Search results for "Rectal Cancer"

showing 10 items of 978 documents

Signaling molecules: the pathogenic role of the IL-6/STAT-3 trans signaling pathway in intestinal inflammation and in colonic cancer.

2008

Although the precise etiology of inflammatory bowel diseases (IBD) still remains unclear, considerable progress has been made in the identification of novel signal transduction pathways that elucidate the immunopathogenesis involved in the perpetuation of the inflammatory process. As both ulcerative colitis and Crohn's disease are associated with an increased risk for developing colorectal cancer (CRC) and precancerous dysplastic epithelial changes, further studies have concentrated on finding a common signaling pathway that could serve as a mechanistic link between inflammation and associated colonic cancer in IBD. This review presents the current data concerning the pathogenic role of the…

STAT3 Transcription FactorCell signalingColorectal cancerClinical BiochemistryAnti-Inflammatory AgentsInflammationAntineoplastic AgentsDiseaseSuppressor of cytokine signallingDrug DiscoverymedicineAnimalsHumansAutocrine signallingPharmacologybusiness.industryInterleukin-6medicine.diseaseInflammatory Bowel DiseasesUlcerative colitisdigestive system diseasesCell Transformation NeoplasticColonic NeoplasmsCancer researchMolecular Medicinemedicine.symptomSignal transductionbusinessSignal TransductionCurrent drug targets
researchProduct

Tumor-Derived Small Extracellular Vesicles Induce Pro-Inflammatory Cytokine Expression and PD-L1 Regulation in M0 Macrophages via IL-6/STAT3 and TLR4…

2021

Tumor-associated macrophages play a key role in promoting tumor progression by exerting an immunosuppressive phenotype associated with the expression of programmed cell death ligand 1 (PD-L1). It is well known that tumor-derived small extracellular vesicles (SEVs) affect the tumor microenvironment, influencing TAM behavior. The present study aimed to examine the effect of SEVs derived from colon cancer and multiple myeloma cells on macrophage functions. Non-polarized macrophages (M0) differentiated from THP-1 cells were co-cultured with SEVs derived from a colorectal cancer (CRC) cell line, SW480, and a multiple myeloma (MM) cell line, MM1.S. The expression of PD-L1, interleukin-6 (IL-6), a…

STAT3 Transcription FactorPD-L1QH301-705.5colorectal cancersmall extracellular vesiclesB7-H1 AntigenArticleCatalysisStat3 Signaling PathwayProinflammatory cytokineM0 macrophageInorganic ChemistryExtracellular VesiclesSettore BIO/13 - Biologia ApplicataCell Line TumorPD-L1Tumor-Associated Macrophagessmall extracellular vesicleHumansMacrophageTLR4Biology (General)Physical and Theoretical ChemistryM0 macrophagesQD1-999Molecular BiologySpectroscopyInflammationTumor microenvironmentbiologyInterleukin-6ChemistryOrganic ChemistryGeneral MedicineComputer Science ApplicationsGene Expression Regulation NeoplasticToll-Like Receptor 4multiple myelomaChemistryCell cultureTumor progressionColonic Neoplasmsbiology.proteinCancer researchTLR4Signal TransductionInternational Journal of Molecular Sciences
researchProduct

HSP110 : role in colorectal cancer development and immunogenicity

2015

Our team studies HSPs, including HSP110. HSPs are chaperones involved in the folding of newly synthesized and denatured proteins. HSPs are overexpressed under stress conditions and are involved in cell survival thanks to their anti-apoptotic and anti-aggregation functions. HSP110 is overexpressed in colorectal cancer and is associated with a poor prognosis. The expression of a mutant HSP110, named HSP110DE9, has been shown in MSI colorectal cancer. This one was shown to act there as a dominant negative, by binding HSP110 and inhibiting its functions. Its expression sensitizes cancer cells to chemotherapy and is associated with a better prognosis for patients.I was first interested in HSP110…

STAT3Cancer colorectal[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyMacrophage polarizationHSP110DE9Colorectal cancerHSP110
researchProduct

FACS-based protocol to assess cytotoxicity and clonogenic potential of colorectal cancer stem cells using a Wnt/β-catenin signaling pathway reporter

2021

Summary Cancer stem cells (CSCs) play a key role in tumor initiation and progression. A real-time tool to evaluate the activation of CSC-specific signaling pathways is crucial for the study of this cancer cell subset. Here, we present a protocol to monitor, in vitro, the activation of Wnt/β-catenin signaling pathway, which is considered a functional biomarker for colorectal CSCs (CR-CSCs). This flow-cytometry-based protocol allows it to isolate CR-CSCs and to evaluate their cytotoxicity upon anti-tumor treatments. For complete details on the use and execution of this protocol, please refer to Di Franco et al. (2021).

