Search results for "Receptor"

showing 10 items of 6990 documents

Molecular anatomy of the neuro-immune connection.

1991

Light microscopic immunohistochemistry was employed to elucidate and compare the presence, distribution, and coexistence of various peptides, neuroendocrine markers and enzymes of the catecholamine pathway in nerves supplying lymphoid tissues in a variety of mammalian species. All lymphoid organs and tissues receive innervation by fibers containing dopamine-beta-hydroxylase and/or tyrosine hydroxylase, neural markers like protein gene product 9.5, synaptophysin and neurofilament and a varied spectrum of peptides. The prominent peptides were tachykinins (substance P, neurokinin A), calcitonin gene-related peptide (CGRP), neuropeptide Y (NPY), and vasoactive intestinal polypeptide/peptide his…

medicine.medical_specialtyNeurofilamentLymphoid TissueNeuroimmunomodulationSwineVasoactive intestinal peptideGuinea PigsPalatine TonsilFluorescent Antibody TechniqueSubstance PThymus GlandCalcitonin gene-related peptidechemistry.chemical_compoundMiceDogsBone MarrowInternal medicineNeural PathwaysmedicineAnimalsHumansNeuronsSheepTyrosine hydroxylasebiologyChemistryGeneral NeuroscienceGeneral MedicineAnatomyNeuropeptide Y receptorImmunohistochemistryRatsEndocrinologySynaptophysinbiology.proteinCatsNeurokinin ALymph NodesPeptidesSpleenThe International journal of neuroscience
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Migraine and cluster headache – the common link

2018

Abstract Although clinically distinguishable, migraine and cluster headache share prominent features such as unilateral pain, common pharmacological triggers such glyceryl trinitrate, histamine, calcitonin gene-related peptide (CGRP) and response to triptans and neuromodulation. Recent data also suggest efficacy of anti CGRP monoclonal antibodies in both migraine and cluster headache. While exact mechanisms behind both disorders remain to be fully understood, the trigeminovascular system represents one possible common pathophysiological pathway and network of both disorders. Here, we review past and current literature shedding light on similarities and differences in phenotype, heritability…

medicine.medical_specialtyNeurologyCluster headacheImplantable Neurostimulators/statistics & numerical dataPain medicineCalcitonin Gene-Related PeptideDeep Brain StimulationMigraine DisordersNitroglycerin/adverse effectsHypothalamuslcsh:MedicineTriptansReviewCalcitonin gene-related peptideBioinformatics03 medical and health sciencesNitroglycerin0302 clinical medicinemedicineHumans030212 general & internal medicineTryptamines/pharmacologyMigraineTrigeminovascular systembusiness.industryNeuromodulationCluster Headache/bloodCluster headacheAnti-CGRP (receptor) monoclonal antibodies – mAbsMigraine Disorders/bloodTrigeminovascular systemlcsh:RGeneral MedicineCalcitonin gene-related peptide (CGRP)medicine.diseaseDeep Brain Stimulation/statistics & numerical dataNeuromodulation (medicine)Tryptamines3. Good healthCalcitonin Gene-Related Peptide/antagonists & inhibitorsAnesthesiology and Pain MedicineImplantable NeurostimulatorsMigraineNeurology (clinical)business030217 neurology & neurosurgerymedicine.drugThe Journal of Headache and Pain
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Beta-adrenoceptor stimulation enhances transmitter output from the rat phrenic nerve.

1988

Abstract 1. Neurally-evoked output of newly synthesized [3H]-acetylcholine from the rat phrenic nerve was measured in the absence of cholinesterase inhibitors. 2. Noradrenaline and isoprenaline enhanced neurally-evoked transmitter output markedly. Moreover, immediately after the application of noradrenaline the basal tritium efflux increased significantly. 3. Pretreatment with propranolol (0.1 mumol l-1) or atenolol (0.3 mumol l-1) completely prevented the stimulatory effect of noradrenaline and isoprenaline on evoked transmitter output. 4. The facilitatory effect of isoprenaline declined, when the exposure time was increased. This observation supports the assumption that beta-adrenoceptors…

medicine.medical_specialtyNeuromuscular transmissionMotor nerveStimulationIn Vitro Techniqueschemistry.chemical_compoundNorepinephrineInternal medicineIsoprenalineReceptors Adrenergic betamedicineAnimalsNeurotransmitterPhrenic nervePharmacologyNeurotransmitter Agentsbusiness.industryIsoproterenolRats Inbred StrainsAtenololPropranololRatsPhrenic NerveEndocrinologymedicine.anatomical_structurechemistryAtenololPeripheral nervous systembusinessmedicine.drugResearch Article
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Presence of muscarinic inhibitory and absence of nicotinic excitatory receptors at the terminal sympathetic nerves of chicken hearts.

