Search results for "Receptor"

showing 10 items of 6990 documents

Are Toll-like receptors and decoy receptors involved in the immunopathogenesis of systemic lupus erythematosus and lupus-like syndromes?

2011

In this paper we focus our attention on the role of two families of receptors, Toll-like receptors (TLR) and decoy receptors (DcR) involved in the generation of systemic lupus erythematosus (SLE) and lupus-like syndromes in human and mouse models. To date, these molecules were described in several autoimmune disorders such as rheumatoid arthritis, antiphospholipids syndrome, bowel inflammation, and SLE. Here, we summarize the findings of recent investigations on TLR and DcR and their role in the immunopathogenesis of the SLE.

lcsh:Immunologic diseases. AllergyChemokineImmunologyInflammationAutoimmunityReview ArticleCell Communicationmedicine.disease_causeAutoantigensAutoimmunityMiceimmune system diseasesToll-like receptormedicineImmunology and AllergyAnimalsHumansLupus Erythematosus SystemicDecoy receptorsReceptorskin and connective tissue diseasesSettore MED/04 - Patologia GeneraleToll-like receptors decoy receptors systemicic erythematous lupusSystemic lupus erythematosusbiologybusiness.industryToll-Like ReceptorsGeneral Medicinemedicine.diseaseImmunity Innatedecoy receptorDisease Models AnimalTumor Necrosis Factor Decoy ReceptorsRheumatoid arthritisImmunologybiology.proteinsystemicic erythematous lupusmedicine.symptomChemokinesbusinesslcsh:RC581-607Tumor Necrosis Factor Decoy ReceptorsSignal Transduction
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CD11b Regulates Fungal Outgrowth but Not Neutrophil Recruitment in a Mouse Model of Invasive Pulmonary Aspergillosis

2019

Abstract Background and Aims: In immunosuppressed individuals Aspergillus (A.) fumigatus is a frequent cause of invasive pulmonary aspergillosis (IPA) which is highly associated with relevant morbidity and mortality. Moreover, it often occurs in patients suffering from leukocyte-adhesion deficiency type 1 (LAD1) which is triggered by a functional loss of CD18 in ß2 integrin receptors as these receptors consist of an alpha subunit (CD11a-CD11d) and CD18 as the common beta subunit. ß2 integrin receptors are differentially expressed by leukocytes, and are required for cell-cell interaction, transendothelial migration, uptake of opsonized pathogens, and cell signaling processes. Here, we asked …

lcsh:Immunologic diseases. AllergyChemokineNeutrophilsPhagocytosisImmunology610 MedizinMedizinMacrophage-1 AntigenCD18InflammationKaplan-Meier EstimateBronchoalveolar LavageBiochemistryMicrobiologyAspergillus fumigatusProinflammatory cytokinecomplement receptor 3MicePhagocytosis610 Medical sciencesmedicineAnimalspneumoniaCC-chemokine ligand 5LungOriginal ResearchInflammationInvasive Pulmonary AspergillosisMice KnockoutCD11b Antigenbiologymedicine.diagnostic_testAspergillus fumigatusCD11bpolymorphonuclear neutrophilsCell BiologyHematologybiology.organism_classificationMice Inbred C57BLDisease Models AnimalBronchoalveolar lavageNeutrophil InfiltrationIntegrin alpha Mβ2 integrinsbiology.proteinCytokinesFemalemedicine.symptomlcsh:RC581-607
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Prediction of Specific TCR-Peptide Binding From Large Dictionaries of TCR-Peptide Pairs

2019

Abstract The T cell repertoire is composed of T cell receptors (TCR) selected by their cognate MHC-peptides and naive TCR that do not bind known peptides. While the task of distinguishing a peptide-binding TCR from a naive TCR unlikely to bind any peptide can be performed using sequence motifs, distinguishing between TCRs binding different peptides requires more advanced methods. Such a prediction is the key for using TCR repertoires as disease-specific biomarkers. We here used large scale TCR-peptide dictionaries with state-of-the-art natural language processing (NLP) methods to produce ERGO (pEptide tcR matchinG predictiOn), a highly specific classifier to predict which TCR binds to which…

