Search results for "Restriction"

showing 10 items of 527 documents

Role of the Angiotensin Type 2 Receptor Gene in Congenital Anomalies of the Kidney and Urinary Tract, CAKUT, of Mice and Men

1999

Angiotensin type 2 receptor gene null mutant mice display congenital anomalies of the kidney and urinary tract (CAKUT). Various features of mouse CAKUT impressively mimic human CAKUT. Studies of the human type 2 receptor (AGTR2) gene in two independent cohorts found that a significant association exists between CAKUT and a nucleotide transition within the lariat branchpoint motif of intron 1, which perturbs AGTR2 mRNA splicing efficiency. AGTR2, therefore, has a significant ontogenic role for the kidney and urinary tract system. Studies revealed that the establishment of CAKUT is preceded by delayed apoptosis of undifferentiated mesenchymal cells surrounding the urinary tract during key ont…

MaleUrologic Diseasesmedicine.medical_specialtyRNA SplicingUrinary systemApoptosisIn situ hybridizationBiologyKidneyMesodermMiceUreterInternal medicinemedicineAnimalsHumansRNA MessengerUrinary TractReceptorMolecular BiologyGeneIn Situ HybridizationMice KnockoutKidneyReceptors AngiotensinIntronSequence Analysis DNACell BiologyPhenotypePedigreePhenotypemedicine.anatomical_structureEndocrinologyMutationKidney DiseasesPolymorphism Restriction Fragment LengthMolecular Cell
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Association Between Single Nucleotide Polymorphisms in the Cyclooxygenase-2, Tumor Necrosis Factor-α, and Vascular Endothelial Growth Factor-A Genes,…

2011

Cyclooxygenase-2 (COX-2), vascular endothelial growth factor-A (VEGF-A), and tumor necrosis factor-α (TNF-α) are mediators of inflammation and angiogenesis; all of them are produced in liver cirrhosis (LC) and in hepatocellular carcinoma (HCC). It was proposed that there is an association between single nucleotide polymorphisms (SNPs) and HCC. These allelic variants influence the transcriptional activity of these genes, and therefore the proteins levels. The VEGF-A pathway is a potential therapeutic target in HCC, and several antiangiogenic agents have entered clinical trials in HCC. We evaluated the frequency of SNPs of COX-2, TNF-α, and VEGF-A genes in patients with HCC versus LC patients…

MaleVascular Endothelial Growth Factor AHeterozygoteCarcinoma HepatocellularCirrhosisAngiogenesisSingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideBiochemistryCOX-2 TNFa VEGF Hepatocellular Carcinoma SNPsGeneticsmedicineHumansneoplasmsMolecular BiologyTumor Necrosis Factor-alphaHomozygoteLiver Neoplasmsnucleotide polymorphisms cyclooxygenase-2 tumor necrosis factor-α vascular endothelial growth factor-A geneshepatocellular carcinoma.Heterozygote advantagemedicine.diseasedigestive system diseasesVascular endothelial growth factor ACyclooxygenase 2Hepatocellular carcinomaImmunologyCancer researchMolecular MedicineFemaleTumor necrosis factor alphaRestriction fragment length polymorphismBiotechnologyOMICS: A Journal of Integrative Biology
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High prevalence of Blastocystis sp. in pigs reared under intensive growing systems: Frequency of ribotypes and associated risk factors

2007

Three hundred and ninety-five pig fecal samples were analyzed looking for Blastocystis sp. using optical microscopy and PCR. A global prevalence of 46.8% has been observed in this study, although relative values of prevalence differ notably according to the strata examined, ranging from 9.3% in sows to 75% in weaners. Statistic analysis of the data included several risk factors such as different management systems, date of sample collection, fecal consistency, age and sex of the animals. The presence of the parasite was statistically associated to the variables "age" and "date of sample collection", being more prevalent in weaners and grower pigs and warm season. Random fragment-length poly…

MaleVeterinary medicineSwineBlastocystis InfectionsPolymerase Chain ReactionRibotypinglaw.inventionFecesRibotypingRisk FactorslawZoonosesGenotypePrevalencemedicineAnimalsAnimal HusbandryParasite Egg CountPolymerase chain reactionFecesSwine DiseasesBlastocystisGenetic diversityGeneral VeterinarybiologyZoonosisAge FactorsGene AmplificationGeneral MedicineDNA Protozoanmedicine.diseasebiology.organism_classificationBlastocystisFemaleParasitologySeasonsSample collectionPolymorphism Restriction Fragment LengthVeterinary Parasitology
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Deletion of the Hunter gene and both DXS466 and DXS304 in a patient with mucopolysaccharidosis type II.