1976

Nicotine (2 X 10(-4) M) or acetylcholine (5.5 X 10(-4) M) in the presence of 3 X 10(-6) M atropine did not increase the rate or amplitude of contraction in isolated atria or ventricular strips of the chicken heart; both drugs also did not cause an output of noradrenaline or adrenaline and did not evoke antidromic discharges in the right sympathetic nerves of isolated perfused chicken hearts. In contrast, "high K+-solutions" evoked an output of noradrenaline and adrenaline and caused a burst of antidromic discharges. Dimethylphenylpiperazine (DMPP; 3.1 X 10(-4) M), by a tyramine-like action, elicited a small output of noradrenaline and increased rate and amplitude of contraction" but did not…

medicine.medical_specialtyNicotineSympathetic Nervous SystemEpinephrineTyramineStimulationIn Vitro TechniquesReceptors NicotinicInhibitory postsynaptic potentialNorepinephrineHeart RateInternal medicineMuscarinic acetylcholine receptormedicineAnimalsReceptors CholinergicEvoked PotentialsPharmacologyChemistryMyocardiumHeartGeneral MedicineMyocardial ContractionReceptors MuscarinicAcetylcholineAntidromicAtropineNicotinic agonistEndocrinologyExcitatory postsynaptic potentialCatsPotassiumDimethylphenylpiperazinium IodideChickensAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Differential blockade by nifedipine and ω-conotoxin GVIA of α1- and β1-adrenoceptor-controlled calcium channels on motor nerve terminals of the rat

1990

Electrically evoked release of [3H]acetylcholine ([3H]ACh) from the rat phrenic nerve and its facilitation by stimulation of presynaptic alpha 1- and beta 1-adrenoceptors were investigated in the absence and presence of nifedipine and omega-conotoxin GVIA. Both calcium channel antagonists did not modify electrically evoked [3H]ACh release, but selectively blocked the effect triggered by both facilitatory adrenergic receptors. The increase in [3H]ACh release mediated via beta 1-adrenoceptor activation was abolished by low concentrations (1 nM) of omega-conotoxin GVIA, whereas nifedipine (100 nM) abolished the facilitatory effect mediated via alpha 1-adrenoceptor stimulation. Therefore, the b…

medicine.medical_specialtyNifedipineNeuromuscular JunctionMollusk Venomschemistry.chemical_elementIn Vitro TechniquesCalciumN-type calcium channelMotor Endplatecomplex mixturesNeuromuscular junctionNifedipineomega-Conotoxin GVIAInternal medicinemedicineAnimalsL-type calcium channelVoltage-dependent calcium channelChemistryGeneral NeuroscienceCalcium channelRats Inbred StrainsCalcium Channel BlockersAcetylcholineElectric StimulationRatsReceptors AdrenergicPhrenic Nervemedicine.anatomical_structureEndocrinologyCalcium ChannelsAcetylcholinemedicine.drugNeuroscience Letters
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Effect of cocaine and related drugs on the uptake of noradrenaline by heart and spleen

1961

Noradrenaline uptake by heart and spleen after intravenous infusion of noradrenaline was measured in the pithed rat. Cocaine, given before the infusion, inhibited the noradrenaline uptake in relation (a) to the dose administered and (b) to the amount of noradrenaline infused. There was an association between increase in the pressor response to a test dose of noradrenaline and inhibition of the uptake by the heart. Drugs related chemically to cocaine, such as alpha-cocaine, amethocaine, and atropine, did not alter the noradrenaline uptake or potentiate the blood pressure response to noradrenaline. The noradrenaline uptake by the heart was unchanged after dibenamine, but blocked by the dichlo…

medicine.medical_specialtyNoradrenaline uptakeAdrenergic receptorAdrenergicSpleenPharmacologyNorepinephrineCocaineIsoprenalineInternal medicinemedicineAnimalsVasoconstrictor AgentsReceptorChemistryMyocardiumIsoproterenolHeartGeneral MedicineArticlesRatsReceptors AdrenergicAtropinemedicine.anatomical_structureEndocrinologyBlood pressureSpleenmedicine.drug
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Hysteroscopic and ultrasonographic evaluation of ulipristal acetate treatment for symptomatic myomas in premenopausal women: a prospective study