lcsh:Immunologic diseases. AllergyComputer scienceevaluation methodsT-LymphocytesT cellImmunologyReceptors Antigen T-CellEpitopes T-LymphocyteTarget peptidePeptide bindingPeptidechemical and pharmacologic phenomenaComputational biologyLigandsSoftware implementationautoencoder (AE)AntigenEvaluation methodsmedicineImmunology and AllergyHumansProtein Interaction Domains and MotifsEpitope specificityAntigensDatabases ProteinOriginal Researchchemistry.chemical_classificationBinding SitesT cell repertoireChemistryRepertoirelong short-term memory (LSTM)T-cell receptorepitope specificitydeep learninghemic and immune systemsmedicine.anatomical_structuremachine learningPeptidesSequence motiflcsh:RC581-607SoftwareProtein BindingSignal TransductionTCR repertoire analysisFrontiers in Immunology
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eIF2α confers cellular tolerance to S. aureus α-toxin

2015

We report on the role of conserved stress-response pathways for cellular tolerance to a pore forming toxin. First, we observed that small molecular weight inhibitors including of eIF2α-phosphatase, jun-N-terminal kinase (JNK), and PI3-kinase sensitized normal mouse embryonal fibroblasts (MEFs) to the small pore forming S. aureus α-toxin. Sensitization depended on expression of mADAM10, the murine ortholog of a proposed high-affinity receptor for α-toxin in human cells. Similarly, eIF2α (S51A/S51A) MEFs, which harbor an Ala knock-in mutation at the regulated Ser51 phosphorylation site of eukaryotic translation initiation factor 2α, were hyper-sensitive to α-toxin. Inhibition of translation w…

lcsh:Immunologic diseases. AllergyMAPK/ERK pathwayImmunologyeIF2αBiologyCycloheximide03 medical and health scienceschemistry.chemical_compoundCellular toleranceImmunology and AllergyInitiation factorpore forming toxinsReceptorOriginal Research030304 developmental biologyGenetics0303 health sciencesKinase030302 biochemistry & molecular biologyJNK Mitogen-Activated Protein KinasesADAM10Translation (biology)MAPKCell biologyEIF2AK4chemistryPhosphorylationCytolysinS. aureus α-toxinlcsh:RC581-607Frontiers in Immunology
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IL-2 Expression in Activated Human Memory FOXP3(+) Cells Critically Depends on the Cellular Levels of FOXP3 as Well as of Four Transcription Factors …

2012

The human CD4(+)FOXP3(+) T cell population is heterogeneous and consists of various subpopulations which remain poorly defined. Anergy and suppression are two main functional characteristics of FOXP3(+)Treg cells. We used the anergic behavior of FOXP3(+)Treg cells for a better discrimination and characterization of such subpopulations. We compared IL-2-expressing with IL-2-non-expressing cells within the memory FOXP3(+) T cell population. In contrast to IL-2-non-expressing FOXP3(+) cells, IL-2-expressing FOXP3(+) cells exhibit intermediate characteristics of Treg and Th cells concerning the Treg cell markers CD25, GITR, and Helios. Besides lower levels of FOXP3, they also have higher levels…

lcsh:Immunologic diseases. AllergyT cellLymphocytePopulationImmunologychemical and pharmacologic phenomenaBiologylymphocyteFlow cytometrytranscription factorsmedicineImmunology and Allergycytokine expressionIL-2 receptorddc:610educationTranscription factorOriginal Researcheducation.field_of_studyIL-2 expressionmedicine.diagnostic_testT cell activationflow cytometryhuman Treg cellsFOXP3T cellhemic and immune systemsmemory Th cellsPhenotypeCell biologymedicine.anatomical_structureImmunologylcsh:RC581-607610 Medizin und GesundheitFrontiers in immunology
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Quantitative Prediction of the Landscape of T Cell Epitope Immunogenicity in Sequence Space

