Search results for "Regulatory"

showing 10 items of 740 documents

The role of recent thymic emigrant-regulatory T-cell (RTE-Treg) differentiation during pregnancy.

2014

During pregnancy, regulatory T cells (Tregs) have a key role in maternal immune tolerance to the semi-allogeneic fetus. Our previous results showed that the naive CD45RA(+)-Treg pool is functionally improved in pregnant women compared with non-pregnant women. Therefore, we examined the thymic output and differentiation of CD45RA(+)CD31(+) recent thymic emigrant (RTE)-Tregs during normal pregnancy and in the presence of preeclampsia. With the onset of pregnancy, the composition of the total CD4(+)CD127(low+/-)FoxP3(+)-Treg pool changed in the way that its percentage of RTE- and CD45RA(-)CD31(+)-memory Tregs decreased strongly, whereas that of the CD45RA(+)CD31(-)-mature naive (MN)-Tregs did …

Adultmedicine.medical_specialtyAdolescentRegulatory T cellImmunologyRecent Thymic Emigrantchemical and pharmacologic phenomenaThymus GlandT-Lymphocytes RegulatoryPreeclampsiaImmune toleranceYoung AdultPre-EclampsiaPregnancyT-Lymphocyte SubsetsInternal medicineImmune ToleranceImmunology and AllergyMedicineAnimalsHumansInterleukin-7 receptorFetusPregnancybusiness.industryFOXP3hemic and immune systemsCell DifferentiationForkhead Transcription FactorsCell BiologyMiddle Agedmedicine.diseasemedicine.anatomical_structureEndocrinologyLeukocyte Common AntigensFemalebusinessImmunologic MemoryImmunology and cell biology
researchProduct

The role of regulatory T cell (Treg) subsets in gestational diabetes mellitus.

2014

Physiological changes during normal pregnancy are characterized by an inflammatory immune response and insulin resistance. Therefore, we hypothesize that gestational diabetes mellitus (GDM) may be caused by an inappropriate adaption of the maternal immune system to pregnancy. In this study we examined the role of regulatory T cell (Treg) differentiation for the development of GDM during pregnancy. We used six-colour flow cytometric analysis to demonstrate that the total CD4(+) CD127(low+/-) CD25(+) forkhead box protein 3 (FoxP3(+)) T(reg) pool consists of four different T(reg) subsets: naive CD45RA(+) T(regs), HLA-DR(-) CD45RA(-) memory T(regs) (DR(-) T(regs)) and the highly differentiated …

Adultmedicine.medical_specialtyendocrine system diseasesRegulatory T cellImmunologychemical and pharmacologic phenomenaCell SeparationLymphocyte ActivationT-Lymphocytes RegulatoryImmunophenotypingYoung AdultImmune systemInsulin resistanceimmune system diseasesPregnancyT-Lymphocyte Subsetshemic and lymphatic diseasesInternal medicinemedicineImmune ToleranceImmunology and AllergyHomeostasisHumansIL-2 receptorInterleukin-7 receptorbusiness.industryFOXP3hemic and immune systemsCell DifferentiationForkhead Transcription FactorsT helper cellOriginal Articlesmedicine.diseaseFlow CytometryGestational diabetesDiabetes GestationalEndocrinologymedicine.anatomical_structureImmunologyCD4 AntigensFemalebusinessImmunologic MemoryClinical and experimental immunology
researchProduct

C4-dicarboxylate carriers and sensors in bacteria

2002

AbstractBacteria contain secondary carriers for the uptake, exchange or efflux of C4-dicarboxylates. In aerobic bacteria, dicarboxylate transport (Dct)A carriers catalyze uptake of C4-dicarboxylates in a H+- or Na+-C4-dicarboxylate symport. Carriers of the dicarboxylate uptake (Dcu)AB family are used for electroneutral fumarate:succinate antiport which is required in anaerobic fumarate respiration. The DcuC carriers apparently function in succinate efflux during fermentation. The tripartite ATP-independent periplasmic (TRAP) transporter carriers are secondary uptake carriers requiring a periplasmic solute binding protein. For heterologous exchange of C4-dicarboxylates with other carboxylic …