2020

Objective: This study aimed to analyze the effects of a six-month therapy with ulipristal acetate (UPA) on myoma size and endometrial thickness in premenopausal women. Material and methods: Seventy-four women undergoing conservative therapy with UPA were enrolled for this study. All women underwent transvaginal ultrasound evaluation to assess the endometrial thickness, and the number and size of myomas at the beginning and after six months. Hysteroscopy and biopsy were performed after six months, if necessary. Results: After six months of treatment, sonographic examination showed a statistically significant (p <.05) reduction of the size of the largest myoma (56.3 ± 5.1 vs. 31.7 ± 10.1 m…

medicine.medical_specialtyNorpregnadienesUterine fibroidsHysteroscopy03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePregnancyUlipristal acetateSelective progesterone receptor modulatorulipristal acetatemedicineHumansProspective Studiesuterine fibroidsProspective cohort studyGynecologyLeiomyomamedicine.diagnostic_testbusiness.industryMetrorrhagiaMyomaselective progesterone receptor modulatormedicine.diseaseMyomachemistryHysteroscopy030220 oncology & carcinogenesisUterine NeoplasmsFemale030211 gastroenterology & hepatologySurgerymedicine.symptombusinessmetrorrhagia
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Lack of binding of gestodene to estrogen receptor in human breast cancer tissue

1990

Competition studies with progesterone and estradiol receptors of human myometrial tissue as well as of mammary cancer tissue showed that gestodene bound with high affinity to the progesterone receptor, as did other synthetic and natural progestogens. However, gestodene did not bind to the estradiol receptor. The relative binding affinities of all tested synthetic and natural ligands showed no organ-specific differences and no differences between neoplastically transformed and normal tissues.

medicine.medical_specialtyNorpregnenesNormal tissueEstrogen receptorBreast NeoplasmsIn Vitro TechniquesBiologyGestodeneBinding CompetitivePromegestoneInternal medicineProgesterone receptormedicineHumansReceptorBinding affinitiesProgesterone CongenersCancermedicine.diseaseEndocrinologyReceptors EstrogenOncologyMyometriumFemaleReceptors ProgesteroneHuman breasthormones hormone substitutes and hormone antagonistsmedicine.drugEuropean Journal of Cancer and Clinical Oncology
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Histamine-sensitive adenylate cyclase of human gastric mucosa: a model for H2-receptor excitation.

1978

Recent studies revealed that human gastric mucosa contains a histamine-sensitive adenylate cyclase which is almost completely localized within the acid-secreting area of the stomach. In an attempt to further characterize the effector system of histamine’s action, we compared the effects of H 1 — and H 2-receptor agonists upon the adenylate cyclase in human fundic gastric mucosa.

medicine.medical_specialtyNucleotidase activityAdenylate kinaseIn Vitro TechniquesCyclasechemistry.chemical_compoundHistamine H2 receptorInternal medicinemedicineGastric mucosaEthylaminesHumansheterocyclic compoundsPharmacology (medical)Receptors Histamine H2Receptors Histamine H1Pharmacologybusiness.industryEffectorStomachMethylhistaminesdigestive system diseasesStimulation Chemicalmedicine.anatomical_structureEndocrinologychemistryGastric MucosaReceptors HistaminebusinessHistamineResearch ArticleAdenylyl CyclasesHistamine
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Toll-like receptor 5 in obesity: The role of gut microbiota and adipose tissue inflammation

2015

Objective This study aimed at establishing bacterial flagellin-recognizing toll-like receptor 5 (TLR5) as a novel link between gut microbiota composition, adipose tissue inflammation, and obesity. Methods An adipose tissue microarray database was used to compare women having the highest (n = 4, H-TLR) and lowest (n = 4, L-TLR) expression levels of TLR5-signaling pathway genes. Gut microbiota composition was profiled using flow cytometry and FISH. Standard laboratory techniques were used to determine anthropometric and clinical variables. In vivo results were verified using cultured human adipocytes. Results The H-TLR group had higher flagellated Clostridium cluster XIV abundance and Firmicu…

medicine.medical_specialtyNutrition and DieteticsbiologyAdiponectinEndocrinology Diabetes and MetabolismAdipose tissue macrophagesLeptinMedicine (miscellaneous)Adipose tissueInflammationGut florabiology.organism_classification3. Good healthInsulin receptorEndocrinologyEndocrinologyTLR5Internal medicinemedicinebiology.proteinmedicine.symptomObesity
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