1992

Hunter syndrome is an X-linked mucopoly-saccharidosis due to deficiency of the lysosomal enzyme iduronate-2-sulfatase (IDS). A cDNA clone containing the entire coding region of the human IDS gene, mapped in Xq28, has been used as molecular probe to study a patient with Hunter syndrome. A submicroscopic deletion has been detected that spans the IDS gene as well as DXS466 and DXS304, 2 loci mapped probably not more than 900 kb from the IDS locus. A detailed clinical description of the patient is provided and his phenotype is compared to that of other patients with IDS deletion described recently. By following the segregation of a restriction fragment length polymorphism at the IDS locus in th…

MaleX ChromosomeLocus (genetics)Iduronate SulfataseBiologyGene mappingmedicineHumansMucopolysaccharidosis type IIChildGenetics (clinical)X chromosomeMucopolysaccharidosis IIGeneticsIduronate-2-sulfataseChromosome MappingHunter syndromeDNAmedicine.diseaseXq28PedigreeBlotting SouthernFemaleRestriction fragment length polymorphismChromosome DeletionPolymorphism Restriction Fragment LengthAmerican journal of medical genetics
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Gene diagnosis and carrier detection in Hunter syndrome by the iduronate-2-sulphatase cDNA probe.

1992

Hunter disease (McKusick 309900) is an X-chromosomal mucopolysaccharidosis due to deficiency of the lysosomal enzyme iduronate-2-sulphatase (IDS; EC 3.1.6.13). Diagnosis is based on both the typical clinical features of patients and the lack/reduction of IDS activity. Female carriers show no symptoms of the disease. In the past, several different assays were elaborated for measuring enzyme activity in carriers but none of them proved to be suitable for detecting heterozygotes reliably (Zlotogora and Bach 1984)

MaleX ChromosomeMucopolysaccharidosisIduronate SulfataseBiologyGene mappingComplementary DNAGenotypeGeneticsmedicineHumansAlleleChildDeoxyribonucleases Type II Site-SpecificGenetics (clinical)Mucopolysaccharidosis IIGeneticsGenetic Carrier ScreeningHunter syndromeHeterozygote advantagemedicine.diseaseMolecular biologyEnzyme assayPedigreeBlotting Southernbiology.proteinDNA ProbesPolymorphism Restriction Fragment LengthJournal of inherited metabolic disease
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Molecular analysis in patients with mucopolysaccharidosis type II suggests that DXS466 maps within the Hunter gene

1993

Hunter disease is an X-linked mucopolysaccharidosis caused by deficiency of the lysosomal enzyme iduronate-2-sulfatase (IDS). Using the IDS cDNA and DNA probes corresponding to loci flanking the IDS locus, we performed molecular genetic studies in two patients with Hunter syndrome. An interstitial deletion spanning the middle part of the IDS gene was found in the first patient. The second patient carries a gross gene rearrangement that can be detected after HindIII or EcoRI digestion of genomic DNA, and is similar to that found recently in seven unrelated Hunter patients. Our data suggest that the structural aberration observed is a partial intragenic inversion. As the same altered hybridiz…

MaleX ChromosomeRestriction MappingLocus (genetics)Iduronate SulfataseHindIIIDeoxyribonuclease EcoRIGeneticsmedicineHumansMucopolysaccharidosis type IIChildDeoxyribonucleases Type II Site-SpecificGenetics (clinical)Mucopolysaccharidosis IIGeneticsbiologyHybridization probeHunter syndromeGene rearrangementmedicine.diseaseMolecular biologyBlotting Southerngenomic DNAChild Preschoolbiology.proteinRestriction fragment length polymorphismDNA ProbesPolymorphism Restriction Fragment LengthHuman Genetics
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Parental practices perceived by children using a French version of the Kids’ Child Feeding Questionnaire

2011

WOS:000293677900024; International audience; About 18% of 6-11-year-old French children are overweight, of whom 3.3% are obese. Parental feeding practices, especially restriction and pressure-to-eat, seem to promote overeating in children. Since no tool was available for the perception of parental feeding practices of French children, our aim was to validate a French version of the Kids'Child Feeding Questionnaire (KCFQ, Carper, Orlet Fischer, & Birch, 2000), and to determine the relationship between KCFQ's dimensions and children's standardised body mass index (BMI z-scores). The questionnaire was completed by 240 normal-weighted or overweight children between the age of 9 and 11. The vali…

Maleconfirmatory factor analysisobesityfood intakeMESH: Parenting030309 nutrition & dieteticsCross-sectional study[SDV]Life Sciences [q-bio]restrictionOverweightMESH: Regression AnalysisMESH: EatingDevelopmental psychologyEatingMESH : Cross-Sectional Studies0302 clinical medicineMESH : ChildMESH : Regression AnalysisSurveys and QuestionnairesMESH: ChildMESH: ObesityMESH : FemaleMass indexOvereatingMESH : Body WeightGeneral PsychologyLanguageMESH : Parenting2. Zero hungerchild0303 health sciencesNutrition and DieteticsParentinggirlMESH : QuestionnairesMESH : Feeding BehaviorbodyMESH: Interviews as TopicConfirmatory factor analysisstyleMESH: Languagemass indexMESH: Feeding BehaviorRegression AnalysisMESH : ObesityFemalemedicine.symptomPsychologyClinical psychologyPsychopathologyMESH : EatingMESH : MaleMESH: Factor Analysis StatisticalMESH : Factor Analysis Statisticalbody mass index030209 endocrinology & metabolismMESH: Body Mass IndexInterviews as Topic03 medical and health sciencesMESH: Cross-Sectional StudiesCronbach's alphaMESH : Languageparental feeding practicemedicineHumansoverweightkids' child feedingMESH: Humans[ SDV ] Life Sciences [q-bio]questionnaireMESH: QuestionnairesMESH : HumansBody WeightassociationFeeding BehaviorMESH: MaleMESH: Body Weightweight statusMESH : Body Mass Indexpressure to eatCross-Sectional StudiesMESH : Interviews as TopicadolescentFactor Analysis StatisticalMESH: FemaleBody mass indexAppetite
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Association of SUMO4 M55V polymorphism with autoimmune diabetes in Latvian patients.

