Search results for "Receptor"

showing 10 items of 6990 documents

The estrogen receptor α:insulin receptor substrate 1 complex in breast cancer: structure–function relationships

2007

Background: Insulin receptor substrate 1 (IRS-1) is a signaling molecule that exerts a key role in mediating cross talk between estrogen receptor a (ERa) and insulin-like growth factor 1 (IGF-1) in breast cancer cells. Previously, we demonstrated that a fraction of IRS-1 binds ERa, translocates to the nucleus, and modulates ERa-dependent transcription at estrogen response elements (ERE). Here, we studied structure-function relationships of the ER-a:IRS-1 complex under IGF-1 and/or estradiol (E 2 ) stimulation. Materials and methods: ERa and IRS-1 deletion mutants were used to analyze structural and functional ERα/IRS-1 interactions. IRS-1 binding to ERE and IRS-1 role in ERa-dependent ERE t…

medicine.medical_specialtyInsulin Receptor Substrate ProteinsActive Transport Cell NucleusEstrogen receptorRepressorBreast NeoplasmsBiologyStructure-Activity Relationshipestrogen receptor alpha (ERa) Insulin receptor substrate 1 (IRS-1) breast cancerCell Line TumorInternal medicineCoactivatormedicineHumansInsulin-Like Growth Factor IReceptors InterferonEstradiolEstrogen Receptor alphaHematologyDNA-binding domainPhosphoproteinsPeptide FragmentsReceptor InsulinProtein Structure TertiaryCell biologyIRS1Repressor ProteinsPleckstrin homology domainEndocrinologyOncologyInsulin Receptor Substrate ProteinsFemaleChromatin immunoprecipitationProtein BindingAnnals of Oncology
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NMDA-induced acetylcholine release in mouse striatum: role of NO synthase isoforms

2002

Striatal cholinergic interneurons are stimulated by glutamatergic inputs from thalamus and cortex via NMDA receptors. The present microdialysis study was designed to characterize the role of nitric oxide (NO) in this process and to identify the NO synthase (NOS) isoform responsible for this effect. For this purpose, we studied the effects of NMDA and 3-morpholino sydnonimine (SIN-1) perfusions on the release of acetylcholine (ACh) in mouse striatum. In wild-type C57/Bl6 mice, perfusion of NMDA (100 µm) induced a two-fold stimulation of ACh release. This effect was attenuated in mice lacking endothelial NOS but was completely absent in mice lacking neuronal NOS. Local perfusion of SIN-1 (300…

medicine.medical_specialtyInterneuronGlutamate receptorBiologyEndothelial NOSBiochemistryCellular and Molecular NeuroscienceGlutamatergicEndocrinologymedicine.anatomical_structurenervous systemInternal medicinemedicineNMDA receptorCholinergicCholinergic neuronAcetylcholinemedicine.drugJournal of Neurochemistry
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Impaired border zone formation and adverse remodeling after reperfused myocardial infarction in cannabinoid CB2 receptor deficient mice.

2014

Abstract Aims Reperfusion of myocardial infarction is associated with inflammatory reaction and subsequent myocardial remodeling with a rapid scar formation in mice. The cannabinoid receptor CB2 has been associated with cardioprotection and regulation of macrophage function. We investigated its role in remodeling of reperfused infarction. Main methods One hour LAD-occlusion was followed by reperfusion over 6 h and 1, 3 and 7 days in wild-type C57/BL6J (WT) and CB2 receptor-deficient (Cnr2 −/− ) mice (n = 8/group). Hearts were processed for functional, morphological and mRNA/protein analysis, and tissue concentration of endocannabinoids was determined using liquid chromatography-multiple rea…

medicine.medical_specialtyIschemiaMyocardial InfarctionInfarctionMyocardial Reperfusion InjuryGeneral Biochemistry Genetics and Molecular BiologyReceptor Cannabinoid CB2MiceInternal medicinemedicineCannabinoid receptor type 2AnimalsMyocytes CardiacMyocardial infarctionGeneral Pharmacology Toxicology and PharmaceuticsCardioprotectionInflammationMice KnockoutbiologyChemistryMyocardiumTenascin CHemodynamicsGranulation tissueGeneral Medicinemedicine.diseaseEndocannabinoid systemMice Inbred C57BLEndocrinologymedicine.anatomical_structureCardiologybiology.proteinGranulation TissueCytokinesLife sciences
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The CB1 cannabinoid receptor mediates excitotoxicity-induced neural progenitor proliferation and neurogenesis.