Aerobic bacteriaAntiporterSuccinic AcidBiophysicsOrganic Anion TransportersReceptors Cell Surfacemedicine.disease_causeBiochemistryFumarate (succinate) sensorTwo-component systemBacterial ProteinsFumaratesEscherichia colimedicineAmino Acid SequenceEscherichia coliDicarboxylate uptake SHistidine protein kinasePhylogenyHistidineDicarboxylic Acid TransportersDicarboxylate transport BbiologyEscherichia coli ProteinsBiological TransportPeriplasmic spaceCell Biologybiology.organism_classificationTwo-component regulatory systemBacteria AerobicModels ChemicalBiochemistryAntiportFumarate/succinate transportEffluxDicarboxylate uptake carrierProtein KinasesDicarboxylate transport A carrierBacteriaSignal TransductionBiochimica et Biophysica Acta (BBA) - Bioenergetics
researchProduct

Negative Regulation of β Enolase Gene Transcription in Embryonic Muscle Is Dependent upon a Zinc Finger Factor That Binds to the G-rich Box within th…

1998

We have previously identified a muscle-specific enhancer within the first intron of the human beta enolase gene. Present in this enhancer are an A/T-rich box that binds MEF-2 protein(s) and a G-rich box (AGTGGGGGAGGGGGCTGCG) that interacts with ubiquitously expressed factors. Both elements are required for tissue-specific expression of the gene in skeletal muscle cells. Here, we report the identification and characterization of a Kruppel-like zinc finger protein, termed beta enolase repressor factor 1, that binds in a sequence-specific manner to the G-rich box and functions as a repressor of the beta enolase gene transcription in transient transfection assays. Using fusion polypeptides of b…

AgingTranscription GeneticMolecular Sequence DataDown-RegulationRepressorRegulatory Sequences Nucleic AcidBiologyBiochemistryDNA-binding proteinGene Expression Regulation EnzymologicMiceGene expressionAnimalsHumansAmino Acid SequenceCloning MolecularMuscle SkeletalEnhancerMolecular BiologyCell NucleusRegulation of gene expressionZinc fingerSp1 transcription factorBinding SitesSequence Homology Amino AcidZinc FingersCell BiologyMolecular biologyDNA-Binding ProteinsEnhancer Elements GeneticRegulatory sequencePhosphopyruvate HydrataseJournal of Biological Chemistry
researchProduct

Interruption of CD28-mediated costimulation during allergen challenge protects mice from allergic airway disease

2012

Background Allergic asthma is a T H 2-promoted hyperreactivity with an immediate, IgE, and mast cell–dependent response followed by eosinophil-dominated inflammation and airway obstruction. Objective Because costimulation by CD28 is essential for T H 2 but not T H 1 responses, we investigated the effect of selective interference with this pathway in mice using the models of ovalbumin and house dust mite–induced airway inflammation. Methods To study the role of CD28 in the effector phase of allergic airway inflammation, we developed an inducibly CD28-deleting mouse strain or alternatively used a CD28 ligand-binding site–specific mouse anti-mouse mAb blocking CD28 engagement. Results We show …

Allergic asthmaLymphocyte ActivationImmunoglobulin Emedicine.disease_causeT-Lymphocytes RegulatoryMice0302 clinical medicineAirway resistanceAllergenImmunology and AllergySensitizationMice Inbred BALB C0303 health sciencesbiologyAntibodies Monoclonalovalbuminrespiratory system3. Good healthmedicine.anatomical_structurecostimulationconditional CD28 knockout miceFemalemedicine.symptomImmunologyInflammation03 medical and health sciencesTh2 CellsCD28 AntigensRespiratory HypersensitivitymedicineAnimalsHumansAntigens DermatophagoidesLymphocyte CountAntibodies Blocking030304 developmental biologyHouse dust miteCD28-specific mAbbusiness.industryReceptor Cross-TalkAirway obstructionmedicine.diseasebiology.organism_classificationMice Mutant Strainsrespiratory tract diseasesDisease Models AnimalCTLA-4Immunologybiology.proteinbusiness030215 immunology
researchProduct