2019

Immunodominant T cell epitopes preferentially targeted in multiple individuals are the critical element of successful vaccines and targeted immunotherapies. However, the underlying principles of this "convergence" of adaptive immunity among different individuals remain poorly understood. To quantitatively describe epitope immunogenicity, here we propose a supervised machine learning framework generating probabilistic estimates of immunogenicity, termed "immunogenicity scores," based on the numerical features computed through sequence-based simulation approximating the molecular scanning process of peptides presented onto major histocompatibility complex (MHC) by the human T cell receptor (T…

lcsh:Immunologic diseases. AllergyT cellT-LymphocytesImmunologyReceptors Antigen T-CellDatasets as TopicEpitopes T-Lymphocytechemical and pharmacologic phenomenaComputational biologyBiologyAdaptive ImmunityimmunogenicityMajor histocompatibility complexEpitopeMajor Histocompatibility ComplexmedicineImmunology and AllergyHumansComputer SimulationAntigen PresentationImmunodominant EpitopesRepertoireImmunogenicityT-cell receptorComputational BiologyAcquired immune systemmedicine.anatomical_structuremachine learningescape mutationbiology.proteinThermodynamicsT cell receptor repertoireSequence space (evolution)lcsh:RC581-607T cell epitopeFrontiers in Immunology
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γδ T Cells Cross-Link Innate and Adaptive Immunity in Mycobacterium tuberculosis Infection

2011

Protective immunity against mycobacterial infections such asMycobacterium tuberculosisis mediated by interactions between specific T cells and activated antigen presenting cells. To date, many aspects of mycobacterial immunity have shown that innate cells could be the key elements that substantially may influence the subsequent adaptive host response. During the early phases of infection, innate lymphocyte subsets play a pivotal role in this context. Here we summarize the findings of recent investigations onγδT lymphocytes and their role in tuberculosis immunity.

lcsh:Immunologic diseases. AllergyT-LymphocytesT cellImmunologyReview ArticleAdaptive ImmunityLymphocyte ActivationMycobacterium tuberculosisImmune systemAntigenImmunitymedicineAnimalsHumansTuberculosisImmunology and AllergyIL-2 receptorAntigen-presenting cellbiologyReceptors Antigen T-Cell gamma-deltaMycobacterium tuberculosisGeneral MedicineAcquired immune systembiology.organism_classificationVirologyImmunity Innategamma delta T cells Mycobacterium tuberculosismedicine.anatomical_structureImmunologylcsh:RC581-607Immunologic Memory
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Instant kit preparation of 68Ga-radiopharmaceuticals via the hybrid chelator DATA: clinical translation of [68Ga]Ga-DATA-TOC

2019

Purpose The widespread use of 68Ga for positron emission tomography (PET) relies on the development of radiopharmaceutical precursors that can be radiolabelled and dispensed in a simple, quick, and convenient manner. The DATA (6-amino-1,4-diazapine-triacetate) scaffold represents a novel hybrid chelator architecture possessing both cyclic and acyclic character that may allow for facile access to 68Ga-labelled tracers in the clinic. We report the first bifunctional DATA chelator conjugated to [Tyr3]octreotide (TOC), a somatostatin subtype 2 receptor (SST2)-targeting vector for imaging and functional characterisation of SSTR2 expressing tumours. Methods The radiopharmaceutical precursor, DATA…

lcsh:Medical physics. Medical radiology. Nuclear medicineNET540 Chemistry and allied sciencesDOTA-TOC540 Chemielcsh:R895-920Gallium-68PET-CTDATA-TOCMolecular imagingOriginal ResearchSomatostatin receptorEJNMMI Research
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Progenitor death drives retinal dysplasia and neuronal degeneration in a mouse model of Atrip-Seckel syndrome

2020

ABSTRACT Seckel syndrome is a type of microcephalic primordial dwarfism (MPD) that is characterized by growth retardation and neurodevelopmental defects, including reports of retinopathy. Mutations in key mediators of the replication stress response, the mutually dependent partners ATR and ATRIP, are among the known causes of Seckel syndrome. However, it remains unclear how their deficiency disrupts the development and function of the central nervous system (CNS). Here, we investigated the cellular and molecular consequences of ATRIP deficiency in different cell populations of the developing murine neural retina. We discovered that conditional inactivation of Atrip in photoreceptor neurons …