2007

Endocannabinoids are lipid signaling mediators that exert an important neuromodulatory role and confer neuroprotection in several types of brain injury. Excitotoxicity and stroke can induce neural progenitor (NP) proliferation and differentiation as an attempt of neuroregeneration after damage. Here we investigated the mechanism of hippocampal progenitor cell engagement upon excitotoxicity induced by kainic acid administration and the putative involvement of the CB1 cannabinoid receptor in this process. Adult NPs express kainate receptors that mediate proliferation and neurosphere generation in vitro via CB1 cannabinoid receptors. Similarly, in vivo studies showed that excitotoxicity-induce…

medicine.medical_specialtyKainic acidCannabinoid receptorNeurotoxinsExcitotoxicityKainate receptorBiologymedicine.disease_causeBiochemistryNeuroprotectionHippocampuschemistry.chemical_compoundMiceReceptor Cannabinoid CB1Epidermal growth factorInternal medicinemedicineAnimalsMolecular BiologyCell ProliferationMice KnockoutNeuronsKainic AcidStem CellsNeurogenesisCell BiologyEndocannabinoid systemCell biologyNerve RegenerationEndocrinologynervous systemchemistrylipids (amino acids peptides and proteins)Fibroblast Growth Factor 2The Journal of biological chemistry
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Estrogenic Modulation of Longevity by Induction of Antioxidant Enzymes

2010

In many species including humans, females live longer than males. We and others have observed that mitochondria from females of Wistar rats and of OF1 mice produce half the amount of peroxide produced by males. We attributed this to a change in the expression of antioxidant, longevity-related genes. We have found that in those species in which females live longer than males, estrogens activate longevity-related genes, particularly antioxidant ones. It should be emphasized that estrogens do not act as antioxidants because of their phenolic ring but rather they act indirectly; that is, they behave as hormones and bind to estrogen receptors, which eventually leads to the upregulation of the ex…

medicine.medical_specialtyKinaseFeminization (biology)Estrogen receptorBiologychemistry.chemical_compoundEndocrinologychemistryDownregulation and upregulationInternal medicinemedicinePhytoestrogensEstrogen receptor alphaEstrogen receptor betaHormone
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Developmental activation and segment-specific co-expression of kininogen and bradykinin (BK) B2-receptors during rat nephrogenesis. • 2143

1996

Developmental activation and segment-specific co-expression of kininogen and bradykinin (BK) B 2 -receptors during rat nephrogenesis. • 2143

medicine.medical_specialtyKininogenchemistry.chemical_compoundEndocrinologychemistryInternal medicinePediatrics Perinatology and Child HealthmedicineBradykininBiologyReceptorcirculatory and respiratory physiologyPediatric Research
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Sinusoidal endothelial liver cells in vitro release endothelin — Augmentation by transforming growth factorβ and Kupffer cell-conditioned media

1991

Endothelin is the most potent vasoconstrictor peptide known today. Using a radioimmunoassay for endothelin, we measured immunoreactive endothelin in culture media of guinea pig sinusoidal endothelial liver cells and human umbilical vein endothelial cells. A time-dependent release of immunoreactive endothelin by confluent sinusoidal endothelial liver cells in culture was found. Sinusoidal endothelial liver cells produced similar amounts of immunoreactive endothelin as umbilical vein endothelial cells, about 900 pg/microgram DNA per 24 h. In the presence of transforming growth factor beta a dose-dependent increase of immunoreactive endothelin release was measured. The maximal increase of 50% …