Regulation of IgE production and airway reactivity by CD4(-)CD8(-) regulatory T cells

2015

The mechanisms of tolerance induction occurring in the course of allergen-specific immunotherapy have not been elucidated in full detail. Our study aimed to characterize high zone tolerance in mouse models of type I allergy and of allergic airway inflammation induced by subcutaneous sensitization of mice with high doses of the model allergen ovalbumin (OVA) without the use of adjuvant. Mice were immunized by subcutaneous injection of high doses (HD) of OVA or, for comparison, low doses (LD) of OVA in saline. HD-mice showed lower specific IgE, but augmented IgG in sera than LD-mice. Pre-treatment of mice with HD-OVA antigen-specifically inhibited IgE production subsequently induced by LD-OVA…

AllergyAdoptive cell transferAllergyOvalbuminImmunologyGene ExpressionCD4-CD8-double-negative T cellsLymphocyte ActivationImmunoglobulin EAirway hyperreactivityT-Lymphocytes RegulatoryImmunophenotypingMouse modelImmunomodulationMiceSubcutaneous injectionAntibody SpecificityT-Lymphocyte SubsetsRespiratory HypersensitivitymedicineAnimalsImmunology and AllergyAntigen doseSensitizationbiologymedicine.diagnostic_testbusiness.industryReceptors Antigen T-Cell gamma-deltaHematologyImmunoglobulin Erespiratory systemmedicine.diseaseAdoptive TransferTolerance inductionOvalbuminImmunoglobulin (Ig)EBronchoalveolar lavagemedicine.anatomical_structureAntibody FormationImmunologybiology.proteinCytokinesFemaleImmunizationbusinessBronchoalveolar Lavage Fluid
researchProduct

Understanding the complexity of IgE-related phenotypes from childhood to young adulthood: A Mechanisms of the Development of Allergy (MeDALL) seminar.

2012

Mechanisms of the Development of Allergy (MeDALL), a Seventh Framework Program European Union project, aims to generate novel knowledge on the mechanisms of initiation of allergy. Precise phenotypes of IgE-mediated allergic diseases will be defined in MeDALL. As part of MeDALL, a scientific seminar was held on January 24, 2011, to review current knowledge on the IgE-related phenotypes and to explore how a multidisciplinary effort could result in a new integrative translational approach. This article provides a summary of the meeting. It develops challenges in IgE-related phenotypes and new clinical and epidemiologic approaches to the investigation of allergic phenotypes, including cluster a…

AllergyAllergyWORLD-HEALTH-ORGANIZATIONBioinformaticsEpigenesis Genetic0302 clinical medicineRisk FactorsImmunology and AllergyMedicineYoung adultChildEpigenesismedia_commonMechanisms of the Development of Allergy0303 health scienceseducation.field_of_studyphenotypesAllergy; Mechanisms of the Development of Allergy; Seventh Framework Program; phenotypes; IgE; asthmaRUSSIAN KARELIAPhenotype3. Good healthLUNG-FUNCTIONPhenotypeChild PreschoolBRONCHIAL HYPERRESPONSIVENESSIgEBIRTH-COHORTAdolescentASTHMA RESEARCH-PROGRAMSystems biologyImmunologyPopulationOBSTRUCTIVE PULMONARY-DISEASEYoung Adult03 medical and health sciencesDIAGNOSTIC GATEKEEPERSHypersensitivityAnimalsHumansmedia_common.cataloged_instanceEuropean unioneducation030304 developmental biologyCLUSTER-ANALYSISbusiness.industryMechanism (biology)ResearchImmunoglobulin Easthmamedicine.disease030228 respiratory systemSeventh Framework ProgramImmunologybusinessT-REGULATORY-CELLS
researchProduct

