyzed the t…

KIT proto-oncogene receptor tyrosine kinaseGiSTbusiness.industryplatelet-derived growth factor receptor alphaPlatelet-Derived Growth Factor Receptor AlphaNeoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseasegastrointestinal stromal tumorExon557/558 deletionOncologyRecurrence free survivalGenotypeCancer researchmedicinePrognostic biomarkerGastrointestinal stromal tumors (GISTs)Stromal tumorprognostic biomarkerbusinessRC254-282Therapeutic Advances in Medical Oncology
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LS104, a Novel Kinase Inhibitor, Induces Apoptosis, Synergizes with Cytostatic Drugs and Is Targeting the Receptor Tyrosine Kinase FLT3.

2005

Abstract Fms-like tyrosine kinase 3 (FLT3), a member of the class III tyrosine kinase receptor family, is expressed in up to 90% of acute myeloid leukemia (AML). Activating mutations like internal tandem duplication (ITD) of the juxtamembrane domain and kinase domain point mutations are found in approximately 35% of AML-cases and are considered to represent an attractive therapeutic target. In this study, we report that the novel hydroxystyryl-acrylonitrile compound LS104 induces potent cytotoxic effects in FLT3 ITD-positive leukemic cells. As a cellular model to investigate FLT3-ITD specific effects we used 32D myeloid cells stably transfected with FLT3-ITD and wt-FLT3, respectively. In MT…

KinaseCell growthImmunologyTyrosine phosphorylationCell BiologyHematologyBiologymedicine.diseaseBiochemistryMolecular biologyReceptor tyrosine kinaseLeukemiachemistry.chemical_compoundchemistryAnnexinhemic and lymphatic diseasesembryonic structuresmedicinebiology.proteinKinase activityTyrosineBlood
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