2006

Small ubiquitin-related modifier (SUMO4), located in IDDM5, has been identified as a potential susceptibility gene for type 1 diabetes mellitus (T1DM). The novel polymorphism M55V, causing an amino acid change in the evolutionarily conserved met55 residue has been shown to activate the nuclear factor kappaB (NF-kappaB), hence the suspected role of SUMO4 in the pathogenicity of T1DM. The M55V polymorphism has been shown to be associated with susceptibility to T1DM in Asians, but not in Caucasians. Latent autoimmune diabetes in adults (LADA) is a slowly progressive form of T1DM and SUMO4 M55V has not been studied in LADA to date. The current study aims to test whether Latvians are similar to …

Maleendocrine system diseasesAdolescentHuman leukocyte antigenBiologyPolymerase Chain ReactionPolymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular Biologylaw.inventionAutoimmune DiseasesMethionineHistory and Philosophy of ScienceGene Frequencyimmune system diseaseslawmedicineHumansGenetic Predisposition to DiseaseAlleleChildPolymerase chain reactionAllelesGeneticsType 1 diabetesGeneral NeuroscienceInfantmedicine.diseasePathogenicityLatviaDiabetes Mellitus Type 1Autoimmune diabetesCase-Control StudiesChild PreschoolSmall Ubiquitin-Related Modifier ProteinsAmino acid changeFemaleRestriction fragment length polymorphismPolymorphism Restriction Fragment LengthAnnals of the New York Academy of Sciences
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Estimating brain load from the EEG.

2009

Modern work requires cognitively demanding multitasking and the need for sustained vigilance, which may result in work-related stress and may increase the possibility of human error. Objective methods for estimating cognitive overload and mental fatigue of the brain on-line, during work performance, are needed. We present a two-channel electroencephalography (EEG)–based index, theta Fz/alpha Pz ratio, potentially implementable into a compact wearable device. The index reacts to both acute external and cumulative internal load. The index increased with the number of tasks to be performed concurrently (p= 0.004) and with increased time awake, both after normal sleep (p= 0.002) and sleep restr…

Malemedicine.medical_specialtyHeartbeatArticle SubjectMental fatiguemedia_common.quotation_subjectbrainlcsh:MedicineWorkloadElectroencephalographyAudiologylcsh:TechnologyGeneral Biochemistry Genetics and Molecular BiologycognitiveYoung Adultwork loadTask Performance and AnalysismedicineReaction TimeHuman multitaskingHumansEEGlcsh:ScienceEvoked PotentialsSimulationGeneral Environmental ScienceSleep restrictionmedia_commonAnalysis of Variancemedicine.diagnostic_testbusiness.industrylcsh:Tlcsh:RElectroencephalographyGeneral Medicineassessment of workloadBrain stateSleep Deprivationlcsh:QbusinessSleepCognitive loadPsychomotor PerformanceVigilance (psychology)Research ArticleTheScientificWorldJournal
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Low Protein Intake Is Associated with a Major Reduction in IGF-1, Cancer, and Overall Mortality in the 65 and Younger but Not Older Population

2014

SummaryMice and humans with growth hormone receptor/IGF-1 deficiencies display major reductions in age-related diseases. Because protein restriction reduces GHR-IGF-1 activity, we examined links between protein intake and mortality. Respondents aged 50–65 reporting high protein intake had a 75% increase in overall mortality and a 4-fold increase in cancer death risk during the following 18 years. These associations were either abolished or attenuated if the proteins were plant derived. Conversely, high protein intake was associated with reduced cancer and overall mortality in respondents over 65, but a 5-fold increase in diabetes mortality across all ages. Mouse studies confirmed the effect…

Malemedicine.medical_specialtyLow proteinnutrition protein intake caloric restriction nutrientsPhysiologymedicine.medical_treatmentLongevityCalorie restrictionBreast NeoplasmsGrowth hormone receptorBiologyArticleMiceLow-protein dietNeoplasmsDiabetes mellitusInternal medicineDiabetes MellitusDiet Protein-RestrictedmedicineAnimalsHumansInsulin-Like Growth Factor IMelanomaMolecular BiologyAgedProportional Hazards ModelsMice KnockoutMice Inbred BALB CIncidence (epidemiology)CancerCell BiologyMiddle Agedmedicine.diseaseMiddle ageMice Inbred C57BLCross-Sectional StudiesEndocrinologyFemaleCarrier ProteinsFollow-Up StudiesSignal TransductionCell Metabolism
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