Search results for "Regulatory"

showing 10 items of 740 documents

In silico characterization of the neural alpha tubulin gene promoter of the sea urchin embryo Paracentrotus lividus by phylogenetic footprinting

2011

During Paracentrotus lividus sea urchin embryo development one alpha and one beta tubulin genes are expressed specifically in the neural cells and they are early end output of the gene regulatory network that specifies the neural commitment. In this paper we have used a comparative genomics approach to identify con- served regulatory elements in the P. lividus neural alpha tubulin gene. To this purpose, we have first isolated a genomic clone containing the entire gene plus 4.5 Kb of 5 0 upstream sequences. Then, we have shown by gene transfer experiments that its non-coding region drives the spatio- temporal gene expression corresponding substantially to that of the endogenous gene. In addi…

Embryo NonmammalianMicroinjectionsSequence analysisGreen Fluorescent ProteinsDNA FootprintingNerve Tissue ProteinsSettore BIO/11 - Biologia MolecolarePhylogenetic footprintingParacentrotus lividusGenes ReporterTubulinGeneticsAnimalsPromoter Regions GeneticMolecular BiologyGeneDNA PrimersExpressed Sequence TagsComparative genomicsGeneticsBinding SitesbiologyGene Transfer TechniquesComputational BiologyMolecular Sequence AnnotationPromoterGenomicsGeneral MedicineSea urchin Neural development Gene expression Phylogenetic footprint Cis-regulatory analysisbiology.organism_classificationGene Expression RegulationRegulatory sequenceParacentrotusOrthologous GeneMolecular Biology Reports
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Early asymmetric cues triggering the dorsal/ventral gene regulatory network of the sea urchin embryo

2014

Dorsal/ventral (DV) patterning of the sea urchin embryo relies on a ventrally-localized organizer expressing Nodal, a pivotal regulator of the DV gene regulatory network. However, the inceptive mechanisms imposing the symmetry-breaking are incompletely understood. In Paracentrotus lividus, the Hbox12 homeodomain-containing repressor is expressed by prospective dorsal cells, spatially facing and preceding the onset of nodal transcription. We report that Hbox12 misexpression provokes DV abnormalities, attenuating nodal and nodal-dependent transcription. Reciprocally, impairing hbox12 function disrupts DV polarity by allowing ectopic expression of nodal. Clonal loss-of-function, inflicted by b…

Embryo NonmammalianTranscription GeneticEctodermp38 Mitogen-Activated Protein Kinasessymmetry breakingdorsal ventral axis sea urchin embryo nodal homeodomain repressor p38 MAPKAnimals Genetically ModifiedCell polarityMorphogenesisGene Regulatory NetworksBiology (General)ZebrafishSea urchinsea urchin embryoGeneticsbiologyGeneral NeuroscienceQRdorsal/ventral polarityCell PolarityGene Expression Regulation DevelopmentalEmbryoGeneral MedicineCell biologymedicine.anatomical_structureGene Knockdown Techniquesembryonic structuresParacentrotusMedicineCuesResearch Articleanimal structuresQH301-705.5Nodal ProteinScienceEmbryonic DevelopmentSettore BIO/11 - Biologia Molecolarep38 MAPKModels BiologicalGeneral Biochemistry Genetics and Molecular Biologybiology.animalEctodermmedicineAnimalsBody PatterningHomeodomain ProteinsGeneral Immunology and MicrobiologyotherCell Biologybiology.organism_classificationEmbryonic stem cellhomeodomain repressorRepressor ProteinsDevelopmental Biology and Stem CellsnodalNODALDevelopmental biologyeLife
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Defining the genomic signature of totipotency and pluripotency during early human development.

2013

The genetic mechanisms governing human pre-implantation embryo development and the in vitro counterparts, human embryonic stem cells (hESCs), still remain incomplete. Previous global genome studies demonstrated that totipotent blastomeres from day-3 human embryos and pluripotent inner cell masses (ICMs) from blastocysts, display unique and differing transcriptomes. Nevertheless, comparative gene expression analysis has revealed that no significant differences exist between hESCs derived from blastomeres versus those obtained from ICMs, suggesting that pluripotent hESCs involve a new developmental progression. To understand early human stages evolution, we developed an undifferentiation netw…

EmbryologyBlastomeresMicroarraysCellular differentiationGene ExpressionCell Fate DeterminationMolecular Cell BiologyGene Regulatory NetworksInduced pluripotent stem cellreproductive and urinary physiologyGeneticsMultidisciplinarySystems BiologyStem CellsQTotipotentRGenomic signatureCell DifferentiationGenomicsCell biologyFunctional GenomicsBlastocyst Inner Cell MassBlastocyst Inner Cell Massembryonic structuresMedicineResearch ArticlePluripotent Stem CellsSystems biologyCell PotencyScienceEmbryonic DevelopmentBiologyMolecular GeneticsGeneticsHumansGene NetworksBiologyEmbryonic Stem CellsGenome HumanGene Expression ProfilingBio-OntologiesComputational BiologyMolecular Sequence AnnotationComparative GenomicsMolecular DevelopmentEmbryonic stem cellSignalingSignaling NetworksGene expression profilingGenome Expression AnalysisTotipotent Stem CellsDevelopmental BiologyPLoS ONE
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Regulation Support to Industrial Clusters: A Cross-Country Analysis