lcsh:MedicineMedicine (miscellaneous)315BlindnessMicechemistry.chemical_compoundImmunology and Microbiology (miscellaneous)Cell DeathneurodevelopmentStem CellsNeurodegenerationapoptosisneurodegenerationSyndromeCell biologyDNA-Binding Proteinsdna damage responsemedicine.anatomical_structurePhotoreceptor Cells VertebrateResearch Articlelcsh:RB1-214NeurogenesisNeuroscience (miscellaneous)Embryonic DevelopmentBiologyRetinaGeneral Biochemistry Genetics and Molecular Biologylcsh:PathologymedicineAnimalsAbnormalities MultipleProgenitor cellVision OcularAdaptor Proteins Signal TransducingCell ProliferationProgenitorRetinalcsh:RRetinalEmbryo Mammalianmedicine.diseasephotoreceptorDisease Models AnimalSeckel syndromechemistryvisual system developmentNerve DegenerationRetinal dysplasiaRetinal DysplasiaTumor Suppressor Protein p53Primordial dwarfismDNA DamageDisease Models & Mechanisms
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Modulation of the Endocannabinoids N-Arachidonoylethanolamine (AEA) and 2-Arachidonoylglycerol (2-AG) on Executive Functions in Human.

2013

Animal studies point to an implication of the endocannabinoid system on executive functions. In humans, several studies have suggested an association between acute or chronic use of exogenous cannabinoids (Δ9-tetrahydrocannabinol) and executive impairments. However, to date, no published reports establish the relationship between endocannabinoids, as biomarkers of the cannabinoid neurotransmission system, and executive functioning in humans. The aim of the present study was to explore the association between circulating levels of plasma endocannabinoids N-arachidonoylethanolamine (AEA) and 2-Arachidonoylglycerol (2-AG) and executive functions (decision making, response inhibition and cognit…

lcsh:MedicineNeuropsychological TestsSocial and Behavioral SciencesPrefrontal cortexReceptores de cannabinoides:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Primates::Haplorhini::Catarrhini::Hominidae::Humans [Medical Subject Headings]Executive FunctionEndocrinologyCognitionWisconsin Card Sorting TestEndocrinologiaHuman PerformanceMedicinePsychologyendocannabinoid systemPrefrontal cortexlcsh:ScienceProblem SolvingPsychiatryMultidisciplinaryCognitive NeurologyCognitive flexibilityPresa de decisionsCognitionMiddle AgedExecutive functionsexecutive functionsHumanos2-arachidonoylglycerolSubstance abuseEndocannabinoidesMental HealthNeurologyCognicióMedicineFemalelipids (amino acids peptides and proteins):Chemicals and Drugs::Amino Acids Peptides and Proteins::Proteins::Membrane Proteins::Receptors Cell Surface::Receptors G-Protein-Coupled::Receptors Cannabinoid [Medical Subject Headings]Animal behaviorClinical psychology:Chemicals and Drugs::Lipids::Fatty Acids::Fatty Acids Unsaturated::Arachidonic Acids [Medical Subject Headings]HumanResearch ArticleAdultN-arachidonoylethanolamine:Psychiatry and Psychology::Mental Disorders::Substance-Related Disorders::Marijuana Abuse [Medical Subject Headings]Polyunsaturated AlkamidesCognitive NeuroscienceDecision MakingArachidonic AcidsGlyceridesYoung AdultCannabinoides -- ReceptorsNeuropsychologyCànnabis:Diseases::Nervous System Diseases::Neurodegenerative Diseases [Medical Subject Headings]Pruebas neuropsicológicasÁcidos araquidónicosHumansFunción ejecutivaBiologyCannabisCannabinoides -- Efectes fisiològics:Anatomy::Nervous System::Central Nervous System [Medical Subject Headings]Behavior:Chemicals and Drugs::Chemical Actions and Uses::Pharmacologic Actions::Molecular Mechanisms of Pharmacological Action::Neurotransmitter Agents::Endocannabinoids [Medical Subject Headings]business.industrylcsh:RCognitive Psychologymedicine.diseaseIowa gambling taskEnfermedades neurodegenerativasAbuso de marihuanaSistema nervioso central:Psychiatry and Psychology::Behavioral Disciplines and Activities::Psychological Tests::Neuropsychological Tests [Medical Subject Headings]:Psychiatry and Psychology::Psychological Phenomena and Processes::Mental Processes::Executive Function [Medical Subject Headings]lcsh:QbusinessDecision makingStroop effectNeuroscienceEndocannabinoids
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