medicine.medical_specialtyKupffer CellsGuinea PigsRadioimmunoassayUmbilical veinTransforming Growth Factor betaInternal medicineDrug DiscoverymedicineAnimalsCells CulturedGenetics (clinical)Dose-Response Relationship DrugbiologyChemistryEndothelinsKupffer cellRadioimmunoassayGeneral MedicineTransforming growth factor betaEndothelin 1In vitroEndocrinologymedicine.anatomical_structureLivercardiovascular systembiology.proteinMolecular MedicineFemaleEndothelium VascularEndothelin receptorTransforming growth factorKlinische Wochenschrift
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Appetite and Obesity

2011

Orexin (hypocretin) plays an important role in promoting wakefulness [1]. It stimulates wakefulness when injected in the cerebral ventricles, in the periventricular nucleus, dorsomedial hypothalamus, or lateral hypothalamus [2, 3]. Orexinergic neurons originate from the lateral hypothalamus and have projections to most parts of the central nervous system including the brain stem. The lack of orexin and/or orexin receptors is linked to narcolepsy [4–6].

medicine.medical_specialtyLateral hypothalamusbusiness.industrymedia_common.quotation_subjectdigestive oral and skin physiologyAppetitemedicine.diseaseOrexin receptorOrexinEndocrinologymedicine.anatomical_structurenervous systemHypothalamusInternal medicinemental disordersmedicineWakefulnessPeriventricular nucleusbusinesshormones hormone substitutes and hormone antagonistspsychological phenomena and processesNarcolepsymedia_common
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Leptin and Leptin Receptor Are Expressed in the Human Endometrium and Endometrial Leptin Secretion Is Regulated by the Human Blastocyst1

2000

Embryonic implantation is a crucial event for the human reproductive function. Cytokines and paracrine molecules have been proposed as putative local regulators of this process. The leptin or the OB protein has been linked to the reproductive function and inflammatory response. In the present study, we describe for the first time the expression of leptin and leptin receptor (long form) in the secretory endometrium and that endometrial leptin secretion is regulated in vitro by the human blastocyst. Leptin and leptin receptor messenger RNA and protein were identified in secretory endometrium and in cultured endometrial epithelial cells (EECs) by RT-PCR, Western blot, and immunohistochemistry.…

medicine.medical_specialtyLeptin receptorEndocrinology Diabetes and MetabolismLeptindigestive oral and skin physiologyBiochemistry (medical)Clinical BiochemistryBiologyEndometriumBiochemistryBlotParacrine signallingEndocrinologyEndocrinologymedicine.anatomical_structureInternal medicineembryonic structuresmedicineSecretionBlastocystReceptorhormones hormone substitutes and hormone antagonistsThe Journal of Clinical Endocrinology & Metabolism
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Impairment of leptin/leptin receptor pathway in nasal epithelium from allergic turbinates

2016

BACKGROUND: Allergic rhinitis (AR) is characterized by a remodeling of nasal epithelium. Leptin adipokine has been already identified as a marker of homeostasis in human bronchial epithelial of asthmatics. TGF-β is a multi-functional cytokine and conflicting findings exist regarding its role in the remodeling responses of the upper airways in allergic rhinitis. OBJECTIVE: We sought to investigate ex-vivo the expression of leptin/leptin receptor pathway and TGF-β in human nasal epithelium. METHODS: 41 biopsies of inferior turbinates obtained from allergic patients with AR (A, n = 20) and from healthy control subjects (C, n = 21) were analyzed for leptin/leptin receptor and for TGF-β 1, 2, 3 …

medicine.medical_specialtyLeptin receptorbusiness.industryLeptinmedicine.medical_treatmentdigestive oral and skin physiologyAdipokineEpitheliummedicine.anatomical_structureEndocrinologyCytokineInternal medicineImmunologyMedicineImmunohistochemistrybusinessReceptorhormones hormone substitutes and hormone antagonistsHomeostasis3.2 Airway Cell Biology and Immunopathology
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