2016

We empirically investigate the role of regulatory governance and industrial clusters to foster the country’s economic performance. Using the ordinary least-square (OLS) regression for a cross section of countries, including the 25 EU Member States, our findings show that the regulation governance has the strongest effects on the employment rate performing a buffering effect with the cluster development. An antagonist effect occurs for the other economic indicators.

EmploymentRegulatory GovernanceClusterSettore SECS-P/03 - Scienza Delle FinanzeIncomeClusters; Employment; Income; Labor Productivity; Regulatory GovernanceLabor ProductivitySettore SECS-P/06 - Economia Applicata
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A noninflammatory mRNA vaccine for treatment of experimental autoimmune encephalomyelitis.

2019

Precision therapy for immune tolerance Autoimmune diseases, such as multiple sclerosis (MS), result from a breach of immunological self-tolerance and tissue damage by autoreactive T lymphocytes. Current treatments can cause systemic immune suppression and side effects such as increased risk of infections. Krienke et al. designed a messenger RNA vaccine strategy that lacks adjuvant activity and delivers MS autoantigens into lymphoid dendritic cells. This approach expands a distinct type of antigen-specific effector regulatory T cell that suppresses autoreactivity against targeted autoantigens and promotes bystander suppression of autoreactive T cells against other myelin-specific autoantigen…

Encephalomyelitis Autoimmune ExperimentalMultiple SclerosisRegulatory T cellEncephalomyelitisAntigen presentationAntigen-Presenting CellsAutoantigensT-Lymphocytes RegulatoryMiceImmune systemAntigenmedicineAnimalsRNA MessengerAntigen-presenting cellImmunosuppression TherapyInflammationVaccines SyntheticMultidisciplinarybusiness.industryEffectorExperimental autoimmune encephalomyelitisBystander Effectmedicine.diseaseMice Inbred C57BLmedicine.anatomical_structureImmunologybusinessPseudouridineScience (New York, N.Y.)
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Smad7 in T cells drives T helper 1 responses in multiple sclerosis and experimental autoimmune encephalomyelitis

2010

Autoreactive CD4+ T lymphocytes play a vital role in the pathogenesis of multiple sclerosis and its animal model, experimental autoimmune encephalomyelitis. Since the discovery of T helper 17 cells, there is an ongoing debate whether T helper 1, T helper 17 or both subtypes of T lymphocytes are important for the initiation of autoimmune neuroinflammation. We examined peripheral blood CD4+ cells from patients with active and stable relapsing-remitting multiple sclerosis, and used mice with conditional deletion or over-expression of the transforming growth factor-beta inhibitor Smad7, to delineate the role of Smad7 in T cell differentiation and autoimmune neuroinflammation. We found that Smad…

Encephalomyelitis Autoimmune ExperimentalMultiple SclerosisT helper 1Regulatory T cellT cellMolecular Sequence DataMice TransgenicBiologySmad7 ProteinMiceInterleukin 21medicineAnimalsHumansCytotoxic T cellAmino Acid SequenceIL-2 receptorAntigen-presenting cellMice Knockoutintegumentary systemEAEimmune regulationCD28Original ArticlesTh1 CellsNatural killer T cellMice Inbred C57BLmedicine.anatomical_structureT cell responsesImmunologyNeurology (clinical)Brain
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Microglial activation milieu controls regulatory T cell responses.

2013

Abstract Although mechanisms leading to brain-specific inflammation and T cell activation have been widely investigated, regulatory mechanisms of local innate immune cells in the brain are only poorly understood. In this study, to our knowledge we show for the first time that MHC class II+CD40dimCD86dimIL-10+ microglia are potent inducers of Ag-specific CD4+Foxp3+ regulatory T cells (Tregs) in vitro. Microglia differentially regulated MHC class II expression, costimulatory molecules, and IL-10 depending on the amount of IFN-γ challenge and Ag dose, promoting either effector T cell or Treg induction. Microglia-induced Tregs were functionally active in vitro by inhibiting Ag-specific prolifer…