Technical advance: Soluble OX40 molecule mimics regulatory T cell modulatory activity on FCεRI-dependent mast cell degranulation

2011

ABSTRACT Tregs play a central role in modulating FcɛRI-dependent MC effector functions in the course of the allergic response. Cellular interaction depends on the constitutive expression of OX40 on Tregs and the OX40L counterpart on MCs. Study of OX40L signaling on MCs is hampered by the need of a highly purified molecule, which triggers OX40L specifically. We now report that sOX40 mimics the physiological activity of Treg interaction by binding to activated MCs. When treated with sOX40, activated MCs showed decreased degranulation and Ca++ influx, whereas PLC-γ2 phosphorylation remained unaffected. Once injected into experimental animals, sOX40 not only located within the endothelium but a…

AllergyCell DegranulationRegulatory T cellImmunologyOX40 LigandAllergy; Cell activation; CostimulationBiologymedicine.disease_causeT-Lymphocytes RegulatoryCell DegranulationMiceHypersensitivitymedicineAnimalsImmunology and AllergyMast CellsPhosphorylationReceptorCell activationMice KnockoutMembrane GlycoproteinsPhospholipase C gammaReceptors IgEDegranulationCell BiologyReceptors OX40humanitiesCell biologymedicine.anatomical_structureCostimulationTechnical AdvanceSolubilityTumor Necrosis FactorsAllergic responsePhosphorylationSignal transductionCell activation
researchProduct

Effective treatment of allergic airway inflammation with Helicobacter pylori immunomodulators requires BATF3-dependent dendritic cells and IL-10

2014

The prevalence of allergic asthma and other atopic diseases has reached epidemic proportions in large parts of the developed world. The gradual loss of the human indigenous microbiota has been held responsible for this trend. The bacterial pathogen Helicobacter pylori is a constituent of the normal gastric microbiota whose presence has been inversely linked to allergy and asthma in humans and experimental models. Here we show that oral or i.p. tolerization with H. pylori extract prevents the airway hyperresponsiveness, bronchoalveolar eosinophilia, pulmonary inflammation, and Th2 cytokine production that are hallmarks of allergen-induced asthma in mice. Asthma protection is not conferred by…

Allergybacterial persistence determinantsT-Lymphocytes Regulatorybacterial immunomodulationImmune toleranceMiceBacterial ProteinsImmune TolerancemedicineAnimalsHumansImmunologic FactorsEosinophiliaAsthmaMice KnockoutAntigens Bacterial1000 MultidisciplinaryMultidisciplinaryHelicobacter pyloribiologyallergy and asthma prevention10061 Institute of Molecular Cancer ResearchInterleukin-18tolerogenic dendritic cellsDendritic Cellsgamma-GlutamyltransferaseDendritic cellBiological SciencesAllergensHelicobacter pyloribacterial infections and mycosesmedicine.diseasebiology.organism_classificationAsthmaInterleukin-10respiratory tract diseases3. Good healthMice Inbred C57BLRepressor ProteinsDisease Models AnimalInterleukin 10Basic-Leucine Zipper Transcription FactorsImmunology570 Life sciences; biologyInterleukin 18medicine.symptom
researchProduct

Chemotherapy and immunomodulation: from immunogenic chemotherapies to novel therapeutic strategies.

2013

Anticancer immunityCancer ResearchOrganoplatinum Compoundsmedicine.medical_treatmentBreast NeoplasmsT-Lymphocytes RegulatoryImmunomodulationChemoimmunotherapyNeoplasmsAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansAnthracyclinesCyclophosphamideChemotherapyCell Deathbusiness.industryImmunosuppressionInflammasomeGeneral MedicineOxaliplatinOncologyImmunologyMyeloid-derived Suppressor CellFemaleFluorouracilbusinessImmunosuppressive Agentsmedicine.drugFuture oncology (London, England)
researchProduct