Science (General)Colorectal cancerTumor initiationBiologyGeneral Biochemistry Genetics and Molecular BiologyQ1-390Cancer stem cellmedicineProtocolHumansFlow Cytometry/Mass CytometryClonogenic assayWnt Signaling PathwayCancerGeneral Immunology and MicrobiologyGeneral NeuroscienceStem CellsWnt signaling pathwayCancerCell Biologymedicine.diseaseFlow CytometryMolecular/Chemical ProbesCancer cellcolorectal cancers cancer stem cells FACS Wnt.Cell isolationCancer researchNeoplastic Stem CellsCell-based AssaysStem cellSettore MED/46 - Scienze Tecniche Di Medicina Di LaboratorioColorectal NeoplasmsSignal TransductionSTAR Protocols
researchProduct

Working with (too) Few Samples

2018

This paper is concerned with gene set differential expression analysis. We compare the transcriptomic behaviour of each gene set between different experimental conditions.The gene set is previously defined. It has been used a gene set collection downloaded from Gene Ontology.A randomization test is proposed and compared with other previous procedures using a RNA-seq experiment of colorectal cancer (CRC).

Set (abstract data type)TranscriptomeDifferential expression analysisColorectal cancerResamplingmedicineComputational biologyBiologymedicine.diseaseGene
researchProduct

Novel biomarker panel for colon cancer unveiled by proteomics

2016

Settore BIO/06 - Anatomia Comparata E CitologiaColorectal cancer MALDI-TOF-MS/MS proteomics transgelin
researchProduct

Expression of a specific Thymidylate synthase polimorfic allele in metastatic colorectal patients is regulated by Myeloid Zinc Finger 1.

2013

Thymidylate Synthase (TS) is the target enzyme for fluoropyrimidine anticancer drugs. Its expression is regulated by the number of functional upstream stimulatory factor (USF) E box consensus elements present on its 5’ untranslated region. To date are known different polymorphisms, the first one consisting of 2 or 3 repeat of a 28 bp sequence, a further single nucleotide polymorphism (SNP) consisting in a G>C substitution within the second repeat of 3R (3RG>3RC) and recently it has been identified an additional SNP a G>C substitution at the 12th nucleotide in the first repeat of the 2R allele (2RG>2RC). These polymorphisms can influence TS expression, in particular 3R/3R genotype and the pr…

Settore BIO/14 - FarmacologiaThymidylate synthase colorectal cancer polymorphism
researchProduct

Analysis of UGT1A1*28 and DPYD*2A polymorphisms in Sicilians patients with metastatic colorectal cancer treated with Irinotecan and 5-fluorouracil.

2013

Settore BIO/14 - Farmacologiapolymorphisms metastatic colorectal cancer
researchProduct

DISMICROBISM IN INFLAMMATORY BOWEL DISESE AND COLORECTAL CANCER: CHANGES IN RESPONSE OF COLOCYTES

2014

Patients with inflammatory bowel disease (IBD) have an increased risk of 10%-15% developing colorectal cancer (CRC) that is a common disease of high economic costs in developed countries. the CRC has been increasing in recent years and its mortality rates are very high. Multiple biological and biochemical factors are responsible for the onset nad progression of this pathology. moreover, it appears absolutely necessary to investigate the environmental factors favoring the onset of CRC and the production of colonic healt. the gut microflora, or microbiota, has an extensive diversity both quantitatively and qualitatively. in utero, the intestine of the mammalian fetus is sterile. Al birth, the…

Settore BIO/17 - IstologiaSettore MED/18 - Chirurgia GeneraleSettore MED/09 - Medicina InternaSettore MED/06 - Oncologia MedicaCOLORECTAL CANCER INTESTINAL BOWEL DISEASE DISMICROBISM
researchProduct

Expression of angiogenic regulators. VEGF and leptin, is regulated by the EGF/P13K/STAT3 pathway in colorectal cancer cells

2009

Settore BIO/17 - IstologiaVEGF STAT3 EGF Colorectal cancer cell
researchProduct