Encephalomyelitis Autoimmune ExperimentalRegulatory T cellT cellImmunologychemical and pharmacologic phenomenaMice TransgenicLymphocyte ActivationT-Lymphocytes RegulatoryImmune toleranceInterferon-gammaMiceImmune systemT-Lymphocyte SubsetsmedicineImmune ToleranceImmunology and AllergyAnimalsCells CulturedCD86MHC class IIbiologyMicrogliaHistocompatibility Antigens Class IIFOXP3Brainhemic and immune systemsForkhead Transcription FactorsCoculture TechniquesCell biologyInterleukin-10Mice Inbred C57BLmedicine.anatomical_structureCellular Microenvironmentbiology.proteinMicrogliaJournal of immunology (Baltimore, Md. : 1950)
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Dendritic Cells Ameliorate Autoimmunity in the CNS by Controlling the Homeostasis of PD-1 Receptor+ Regulatory T Cells

2012

SummaryMature dendritic cells (DCs) are established as unrivaled antigen-presenting cells (APCs) in the initiation of immune responses, whereas steady-state DCs induce peripheral T cell tolerance. Using various genetic approaches, we depleted CD11c+ DCs in mice and induced autoimmune CNS inflammation. Unexpectedly, mice lacking DCs developed aggravated disease compared to control mice. Furthermore, when we engineered DCs to present a CNS-associated autoantigen in an induced manner, we found robust tolerance that prevented disease, which coincided with an upregulation of the PD-1 receptor on antigen-specific T cells. Additionally, we showed that PD-1 was necessary for DC-mediated induction o…

Encephalomyelitis Autoimmune ExperimentalT cellProgrammed Cell Death 1 ReceptorImmunologyAntigen presentationCD11cAutoimmunity610 Medicine & healthchemical and pharmacologic phenomenaBiologymedicine.disease_causeAutoantigensT-Lymphocytes RegulatoryB7-H1 AntigenAutoimmunityImmune toleranceMiceImmune systemDownregulation and upregulationImmune TolerancemedicineAnimalsImmunology and AllergyReceptorMice KnockoutAntigen Presentation2403 Immunologyhemic and immune systemsDendritic Cells2725 Infectious DiseasesTh1 CellsCD11c AntigenMice Inbred C57BLInfectious Diseasesmedicine.anatomical_structure10032 Clinic for Oncology and HematologyImmunology2723 Immunology and AllergyTh17 CellsImmunity
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Regulatory T cells--the renaissance of the suppressor T cells.

2007

Immune reactions are stringently regulated and balanced by complex interactions of stimulating and suppressing mechanisms. Dysfunctions of this sophisticated immune regulatory network can lead to a variety of diseases such as autoimmunity, allergy, cancer, and pregnancy disorders. The rediscovery of suppressor T cells a decade ago--now designated as T regulatory cells--set off a huge avalanche of research activities leading to a multitude of preclinical and clinical studies. Herein, we give a comprehensive review about this research on T regulatory cells and the relevance of this suppressive T cell population for the development of innovative immune therapeutic strategies.

Encephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentT cellPopulationAutoimmunitymedicine.disease_causeInfectionsT-Lymphocytes RegulatoryAutoimmunitylaw.inventionMiceImmune systemlawPregnancyT-Lymphocyte SubsetsTransplantation ImmunologyNeoplasmsmedicineSuppressor Factors ImmunologicAnimalsHumanseducationeducation.field_of_studybusiness.industryModels ImmunologicalGeneral MedicineT lymphocyteImmunotherapyInflammatory Bowel DiseasesTransplantationDisease Models Animalmedicine.anatomical_structureDiabetes Mellitus Type 1ImmunologySuppressorFemaleImmunotherapybusinessAnnals of medicine
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IL-6 controls Th17 immunity in vivo by inhibiting the conversion of conventional T cells into Foxp3+regulatory T cells

2008

The conditions leading to the induction of adaptive Foxp3+regulatory T cells (T-regs) from peripheral T cells in vivo are incompletely understood. Here, we show that unresponsiveness of T cells to IL-6 by T cell-selective deletion of gp130 or immunization of wild-type mice with antigen in incomplete Freund's adjuvant (IFA), which fails to induce IL-6, promotes the conversion of peripheral CD4+T cells into adaptive Foxp3+T-regs. Thus, both T cell-conditional gp130 knockout (KO) mice immunized with MOG35-55 in complete Freund's adjuvant (CFA) and wild-type mice immunized with MOG35-55 in IFA develop overwhelming antigen-specific T-reg responses and are protected from experimental autoimmune e…

Encephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentchemical and pharmacologic phenomenaInflammationBiologyT-Lymphocytes RegulatoryMiceInterleukin 21Antigenimmune system diseasesmedicineAnimalsCytotoxic T cellIL-2 receptorMice KnockoutMultidisciplinaryInterleukin-6Experimental autoimmune encephalomyelitisFOXP3Forkhead Transcription Factorshemic and immune systemsT-Lymphocytes Helper-InducerBiological Sciencesmedicine.diseaseCytokineImmunologymedicine.symptomProceedings of the National Academy of Sciences
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